The document provides details on the medical operations and staffing for a large Ironman triathlon event. It discusses:
1. The medical tent serves a large role in the event and takes 5 months to organize, staffing over 100 medical professionals across various medical stations along the swim, bike, run courses and finish line.
2. The medical team prepares for common injuries and medical issues among athletes including heat illness, hyponatremia, cardiac problems, and provides protocols for rapid response and treatment.
3. On average, over 400 athletes finish the full ironman event each day, with most completing the run portion in the evening hours from 6-10pm. The medical operations aim to safely
Calm Computing 2016: La rencontre de l'internet industriel et des objets av...calmr.io
Dans le cadre de la 6e Édition de ECOM MTL.
Réalité virtuelle et objets connectés - Application concrète
Au cours de la dernière annnée, de nouvelles technologies et de nouveaux produits prendre de plus en plus de place sur le marché. Conséquence : un intérêt de plus en plus grandissant pour pour des technolgies comme l'augmented reality et le virtual reality. Qu'ont ils en commun? Ils font partie d'un système intégré où le monde réèl devient plus connecté que jamais :
Le wearable tech a rejoint le maché de masse.
Les possibilités de mesure ont atteint de nouveaux sommets (sensor tech).
Les applications sont plus concrète et utiles que jamais.
Calm Computing 2016: La rencontre de l'internet industriel et des objets av...calmr.io
Dans le cadre de la 6e Édition de ECOM MTL.
Réalité virtuelle et objets connectés - Application concrète
Au cours de la dernière annnée, de nouvelles technologies et de nouveaux produits prendre de plus en plus de place sur le marché. Conséquence : un intérêt de plus en plus grandissant pour pour des technolgies comme l'augmented reality et le virtual reality. Qu'ont ils en commun? Ils font partie d'un système intégré où le monde réèl devient plus connecté que jamais :
Le wearable tech a rejoint le maché de masse.
Les possibilités de mesure ont atteint de nouveaux sommets (sensor tech).
Les applications sont plus concrète et utiles que jamais.
Summary and illustrations of various traumatic brain injury including primary and secondary lesions as well as limited information on indications of brain imaging in trauma
An overview of the most commonly encountered emergencies in endurance athletes. The Baker to Vegas Law Enforcement Relay Race is the Largest of its kind in the world. This Year over 7000 runners will be competing in the 120 mile race.
Summary and illustrations of various traumatic brain injury including primary and secondary lesions as well as limited information on indications of brain imaging in trauma
An overview of the most commonly encountered emergencies in endurance athletes. The Baker to Vegas Law Enforcement Relay Race is the Largest of its kind in the world. This Year over 7000 runners will be competing in the 120 mile race.
Nusing Management of CAD Symposia (French) presented at Hôpital Sacré Coeur in Milot, Haiti.
CRUDEM’s Education Committee (a subcommittee of the Board of Directors) sponsors one-week medical symposia on specific medical topics, i.e. diabetes, infectious disease. The classes are held at Hôpital Sacré Coeur and doctors and nurses come from all over Haiti to attend.
Nusing Management of CAD Symposia (English) presented at Hôpital Sacré Coeur in Milot, Haiti.
CRUDEM’s Education Committee (a subcommittee of the Board of Directors) sponsors one-week medical symposia on specific medical topics, i.e. diabetes, infectious disease. The classes are held at Hôpital Sacré Coeur and doctors and nurses come from all over Haiti to attend.
Similar to IRONMAN ARIZONA MEDICAL ORIENTATION 2013 (20)
ABDOMINAL TRAUMA in pediatrics part one.drhasanrajab
Abdominal trauma in pediatrics refers to injuries or damage to the abdominal organs in children. It can occur due to various causes such as falls, motor vehicle accidents, sports-related injuries, and physical abuse. Children are more vulnerable to abdominal trauma due to their unique anatomical and physiological characteristics. Signs and symptoms include abdominal pain, tenderness, distension, vomiting, and signs of shock. Diagnosis involves physical examination, imaging studies, and laboratory tests. Management depends on the severity and may involve conservative treatment or surgical intervention. Prevention is crucial in reducing the incidence of abdominal trauma in children.
DISSERTATION on NEW DRUG DISCOVERY AND DEVELOPMENT STAGES OF DRUG DISCOVERYNEHA GUPTA
The process of drug discovery and development is a complex and multi-step endeavor aimed at bringing new pharmaceutical drugs to market. It begins with identifying and validating a biological target, such as a protein, gene, or RNA, that is associated with a disease. This step involves understanding the target's role in the disease and confirming that modulating it can have therapeutic effects. The next stage, hit identification, employs high-throughput screening (HTS) and other methods to find compounds that interact with the target. Computational techniques may also be used to identify potential hits from large compound libraries.
Following hit identification, the hits are optimized to improve their efficacy, selectivity, and pharmacokinetic properties, resulting in lead compounds. These leads undergo further refinement to enhance their potency, reduce toxicity, and improve drug-like characteristics, creating drug candidates suitable for preclinical testing. In the preclinical development phase, drug candidates are tested in vitro (in cell cultures) and in vivo (in animal models) to evaluate their safety, efficacy, pharmacokinetics, and pharmacodynamics. Toxicology studies are conducted to assess potential risks.
Before clinical trials can begin, an Investigational New Drug (IND) application must be submitted to regulatory authorities. This application includes data from preclinical studies and plans for clinical trials. Clinical development involves human trials in three phases: Phase I tests the drug's safety and dosage in a small group of healthy volunteers, Phase II assesses the drug's efficacy and side effects in a larger group of patients with the target disease, and Phase III confirms the drug's efficacy and monitors adverse reactions in a large population, often compared to existing treatments.
After successful clinical trials, a New Drug Application (NDA) is submitted to regulatory authorities for approval, including all data from preclinical and clinical studies, as well as proposed labeling and manufacturing information. Regulatory authorities then review the NDA to ensure the drug is safe, effective, and of high quality, potentially requiring additional studies. Finally, after a drug is approved and marketed, it undergoes post-marketing surveillance, which includes continuous monitoring for long-term safety and effectiveness, pharmacovigilance, and reporting of any adverse effects.
Basavarajeeyam is an important text for ayurvedic physician belonging to andhra pradehs. It is a popular compendium in various parts of our country as well as in andhra pradesh. The content of the text was presented in sanskrit and telugu language (Bilingual). One of the most famous book in ayurvedic pharmaceutics and therapeutics. This book contains 25 chapters called as prakaranas. Many rasaoushadis were explained, pioneer of dhatu druti, nadi pareeksha, mutra pareeksha etc. Belongs to the period of 15-16 century. New diseases like upadamsha, phiranga rogas are explained.
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Integrating Ayurveda into Parkinson’s Management: A Holistic ApproachAyurveda ForAll
Explore the benefits of combining Ayurveda with conventional Parkinson's treatments. Learn how a holistic approach can manage symptoms, enhance well-being, and balance body energies. Discover the steps to safely integrate Ayurvedic practices into your Parkinson’s care plan, including expert guidance on diet, herbal remedies, and lifestyle modifications.
Here is the updated list of Top Best Ayurvedic medicine for Gas and Indigestion and those are Gas-O-Go Syp for Dyspepsia | Lavizyme Syrup for Acidity | Yumzyme Hepatoprotective Capsules etc
Basavarajeeyam is a Sreshta Sangraha grantha (Compiled book ), written by Neelkanta kotturu Basavaraja Virachita. It contains 25 Prakaranas, First 24 Chapters related to Rogas& 25th to Rasadravyas.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
share - Lions, tigers, AI and health misinformation, oh my!.pptxTina Purnat
• Pitfalls and pivots needed to use AI effectively in public health
• Evidence-based strategies to address health misinformation effectively
• Building trust with communities online and offline
• Equipping health professionals to address questions, concerns and health misinformation
• Assessing risk and mitigating harm from adverse health narratives in communities, health workforce and health system
These lecture slides, by Dr Sidra Arshad, offer a quick overview of the physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar lead (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
6. Describe the flow of current around the heart during the cardiac cycle
7. Discuss the placement and polarity of the leads of electrocardiograph
8. Describe the normal electrocardiograms recorded from the limb leads and explain the physiological basis of the different records that are obtained
9. Define mean electrical vector (axis) of the heart and give the normal range
10. Define the mean QRS vector
11. Describe the axes of leads (hexagonal reference system)
12. Comprehend the vectorial analysis of the normal ECG
13. Determine the mean electrical axis of the ventricular QRS and appreciate the mean axis deviation
14. Explain the concepts of current of injury, J point, and their significance
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. Chapter 3, Cardiology Explained, https://www.ncbi.nlm.nih.gov/books/NBK2214/
7. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
4.
Swim
Transition
South Side Main Medical
North Side Warming
Men’s and Women’s Change Tent
Main Medical
Finish
72 bed MASH unit
Teams comprised of 3-4 staff per 6 bed section
Evaluation/oral suport/IV support/Meds/Istat
2 ALS ambulances and PM staff
5.
The Medical tent serves a
large role in the Ironman
and thus organization of
the medical team is
important and takes
shape nearly 5 months
before each race every
year.
The Medical team is
comprised of doctors,
nurses, paramedics,
EMT’s, medical students,
athletic trainers, and
other personnel such as
IV technicians and
communications,
security, and supply
volunteers.
6.
Catchers are placed at the finish line. They
are educated on cooling down the athlete and
escorting those that need medical care.
At the beginning of the tent, we have a
computer check-in. Personnel will take the
athlete’s chip and the athlete is checked in.
7.
Swim Medical 5:30-9:00 am
South Main Tent (Swim Exit)
Hasan Chaudry MD/Gina Ferguson RN
Joe Garcia MD/Susan Girard RN
Alex Espinoza MD/Kelley Deeny RN/Terra Marr RN
Lainie Yarris MD/Sue White RN/Brad Williams PM
Jeff Jaco PA/Michelle Nelson RN/Mary Ellen Quinn NPEMT
Finish/Catching
Rodney Schnuelle EMT/Katie Sidesinger RN/Milica Mcdowell DPT/Conrad Hilton/Scott Simpson
North Medical Tent
Michelle Miller RN/Billy Burnett EMT/Brian Byars EMT
Main Medical Setup 8:00-10:00 am
Nicole Barreda ND/Maria Santander MD
Al Valle CPU/Craig Wolchover CPU/Alex Zdvorak CPU
Transition Medical 8:00-10:00 am
Brian Crowder RN/Jocelyn Mayes RN
Robyn Crowder/Paul Herberger
Transition Medical 12:00-5:30 pm
Men’s Tent: Savas Petrides MD (Sarah Petrides)/Bob Nelson MD
Ignacio Marquez EMT/Kenneth Zurcher/Michael Bishop PM
Women’s Test: Bridget Haga RN/Katie Hoodjer RN
Jim West PM/Sarah West RN/Lisa Sherry-Kulow RN/Sana
Shuttari NP/Angela Tetschner RN/Lori Vega RN
8.
Main Medical 12:00-3:00 pm
CPU: Jordan Ahern/Kali Bayes
A: Liz Dinsmoor RN/Mark Ford EMT
B: Ann Kelly RN/Judy Lynn PM/Deborah Ackerman RN
C: Tanya Marvin PA/Clare Ryan RN NP
Main Medical 3:00-6:00 pm
CPU: Jordan Ahern/Mikayla Jaraczewski/Jennifer Light
A: Liz Dinsmoor RN/Carly Fellhoelter CAN
B: Lyle Edwards PM/Praise Gunadi RN
C: Terra Marr RN/Jennifer Quint RN
D: Robert Ziltzer MD/Amy Turano RN/Rachel Dickman EMT
Run Medical 3:00-6:00 pm
Steven Cho/Jason Lee
Dina Utter RN/Praise Gunadi RN
9.
Main Medical 6:00-9:00 pm
CPU: Jordan Ahern/Darrin Anderson/Joseph Baxter/Samantha Calvin/Bonnie and Danny Kissinger/Sue Pierson/Annette Paris
A: Ryan Coates DO/Dan Leon PM/Roger Chopelas RN/Andrew Bliss EMT
B Dennis Scribner MD/Lisa Banyasz RN/Richard Alamo PM
C: Benjamin Johansen DO/Cliff Bailey RN/Rodney Schnuelle EMT
E: Amish Shah MD/Tara Peters RN/Dane Morrissey PM/Mark Thornberry PM
F: Mikyong Hand MD/Kimberli Haas RN//Tommy Roads/Kimberly Hansen ATC
G: Donn Hogan MD/Rachael Roberts RN/Desiree Hart-Wilson RN/Pamela Pimentel RN
H: Andrew Utter MD/ /Anna Lipa RN/Loren Swensen EMT
I: Lyle Edwards PM/Neil Martin PM/Kathy Brown RN/David Jordan PM
Walking Wounded: Jay Mellen MD/Brian Moore MD/Carol Holliday RN/ Terra Marr RN/Charles Brown PM/Jennifer Sheneman RN
Donovan Adendorf DC/Megan Cook/Jackie English/Carly Fellhoelter Crna/Cyntia Hurtado/Mikayla Jaraczewski/Bret Jones/Neal Mckimpson/Leonor
Medina/Anna Pastorino/Scott Simpson/Conrad Hilton/Josh Hendrix
Catcher Assist: Ted McCarthy MD/Mike Pierson MD/Marita Mcgoldrick NP/Eric Loiland PT/Sarah Kellerhals/Yoonha Kim/ Matthew Petterson/Sarah
Rubenstein/Amber Sandoval-Menendez
Run Medical 6:00-9:00 pm
Patrick Weldon MD/Gary Young RN/Jude Carza RN/Raiza Carza RN
Charting: Gregg Bassett/Sam Fleischer
Assist: Joanne Donnelly Crna/James Fernandes PTATC/Jose Fonseca PT
Main Medical 9:00-12:30 am
CPU: Jordan Ahern/Cassie Beard/Tamara Brown/Michelle Drietz
A: Mike Brown DO/Lisa Austin RN/Brad Eppelheimer ATC
B: Matthew Duke DO/Zelda Carson RN/Brianna Foote RN
C: Savas Petrides MD (Sarah Petrides)/Carly Fellhoelter crna/Nathan Hill EMT
D: Richard Ernst MD/Yiselle Gonzalez RN/Chris Murray PM
E: Dana Jamison MD/Melissa Kay ATC
F: Amish Shah MD/Terra Marr RN
Walking Wounded: Dennis Scribner MD/ Maya Davidow MD/Peggy Thornhill RN
Tracy Lee/Chung-Kai Liu/
Catcher Assist: Ted McCarthy MD/ Kelsey Picha ATC/Sharon Plummer ND
Run Medical 9:00-12:30 am
Amy Wiser MD/Vicky Tippett MD/Kortney Parman RN
Evan Wolff/Russell McWilliam/Tina Rossi/Gillian Hansen
10. Ironman Average Finish Time
450
400
350
300
250
# of Athletes
Run:
200
150
100
50
0
Until 5pm
5pm-6pm
6pm-7pm
7pm-8pm
8pm-9pm
Time of Day
9pm-10pm
10pm-11pm
11pm+
13. • Compensatory Mechanisms:
– Shivering (which occurs from 37 to 32 degrees)
– Autonomic Nervous system (occurring from 37
to 32 degrees, includes bradycardia and
vasoconstriction)
– Extrapyramidal stimulation of skeletal muscles
– Adaptive Behavioral responses
14. TREATMENT
• Treatment: Focus on diminishing heat loss
• Use of heating devices. This includes
heaters within the tents , warm baths
• Removal of wet clothes. Wet clothing
leads to heat dissipation five times
• Cover and keep patient insulated.
Aluminum foil blankets efficient than wool
blankets
• Warm IV Fluids
• Serious hypothermia , follow Marine
Corps Marathon Hypothermia algorithm
16.
Cardiac Arrest, Heat Stroke, and ExerciseAssociated Hyponatremia.
Initial Approach to the unresponsive athlete:
Initial Evaluation (VS) / C-A-B
Elevate the legs
Assess Mental Status
Assess Volume Status
Fluid Replacement
Continuous Monitoring
17.
Initial Evaluation/ C-A-B
Elevate the Legs
AMS often presents with Heat Stroke and Exercise-Associated Hyponatremia
Assess Volume Status
This will help any conscious athletes that are light-headed
Assess Mental Status
Compressions-Airway-Breathing (check back of bib for Medical Information)
Fluids consumed, vomit/diarrhea, urinated during race, orthostatic pulse
Fluid Replacement
Fluids w/electrolytes such as Sports Drink or Chicken broth
An IV may be started based on clinical judgment
Continuous Monitoring
Any athlete needing to go to the hospital MUST be seen by the Medical
Director
18.
Heat Exhaustion- more common in warmer environments
During race blood vessels maximally dilated in legs for cooling
and delivery of O2 to working muscles
Adrenaline released to regulate BP and maintain cardiac output
When athlete stops pump action is lost and adrenaline no longer
released
Vessels remain dilated leading to pooling of blood in lower
extremity
TREATMENT
Have athlete continue “cool down” walk after race
Lie the athlete down and elevate the legs
If no nausea present offer PO fluids or possible IV
19.
During exercise runners often sweat >1.5 L/Hr while maximally
absorbing approximately 1 L (negative fluid balance)
With as little as 3% loss of body weight a marathon runner
performance is impaired
Volume depletion leads to hyperthermia due to the limiting of
blood shunted to the skin
Runners present:
Hypotension, tachycardia, weight loss, decreased skin turgor, hypernatremia
TREATMENT
Oral fluids such as sports drink or chicken broth
IV NS, first w/500 ml bolus, then 500 ml over 30-60 min
Any sign of AMS must have electrolytes drawn
20.
Most common mechanism due to over-hydration
Trained athletes should have calculated fluid loss during exercise
Risk factors for developing EAH
Finish under 4 hrs, inexperience, small stature, female gender
Hallmark for EAH: AMS or unusual neurologic complaint
Recognizing Hyponatremia
Stage 1: dizziness, nausea, vomiting, headache
Stage 2: mental status changes (confusion, disorientation)
Stage 3: altered consciousness (delirium, seizures, coma)
(Na+ 130-135 mEq/L)
(Na+ 125-130 mEq/L)
(Na+ <125 mEq/L)
21.
Stage 1
If vitals stable, fluid restriction (4oz max) and observe until urination
Fluid replacement: add 3 salt packs (3/8 tsp) to half cup electrolyte drink, this
will provide 1 g Na or a 3%saline solution
Should athlete continue fluid restriction of 4oz max/ mile adding salt as above
Stage 2/Stage 3
Consider transferring to ER
Give 3% oral saline solution, if can tolerate PO fluids
Transport via EMT/Paramedics, advise NO IV fluids until hyponatremia
excluded
ALL runners w/EAH must receive discharge from Medical director
22.
Stage 1
Draw blood for Na+ concentration and give no IV fluids
Limit fluid to 4oz (preferably broth) or 3% saline or salty snacks
Stage 2
Draw blood for Na+ concentration, if >125 mEq/L offer PO broth
Oral hypertonic saline: 4 cubes of bouillon in 4oz hot water for a 9% solution
Limit fluid to 4oz, continue observation until they urinate
If not improved 15-20 min repeat oral solution or give 100 ml bolus 3%NaCl
Begin cardiac monitoring and pulse oximetry
A 2nd IV bolus given 10-15 min later
Any signs of neuro must go to ER, transport on IV=3%NaCl at 1 ml/min
Stage 3
Draw blood for Na+ concentration, if <125 or delirium present must go to ER
Begin cardiac monitoring and pulse oximetry
Give 100 ml bolus 3% NaCl and a 2nd in 10-15 min
Both Valium and Ativan for IV injection are available to treat seizures
23.
With a core temp >104°F organ/brain cells begin to die
Goal is Rapid cooling to < 104°F within 30 min
Athlete presents w/CNS changes, altered cognition/behavior
If oral reading >100.5°F, then rectal temp should be taken
Early Warning signs
Irritability, confusion, apathy, belligerence, emotional instability, irrational
behavior, giddiness , vomiting, numbness ,tingling, collapse, seizures, coma
Paradoxical chills and goose bumps signal shutdown of peripheral circulation
Follow Mantra: “cool first, transport second”
24.
Cool First
Rapid cooling by placing athlete into ice-water submersion tub (immerse trunk, both
arms and both legs) has 100% survival rate when athlete immersed within 10 min of
collapse (key to maximal cooling is constant stirring of the ice)
May also cold water douse the body while patient lying on porous stretcher, while
massaging major muscle groups w/ice bags
May also place wet ice towels over entire body, rotate w/cooler towels every 2 min
Check rectal temp/vitals/CNS status every few min
At athlete can be cooled from 108-110°F to 102°F within 15-30 min, average rate of
cooling drop is 1°F every 3 min
Transport Second
Once temp reaches 102°F, remove from immersion tub
Observe for 30-60 min to ensure drinking fluids, normal vitals, good cognition
Physicians recommended to follow up and evaluate patients w/sever hyperthermia
If rectal cannot be measured, cold water immersion for 10-15 min then transport to
ER
25.
Usually occurs near the finish, theory is increased adrenaline
release precipitates arrhythmia, when runner sees finish line and
sprints to improve position
Any athlete w/Cardiac arrest should prompt notification Medical
Command and activate 911 system and initiate ambulance
TREATMENT
Good quality chest compressions
Early defibrillation when AED available
Follow the Marine Corps Marathon algorithm for Emergency Cardiac Care
26. 1. 10.9 million runners, 59 (mean [±SD] age, 42±13 years; 51 men) had
cardiac arrest 0.54 per 100,000 participants; interval and among
(men 0.90 per 100,000, women 0.16).
2. Cardiovascular disease accounted for the majority of cardiac
arrests. The incidence rate was significantly higher during
marathons (1.01 per 100,000) than during half-marathons
3. Of the 59 cases of cardiac arrest, 42 (71%) were fatal (incidence,
0.39 per 100,000).
4. Among the 31 cases with complete clinical data, initiation of
bystander-administered cardiopulmonary resuscitation and an
underlying diagnosis other than hypertrophic cardiomyopathy
were the strongest predictors of survival.
27.
Most important factor pre-disposing to this condition is
hypovolemia
Damage to skeletal muscle and release CK-MM, myoglobin and
aldolase into circulation
High levels of myoglobin can cause acute renal failure, DIC, lactic
acidosis and cardiac dysrhythmias
Symptoms
Risk Factors
myalgias, muscular swelling, tenderness, dark colored urine, lower back or
abdominal pain
Poor fitness level, high temp/humidity, sickle cell trait, viral illness, hx of
myopathies, prior renal insufficiency
TREATMENT
Fluid replacement, maintain good urine output, transport to Hospital
28.
Check athlete for Hx of Diabetes
Exercise acts like regular insulin and this must be calculated
In non-diabetics may occur due to complete depletion of glycogen stores
Symptoms
Fatigue, dizziness, tachycardia, nausea, vomiting, impaired mental status
*Any of these symptoms a “finger stick” glucose should be checked
TREATMENT (if glucose <55)
PO glucose if no nausea or vomiting
Bolus of D50 IV
ER transport for those who fail to respond or remain hypoglycemic
29.
Exercise=Sympathetic Nervous System
Digestion= Parasympathetic Nervous System
Athletes must train their guts to digest on the run
Under ideal conditions the gut can process 1.5 L/hr
Many novice runners will over-feed or over-hydrate, and gut cannot handle
this volume leading to nausea/vomit
Symptoms
Diarrhea (possible osmotic diarrhea), nausea, vomit, abdominal pain
*Be cautious w/abdominal pain, during exercise 70% of splanchnic circulation
is shunted to working muscles, this can result in ischemia and eventually
necrosis of intestinal lining cells
30.
15% of general population and 40% athletes w/allergies will
develop Exercise-Induced bronchospasm
If patient has true asthma, number rises to over 70%
Common on days w/ increased pollutants such as smoke, dust,
pollen
TREATMENT
Replacing volume, administer breathing treatments w/inhaled bronchodilators
Albuterol is effective as rescue inhaler
31.
Treatment is two fold: addressing metabolic issues and
mechanical stretching of injured muscle
First begin IV and check serum sodium and magnesium
TREATMENT
EAH? Draw iSTAT
2-4 g Magnesium sulfate give slow IV push while watching reflexes and
respirations
Ativan 0.5 mg IM or IV
2-4 g Magnesium sulfate can be added to 1 L IV NS and run at 500-1,000 ml/hr
Sickle cell trait can produce muscle ischemia and should be treated w/O2 and
IV fluids, transport to ER
Use of the Massage therapist may be helpful
32.
Remember sunburned skin does not sweat well, SPF >30 should be
used
Abrasions should be cleaned w/Hydrogen peroxide or Hibiclens
Avoid waterproof products which block pores and reduce sweating
Betadyne retards wound healing
Small, intact blisters that are minimally painful usually don’t need
any treatment. Cover it with a small bandaid, and triple antibiotic
ointment.
Larger or painful blisters that are intact should be carefully drained,
without removing the skin.
clean the skin over the blister with rubbing alcohol or soap and water.
sterilized needle or pin, puncture a small hole at the edge of the blister.
Drain the fluid with gentle pressure and apply a bandaid with some triple antibiotic
ointment.
Keep it clean and dry for 2-3 days, changing the dressing daily. You may then begin
to carefully trim away the dead skin. Use second skin, or blister pads for protection,
until the new skin matures.
34. HOME CARE
Stretch tight muscles, and ice sore tendons and joints.
Anti-inflammatory medications, such as ibuprofen or
naproxen may help speed recovery.
Eat a light diet with foods that are easy to digest, such
as pasta, rice, or potatoes. Adding some protein
actually helps your muscles recovers faster.
Drink plenty of fluids. It is a good idea to salt your
food or use a Sports Drink to replace the electrolytes
(such as sodium) you have lost. Avoid drinking large
quantities of water.
35. SEEK MEDICAL ATTENTION IF YOU EXPERIENCE:
Severe leg or calf pain not improved with elevation
Abdominal pain with or without vomiting or diarrhea
Persistent temperature over 100.50 F (37.80 C) orally
Blood in vomit or stool (even dark red or black stool)
Unusual weakness, dizziness or fainting
Decreased urine output, bloody or very dark urine
Unusual chest pain, muscle cramping or edema
[puffiness]
Coughing, trouble breathing, colored or bloody
phlegm
Severe headache, vision changes or confusion