+
Otitis media
Abdullatif Sami Al Rashed
College of Medicine, King Faisal University
Al Ahsa, KSA
+
ACUTE SUPPURATIVE
OTITIS MEDIA
+
Introduction
 more common especially in infants and children of lower
socioeconomic group.
 the disease follows viral infection of upper respiratory tract but
soon the pyogenic organisms invade the middle ear.
 Breast or bottle feeding in a young infant in horizontal position,
Swimming and diving are risk factors
 Streptococcus pneumoniae (30%), Haemophilus influenzae
(20%) and Moraxella catarrhalis
+
CLINICAL FEATURES
 1. Stage of tubal occlusion:
 Symptoms.
 Deafness and otalgia
 no fever.
 Signs.
 Tympanic membrane is retracted with handle of malleus assuming a
more horizontal position,
 prominence of lateral process of malleus
 loss of light reflex.
 Tuning fork tests show conductive deafness.
+
CLINICAL FEATURES
 2. Stage of presuppuration
 Symptoms.
 Severe otalgia affects the sleep, throbbing in nature.
 Deafness and tinnitus but complained only by adults.
 children runs high degree of fever and is restless.
 Signs.
 congestion of pars tensa.
 Leash of blood vessels appear along the handle of malleus and at the
periphery of tympanic membrane imparting it a cart-wheel appearance.
 then, whole of tympanic membrane including pars flaccida becomes
uniformly red.
 Conductive hearing loss.
+
CLINICAL FEATURES
 3. Stage of suppuration.
 Symptoms.
 Otalgia becomes very severe.
 Deafness increases,
 Children may run fever of 39-40 C A/W vomiting and convulsions.
 Pain disappear when there is discharge
 Signs.
 Tympanic membrane appears red and bulging with loss of landmarks.
 Handle of malleus may be engulfed by the swollen and protruding tympanic
membrane and may not be discernible.
 A yellow spot may be seen on the tympanic membrane where rupture is clear.
 Tenderness may be elicited over the mastoid antrum.
 X-rays of mastoid will show clouding of air cells because of exudate.
+
CLINICAL FEATURES
 4. Stage of resolution.
 Symptoms.
 evacuation of pus, relieve otalgia,
 fever comes down and child feels better.
 Signs.
 External auditory canal may contain blood-tinged discharge which
later becomes mucopurulent.
 small perforation is seen in anteroinferior quadrant of pars tensa.
 Hyperaemia of tympanic membrane subside with return to normal
colour and landmarks.
+
CLINICAL FEATURES
 5. Stage of complication.
 If virulence of organism is high or resistance of patient poor,
resolution may not take place and disease spreads beyond the
confines of middle ear.
 It may lead to:
 acute mastoiditis, labyrinthitis
 subperiosteal abscess, extradural abscess, brain abscess
 facial paralysis
 meningitis
+
TREATMENT
 Antibiotics:
 amoxicillin (40 mg/kg/day in three divided doses)
 When there is discharge we give topical antibiotic
 Decongestant nasal drops: Ephedrine
 Oral nasal decongestants: Pseudoephedrine
 Analgesics: paracetamol
 Ear toilet. If there is discharge in the ear
 Myringotomy: indicated when
 Failure of medical TTT
 drum is bulging and there is acute pain,
 persistent effusion more the an 12 weeks.
+
OTITIS MEDIA WITH
EFFUSION
+
ETIOLOGY
 Adenoid hyperplasia
 Chronic rhinitis and sinusitis
 Chronic tonsillitis
 Benign and malignant tumours of nasopharynx
 Unresolved acute otitis media
 Viral
+
CLINICAL FEATURES
 Hearing loss
 Delayed and defective speech
 Discomfort in the ear or very mild pain
+
CLINICAL FEATURES
 Otoscopic findings
 Tympanic membrane is often dull and opaque
 loss of light reflex
 Yellow, grey or bluish in colour.
 blood vessels may be seen along the handle of malleus
 Tympanic membrane having retraction.
 may appear full or slightly bulging in its posterior part due to
effusion.
 Mobility of the tympanic membrane is restricted
+
TESTS
 Tuning fork tests show conductive hearing loss.
 Audiometry. conductive hearing loss of 20–40 dB
 Impedance audiometry (tympanometry).
 It is useful in infants and children.
 B Curve:
 reduced compliance and flat curve with a shift to negative side.
 X-ray mastoids. There is clouding of air cells due to fluid.
+
TREATMENT
 Decongestants.
 Antihistaminics or sometimes steroids in cases of allergy.
 Antibiotics.
 Valsalva manoeuvre, Children can be given chewing gum to
encourage repeated swallowing to open the tube.
 Myringotomy and aspiration of fluid with Grommet insertion
 Tympanotomy or cortical mastoidectomy
 required for removal of loculated thick fluid or associated with
cholesterol granuloma
+
SEQUELAE OF CHRONIC OTITIS
MEDIA with effusion
 Atrophy of tympanic membrane and atelectasis of the middle
ear.
 Ossicular necrosis
 Most commonly, long process of incus gets necrosed.
 Tympanosclerosis
 Hyalinized collagen with chalky deposits may be seen
 Retraction pockets and cholesteatoma
 Cholesterol granuloma
+
CHRONIC SUPPURATIVE
OTITIS MEDIA
+
TYPES
 Tubotympanic. Also called the safe or benign type. There is no
risk of serious complications.
 Atticoantral. Also called unsafe or dangerous type. risk of
serious complications is high.
+
TUBOTYMPANIC TYPE
 After recurrent or untreated acute OM
 Pseudomonas aeruginosa, Proteus, Escherichia coli and
Staphylococcus aureus, while anaerobes include Bacteroides
fragilis and anaerobic Streptococci
+
TUBOTYMPANIC TYPE
 CLINICAL FEATURES:
 Otorrhea:
 nonoffensive, mucoid or mucopurulent, constant or intermittent
 Hearing loss. Conductive
 Perforation. Always central by otoscope
 Middle ear mucosa. It is seen when the perforation is large.
Red, oedematous and swollen. Occasionally, a polyp may be
seen
+
TUBOTYMPANIC TYPE
 INVESTIGATIONS
 Examination under microscope
 To check presence of granulations, in-growth of squamous
epithelium from the edges of perforation, status of ossicular chain,
tympanosclerosis and adhesions
 Audiogram. Conductive mainly but SNHL can A/W
 Culture and sensitivity of ear discharge
 Mastoid X-rays/CT scan temporal bone
+
TUBOTYMPANIC TYPE
 TREATMENT
 Ear toilet.
 Antibiotic ear drops containing neomycin
 Systemic antibiotics. They are useful only in acute exacerbation
of CSOM
 Instructions to patients:
 keep water out of the ear during bathing, and swimming.
 Rubber inserts can be used.
 Hard nose blowing ( ‫ينفخ‬‫بخشمه‬‫بقوه‬ )can also push the infection from
nasopharynx to middle ear and should b e avoided.
+
TUBOTYMPANIC TYPE
 TREATMENT
 Treat other causes such as adenoid or tonsill
 Aural polyp or granulations, if present, should be removed by
surgical excision.
 Myringoplasty with or without ossicular reconstruction can be
done to restore hearing when ear is dry
+
ATTICOANTRAL TYPE
 A/W:
 Cholesteatoma.
 Osteitis and granulation tissue
 Ossicular necrosis.
 Cholesterol granuloma
 SYMPTOMS
 Otorrhea; Usually scanty, but always foul-smelling due to bone
destruction.
 Hearing loss.
 Bleeding.
+
ATTICOANTRAL TYPE
 SIGNS
 Perforation either attic or posterosuperior marginal type
 Retraction pocket
 Cholesteatoma: Pearly-white flakes
 INVESTIGATIONS
 Examination under microscope
 Tuning fork tests and audiogram
 X-ray mastoids/CT scan temporal bone
 Culture and sensitivity of ear discharge
Only study the yellow color
+
ATTICOANTRAL TYPE
 TREATMENT: surgery is main TTT
 Mastiodectomy
 Canal wall down procedures
 Canal wall up procedures
 Hearing can be restored by myringoplasty or tympanoplasty
+
adhesive otitis media.
 Tympanic membrane is very thin and wraps the promontory
and ossicles.
 There is no middle ear space, mucosal lining of the middle ear
is absent and tympanic membrane gets adherent to the
promontory.
 Retraction pockets are formed which may collect keratin plugs
and form cholesteatoma.
 Erosion of the long process of incus and stapes superstructure
is common in such cases
+
Complications of Suppurative
Otitis Media
+
INTRATEMPORAL
COMPLICATIONS
 (I) ACUTE MASTOIDITIS
 Pain behind the ear after ttt of OM
 Increased in its intensity or recurrence of pain
 Persistent fever
 Discharge profuse, pulsatile and increases in purulence after OM
TTT
 Mastoid Tenderness.
 Perforation of tympanic membrane.
 Swelling over the mastoid
 Hearing loss
 Mastoid fistula
+
INTRATEMPORAL
COMPLICATIONS
 (I) ACUTE MASTOIDITIS
 CBC, ESR, X Ray Mastoid, Ear swab
 Hospitalization of the patient, Antibiotics, Myringotomy.
+
INTRATEMPORAL
COMPLICATIONS
 2. Gradenigo syndrome
 triad of
 (i) external rectus palsy (VIth nerve palsy),
 (ii) deep-seated ear or retro-orbital pain (Vth nerve involvement)
 and (iii) persistent ear discharge
 CT scan and MRI For Dx.
 CT scan of temporal bone will show bony details of the petrous apex
and the air cells
 MRI helps to differentiate diploic marrow-containing apex from the
fluid or pus.
 TTT  Cortical, modified radical or radical mastoidectomy
+
INTRATEMPORAL
COMPLICATIONS
 3. FACIAL PARALYSIS
 Both OM and Facial palsy must be treated
 Nerve decompression + OM TTT
 4. LABYRINTHITIS
 Fistula of labyrinth TTT  mastoid exploration to eliminate the
cause + Systemic antibiotic
 Diffuse Serous Labyrinthitis and Diffuse suppurative
Labyrinthitis TTT  same SNHL lecture + OM TTT
+
INTRACRANIAL COMPLICATIONS
Of OTITIS MEDIA
 EXTRADURAL ABSCESS
 Persistent headache on the side of otitis media
 Severe pain in the ear
 General malaise with low-grade fever
 Pulsatile purulent ear discharge.
 Dx by contrast-enhanced CT or MRI.
 TTT  Cortical or modified radical or radical mastoidectomy with
antibiotic cover
+
INTRACRANIAL COMPLICATIONS
Of OTITIS MEDIA
 SUBDURAL ABSCESS
 Meningeal irritation symptoms
 Aphasia, hemiplegia and hemianopia.
 Jacksonian type of epileptic fits
 Raised intracranial tension Symptoms and signs
 Lumbar puncture should not be done as it can cause herniation of
the cerebellar tonsils.
 TTT  A series of burr holes or a craniotomy is done to drain
subdural empyema. Intravenous antibiotics are administered to
control infection then AFTER THAT MASTIODECTOMY
+
INTRACRANIAL COMPLICATIONS
Of OTITIS MEDIA
 MENINGITIS
 Fever, chills and rigors.
 Headache.
 Neck rigidity.
 Photophobia and mental irritability.
 Nausea and vomiting (sometimes projectile).
 positive Kernig’s Sign (extension of leg with thigh flexed on
abdomen causing pain)
 positive Brudzinski’s sign (flexion of neck causes flexion of hip and
knee)
 CT with contrast or MRI and LP for Dx
 TTT  antibiotics + OM TTT (Mastoidectomy)
+
 ANY ABSCESS you should TTT by:
 Antibiotic
 Craniotomy Drainage
 OM TTT (Mastoidectomy)
THANK YOU

Otitis Media

  • 1.
    + Otitis media Abdullatif SamiAl Rashed College of Medicine, King Faisal University Al Ahsa, KSA
  • 2.
  • 3.
    + Introduction  more commonespecially in infants and children of lower socioeconomic group.  the disease follows viral infection of upper respiratory tract but soon the pyogenic organisms invade the middle ear.  Breast or bottle feeding in a young infant in horizontal position, Swimming and diving are risk factors  Streptococcus pneumoniae (30%), Haemophilus influenzae (20%) and Moraxella catarrhalis
  • 4.
    + CLINICAL FEATURES  1.Stage of tubal occlusion:  Symptoms.  Deafness and otalgia  no fever.  Signs.  Tympanic membrane is retracted with handle of malleus assuming a more horizontal position,  prominence of lateral process of malleus  loss of light reflex.  Tuning fork tests show conductive deafness.
  • 5.
    + CLINICAL FEATURES  2.Stage of presuppuration  Symptoms.  Severe otalgia affects the sleep, throbbing in nature.  Deafness and tinnitus but complained only by adults.  children runs high degree of fever and is restless.  Signs.  congestion of pars tensa.  Leash of blood vessels appear along the handle of malleus and at the periphery of tympanic membrane imparting it a cart-wheel appearance.  then, whole of tympanic membrane including pars flaccida becomes uniformly red.  Conductive hearing loss.
  • 6.
    + CLINICAL FEATURES  3.Stage of suppuration.  Symptoms.  Otalgia becomes very severe.  Deafness increases,  Children may run fever of 39-40 C A/W vomiting and convulsions.  Pain disappear when there is discharge  Signs.  Tympanic membrane appears red and bulging with loss of landmarks.  Handle of malleus may be engulfed by the swollen and protruding tympanic membrane and may not be discernible.  A yellow spot may be seen on the tympanic membrane where rupture is clear.  Tenderness may be elicited over the mastoid antrum.  X-rays of mastoid will show clouding of air cells because of exudate.
  • 7.
    + CLINICAL FEATURES  4.Stage of resolution.  Symptoms.  evacuation of pus, relieve otalgia,  fever comes down and child feels better.  Signs.  External auditory canal may contain blood-tinged discharge which later becomes mucopurulent.  small perforation is seen in anteroinferior quadrant of pars tensa.  Hyperaemia of tympanic membrane subside with return to normal colour and landmarks.
  • 8.
    + CLINICAL FEATURES  5.Stage of complication.  If virulence of organism is high or resistance of patient poor, resolution may not take place and disease spreads beyond the confines of middle ear.  It may lead to:  acute mastoiditis, labyrinthitis  subperiosteal abscess, extradural abscess, brain abscess  facial paralysis  meningitis
  • 9.
    + TREATMENT  Antibiotics:  amoxicillin(40 mg/kg/day in three divided doses)  When there is discharge we give topical antibiotic  Decongestant nasal drops: Ephedrine  Oral nasal decongestants: Pseudoephedrine  Analgesics: paracetamol  Ear toilet. If there is discharge in the ear  Myringotomy: indicated when  Failure of medical TTT  drum is bulging and there is acute pain,  persistent effusion more the an 12 weeks.
  • 10.
  • 11.
    + ETIOLOGY  Adenoid hyperplasia Chronic rhinitis and sinusitis  Chronic tonsillitis  Benign and malignant tumours of nasopharynx  Unresolved acute otitis media  Viral
  • 12.
    + CLINICAL FEATURES  Hearingloss  Delayed and defective speech  Discomfort in the ear or very mild pain
  • 13.
    + CLINICAL FEATURES  Otoscopicfindings  Tympanic membrane is often dull and opaque  loss of light reflex  Yellow, grey or bluish in colour.  blood vessels may be seen along the handle of malleus  Tympanic membrane having retraction.  may appear full or slightly bulging in its posterior part due to effusion.  Mobility of the tympanic membrane is restricted
  • 14.
    + TESTS  Tuning forktests show conductive hearing loss.  Audiometry. conductive hearing loss of 20–40 dB  Impedance audiometry (tympanometry).  It is useful in infants and children.  B Curve:  reduced compliance and flat curve with a shift to negative side.  X-ray mastoids. There is clouding of air cells due to fluid.
  • 15.
    + TREATMENT  Decongestants.  Antihistaminicsor sometimes steroids in cases of allergy.  Antibiotics.  Valsalva manoeuvre, Children can be given chewing gum to encourage repeated swallowing to open the tube.  Myringotomy and aspiration of fluid with Grommet insertion  Tympanotomy or cortical mastoidectomy  required for removal of loculated thick fluid or associated with cholesterol granuloma
  • 16.
    + SEQUELAE OF CHRONICOTITIS MEDIA with effusion  Atrophy of tympanic membrane and atelectasis of the middle ear.  Ossicular necrosis  Most commonly, long process of incus gets necrosed.  Tympanosclerosis  Hyalinized collagen with chalky deposits may be seen  Retraction pockets and cholesteatoma  Cholesterol granuloma
  • 17.
  • 18.
    + TYPES  Tubotympanic. Alsocalled the safe or benign type. There is no risk of serious complications.  Atticoantral. Also called unsafe or dangerous type. risk of serious complications is high.
  • 20.
    + TUBOTYMPANIC TYPE  Afterrecurrent or untreated acute OM  Pseudomonas aeruginosa, Proteus, Escherichia coli and Staphylococcus aureus, while anaerobes include Bacteroides fragilis and anaerobic Streptococci
  • 21.
    + TUBOTYMPANIC TYPE  CLINICALFEATURES:  Otorrhea:  nonoffensive, mucoid or mucopurulent, constant or intermittent  Hearing loss. Conductive  Perforation. Always central by otoscope  Middle ear mucosa. It is seen when the perforation is large. Red, oedematous and swollen. Occasionally, a polyp may be seen
  • 22.
    + TUBOTYMPANIC TYPE  INVESTIGATIONS Examination under microscope  To check presence of granulations, in-growth of squamous epithelium from the edges of perforation, status of ossicular chain, tympanosclerosis and adhesions  Audiogram. Conductive mainly but SNHL can A/W  Culture and sensitivity of ear discharge  Mastoid X-rays/CT scan temporal bone
  • 23.
    + TUBOTYMPANIC TYPE  TREATMENT Ear toilet.  Antibiotic ear drops containing neomycin  Systemic antibiotics. They are useful only in acute exacerbation of CSOM  Instructions to patients:  keep water out of the ear during bathing, and swimming.  Rubber inserts can be used.  Hard nose blowing ( ‫ينفخ‬‫بخشمه‬‫بقوه‬ )can also push the infection from nasopharynx to middle ear and should b e avoided.
  • 24.
    + TUBOTYMPANIC TYPE  TREATMENT Treat other causes such as adenoid or tonsill  Aural polyp or granulations, if present, should be removed by surgical excision.  Myringoplasty with or without ossicular reconstruction can be done to restore hearing when ear is dry
  • 25.
    + ATTICOANTRAL TYPE  A/W: Cholesteatoma.  Osteitis and granulation tissue  Ossicular necrosis.  Cholesterol granuloma  SYMPTOMS  Otorrhea; Usually scanty, but always foul-smelling due to bone destruction.  Hearing loss.  Bleeding.
  • 26.
    + ATTICOANTRAL TYPE  SIGNS Perforation either attic or posterosuperior marginal type  Retraction pocket  Cholesteatoma: Pearly-white flakes  INVESTIGATIONS  Examination under microscope  Tuning fork tests and audiogram  X-ray mastoids/CT scan temporal bone  Culture and sensitivity of ear discharge
  • 27.
    Only study theyellow color
  • 28.
    + ATTICOANTRAL TYPE  TREATMENT:surgery is main TTT  Mastiodectomy  Canal wall down procedures  Canal wall up procedures  Hearing can be restored by myringoplasty or tympanoplasty
  • 29.
    + adhesive otitis media. Tympanic membrane is very thin and wraps the promontory and ossicles.  There is no middle ear space, mucosal lining of the middle ear is absent and tympanic membrane gets adherent to the promontory.  Retraction pockets are formed which may collect keratin plugs and form cholesteatoma.  Erosion of the long process of incus and stapes superstructure is common in such cases
  • 30.
  • 31.
    + INTRATEMPORAL COMPLICATIONS  (I) ACUTEMASTOIDITIS  Pain behind the ear after ttt of OM  Increased in its intensity or recurrence of pain  Persistent fever  Discharge profuse, pulsatile and increases in purulence after OM TTT  Mastoid Tenderness.  Perforation of tympanic membrane.  Swelling over the mastoid  Hearing loss  Mastoid fistula
  • 32.
    + INTRATEMPORAL COMPLICATIONS  (I) ACUTEMASTOIDITIS  CBC, ESR, X Ray Mastoid, Ear swab  Hospitalization of the patient, Antibiotics, Myringotomy.
  • 35.
    + INTRATEMPORAL COMPLICATIONS  2. Gradenigosyndrome  triad of  (i) external rectus palsy (VIth nerve palsy),  (ii) deep-seated ear or retro-orbital pain (Vth nerve involvement)  and (iii) persistent ear discharge  CT scan and MRI For Dx.  CT scan of temporal bone will show bony details of the petrous apex and the air cells  MRI helps to differentiate diploic marrow-containing apex from the fluid or pus.  TTT  Cortical, modified radical or radical mastoidectomy
  • 36.
    + INTRATEMPORAL COMPLICATIONS  3. FACIALPARALYSIS  Both OM and Facial palsy must be treated  Nerve decompression + OM TTT  4. LABYRINTHITIS  Fistula of labyrinth TTT  mastoid exploration to eliminate the cause + Systemic antibiotic  Diffuse Serous Labyrinthitis and Diffuse suppurative Labyrinthitis TTT  same SNHL lecture + OM TTT
  • 37.
    + INTRACRANIAL COMPLICATIONS Of OTITISMEDIA  EXTRADURAL ABSCESS  Persistent headache on the side of otitis media  Severe pain in the ear  General malaise with low-grade fever  Pulsatile purulent ear discharge.  Dx by contrast-enhanced CT or MRI.  TTT  Cortical or modified radical or radical mastoidectomy with antibiotic cover
  • 38.
    + INTRACRANIAL COMPLICATIONS Of OTITISMEDIA  SUBDURAL ABSCESS  Meningeal irritation symptoms  Aphasia, hemiplegia and hemianopia.  Jacksonian type of epileptic fits  Raised intracranial tension Symptoms and signs  Lumbar puncture should not be done as it can cause herniation of the cerebellar tonsils.  TTT  A series of burr holes or a craniotomy is done to drain subdural empyema. Intravenous antibiotics are administered to control infection then AFTER THAT MASTIODECTOMY
  • 39.
    + INTRACRANIAL COMPLICATIONS Of OTITISMEDIA  MENINGITIS  Fever, chills and rigors.  Headache.  Neck rigidity.  Photophobia and mental irritability.  Nausea and vomiting (sometimes projectile).  positive Kernig’s Sign (extension of leg with thigh flexed on abdomen causing pain)  positive Brudzinski’s sign (flexion of neck causes flexion of hip and knee)  CT with contrast or MRI and LP for Dx  TTT  antibiotics + OM TTT (Mastoidectomy)
  • 40.
    +  ANY ABSCESSyou should TTT by:  Antibiotic  Craniotomy Drainage  OM TTT (Mastoidectomy)
  • 41.