IHD also known as coronary artery diseases is a condition in which there is inadequate supply of blood and oxygen to a portion of myocardium. Imbalance between myocardial oxygen supply and demand causes Angina, MI, Hear failure, and Arrhythmia
Acute heart failure (AHF) is defined as rapid onset of new or worsening signs and symptoms of heart failure. It represents a life-threatening condition requiring treatment for fluid overload and hemodynamic compromise. Presentation may be initial diagnosis with symptoms and signs of AHF or acute decompensation of pre-existing cardiomyopathy. Hemodynamic instability results from disorders of the myocardium, valves, conduction system or pericardium, in isolation or combination. Potentially treatable causes, e.g. acute coronary syndromes, must be diagnosed and managed early for restoration of function.
Physiological changes associated with AHF result in reduced cardiac output and end-organ hypoperfusion. Once potentially treatable causes are managed, stratification of patients by clinical presentation guides further therapeutic intervention. AHF patients can be categorized as either ‘wet’ or ‘dry’ by clinical fluid status assessment, and either ‘cold’ or ‘warm’ according to perfusion status. In combination, these features identify four patient groups (‘warm-wet’, ‘warm-dry’, ‘cold-dry’, ‘cold-wet’) that guide therapy and facilitate prognostication. ‘Warm-dry’ patients rarely require intensive care for AHF treatment but may benefit from escalation of oral therapeutic regimen. Patients who examine as ‘cold-dry’ may benefit from fluid challenge, and/or inotropic agent infusion. ‘Warm-wet’ patients present with predominantly congestive or hypertensive symptoms which benefit from diuresis and vasodilatation. Patients who present ‘wet-cold’ with normal blood pressure (SBP >90) may benefit from vasodilators and diuretics, with inotropic agents for refractory symptoms. Hypotensive ‘wet-cold’ patients (classic cardiogenic shock) require inotropy with or without vasopressor agents, effective diuresis and early consideration of mechanical circulatory support (MCS).
Definitive therapies for AHF depend on underlying cause, and may include coronary artery intervention, valve repair, rhythm control to restore atrio-ventricular synchrony or management of pericardial tamponade. Patients with severe AHF not responsive to standard therapies should be considered for temporary MCS while candidacy for more durable option is explored by the multi-disciplinary team.
IHD also known as coronary artery diseases is a condition in which there is inadequate supply of blood and oxygen to a portion of myocardium. Imbalance between myocardial oxygen supply and demand causes Angina, MI, Hear failure, and Arrhythmia
Acute heart failure (AHF) is defined as rapid onset of new or worsening signs and symptoms of heart failure. It represents a life-threatening condition requiring treatment for fluid overload and hemodynamic compromise. Presentation may be initial diagnosis with symptoms and signs of AHF or acute decompensation of pre-existing cardiomyopathy. Hemodynamic instability results from disorders of the myocardium, valves, conduction system or pericardium, in isolation or combination. Potentially treatable causes, e.g. acute coronary syndromes, must be diagnosed and managed early for restoration of function.
Physiological changes associated with AHF result in reduced cardiac output and end-organ hypoperfusion. Once potentially treatable causes are managed, stratification of patients by clinical presentation guides further therapeutic intervention. AHF patients can be categorized as either ‘wet’ or ‘dry’ by clinical fluid status assessment, and either ‘cold’ or ‘warm’ according to perfusion status. In combination, these features identify four patient groups (‘warm-wet’, ‘warm-dry’, ‘cold-dry’, ‘cold-wet’) that guide therapy and facilitate prognostication. ‘Warm-dry’ patients rarely require intensive care for AHF treatment but may benefit from escalation of oral therapeutic regimen. Patients who examine as ‘cold-dry’ may benefit from fluid challenge, and/or inotropic agent infusion. ‘Warm-wet’ patients present with predominantly congestive or hypertensive symptoms which benefit from diuresis and vasodilatation. Patients who present ‘wet-cold’ with normal blood pressure (SBP >90) may benefit from vasodilators and diuretics, with inotropic agents for refractory symptoms. Hypotensive ‘wet-cold’ patients (classic cardiogenic shock) require inotropy with or without vasopressor agents, effective diuresis and early consideration of mechanical circulatory support (MCS).
Definitive therapies for AHF depend on underlying cause, and may include coronary artery intervention, valve repair, rhythm control to restore atrio-ventricular synchrony or management of pericardial tamponade. Patients with severe AHF not responsive to standard therapies should be considered for temporary MCS while candidacy for more durable option is explored by the multi-disciplinary team.
Cardiomyopathy is a group of disease that affect the heart muscle. Early on there may be few or no symptoms. As the disease worsens, shortness of breath, feeling tired, and swelling of legs may occur, due to the onset of heart failure. An irregular heart beat and fainting may occur.
Cardiomyopathy is a disease of the heart muscles that makes it harder for your heart to pump blood to the rest of your body. Cardiomyopathy can lead to heart failure.
According to the structural and functional abnormalities of the heart muscle
Dilated cardiomyopathy
Hypertrophic cardiomyopathy
Restrictive cardiomyopathy
Arrhythmogenic right ventricular cardiomyopathy
Unclassified cardiomyopathy
This is a power point presentation titled "Chronic Stable Angina" . For more medical power points, PDFs, ECGs, X-rays, please visit www.medicaldump.com
Cardiomyopathy is a group of disease that affect the heart muscle. Early on there may be few or no symptoms. As the disease worsens, shortness of breath, feeling tired, and swelling of legs may occur, due to the onset of heart failure. An irregular heart beat and fainting may occur.
Cardiomyopathy is a disease of the heart muscles that makes it harder for your heart to pump blood to the rest of your body. Cardiomyopathy can lead to heart failure.
According to the structural and functional abnormalities of the heart muscle
Dilated cardiomyopathy
Hypertrophic cardiomyopathy
Restrictive cardiomyopathy
Arrhythmogenic right ventricular cardiomyopathy
Unclassified cardiomyopathy
This is a power point presentation titled "Chronic Stable Angina" . For more medical power points, PDFs, ECGs, X-rays, please visit www.medicaldump.com
Angina also known as angina pectoris is a medical condition characterized by chest pain usually left sided due to inadequate blood supply (ischemia) to the heart muscles due to obstruction (like presence of blood clot), narrowing or contraction (vasospasm) of the supplying coronary arteries.
A brief description for 2nd year MBBS students about IHD- MI,Unstable Angina by Dr Sabu Augustine. content from other presentations (ppts)and text books
Angina pectoris is a clinical syndrome usually characterized by episodes of pain or pressure in the anterior chest . The cause is usually insufficient coronary blood flow which results in a decreased oxygen supply to meet an increased myocardial demand for oxygen in response to physical exertion or emotional stress.
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Ischemic heart disease is a condition of recurring chest pain or discomfort that occurs when a part of the heart does not receive enough blood. This condition occurs most often during exertion or excitement, when the heart requires greater blood flow.
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June 3, 2024 Anti-Semitism Letter Sent to MIT President Kornbluth and MIT Cor...Levi Shapiro
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Dear Dr. Kornbluth and Mr. Gorenberg,
The US House of Representatives is deeply concerned by ongoing and pervasive acts of antisemitic
harassment and intimidation at the Massachusetts Institute of Technology (MIT). Failing to act decisively to ensure a safe learning environment for all students would be a grave dereliction of your responsibilities as President of MIT and Chair of the MIT Corporation.
This Congress will not stand idly by and allow an environment hostile to Jewish students to persist. The House believes that your institution is in violation of Title VI of the Civil Rights Act, and the inability or
unwillingness to rectify this violation through action requires accountability.
Postsecondary education is a unique opportunity for students to learn and have their ideas and beliefs challenged. However, universities receiving hundreds of millions of federal funds annually have denied
students that opportunity and have been hijacked to become venues for the promotion of terrorism, antisemitic harassment and intimidation, unlawful encampments, and in some cases, assaults and riots.
The House of Representatives will not countenance the use of federal funds to indoctrinate students into hateful, antisemitic, anti-American supporters of terrorism. Investigations into campus antisemitism by the Committee on Education and the Workforce and the Committee on Ways and Means have been expanded into a Congress-wide probe across all relevant jurisdictions to address this national crisis. The undersigned Committees will conduct oversight into the use of federal funds at MIT and its learning environment under authorities granted to each Committee.
• The Committee on Education and the Workforce has been investigating your institution since December 7, 2023. The Committee has broad jurisdiction over postsecondary education, including its compliance with Title VI of the Civil Rights Act, campus safety concerns over disruptions to the learning environment, and the awarding of federal student aid under the Higher Education Act.
• The Committee on Oversight and Accountability is investigating the sources of funding and other support flowing to groups espousing pro-Hamas propaganda and engaged in antisemitic harassment and intimidation of students. The Committee on Oversight and Accountability is the principal oversight committee of the US House of Representatives and has broad authority to investigate “any matter” at “any time” under House Rule X.
• The Committee on Ways and Means has been investigating several universities since November 15, 2023, when the Committee held a hearing entitled From Ivory Towers to Dark Corners: Investigating the Nexus Between Antisemitism, Tax-Exempt Universities, and Terror Financing. The Committee followed the hearing with letters to those institutions on January 10, 202
The French Revolution, which began in 1789, was a period of radical social and political upheaval in France. It marked the decline of absolute monarchies, the rise of secular and democratic republics, and the eventual rise of Napoleon Bonaparte. This revolutionary period is crucial in understanding the transition from feudalism to modernity in Europe.
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Macroeconomics- Movie Location
This will be used as part of your Personal Professional Portfolio once graded.
Objective:
Prepare a presentation or a paper using research, basic comparative analysis, data organization and application of economic information. You will make an informed assessment of an economic climate outside of the United States to accomplish an entertainment industry objective.
Read| The latest issue of The Challenger is here! We are thrilled to announce that our school paper has qualified for the NATIONAL SCHOOLS PRESS CONFERENCE (NSPC) 2024. Thank you for your unwavering support and trust. Dive into the stories that made us stand out!
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2. Students will
Conceptualize ischemic heart disease with its etiology
and pathogenesis
Explain syndrome of CAD
Discuss the risk factors and relate sign and symptoms,
investigations to pathophysiology of IHD
Discuss about treatment and prognosis
Discuss about the nursing management of patient
with IHD
3.
4. IHD – A group of closely related syndromes caused by an
imbalance between the myocardial oxygen demand and blood
supply.
Usually caused by decreased coronary artery flow
Four syndromes :
Angina pectoris ( chest pain )
Acute myocardial infarction
Chronic ischemic heart disease and congestive heart failure
Sudden cardiac death
The most common cause of IHD is coronary atherosclerosis
Less commonly it is due to vasospasm and vasculitis
5.
6. Age – risk increases with age
Sex – males are more prone and females are prone after 10 yrs as
postmenopausal decrease in cardiac protective estrogen
Race – non whites increased risk
Modifiable risk factors
Elevated lipid levels
Hypertension
Obesity
Tobacco use
Diabetes mellitus
Sedentary lifestyle and stress
7.
8. Role of clinical stenosis and obstruction
Role of acute plaque change
Role of coronary thrombus
Role of vasoconstriction
Role of inflammation
9.
10.
11.
12.
13.
14. Angina Pectoris
Acute MI
Chronic IHD with Congestive Heart Failure
Sudden cardiac deadth.
16. STABLE ANGINA PECTORIS
It is relieved by rest or nitroglycerine
> Character - substernal chest pain,pressure,heaviness
including squezing,burning,choking
- may produce in numbness or weakness in arms, wrists or
hands
- associated symptoms include diaphoresis, nausea,
dypsnea,tachycardia,increased BP
- women may experience chest pain,jawpain,shortness of
breath
Location – behind middle or upper third of sternum
Radiation – radiates to neck , jaw , shoulder , arms ,hands and
posterior intrascapular area
Duration – lasts 2 to 15 min after stopping activity
17.
18. Chest pain occuring at rest
No increase on oxygen demand
Lack of blood flow to heart due to plaque
Lasts longer than 10 min
Unrelieved by rest or sublingual nitroglycerine
SILENT ISCHEMIA
Absence of ischemia with imbalance between myocardial
oxygen demand and bkood supply
ST elevation in ECG
Increase heart rate , blood pressure and blood viscosity.
19. ANTIANGINAL
i. Nitrates : vasodilation of arteries
ii. Cardioselective Beta Adrenergic Blockers : inhibit sympathetic stimulation
iii. Calcium channel blockers : inhibit movement of calcium with heart muscle
and coronary vessel
ACE INHIBITORS - Remodelling the vacular endothelium
ANTILIPID AGENTS – Reduce total cholesterol and triglyceride level
ANTIPLATELET AGENTS – Decrease platelet aggregation
FOLIC ACID AND B COMPLEX VIT – Treat increased homocystiene levels
20. Characteristic chest pain and clinical history
Nitroglycerin test : relief of pain with nitroglycerine
Blood tests : cardiac markers , creatine kinase and its
isoenzyme CK- MB , homocycteine and lipoprotien
,hemoglobin to rule out anemia
12 lead ECG which may show LVH , ST elevation, arrythmias
ECG stress testing
21. Treadmill test ( TMT ) : changes in ST elevation
Cardiac cathetorization : determines the presence ,
location and extent of coronary lesions.
Position emission tomography ( PET Scan ) : detect very
small perfusion differences caused by arteries.
Computed tomography ( CT Scan ) : detects coronary
calcium
Radionuclide imaging : indicates regions of ischemia
induced by exercise
22.
23. Participate in normal daily program of activities that
do not produce chest discomfort and shortness of
breath
Avoid activities like walking against the wind,
extremes of temperature , emotionally stressful
situations
Rest after each meal
Do not carry heavy objects
24. Carry nitroglycerine at all time
Place nitroglycerine under the tongue at first sign of chest
discomfort
Stop all efforts or activity, sit and take nitroglycerine tablet
Repeat dosage in 5 min for total of 3 tablets if relief is not
obtained
Keep a record of the number of tablets taken to evaluate
change in anginal pattern
Take nitroglycerine prophylactically to avoid pain known to
occur with certain activities
25. Instruct patient on administration of transdermal
nitroglycerine patches
a) Remove previous patch ; fold in half so that the medication
does not touch your fingertips and will not be accessible in
trash
b) Wipe area with tissue to remove any residual medication
c) Apply patch to a clean ,dry and non hairy area of body
d) Rotate administrationn sites
e) Instruct patient not to remove patch for swimming or
bathing
Teach patient about potential adverse effects of medications
a) Constipation
b) Ankle edema
c) Dizziness
27. Inform patient the methods of stress reduction
Review low fat and low cholesterol diet
a. Omega 3 improve arterial health and decrease BP,
triglycerides and the growth of atherosclerosis
plaque.
b. Omega 3 oils can be found in fatty fish.
Instruct patient to avoid excessive caffeine intake s it
increases heart rate and produce angina
Encourage patient to avoid alcohol
28.
29. ACUTE PAIN related to an imbalance in oxygen supply
and demand
DECREASED CARDIAC OUTPUT related to reduced
heart rate
ANXIETY related to chest pain
31. RELIEVING PAIN
Determine intensity of patients angina
Fowlers position
Administer oxygen , if appropriate
Obtain BP , HR , RR
Administer anti-anginal drug
Take vital signs every 5 to 10 min until angina pain subsides
Monitor progression of stable angina to unstable angina
32. MAINTAINING CARDIAC OUTPUT
Take BP and HR in sitting position {evaluate
orthostatic hypotension which occurs during drug therapy
}.
Note changes in BP more than 10 mmHg and changes in
HR of more than 10 beats/min
Note patients complaints of headache and dizziness
Continuous ECG monitoring
33. DECREASING ANXIETY
Explain to patient the importance of anxiety reduction to assist
in control of angina
{ Angina and fear increase stress on heart, requiring the heart to use more oxygen }