This document provides an overview of local anaesthesia techniques for children. It discusses definitions of local anaesthesia and various techniques including surface anaesthesia, infiltration, nerve blocks, and recent advances. It covers local anaesthetic solutions, pharmacological and non-pharmacological pain control methods, and complications of local anaesthesia such as allergic reactions and toxicity when using local anaesthetics in children. The goal is to provide effective pain control while minimizing risks for paediatric dental procedures.
brief description on posterior superior alveolar nerve block.
its uses in dentistry, technique and action. locating PSA nerve is easy and this is the most used nerve block in dentistry.
Smear layer is a controversial topic in the field of operative dentistry and endodontics. This presentation includes composition, concepts, structure, advantages, disadvantages, and removal methods of smear layer.
Dental Management of a Medically Compromised Patients - Presented by Dr. Shweta and Parray as a part of Dhaka Dental COllege OMS Department Weekly Presentation Program
Non –pharmacological behavior management in childrenDr. Harsh Shah
Overview on nonpharmacological managent of behaviour in children
Presented by : Mayuri Karad
SDDCH Parbhani
Guided by : Dr. Rehan Khan
Dept, of Pediatric and preventive dentistry
brief description on posterior superior alveolar nerve block.
its uses in dentistry, technique and action. locating PSA nerve is easy and this is the most used nerve block in dentistry.
Smear layer is a controversial topic in the field of operative dentistry and endodontics. This presentation includes composition, concepts, structure, advantages, disadvantages, and removal methods of smear layer.
Dental Management of a Medically Compromised Patients - Presented by Dr. Shweta and Parray as a part of Dhaka Dental COllege OMS Department Weekly Presentation Program
Non –pharmacological behavior management in childrenDr. Harsh Shah
Overview on nonpharmacological managent of behaviour in children
Presented by : Mayuri Karad
SDDCH Parbhani
Guided by : Dr. Rehan Khan
Dept, of Pediatric and preventive dentistry
Presentation on local anaesthetics - Chandragiri Siva sai
Includes: Introduction, Classification, Mechanism of action, Duration of action, side effect and different phenomena of anesthetic agent.
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
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MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
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Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
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Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
1. LOCAL ANAESTHESIA
FOR CHILDREN
Dr. Mutyala Jhansi(JR-1)
Department of pediatric and preventive
dentistry
King george’s medical university
2. CONTENTS
• Introduction
• Surface anaesthesia
• Non Pharmacological pain control
• Local anaesthetic solutions
• Techniques of local anaesthesia
• Recent advances in LA
• Complications of LA
• Contraindications to LA
• References
4. SURFACE ANAESTHESIA
Surface
anaesthesia
Physical
method
Application of volatile liquids such as ethyl chloride.
The latent heat of evaporation of this material reduces
the temperature of the surface tissue and this produces
anaesthesia.
Rarely used in children.
Pharmacological
method(topical
anaesthetics)
5. INTRA-ORAL TOPICAL AGENT
• The success depends on the technique.
• Ethyl aminobenzoate (benzocaine), butacaine sulfate, cocaine,
dyclonine, Lidocaine are used.
Time of
application
-5min
Anesthetise
2-3 mm depth
Spray,
solution ,
cream &
ointment
7. TOPICAL ANAESTHETICS FOR SKIN
• EMLA cream (5% Eutectic mixture of the prilocaine and lidocaine)
• Clinical trial of intra oral use of EMLA shown to be more effective
then conventional local anaesthesia
9. • It is shown to be effective in providing symptomatic pain
relief.
ELECTROANALGESIA(TENS)
10. MECHANISM
Acute pain
Threshold for electrical
stimulation Large
myelinated nerve fiber
smaller is less than
unmyelinated pain fibers
Stimulation of these fibres
by the current from the
TENS machine closes the
‘gate’ to central
transmission of the signal
from the pain fibres.
Chronic
pain
Where the release of
endogenous painkillers
such as β-endorphins is
stimulated
11. • HYPNOSIS It can be used as an adjuctive to LA in children and most
effective in young children by decreasing the pulse rate and the
incidence of crying.
• RADIOWAVES high frequency waves are released which inturn
causes the release of endorphins.
ADVANTAGES
1) Systemic toxicity
2) Chance of self-inflected trauma
12. LOCAL ANAESTHETIC SOLUTIONS
• A number of LA solutions lasting from 10mins to 6 hrs are
available.
2%
LIDOCAINE
+
ADRENALINE
17. METHODS OF LA ADMINISTRATION
Infiltration anaesthesia
Regional block anaesthesia
Intra-ligamentary anaesthesia
Intra osseous, inter septal and intrapulpal
18. INFILTRATION ANAESTHESIA
• Method of choice in the maxilla.
• Infiltration of 0.5 to 1.0ml of local
anaesthetic is sufficient for pulpal
anaesthesia
• The objective is to deposit LA solution as
close as possible to the apex of tooth of
interest ( supra-periostelly).
19. • Buccal infiltration in the mandible is reliable for pulpal anaesthesia of
primary teeth and unreliable on permanent teeth, with exception of
lower incisors teeth (jaber et al. 2010)
• Oulis and association compared the effectiveness of mandibular
infiltration anaesthesia with mandibular block. Results reported that
manibular infiltration was less effective than mandibular block for
pulpotomy and extraction (p=0.05)
20. REGIONAL BLOCK ANAESTHESIA
INFERIOR ALVEOLAR AND LINGUAL NERVE BLOCKS
Administration of this blocks are easier to perform successfully
in children than adults (because of the relative position
mandibular foramen to the occlusal level)
Best performed with child’s mouth fully open.
21. LANDMARKS
Mucobuccal fold
Anterior border of ramus of the mandible
External oblique ridge
Retromolar triangle
Internal oblique ridge
Pterygomandibular liigament
Buccal sucking pad
Pterygomandibular space
22.
23. LINGUAL NERVE BLOCK
• The lingual nerve is blocked by withdrawing the needle halfway,
aspirating again, and depositing most of the remaining solution at
this point.
• The final contents of the cartridge are expelled as the needle is
withdrawn through the tissue.
• A common fault is to contact bone only a few millimeters following
insertion.
• This lead to unsuccessful anaesthesia, occur because the angle of
entry is too obtuse
24. LONG BUCCAL, MENTAL AND INCISIVE
NERVE BLOCKS
LONG BUCCAL INJECTIONS usually equates to a buccal infiltration in
children.
MENTAL AND INCISIVE NERVE BLOCKS readily administered in children
as the orientation of the mental foramen is such that it faces forward
rather than posteriorly as in adult.
25. • Blocked of transmission in the mental nerve provides
excellent soft tissue anaesthesia.
• The method of choice for pulpal anaesthesia in the
permanent lower incisors is a combination of buccal and
lingual infiltration.
26. MAXILLARY NERVE BLOCK
• Regional block techniques are seldom required in a child’s
maxilla .
• Infraorbital, Greater palatine and nasopalatine nerve blocks
INFILTRATION
27. INTRA-LIGAMENTARY ANAESTHESIA
• This is a method of intra osseous injection with LA reaching the
cancellous space in the bone via the periodontal ligament.
• The recommended dose per root is 0.2ml.
• Advantage Reduces the occurence of self mutilation of lip & tongue.
29. • It is important not to inject too quickly:
about 15 sec per depression of the
specialized syringe lever is needed.
Wait for
5sec before
needle
withdrawal
30. INTRA-OSSEOUS, INTER-SEPTAL AND
INTRAPULPAL INJECTION
• INTRA PULPAL it often provides the desired anaesthesia, but
the technique has the disadvantage of being initially painful,
although the onset of anaesthesia is usually rapid.
31. • INTRAOSSEOUS INJECTION techniques(of which interseptal injection
is one type)require the deposition of LA solution in the porous
alveolar bone.
32. • This technique is not particularly difficult in children because
their cortical bone is less dense than that of adult.
INDICATION
• This method is useful when the use of periodontal injection is
contraindicated due to periodontal ligament space infection.
34. A. COMPUTERED-CONTROLLED LA DELIVERY
SYSTEM (WAND)
• The system includes conventional LA needle and a disposable
wand-like syringe held by a pen grasp.
35. • PRINCIPLE: To deliver local anesthetic solution at a constant
rate and slower speed to avoid causing discomfort to the
patient
• The system includes an aspiration cycle for use when
necessary.
• There are three modes of flow rate available: slow, fast and
turbo mode.
• Block, infiltration, palatal and periodontal ligament injections
are all reported to be more comfortable for the patient with
the wand than conventional injection techniques.
36. COMFORT CONTROL SYRINGE
• It was marketed as an alternative to the Wand and has two
components; base unit and syringe no foot pedal.
• The most important functions of this unit is injection and
aspiration can be controlled directly from the syringe.
37. • Five different rate settings for specific applications like block,
infiltration, PDL, IO and Palatal regions.
• The unit uses two stage delivery rates for every injection.
• It initially expresses the LA solution at an extremely low rate and
after 10 seconds the rate slowly increases to the pre-
programmed value for the selected injection technique.
• Disadvantage The syringe is bulky and cumbersome to use when
compared to the wand hand piece.
38. SINGLE-TOOTH ANESTHESIA [STA]
• In 2006, the manufacturers of the original CCLAD, introduced a
new device, Single Tooth Anesthesia (STA)
• PRINCIPLE It incorporates dynamic pressure-sensing (DPS)
technology that provides a constant monitoring of the pressure of
local anaesthetic solution during the drug administration.
39. • Since the pressure of the LA is strictly regulated by the STA system,
a greater volume of LA can be administered with increased comfort
and less tissue damage
• It has 3 modes for rate of injection: STA mode, normal mode and
turbo mode.
40.
41. NEW INJECTION TECHNIQUES
• With the development of CCLAD 2 new injection techniques have
evolved
1. Anterior middle superior alveolar nerve block (AMSA)
2. Posterior approach to anterior superior alveolar nerve block
(P-ASA)
• Though either may be administered with a traditional local
anesthetic syringe.
42. • For the AMSA, you slowly inject in the area between the
maxillary premolars and the palatal suture. You will see a
blanching of the palatal tissues. This will give you anesthesia
from second premolar to central incisor.
43. B. JET INJECTORS
• PRINCIPLE It is based on the principle of using a mechanical
energy source to create a pressure ,sufficient to push a liquid
medication through a very small orifice so that it can penetrate
into the subcutaneous tissues without a needle.
• The solution is injected through orifice which is 7 times smaller
than the smallest available needle.
44. ADVANTAGES
a. Painless injection
b. Less tissue damage
c. Faster injection
d. Faster rate of drug absorption into the tissues
e. Successful in children with bleeding diatheses.
45. DRAWBACKS
a. Expensive equipments
b. Specialized syringes
c. Can’t be used for nerve blocks, only infiltration and surface
anesthesia are possible.
46. C.INTRA-OSSEOUS ANESTHESIA
• Stabident, an Intraosseous Injection delivery system has a
disadvantage that it can be used only in visible and readily
accessible area because while giving intraoral injection once the
perforator is withdrawn, it can be extremely difficult to locate the
perforation site with the anesthetic needle.
STABIDENT
47. • X-Tip uses the pilot drill which is a hollow tube through which a
27-gauge needle can pass.
• The initial drill stays in place, allowing the anesthetic to be placed
without hunting for the perforation that was just created.
X-TIP
48. • IntraFlow anesthesia system uses a single-step method which
allows entry into the penetration zone, injection, and
withdrawal in one continuous step, without the need to
relocate the perforation site.
• Reemers et al. reported that the IntraFlow system as a
primary technique provide reliable anesthesia of posterior
mandibular teeth compared with an inferior alveolar nerve
block.
INTRAFLOW
49. D. VIBROTACTILE DEVICES
• These devices work on the principle of ‘gate control’
theory thereby reduces pain.
VIBRAJECT
Vibraject has a battery operated device which is
attached to the standard anaesthetic syringe, causing
the syringe and needle apparatus to vibrate
50. DENTAL VIBE
• Dental Vibe is a cordless hand held device which gently stimulates
the sensory receptors at the injection site causing the neural pain
gate to close.
• The tissues are vibrated before the needle penetration.
DISADVANTAGE
it is not directly attached to the syringe
and a separate unit is required,
so both hands are engaged.
51. ACCUPAL
• Accupal is a cordless device which applies both vibration and
pressure at the injection site.
52. E. SAFETY DENTAL SYRINGES
• It prevent the risk of accidental needle stick injury
occurring with a contaminated needle after local
anaesthesia administration.
• These syringes possess a sheath that locks over the
needle when it is removed from the patient’s tissues
preventing accidental needle stick injury.
53.
54. F. DENTIPATCH [INTRAORAL LIGNOCAINE
PATCH]
• Dentipatch contains 10-20% lidocaine, which is
placed on dried mucosa for 15 minutes.
• Disadvantages include central nervous system and
cardiovascular system complications.
55. CONTENTS OF LA
INGREDIENT FUNCTION
• L A agent conduction blocked
• Vasoconstrictor LA absorption into blood
• Sodium metabisulfite antioxident
• Methylparaben preservative, bacteriostatic
• Sodium chloride isotonicity of solution
• Sterile water diluent
57. GENERALIZED COMPLICATIONS
PSYCHOGENIC
• The most common psychogenic complication of LA is fainting.
• MANAGEMENT- sympathetic management and supine
position with legs slightly elevated.
58. ALLERGY
• Very rare complication.
• Allergy can manifest in a verity of forms, ranging from a minor
localized reaction to the emergency of anaphylatic shock.
• If any suggestion that a child is allergic to LA they should be referred
to local dermatology or clinical pharmacology department.
• Taken advice for which alternative LA can be safely given to the child.
59. TOXICITY
• Overdosage of LA leading to
toxicity is rarely a problem in adult
but can occur In children.
• Doses which are well below toxic
level in adult can produce
problem in children.
• All the drugs , dosages should be
related to body weight.
60. CLARK'S RULE
• Clark's Rule uses Weight in Lbs
FORMULA
Adult Dose X (Weight ÷ 150) = Childs Dose
Example
11 year old girl / 70 Lbs
500mg X (70 ÷ 150) = Child's Dose
500mg X .47 = 235mg
61. YOUNG'S RULE
• Youngs Rule uses age.
FORMULA
Adult Dose X (Age ÷ (Age+12)) = Child's Dose
DOSAGE BASED ON WEIGHT
• Based on weight in kgs.
• Example:
The prescription calls for 5mg per kg
20 x 5mg = 100mg
62. CARDIOVASCULAR
EFFECTS
• Cardiovascular effects caused by the combined action of the
anaesthetic agent and vasoconstrictor.
• Their direct action on cardiac tissue and the peripheral vasculature.
• Indirectly via inhibition of the autonomic nerves that regulate
cardiac and peripheral vascular function.
63. CNS EFFECTS
• The CNS is not immune to local anaesthetic agent.
• At low doses the effect is excitatory as CNS inhibitory fibers are
blocked.
• At high doses the effect is depressant and can lead to
unconsciousness and respiratory arrest.
• Fatalities due to LA overdose in children are generally the result
of central nervous tissue depression.
64. METHAEMOGLOBINAEMIA
• Prilocaine causes cyanosis due to
methaemoglobinaemia.
• In this the ferrous iron of normal
haemoglobin is converted to the ferric
form, which cannot combine with
oxygen.
65. TREATMENT OF TOXICITY
• The best treatment of toxicity is prevention; aided by
1. Aspiration
2 . Slow injection
3. Dose limitation.
66. • When toxic reaction occurs, the procedure is as follows:
1. Stop the dental treatment
.
2. Provide basic life support.
3. Call for medical assistance.
4. Protect the patient from injury.
5. Monitor vital signs
67. DRUG INTERACTIONS
• Apparently innocuous drug combinations can interact and cause
significant problem in children.
• Example, an episode of methaemoglobinaemia has been
reported in a 3 months old child following the application of
EMLA.
• It was concluded in this case that prilocaine(in EMLA) had
interacted with a sulfonamide that the child was already
receiving.
68. INFECTIONS
• The introduction of agent capable of producing a generalized
infection, such as human immunodeficiency virus(HIV)
infection and Hepatitis is a complication that should not occur
when appropriate cross-infection control measures are
employed.
69. LOCALIZED COMPLICATIONS
NEEDLE BREAKAGE
• Most common with IAN block and then with PSA block.
CAUSES
1. Weakening of needle by bending
2. Unexpected movements by patient
3. Smaller gauge needles
71. PARESTHESIA
• It is defined persistent anaesthesia or altered sensation well
beyond the expected duration of anaesthesia.
CAUSES
1. Trauma to any nerve or nerve sheath.
2. LA solution contaminated by alcohol.
3. Hemorrhage
72. MANAGEMENT
1. Be reassuring the pt, explain that it is not uncommon.
2. It normally persists for at least 2 months and may last upto 1
year.
3. Consultation with an oral surgeon or neurologist still the
sensory deficit is evident after 1 yr.
4. Dental treatment may continue.
73. FACIAL NERVE PARALYSIS
• It occurs when anaesthetic introduced into the deep lobe of
parotid gland.
74. • It lasts no more than several hours depending on the LA
formulation, volume injected and proximity to the facial
nerve.
• Primary problem associated is persons face appears lopsided,
Unable to voluntarily close one eye.
75. MANAGEMENT
1. Reassure the patient, explain the situation is transient.
2. Contact lens should be removed.
3. An eye patch should be applied to the affected eye.
4. No contraindication for reanaesthetizing the pt.
76. TRISMUS
• It is a prolonged tetanic spasm of the jaw muscle(locked jaw).
CAUSES
1. Most common etiologic factor is trauma to muscle or blood
vessels.
2. LA solution contaminated with alcohol.
3. Hemorrhage
4. Low grade infections after injection.
5. Multiple needle penetrations.
77. MANAGEMENT
• Heat therapy
• Warm saline rinses
• Analgesic
• Muscle relaxants
• Initiate physiotherapy
• Complete recovery may take about 6 weeks (4-20weeks)
• Surgical intervention to correct chronic dysfunction.
78. SOFT TISSUE INJURY
• Lip and tongue are the most frequent sites involved.
• Caused by biting and chewing these tissues while still
anaesthetized.
79. • Trauma to anaesthetized tissue can lead to swelling and
significant pain when the anaesthetic effect resolves.
PREVENTION
• A cotton roll can be placed between the lip and teeth if they
are still anaesthetized at the time of discharge.
80. MANAGEMENT
It involves symptomatic treatment:
1. Analgesics for pain
2. Antibiotics
3. Lukewarm saline rinses
4. Petroleum jelly or other lubricants to minimize irritation.
81. HEMATOMA
• The effusion of blood into extravascular spaces.
• Hematoma after the nicking of artery increases rapidly in size
then vein.
• Size also depends on the density of the surrounding tissue.
82. PROBLEM
• Complications include trismus and pain.
• Discoloration and swelling subside within 7-14 days.
• Hematoma associated with PSA block can be avoided by using
shorter needles.
MANAGEMENT
Immediate
• Direct pressure applied to the site of bleeding.
• Pressure applied should not be less then 2mins.
83. • IAN block pressure applied to the medial aspect of the
mandibular ramus.
• Infraorbital pressure applied to the skin over the foramen.
• Mental block pressure applied on mucosa or skin over the
foramen.
• PSA block apply pressure in medial and superior direction.
84. Subsequent
• Advice the pt not to apply heat for at least 4-6 hrs.
• Ice may be applied to the region immediately on recognition
of developing hematoma( analgesic and vasoconstrictor).
• With or without treatment hematoma will present for 7-14
days.
85. PAIN ON INJECTION
CAUSES
• Careless injection techniques
• Use of dull needles
• Rapid deposition of LA
• Needles with barbs
86. PROBLEM
• Increased pt anxiety lead to sudden movements , risk of
needle breakage.
• No management is necessary.
87. BURNING ON INJECTION
CAUSES
• Primary cause of mild burning sensation is the pH of LA
solution.
• Rapid injection of LA
• Contamination of LA cartridges
• Solutions warmed to normal body temperature.
No treatment ,because it is transient and do not lead to
prolonged tissue involvement.
88. INFECTION
CAUSES
• Contamination of the needle.
• Injecting LA solution into an area of infection.
MANAGEMENT
• Pts usually reports postinjection pain and dysfunction 1 or
more days after dental care.
• Keep the pt on anatibiotics for about 7-10 days.
90. PROBLEM
• Edema is intense enough to produce airway obstruction.
MANAGEMENT
• Edema caused by traumatic injection or irritating solutions it
resolves in several days without formal treatment .
• After hemorrhage 7-14 days
• Edema by infection does not resolve but may become more
progressively intense, antibiotic therapy should be instituted.
• Allergy induced edema is potentially life threatening.
91. SLOUGHING OF TISSUES
• Prolonged irritation or ischemia of gingival soft tissue.
CAUSES
1. Epithelial desqumation
2. Sterile abscess
92. MANAGEMENT
• Reasure the patient
• Symptomatic treatment: for pain aspirine or codeine and a
topically applied ointment(orabase) to reduce the irritation.
• Epithelial desquamation resolves within few days.
• Sterile abscess may run 7 to 10 days
93. POSTANAESTHETIC INTRAORAL LESIONS
• This is the latent form of the disease process that was present
in the tissue before the injection.
• Patient report approximately 2 days after intraoral injection.
CAUSES
• Recurrent aphthous stomatitis
• Herpes simplex
95. CONTRAINDICATIONS OF LA
• In certain children some LA agents will be contraindicated and
in others, specific techniques are not advised.
GENERAL CONTRAINDICATIONS
• Immaturity
• Mental or physical handicap
• Treatment factors
96. SPECIFIC CONTRAINDICATIONS
BISULFITE ALLERGY
• All esters and vasoconstrictor drugs (absolute)
• Amide group without vasoconstrictor
ATYPICAL PLASMA CHOLINESTERASE
• Esters (relative)
• Amide
98. CARDIOVASCULAR AND HYPERTHYROIDISM
• High concentration of vasoconstrictors (relative)
• LA with epinephrine conc. Of 1:100,000 or 1:200,000 or
mepivacaine 3% or prilocaine 4%
RENAL DYSFUNCTION
• Amides or esters (relative)
• Amides or esters
99. POOR BLOOD SUPPLY
• Vasoconstrictor containing LA solutions like after
therapeutic irradiation.
SPECIFIC TECHNIQUES
• Bleeding diatheses
• Incomplete root formation
• Epilepsy : Therapeutic dosages do not interact with standard
antiepileptic drugs.
Electro-analgesia
100. CONCLUSION
When pain free reliable local anaesthesia is achieved
in children confidence is gained by both the child and
operator, and a sound satisfactory professional
relationship is established.
101. References
1. Text book of Pediatric dentistry, 4th edition,
Richard Welbury.
2. Text book of Dentistry for the child and
adolescent, 1st asia edition, Jeffery A. Dean.
3. Text book of Local anaesthesia, 7th edition,
Richard C. Bennet.
4. Text book of Local anaesthesia, malamed, 5th
edition, stanely F. malamed.
5. Hochman MN, Chiarello D, Hochman CB,
Lopatkin R, Pergola S. Computerized Local
Anesthesia Delivery vs. Traditional Syringe
Technique. NY State Dent J. 1997;63:24-9.
6. Ferrari M, Cagidiaco MC, Vichi A, Goracci C.
Efficacy of the Computer-Controlled Injection
System STATM, and the dental syringe for
intraligamentary anesthesia in restorative
patients. Inter Dent SA. 2008;11(1):4-12.
102. 7. Friedman MJ, Hochman MN. P-ASA block injection: a new palatal technique to
anesthetize maxillary anterior teeth. J Esthet Dent. 1999;11(2):63-71. 6.
8. Ran D, Peretz B. Assessing the pain reaction of children receiving periodontal
ligament anesthesia using a computerized device (Wand). J Clin Pediatr Dent.
2003;27(3):247-50.
9. Remmers T, Glickman G, Spears R, He J. The efficacy of IntraFlow intraosseous
injection as a primary anesthesia technique. J Endod 2008;34:280-3.
10. Nanitsos E, Vartuli R, Forte A, Dennison PJ, Peck CC: The effect of vibration on
pain during local anaesthesia injections. Aust Dent J 2010, 54:94-100.
11. Blair J. Vibraject from ITL dental. Dent Econ. 2002;92:90
12. Ogle OE, Mahjoubi G. Advances in local anesthesia in dentistry. Dent Clin
North Am 2011;55:481-99.
103. 1. The most effective topical anaesthetic is
a. Lignocaine
b. Tetracaine
c. Ethyl amino benzoate
d. Dyclonine
104. 2. Jet injection was introduced by
a. Figge and Scherer (1947)
b. Schroeder (1948)
c. Mckay (1952)
d. Frank (1966)
105. 3. Gow gates mandibular block technique
anaesthetizes all EXCEPT
a. mandibular molars
b. Mylohyoid
c. Premolar
d. Mandibular incisors
106. 4. Which of the following are advantages of intra
ligamentary injection
a. provides reliable pain control rapidly and easily
b. It provides pulpal anaesthesia for 30-45 minutes
c. it may be useful in young or disabled patients
where the postoperative trauma is common
d. useful in patients with bleeding disorders
e. all of the above
107. 5. The maximum dose of lignocaine which can
be administered
a. 4.4 mg/kg body weight
b. 2 mg/kg body weight
c. 6.4 mg/kg body weight
d. 2 gm/kg body weight