Mercury poisoning
History of occurrence of Minamata disease
Signs and symptoms of Minamata disease
Treatment of Minamata
Chisso Corporation’s chemical factory
From 1932 to 1968, the Chisso Corporation dumped approximately 27 tons of mercury compounds into Minamata Bay from its chemical factory in Kumamoto, Japan. The mercury bioaccumulated in the local seafood which, when eaten by the local population, caused thousands to develop mercury poisoning symptoms known as Minamata disease. Symptoms included neurological problems like numbness, impaired vision and speech. It was later discovered that the mercury compounds were a byproduct of the factory's production process. Despite early warnings, the Chisso Corporation denied claims and continued polluting until 1959 when researchers conclusively linked the disease to methylmercury poisoning from the factory's waste.
Minamata Bay was heavily polluted in the 1950s and 1960s by wastewater from the Chisso Corporation's factory in Minamata, Japan, particularly by methylmercury. The highly toxic compound bioaccumulated in fish and shellfish in the bay which, when eaten by the local populace, caused Minamata disease. More than 10,000 people were affected, 1,784 of whom had died. While pollution and illnesses continued for decades, compensation to victims has occurred and clean-up efforts have been made, though the issue remains ongoing in Japanese society.
Minamata disease is a nervous system disorder caused by eating fish and shellfish contaminated with methyl mercury, a byproduct discharged from acetaldehyde factories into the water. In Minamata, many people contracted the disease in the 1950s after eating large amounts of contaminated seafood. Since then, the city has worked to regenerate by issuing declarations to improve the environment, health and welfare through garbage sorting, recycling, and eco-friendly shops to prevent future pollution.
Minamata is a city located in the Kumamoto prefecture, Japan. The Chisso Corporation, basically a fertilizer and chemicals producer started to release its waste into the Minamata river. This affected the whole Minamata community since the concentration Mercury became high.
The document discusses the Minamata mercury pollution incident that occurred in Japan between 1908 and 1959. It summarizes that:
1) A chemical factory began dumping mercury-laced wastewater into Minamata Bay in 1908, poisoning the local fishing community through the food chain over decades.
2) "Minamata disease" was discovered and linked to methylmercury poisoning in 1956 when residents developed neurological symptoms.
3) It took until 1959 to officially identify the factory as the source of pollution and order wastewater treatment, by which time many had died or suffered permanent injury from mercury exposure.
Environmental Engineering - Case Study - The Minamata Disease DisasterJohn Paul Espino
The Minamata Bay pollution disaster in Japan from 1932 to 1968 exposed local residents to methylmercury released as a byproduct of a chemical plant. Over time, residents developed neurological symptoms and the cause was traced to eating fish and shellfish contaminated with mercury from the plant's wastewater. While the company denied responsibility, over 2,000 victims were eventually certified with more receiving compensation. Decades later, protests and media coverage helped bring awareness and democratization around the environmental and human impacts of industrial pollution.
Minamata disease was caused by mercury poisoning from industrial wastewater discharged into Minamata Bay in Kumamoto, Japan by Chisso Corporation from 1932 to 1968. Over 17,000 victims suffered symptoms including numbness, hearing and vision loss, and even death. While remediation efforts cleaned the bay, many victims still experience health issues. The disaster demonstrated the importance of responsible waste disposal and protecting the environment and human health from industrial pollution.
The Chisso Corporation dumped mercury waste into Minamata Bay from 1932 to 1968, poisoning the local fishing community and causing "Minamata Disease". Over 3,000 victims suffered physical deformities, brain damage, or death from methylmercury poisoning. It took decades for victims to receive compensation, as Chisso denied responsibility. By 1973, a court ruled Chisso was negligent in its dumping and liable for damages, though compensation continues nearly 50 years later.
From 1932 to 1968, the Chisso Corporation dumped approximately 27 tons of mercury compounds into Minamata Bay from its chemical factory in Kumamoto, Japan. The mercury bioaccumulated in the local seafood which, when eaten by the local population, caused thousands to develop mercury poisoning symptoms known as Minamata disease. Symptoms included neurological problems like numbness, impaired vision and speech. It was later discovered that the mercury compounds were a byproduct of the factory's production process. Despite early warnings, the Chisso Corporation denied claims and continued polluting until 1959 when researchers conclusively linked the disease to methylmercury poisoning from the factory's waste.
Minamata Bay was heavily polluted in the 1950s and 1960s by wastewater from the Chisso Corporation's factory in Minamata, Japan, particularly by methylmercury. The highly toxic compound bioaccumulated in fish and shellfish in the bay which, when eaten by the local populace, caused Minamata disease. More than 10,000 people were affected, 1,784 of whom had died. While pollution and illnesses continued for decades, compensation to victims has occurred and clean-up efforts have been made, though the issue remains ongoing in Japanese society.
Minamata disease is a nervous system disorder caused by eating fish and shellfish contaminated with methyl mercury, a byproduct discharged from acetaldehyde factories into the water. In Minamata, many people contracted the disease in the 1950s after eating large amounts of contaminated seafood. Since then, the city has worked to regenerate by issuing declarations to improve the environment, health and welfare through garbage sorting, recycling, and eco-friendly shops to prevent future pollution.
Minamata is a city located in the Kumamoto prefecture, Japan. The Chisso Corporation, basically a fertilizer and chemicals producer started to release its waste into the Minamata river. This affected the whole Minamata community since the concentration Mercury became high.
The document discusses the Minamata mercury pollution incident that occurred in Japan between 1908 and 1959. It summarizes that:
1) A chemical factory began dumping mercury-laced wastewater into Minamata Bay in 1908, poisoning the local fishing community through the food chain over decades.
2) "Minamata disease" was discovered and linked to methylmercury poisoning in 1956 when residents developed neurological symptoms.
3) It took until 1959 to officially identify the factory as the source of pollution and order wastewater treatment, by which time many had died or suffered permanent injury from mercury exposure.
Environmental Engineering - Case Study - The Minamata Disease DisasterJohn Paul Espino
The Minamata Bay pollution disaster in Japan from 1932 to 1968 exposed local residents to methylmercury released as a byproduct of a chemical plant. Over time, residents developed neurological symptoms and the cause was traced to eating fish and shellfish contaminated with mercury from the plant's wastewater. While the company denied responsibility, over 2,000 victims were eventually certified with more receiving compensation. Decades later, protests and media coverage helped bring awareness and democratization around the environmental and human impacts of industrial pollution.
Minamata disease was caused by mercury poisoning from industrial wastewater discharged into Minamata Bay in Kumamoto, Japan by Chisso Corporation from 1932 to 1968. Over 17,000 victims suffered symptoms including numbness, hearing and vision loss, and even death. While remediation efforts cleaned the bay, many victims still experience health issues. The disaster demonstrated the importance of responsible waste disposal and protecting the environment and human health from industrial pollution.
The Chisso Corporation dumped mercury waste into Minamata Bay from 1932 to 1968, poisoning the local fishing community and causing "Minamata Disease". Over 3,000 victims suffered physical deformities, brain damage, or death from methylmercury poisoning. It took decades for victims to receive compensation, as Chisso denied responsibility. By 1973, a court ruled Chisso was negligent in its dumping and liable for damages, though compensation continues nearly 50 years later.
A case study on minamata disease-By Ritu Chauhanabhimicro19
Minamata disease was caused by mercury poisoning from industrial wastewater dumped into Minamata Bay from 1956-1968. Over 900 people died and thousands suffered from neurological symptoms like numbness, weakness, and loss of coordination. The disease drew attention to environmental pollution issues in Japan. After the victims were recognized, Minamata City took measures like environmental education, promoting sustainable practices, and honoring those affected to prevent future disasters and rebuild their community.
This document discusses marine pollution, including its sources and effects. It defines marine pollution as the discharge of waste substances into the sea, harming living resources and human health. Major pollutants include sewage, pesticides, plastics, metallic wastes, oil, sediment plumes from mining, heat, radioactive waste, and dredge spoils. These pollutants can damage ecosystems, kill wildlife, accumulate in food chains, and threaten human health if consumed from contaminated marine life. Prevention requires controlling pollution at sources, cleaning up waste, and raising awareness of the importance of protecting oceans to maintain Earth's chemical and biological balance.
The hazardous impact of microplastics in aquatic environments calls for more scientific research to understand their occurrence, effects and mitigation strategies. This work describes the analysis of primary and secondary microplastics using FTIR and FTIR microscopy techniques.
This document summarizes the current state of knowledge around microplastics and their trophic transfer in aquatic ecosystems. It outlines background information on microplastics and their sources. It then reviews several case studies that demonstrate trophic transfer of microplastics between invertebrates, fish, and top predators like seals. The studies found microplastics accumulate at higher trophic levels. The document concludes by identifying key knowledge gaps and recommending future research focus on effects of microplastics on human health and standardizing detection methods, while promoting efforts to reduce plastic use.
This document discusses various types of sea water pollution including chemicals, metals, radioactive substances, solid waste, oil, sewage, and agricultural runoff. Major pollutants released into the oceans include billions of tons of ballast water, trillions of gallons of sewage, millions of tons of oil and solid waste. Heavy metals like mercury, lead and copper released from human activities pose serious threats as they accumulate in the food chain. Pollution has severe negative effects on marine life and ecosystems as well as public health. Prevention of pollution through changes in human attitudes and behaviors is important since the impacts of pollution on the environment can be long-lasting and irreversible.
The document discusses sources and consequences of oil pollution. It describes various sources of oil pollution including natural seeps, sea-based sources like oil spills from tankers and pipelines, and land-based sources such as urban and industrial runoff. Sea-based sources are divided into accidental discharges from incidents and operational discharges from regular shipping activities. Land-based sources also include oil in untreated sewage and stormwater. The consequences of oil spills are extensive damage to wildlife from ingestion and coating of feathers/fur, as well as long-term pollution of sediments that can impact burrowing animals for decades. Cleanup and recovery of oil spills is challenging and dependent on various environmental factors.
This document discusses microplastic pollution and its potential threat to marine invertebrates and the food web. It defines microplastics and explains how they enter the marine environment. Studies found microplastics present in sediment samples from beaches around the world. Microplastics can be ingested by small organisms and transferred up the food chain, as demonstrated by studies showing zooplankton ingesting microplastics that were then found in mysid shrimp that ate the zooplankton. The document also discusses how microplastics can be taken up by blue mussels and transferred to shore crabs that eat the mussels, entering the intertidal food web.
This document discusses water pollution, including its definition, types of pollutants, sources, effects, and ways to prevent it. It defines water pollution as any physical or chemical change in water that harms humans or other organisms. The major types of pollutants are pathogens, inorganic materials, organic materials, suspended solids, and radioactive materials. Sources include sewage, agriculture, oil, erosion, and industrial/domestic waste. Effects are risks to human and animal health. Prevention methods center around proper waste disposal, reducing runoff, and recycling/reusing water.
Microplastics (MPs) are small plastic pieces with size less than 5mm that have entered and polluted the environment.
While many investigations including several critical reviews on MPs in the environment have been
conducted, most of them are focused on their occurrences in marine environment
Human activities such as burning coal, medical waste, and other materials containing mercury are major contributors to mercury pollution. Mercury enters the environment through air pollution and can harm human health, especially for fetuses and children. The largest source of mercury air pollution is coal-fired power plants, which emit around 70% of the mercury in the US. Capturing mercury from coal plant emissions is challenging given mercury's various chemical forms.
The Love Canal disaster involved a neighborhood in Niagara Falls, New York that was built on top of a former chemical waste dump containing over 21,000 tons of toxic waste. In the late 1970s, residents noticed high rates of cancer and birth defects, which were eventually traced to the chemical waste leaking into the soil and water supply from the dump located under the neighborhood. Over 800 families were evacuated and reimbursed for their homes after extensive legal battles and media coverage brought attention to the environmental and public health crisis.
Marine pollution and its effect on environmentPramoda Raj
This document discusses various types of marine pollution including oil pollution, eutrophication, conservative pollutants such as metals and halogenated hydrocarbons, thermal pollution, radioactive pollution, and litter and plastic pollution. It outlines sources of each type of pollution and their impacts on the marine environment such as oil spills harming ecosystems, nutrients from eutrophication causing algal blooms, metals and plastics bioaccumulating in marine organisms and entering the food chain, and litter entangling and being ingested by animals. The conclusion states that education on marine pollution through awareness, research, and dissemination is key to reducing pollution in oceans.
This slide presentation is part DYUTI 2010 preconference series. This slides discuss various environmental disasters. Prepared and Presented by Kochubaby Manjorran
The document discusses various types of marine pollution including plastic debris, oil pollution, eutrophication, acidification, thermal pollution, radioactive pollution, and underwater noise pollution. It outlines the sources, impacts and solutions for each type of pollution. The sources discussed include land-based sources like agricultural and municipal runoff, ocean dumping, oil spills, and deep sea mining. The impacts covered are effects on marine life, human health, marine activities, and water quality. Solutions proposed are prevention through regulations, cleanup methods like bioremediation, skimming, burning, and reducing plastic and radioactive waste through various recycling and disposal methods.
Radioactive pollution is defined as the release of radioactive substances or particles into the environment from human activities like nuclear weapon testing, nuclear power plants, or accidents. It can cause serious health effects like cancer due to radiation exposure and remains toxic for centuries. Sources include natural processes like radioactive minerals as well as human activities involving nuclear materials, weapons, power plants, and medical isotopes. Effects range from acute radiation sickness to long-term mutations and increased cancer risks. Monitoring, safe waste disposal, and prevention of leaks and accidents are important for controlling radioactive pollution.
Slides from the Deschutes Land Trust's Nature Night presentation by Dr. Susanne Brander, researcher at Oregon State University considering the impacts of microplastic pollution on our environment.
Radioactive pollution occurs when radioactive materials are released into the environment unintentionally through activities like nuclear weapons production, mining radioactive ores, coal ash, medical waste disposal, and accidents at nuclear power plants. Exposure to radiation from these sources can cause DNA damage leading to cancers or other health issues depending on the level and duration of exposure. Prevention methods include proper disposal of nuclear waste, minimizing production of radioactive materials, and treating contaminated sites using bacteria found in distillery wastewater sludge that convert uranium into a less soluble and more removable form.
The document summarizes the tannery process, associated wastewater treatment, and impact on public health. It describes the various stages of tanning including soaking, liming, pickling, and tanning. This generates wastewater high in salts, chromium, sulfides, and organic matter. Primary treatment includes screening, equalization, coagulation, and sludge dewatering to remove solids and reduce BOD and COD. Effluent standards vary by country but are often exceeded by tanneries. Pollutants from tanneries like chromium, ammonia, and hydrogen sulfide can cause respiratory illness and dermatitis. Alternative treatment methods are evaluated based on cost, technical criteria, and
Fluoride occurs naturally in drinking water and is also added to promote dental health, though high levels can cause health issues. A study found fluoride in 23 of 25 water samples tested, all below EPA and WHO standards. Excess fluoride intake, especially during tooth development, can cause dental and skeletal fluorosis. Dental fluorosis causes tooth discoloration and pitting, while skeletal fluorosis damages bones and joints. Methods for removing fluoride include distillation, reverse osmosis, activated alumina filtration, and bone char carbon filtration, which can remove 90-98% of fluoride but require maintenance.
7. Mushroom ( poisioness) A Series of Presention By Mr Allah Dad Khan Master ...Mr.Allah Dad Khan
A Series of Presention By Mr Allah Dad Khan Master Trainer in Mushroom Technology
Former DG Agriculture Extension KPK , Visiting Professor the University of Agriculture Peshawar Pakistan
A case study on minamata disease-By Ritu Chauhanabhimicro19
Minamata disease was caused by mercury poisoning from industrial wastewater dumped into Minamata Bay from 1956-1968. Over 900 people died and thousands suffered from neurological symptoms like numbness, weakness, and loss of coordination. The disease drew attention to environmental pollution issues in Japan. After the victims were recognized, Minamata City took measures like environmental education, promoting sustainable practices, and honoring those affected to prevent future disasters and rebuild their community.
This document discusses marine pollution, including its sources and effects. It defines marine pollution as the discharge of waste substances into the sea, harming living resources and human health. Major pollutants include sewage, pesticides, plastics, metallic wastes, oil, sediment plumes from mining, heat, radioactive waste, and dredge spoils. These pollutants can damage ecosystems, kill wildlife, accumulate in food chains, and threaten human health if consumed from contaminated marine life. Prevention requires controlling pollution at sources, cleaning up waste, and raising awareness of the importance of protecting oceans to maintain Earth's chemical and biological balance.
The hazardous impact of microplastics in aquatic environments calls for more scientific research to understand their occurrence, effects and mitigation strategies. This work describes the analysis of primary and secondary microplastics using FTIR and FTIR microscopy techniques.
This document summarizes the current state of knowledge around microplastics and their trophic transfer in aquatic ecosystems. It outlines background information on microplastics and their sources. It then reviews several case studies that demonstrate trophic transfer of microplastics between invertebrates, fish, and top predators like seals. The studies found microplastics accumulate at higher trophic levels. The document concludes by identifying key knowledge gaps and recommending future research focus on effects of microplastics on human health and standardizing detection methods, while promoting efforts to reduce plastic use.
This document discusses various types of sea water pollution including chemicals, metals, radioactive substances, solid waste, oil, sewage, and agricultural runoff. Major pollutants released into the oceans include billions of tons of ballast water, trillions of gallons of sewage, millions of tons of oil and solid waste. Heavy metals like mercury, lead and copper released from human activities pose serious threats as they accumulate in the food chain. Pollution has severe negative effects on marine life and ecosystems as well as public health. Prevention of pollution through changes in human attitudes and behaviors is important since the impacts of pollution on the environment can be long-lasting and irreversible.
The document discusses sources and consequences of oil pollution. It describes various sources of oil pollution including natural seeps, sea-based sources like oil spills from tankers and pipelines, and land-based sources such as urban and industrial runoff. Sea-based sources are divided into accidental discharges from incidents and operational discharges from regular shipping activities. Land-based sources also include oil in untreated sewage and stormwater. The consequences of oil spills are extensive damage to wildlife from ingestion and coating of feathers/fur, as well as long-term pollution of sediments that can impact burrowing animals for decades. Cleanup and recovery of oil spills is challenging and dependent on various environmental factors.
This document discusses microplastic pollution and its potential threat to marine invertebrates and the food web. It defines microplastics and explains how they enter the marine environment. Studies found microplastics present in sediment samples from beaches around the world. Microplastics can be ingested by small organisms and transferred up the food chain, as demonstrated by studies showing zooplankton ingesting microplastics that were then found in mysid shrimp that ate the zooplankton. The document also discusses how microplastics can be taken up by blue mussels and transferred to shore crabs that eat the mussels, entering the intertidal food web.
This document discusses water pollution, including its definition, types of pollutants, sources, effects, and ways to prevent it. It defines water pollution as any physical or chemical change in water that harms humans or other organisms. The major types of pollutants are pathogens, inorganic materials, organic materials, suspended solids, and radioactive materials. Sources include sewage, agriculture, oil, erosion, and industrial/domestic waste. Effects are risks to human and animal health. Prevention methods center around proper waste disposal, reducing runoff, and recycling/reusing water.
Microplastics (MPs) are small plastic pieces with size less than 5mm that have entered and polluted the environment.
While many investigations including several critical reviews on MPs in the environment have been
conducted, most of them are focused on their occurrences in marine environment
Human activities such as burning coal, medical waste, and other materials containing mercury are major contributors to mercury pollution. Mercury enters the environment through air pollution and can harm human health, especially for fetuses and children. The largest source of mercury air pollution is coal-fired power plants, which emit around 70% of the mercury in the US. Capturing mercury from coal plant emissions is challenging given mercury's various chemical forms.
The Love Canal disaster involved a neighborhood in Niagara Falls, New York that was built on top of a former chemical waste dump containing over 21,000 tons of toxic waste. In the late 1970s, residents noticed high rates of cancer and birth defects, which were eventually traced to the chemical waste leaking into the soil and water supply from the dump located under the neighborhood. Over 800 families were evacuated and reimbursed for their homes after extensive legal battles and media coverage brought attention to the environmental and public health crisis.
Marine pollution and its effect on environmentPramoda Raj
This document discusses various types of marine pollution including oil pollution, eutrophication, conservative pollutants such as metals and halogenated hydrocarbons, thermal pollution, radioactive pollution, and litter and plastic pollution. It outlines sources of each type of pollution and their impacts on the marine environment such as oil spills harming ecosystems, nutrients from eutrophication causing algal blooms, metals and plastics bioaccumulating in marine organisms and entering the food chain, and litter entangling and being ingested by animals. The conclusion states that education on marine pollution through awareness, research, and dissemination is key to reducing pollution in oceans.
This slide presentation is part DYUTI 2010 preconference series. This slides discuss various environmental disasters. Prepared and Presented by Kochubaby Manjorran
The document discusses various types of marine pollution including plastic debris, oil pollution, eutrophication, acidification, thermal pollution, radioactive pollution, and underwater noise pollution. It outlines the sources, impacts and solutions for each type of pollution. The sources discussed include land-based sources like agricultural and municipal runoff, ocean dumping, oil spills, and deep sea mining. The impacts covered are effects on marine life, human health, marine activities, and water quality. Solutions proposed are prevention through regulations, cleanup methods like bioremediation, skimming, burning, and reducing plastic and radioactive waste through various recycling and disposal methods.
Radioactive pollution is defined as the release of radioactive substances or particles into the environment from human activities like nuclear weapon testing, nuclear power plants, or accidents. It can cause serious health effects like cancer due to radiation exposure and remains toxic for centuries. Sources include natural processes like radioactive minerals as well as human activities involving nuclear materials, weapons, power plants, and medical isotopes. Effects range from acute radiation sickness to long-term mutations and increased cancer risks. Monitoring, safe waste disposal, and prevention of leaks and accidents are important for controlling radioactive pollution.
Slides from the Deschutes Land Trust's Nature Night presentation by Dr. Susanne Brander, researcher at Oregon State University considering the impacts of microplastic pollution on our environment.
Radioactive pollution occurs when radioactive materials are released into the environment unintentionally through activities like nuclear weapons production, mining radioactive ores, coal ash, medical waste disposal, and accidents at nuclear power plants. Exposure to radiation from these sources can cause DNA damage leading to cancers or other health issues depending on the level and duration of exposure. Prevention methods include proper disposal of nuclear waste, minimizing production of radioactive materials, and treating contaminated sites using bacteria found in distillery wastewater sludge that convert uranium into a less soluble and more removable form.
The document summarizes the tannery process, associated wastewater treatment, and impact on public health. It describes the various stages of tanning including soaking, liming, pickling, and tanning. This generates wastewater high in salts, chromium, sulfides, and organic matter. Primary treatment includes screening, equalization, coagulation, and sludge dewatering to remove solids and reduce BOD and COD. Effluent standards vary by country but are often exceeded by tanneries. Pollutants from tanneries like chromium, ammonia, and hydrogen sulfide can cause respiratory illness and dermatitis. Alternative treatment methods are evaluated based on cost, technical criteria, and
Fluoride occurs naturally in drinking water and is also added to promote dental health, though high levels can cause health issues. A study found fluoride in 23 of 25 water samples tested, all below EPA and WHO standards. Excess fluoride intake, especially during tooth development, can cause dental and skeletal fluorosis. Dental fluorosis causes tooth discoloration and pitting, while skeletal fluorosis damages bones and joints. Methods for removing fluoride include distillation, reverse osmosis, activated alumina filtration, and bone char carbon filtration, which can remove 90-98% of fluoride but require maintenance.
7. Mushroom ( poisioness) A Series of Presention By Mr Allah Dad Khan Master ...Mr.Allah Dad Khan
A Series of Presention By Mr Allah Dad Khan Master Trainer in Mushroom Technology
Former DG Agriculture Extension KPK , Visiting Professor the University of Agriculture Peshawar Pakistan
This document provides information about Mycobacterium marinum, including its morphological, biochemical, and epidemiological characteristics. It causes opportunistic infections in humans and fish. In humans, it typically presents as slow-developing cutaneous lesions on areas exposed to contaminated water. In fish, it can cause chronic granulomatous disease. Diagnosis involves histopathology, staining, culturing of lesions, and PCR. Treatment involves antibiotics, sometimes with surgery. Prevention focuses on sanitation of aquariums and proper handling and cooking of fish.
This presentation explains about the concept of food intoxication. The toxins produced by the microbes in food (fungal) and toxins present in the food stuff were provided. The information about the diseases caused by such toxins were disclosed.
Secondary metabolites produced by fungi mycotoxine-190904175035.pdfrahul343481
The document discusses mycotoxins, which are toxic secondary metabolites produced by fungi. It notes that there are six major groups of mycotoxins: aflatoxins, ochratoxins, citrinin, ergot alkaloids, patulin, and Fusarium toxins. Mycotoxins can contaminate agricultural commodities like grains and cause health issues in humans and livestock. Preventing mycotoxin contamination is important for agriculture and food production.
This document discusses various types of marine toxins that can contaminate seafood and cause illness in humans. It describes toxins from fish poisoning including scombrotoxin, ciguatera, and tetrodotoxin poisoning. It also discusses various shellfish poisoning toxins including paralytic shellfish poisoning, diarrhetic shellfish poisoning, amnesic shellfish poisoning, and neurotoxic shellfish poisoning. Finally, it mentions toxins from contaminated water including brevetoxins, cyanobacteria, and Pfiesteria-like organisms that can cause illness.
Schistosoma Mansoni and Schistosoma japonicumAdib Dilshad
This document provides information about Schistosoma mansoni and Schistosoma japonicum, which are parasites that cause schistosomiasis. It details their discovery, distribution, life cycles, clinical manifestations, diagnosis, and treatment. S. mansoni causes intestinal schistosomiasis and is found widely in Africa and South America. S. japonicum causes more severe oriental schistosomiasis in Asia. Both have complex life cycles involving human and snail hosts. Chronic infection can lead to liver and intestinal damage. Diagnosis involves stool and blood tests to detect eggs or antibodies. Praziquantel is the treatment.
fungal diseases of fish(220705180075) 1.pptxDevPanda5
This document discusses several common fungal diseases that affect fish:
Saprolegniasis is the most common fungal infection in pet fish, usually affecting areas of injury. Branchiomycosis is a fungal infection of gill tissue that obstructs blood flow and causes gills to lose color. Ichthyophonus is a systemic fungal disease characterized by lesions on the skin and internal organs with no known cure. Dermatomycosis causes fine hair-like growths on infected areas and can cause fin erosion and hemorrhaging. Good management practices like water quality, nutrition, and handling are important for controlling fungal infections in fish.
This document discusses two genera of parasites, Leishmania and Trypanosoma, that cause diseases in humans. It describes the four major Leishmania pathogens, their life cycles involving sandflies, clinical manifestations including visceral leishmaniasis and cutaneous leishmaniasis, and methods of laboratory diagnosis. It also discusses the three major Trypanosoma pathogens, their life cycles involving reduviid bugs or tsetse flies, diseases caused including Chagas disease and sleeping sickness, clinical features, and diagnosis. The document provides detailed information on the pathogenesis, epidemiology, symptoms and laboratory identification of infections caused by these important parasites.
Natural toxins can be found in foods from three main sources:
1) Naturally occurring in plants and animals, such as cyanogenic glycosides in cassava which can release cyanide, and glucosinolates in cruciferous vegetables which can interfere with iodine uptake.
2) As contaminants, such as mycotoxins from mold growth on crops.
3) Formed during food processing, such as biogenic amines produced by microbial decarboxylation of amino acids in fermented or aged foods.
Chronic ingestion of many natural toxins is generally safe but some can cause acute toxicity, carcinogenicity, or other health issues depending on amount and individual susceptibility
This document provides an overview of medical entomology, which is the study of the relationship between arthropods like insects and arachnids, and their ability to transmit pathogens or parasites that cause disease in humans. It discusses how arthropods can directly cause issues like allergic reactions, annoyance, or myiasis. Additionally, it explains how some arthropods act as vectors, undergoing part of the life cycle of a pathogen and transmitting it between hosts. Knowing the taxonomy, biology and ecology of vectors is important for developing control strategies. As medical professionals, understanding insect-human contact and identifying common vectors can help in treating patients and controlling diseases.
The 1971 Iraq poison grain disaster was caused by grain treated with methylmercury fungicide being imported and used to make bread, poisoning over 6,500 people and killing 459. Methylmercury is a toxic form of mercury that forms when bacteria react with mercury in plants, soil or water. It was used to treat grain but warnings were not understood, and the dyed grain was washed, removing the warnings but not the toxins. Symptoms included numbness, loss of balance, blindness and death from nervous system damage. Over 40,000 were poisoned, with long term effects continuing due to mercury's long half life in the brain.
Toxic seafood ingestions can cause a variety of illnesses. Ciguatera fish poisoning results from eating reef fish contaminated with ciguatoxins and causes gastrointestinal and neurological symptoms. Scombroid poisoning is caused by histamine accumulation in improperly stored fish like tuna and mahi-mahi, resulting in flushing, rash, and headache. Paralytic shellfish poisoning from eating toxic mollusks can progress to paralysis and respiratory failure due to saxitoxin. Treatment focuses on supportive care and respiratory support.
Detectable in almost all internal organs,
Organs rich in adipose tissue are storage depots from which hydrocarbons are gradually released
Gastrointestinal tract contains high levels of hydrocarbon and metabolites, even when PAH are administered by other routes, as a result of mucociliary clearance and swallowing or hepatobiliary excretion (IPCS, 1998)
Tularemia, also known as “rabbit fever,” is a disease caused by the bacterium Francisella tularensis. Tularemia is typically found in animals, especially rodents, rabbits, and hares. Tularemia is usually a rural disease and has been reported in all U.S. states except Hawaii.
Tularemia is a zoonotic infectious disease caused by the bacterium Francisella tularensis. It was first identified in 1911 in Tulare County, California and can be transmitted to humans through tick or deer fly bites, handling infected animals, inhaling contaminated materials, or ingesting contaminated food or water. Symptoms vary depending on the route of infection but may include fever, fatigue, headache, and muscle aches. While treatment involves antibiotics, prevention focuses on avoiding contact with wild animals and insects and properly handling/cooking meat. Currently no approved vaccine exists for human use.
Integrating Ayurveda into Parkinson’s Management: A Holistic ApproachAyurveda ForAll
Explore the benefits of combining Ayurveda with conventional Parkinson's treatments. Learn how a holistic approach can manage symptoms, enhance well-being, and balance body energies. Discover the steps to safely integrate Ayurvedic practices into your Parkinson’s care plan, including expert guidance on diet, herbal remedies, and lifestyle modifications.
Basavarajeeyam is an important text for ayurvedic physician belonging to andhra pradehs. It is a popular compendium in various parts of our country as well as in andhra pradesh. The content of the text was presented in sanskrit and telugu language (Bilingual). One of the most famous book in ayurvedic pharmaceutics and therapeutics. This book contains 25 chapters called as prakaranas. Many rasaoushadis were explained, pioneer of dhatu druti, nadi pareeksha, mutra pareeksha etc. Belongs to the period of 15-16 century. New diseases like upadamsha, phiranga rogas are explained.
- Video recording of this lecture in English language: https://youtu.be/kqbnxVAZs-0
- Video recording of this lecture in Arabic language: https://youtu.be/SINlygW1Mpc
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Rasamanikya is a excellent preparation in the field of Rasashastra, it is used in various Kushtha Roga, Shwasa, Vicharchika, Bhagandara, Vatarakta, and Phiranga Roga. In this article Preparation& Comparative analytical profile for both Formulationon i.e Rasamanikya prepared by Kushmanda swarasa & Churnodhaka Shodita Haratala. The study aims to provide insights into the comparative efficacy and analytical aspects of these formulations for enhanced therapeutic outcomes.
8 Surprising Reasons To Meditate 40 Minutes A Day That Can Change Your Life.pptxHolistified Wellness
We’re talking about Vedic Meditation, a form of meditation that has been around for at least 5,000 years. Back then, the people who lived in the Indus Valley, now known as India and Pakistan, practised meditation as a fundamental part of daily life. This knowledge that has given us yoga and Ayurveda, was known as Veda, hence the name Vedic. And though there are some written records, the practice has been passed down verbally from generation to generation.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of the physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar lead (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
6. Describe the flow of current around the heart during the cardiac cycle
7. Discuss the placement and polarity of the leads of electrocardiograph
8. Describe the normal electrocardiograms recorded from the limb leads and explain the physiological basis of the different records that are obtained
9. Define mean electrical vector (axis) of the heart and give the normal range
10. Define the mean QRS vector
11. Describe the axes of leads (hexagonal reference system)
12. Comprehend the vectorial analysis of the normal ECG
13. Determine the mean electrical axis of the ventricular QRS and appreciate the mean axis deviation
14. Explain the concepts of current of injury, J point, and their significance
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. Chapter 3, Cardiology Explained, https://www.ncbi.nlm.nih.gov/books/NBK2214/
7. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
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Overall life span (LS) was 1671.7±1721.6 days and cumulative 5YS reached 62.4%, 10 years – 50.4%, 20 years – 44.6%. 94 LCP lived more than 5 years without cancer (LS=2958.6±1723.6 days), 22 – more than 10 years (LS=5571±1841.8 days). 67 LCP died because of LC (LS=471.9±344 days). AT significantly improved 5YS (68% vs. 53.7%) (P=0.028 by log-rank test). Cox modeling displayed that 5YS of LCP significantly depended on: N0-N12, T3-4, blood cell circuit, cell ratio factors (ratio between cancer cells-CC and blood cells subpopulations), LC cell dynamics, recalcification time, heparin tolerance, prothrombin index, protein, AT, procedure type (P=0.000-0.031). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and N0-12 (rank=1), thrombocytes/CC (rank=2), segmented neutrophils/CC (3), eosinophils/CC (4), erythrocytes/CC (5), healthy cells/CC (6), lymphocytes/CC (7), stick neutrophils/CC (8), leucocytes/CC (9), monocytes/CC (10). Correct prediction of 5YS was 100% by neural networks computing (error=0.000; area under ROC curve=1.0).
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
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2. HISTORY OF OCCURRENCE OF
MINAMATA DISEASE
•Minamata is a small town facing
Shiranui sea in Kumamoto
Perfecture on Kyushu Island in
southern Japan.
•1956 Spring a 5 year old girl was
found with neurological symptoms.
She had convulsions and difficulty
in speaking and walking.
She was the first well documented
case of Minamata disease and was
officially reported with 3 other
cases including her sister on May
1st 1956.
Minamata disease sometimes
3. FINDING THE CAUSE
•Reseachers from Kumamoto University found
the staple food od victims was invariably fish
and shellfish from manamata bay.
•The cats in the local area which tended to eat
scarps from under the tables died with
similar symptoms those in dying humans.
•The led researches to believe that outbreak
was caused by some kind of food poisoning
with fish and shellfish.
•November 4th 1956 researches officially
informed that initial findings : “Minamata
disease is rather considered to be poisoning
by heavy metal presumably enters human
body mainly through fish and shellfish”.
4. CHISSO CORPORATION’S CHEMICAL
FACTORY
•It was caused by the release of methylmercury in
the industrial wastewater from the Chisso
Corporation’s chemical factory, which continued
from 1932 to 1968.
•This highly toxic chemical bioaccumulated and
biomagnified in shellfish and fish in Minamata
Bay and the Shiranui Sea, which, when eaten by
the local population, resulted in mercury
poisoning.
•Minamata disease has not been confined to
Minamata where the source of the mercury was
primarily from eating fish caught in the
contaminated Bay.
•Other sources of maternal exposure to methyl
mercury have included flour made from seed
grain treated with methyl mercury (which affected
5. WHAT IS MINAMATA DISEASE?
Minamata disease is a
methylmercury poisoning associated
with daily consumption of large
quantities of fish and shellfish
heavily contaminated with the toxic
chemical.
The disease shows variety of clinical
symptoms depending on the
exposure level to chemical.
When its Sever its knows as Hunter-
Russell syndrome.
6. SIGNS AND SYMPTOMS OF
MINAMATA DISEASE
• Ataxia (lack of muscle
coordination on voluntary
movements)
• Numbness in hands and feet
• Loss of peripheral vision
• Damage to hearing and
speech
• In extreme cases, insanity,
paralysis, coma, and death
follow within weeks of the
onset of symptoms.
• A congenital form of the
7.
8. TREATMENT OF MINAMATA
•No fundamental cure for manamata disease yet been discovered
so treatment includes helping the patient with the painful
symptoms.
•The most important aspect is the removal of mercury from the
body using chelating agents.
•Physical rehabilitation for the patients with loss of muscle
function and paralysis this helps some patients to regain little
amount of control over mobility.
•In some cases patients suffer form convulsions in which case
doctors will give anticonvulsant drugs.