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Mumps
Dr. Saurabh Agrawal
Intro
• Acute infec. Disease
• Caused by RNA virus classified as rubulavirus
of family paramyxoviridae
• Non suppurative enlargement and tenderness
of one or both parotid glands
• Negligible mortality, morbidity is high
Problem statement
• Incidence- 100-1000cases / 1 lakh population
Agent fac
• Agent- Myxovirus parotiditis
• Source- clinical & subclinical case
• Communicability- usually 4-6 days before the
onset of symp and a week or more there after.
• Maximum infectivity is just before and at the
onset of parotitis
Host fac
• Age and sex- 5-9yrs, no age is exempt if no
immunity
• More severe in adults
• Immunity- one attack, clinical, sub-clinical life
long immunity
Environment fac
• Peak is winter and spring
• Epidemic is associated with over crowding
Mode of transmission
• Droplet infection and direct contact with
infected person
Incubation period
• Varies from 2-3 weeks
• Usually 18 days
Clinical features
• Generalized viral infection
• 30-40% clinically non- apparent
• Pain and swelling in one or both parotid gland
• Also involve sub lingual and sub mandibular
glands
• Often child complains ear pain prior to onset
of swelling
• Pain and stiffness in opening mouth
• May also affect
– Testis
– Pancreas
– CNS
– Ovaries
– Prostate
• Severe cases might mark fever, headache for
3-5 days
• Swelling subsides over 1-2 weeks
Complication
• Frequent but not serious
• Orchitis, ovaritis, pancreatitis, meningo-
encephalitis, thyroiditis, neuritis, hepatitis,
myocarditis
Prevention
• Vaccination: live attenuated
– Single dose- 0.5ml IM
Mumps
Mumps
Mumps

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Mumps

  • 2. Intro • Acute infec. Disease • Caused by RNA virus classified as rubulavirus of family paramyxoviridae • Non suppurative enlargement and tenderness of one or both parotid glands • Negligible mortality, morbidity is high
  • 3. Problem statement • Incidence- 100-1000cases / 1 lakh population
  • 4. Agent fac • Agent- Myxovirus parotiditis • Source- clinical & subclinical case • Communicability- usually 4-6 days before the onset of symp and a week or more there after. • Maximum infectivity is just before and at the onset of parotitis
  • 5. Host fac • Age and sex- 5-9yrs, no age is exempt if no immunity • More severe in adults • Immunity- one attack, clinical, sub-clinical life long immunity
  • 6. Environment fac • Peak is winter and spring • Epidemic is associated with over crowding
  • 7. Mode of transmission • Droplet infection and direct contact with infected person
  • 8. Incubation period • Varies from 2-3 weeks • Usually 18 days
  • 9. Clinical features • Generalized viral infection • 30-40% clinically non- apparent • Pain and swelling in one or both parotid gland • Also involve sub lingual and sub mandibular glands • Often child complains ear pain prior to onset of swelling • Pain and stiffness in opening mouth
  • 10. • May also affect – Testis – Pancreas – CNS – Ovaries – Prostate • Severe cases might mark fever, headache for 3-5 days • Swelling subsides over 1-2 weeks
  • 11. Complication • Frequent but not serious • Orchitis, ovaritis, pancreatitis, meningo- encephalitis, thyroiditis, neuritis, hepatitis, myocarditis
  • 12. Prevention • Vaccination: live attenuated – Single dose- 0.5ml IM