This document provides an overview of dental implants, including:
- Dental implants are artificial tooth roots placed in the jaw to hold replacement teeth. There are three main types: endosseous, subperiosteal, and transosteal implants.
- Implants are typically made of titanium and fuse with the jawbone through osseointegration. Placement involves raising soft tissue flaps, drilling pilot holes, widening the holes, placing the implant, and adapting the soft tissue around it.
- Risks include injury to nearby structures during surgery and post-operative infection or bleeding. Success rates depend on patient health and bone quality. With proper planning and placement, implants can successfully replace missing
The primary success metric of dental implants is achieving osseointegration, which is influenced by many factors including implant design, surface treatments, as well as treatment method. Implant drilling is also a major influential factor.
Indian Dental Academy: will be one of the most relevant and exciting training
center with best faculty and flexible training programs for dental
professionals who wish to advance in their dental practice,Offers certified
courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry,
Prosthetic Dentistry, Periodontics and General Dentistry.
This seminar deals with implant-related complications that lead to implant failure.this also discus diagnostic criteria and preventive methods for an implant failure.
the dental implant design from the point of view of dental biomaterials and the effect of force factors on choice of implant design in correlation with bone defects and anatomical anomalies
The primary success metric of dental implants is achieving osseointegration, which is influenced by many factors including implant design, surface treatments, as well as treatment method. Implant drilling is also a major influential factor.
Indian Dental Academy: will be one of the most relevant and exciting training
center with best faculty and flexible training programs for dental
professionals who wish to advance in their dental practice,Offers certified
courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry,
Prosthetic Dentistry, Periodontics and General Dentistry.
This seminar deals with implant-related complications that lead to implant failure.this also discus diagnostic criteria and preventive methods for an implant failure.
the dental implant design from the point of view of dental biomaterials and the effect of force factors on choice of implant design in correlation with bone defects and anatomical anomalies
Introduction of Dental implant
What is ossteointegration
Requirement of dental implant
Steps to select proper case of Dental implant
Implant design , diameter in details , bone factor ,biocompatibility.
Materials for dental implant and surface cotting
A dental implant (also known as an endosseous implant or fixture) is interfacing with the bone of the jaw or skull to support a dental prosthesis such as a crown, a bridge or a denture.
Prosthesis is one of the most important component of an implant. There are various prosthetic factors that must be considered for a successful implant. Few of them include prosthesis type and material, the connection between abutment and prosthesis, occlusal factors, etc.
Split ridge and expansion techniques are effective for the correction of moderately resorbed edentulous ridges in selected cases.
Transverse expansion is based on osseous plasticity obtained by corticotomy. It progressively allows for an adequate transversal intercortical diameter large enough to insert one or several dental implants.
The gap created by sagittal osteotomy expansion undergoes spontaneous ossification, following a mechanism similar to that occurring in fractures.
implantology biologic and clinical aspects / dental implant courses by Indian...Indian dental academy
Description :
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
2. What is a dental implant?
• A dental implant is an artificial tooth root that is placed
into jaw to hold a replacement tooth or bridge. Dental
implants may be an option for people who have lost a
tooth or teeth due to periodontal disease, an injury, or
some other reason.
3. What Are the Typesof Implants?
• There are three types of implants, and they can be described
according to their shape and how they are attached to the
jaw.
1. ENDOSSEOUS (en-doss-ee-us)-“within the bone”
2. SUBPERIOSTEAL (sub-pear-ee-oss-tee-al)-“on top of the
bone”
3. TRANSOSTEAL (trans-oss-tee-al)-“through the bone”
4. ENDOSSEOUS “within the bone”
These implants are usually
shaped like a screw or
cylinder and are made
either of metal, metal
covered with ceramic, or
ceramic material. They are
placed within the
jawbone. There are also
blade-shaped endosseous
implants
5. SUBPERIOSTEAL -“on top of the bone”
• These implants consist of a metal framework that
attaches on top of the jawbone but underneath the gum
tissue.
6. TRANSOSTEAL-“through the bone”
• These implants are either a metal pin or a U-shaped frame
that passes through the jawbone and the gum tissue, into the
mouth.
8. What are the implants made of?
• Modern dental implants can fuse with bone through a
biologic process called Osseointegration. Materials such
as titanium, and some ceramics form this bone
integration instead of causing a foreign body reaction as
found with most other materials.
9. OSSEOINTEGRATION
• A direct structural and functional connection between ordered
living bone and the surface of a load carrying implant
• Dental implants work by a process known as osseointegration,
which occurs when bone cells attach themselves directly to
the titanium surface, essentially locking the implant into the
jaw bone. New studies shown Osseointegration will be
completed within 8 weeks.
11. Ideal Implant Spacing
• Remember 2 mm as a key figure. An implant needs about 2 mm of bone
between it and the neighboring tooth to allow normal bone metabolism.
This space allows for normal bone metabolism to maintain the
periodontium around the tooth, and enough space for force-adaptation of
the osseointegrating column of bone around the implant. 2 mm is also
minimal safe distance to plan from the tip of the implant to any anatomical
limitation.
12. Ideal Implant Spacing
• The second important number is 3 mm. This refers to the minimum distance
between implants. Since there is no periodontal ligament around implants,
there is inherently less blood supply around implants than around a natural
tooth, which receives a rich blood supply from the PDL. The effects of a
lower blood supply has manifested over time clinically and in the literature
as more bone loss between implants that are closer than 3 mm.
Cleansability is even more ideal with closer to 4 mm between implants.
13. Ideal Implant Spacing
• The last number to remember is 7 mm, referring to the minimum amount of
space from the top of the implant to the opposing dentition. 9 mm occlusal
space is more ideal, and a little less is acceptable for a short, squatty screw-
retained restoration.
14. Different approachesto placementDI
1. Immediate post-extraction implant placement.
2. Delayed immediate post-extraction implant placement (two
weeks to three months after extraction).
3. Late implantation (three months or more after tooth
extraction).
15. Basicimplant surgicalprocedure
• Most implant systems have five basic
steps for placement of each implant:
• 1. Soft tissue reflection: An incision is made
over the crest of bone, splitting the
thicker attached gingiva roughly in half so that
the final implant will have a thick band of tissue
around it. The edges of tissue, each referred to
as a flap are pushed back to expose the bone.
Flapless surgery is an alternate technique,
where a small punch of tissue (the diameter of
the implant) is removed for implant placement
rather than raising flaps.
Ridge missing tooth
16. Basicimplant surgicalprocedure
2. Drilling at high speed: After reflecting the soft
tissue, and using a surgical guide or stent as
necessary, pilot holes are placed with precision drills
at highly regulated speed to prevent burning or
pressure necrosis of the bone.
3. Drilling at low speed: The pilot hole is expanded by
using progressively wider drills (typically between
three and seven successive drilling steps, depending
on implant width and length). Care is taken not to
damage the osteoblast or bone cells by overheating.
A cooling saline or water spray keeps
the temperature low
Slow speed drill
Tissue opened
17. Basicimplant surgicalprocedure
4. Placement of the implant: The implant screw is
placed and can be self-tapping, otherwise the
prepared site is tapped with an implant analog. It
is then screwed into place at a precise torque so
as not to overload the surrounding bone
(overloaded bone can die, a condition called
osteonecrosis, which may lead to failure of the
implant to fully integrate or bond with the
jawbone).
5. Tissue adaptation: The gingiva is adapted around
the entire implant to provide a thick band of
healthy tissue around the healing abutment. In
contrast, an implant can be "buried", where the
top of the implant is sealed with a cover
screw and the tissue is closed to completely cover
it. A second procedure would then be required to
uncover the implant at a later date.
Healing abutment
Implant fixture
19. loading of dental implants
1. Immediate loading procedure.
2. Early loading (one week to twelve weeks).
3. Delayed loading (over three months)
20. Healing time (oldStudies)
• For an implant to become permanently stable, the body must
grow bone to the surface of the implant (Osseointegration)
• Based on this biologic process, it was thought that loading an
implant during the osseointegration period would result in
movement that would prevent osseointegration, and thus
increase implant failure rates. As a result, three to six months
of integrating time (depending on various factors) was allowed
before placing the teeth on implants.
21. Healing time (NewStudies)
• the initial stability of the implant in bone is a more important
determinant of success of implant integration, rather than a
certain period of healing time. As a result, the time allowed to
heal is typically based on the density of bone the implant is
placed in and the number of implants splinted together, rather
than a uniform amount of time. When implants can withstand
high torque (35 Ncm) and are splinted to other implants, there
are no meaningful differences in long-term implant survival or
bone loss between implants loaded immediately, at three
months, or at six months. The corollary is that single implants,
even in solid bone, require a period of no-load to minimize the
risk of initial failure.( 8 weeks)
22. Risks and complications (Duringsurgery)
• Placement of dental implants is a surgical procedure and
carries the normal risks of surgery including infection,
excessive bleeding and necrosis of the flap of tissue around
the implant. Because the surgeon is blind to the location of
the tip of the drill when it is in the bone, nearby anatomic
structures can also be injured such as the inferior alveolar
nerve, the maxillary sinus and blood vessels.
23. Immediate post-operative risks
1. Infection (pre-op antibiotics reduce the risk of implant
failure by 33 percent but have no impact on the risk of
infection)
2. Excessive bleeding
3. Flap breakdown (less-than 5 percent)
24. General considerations
• Planning for dental implants focuses on the general health
condition of the patient, the local health condition of the
mucous membranes and the jaws and the shape, size, and
position of the bones of the jaws, adjacent and opposing
teeth. There are few health conditions that absolutely
preclude placing implants although there are certain
conditions that can increase the risk of failure. Those with
poor oral hygiene, heavy smokers and diabetics are all at
greater risk for a variant of gum disease that affects implants
called peri-implantitis, increasing the chance of long-term
failures. Long-term steroid use, osteoporosis and other
diseases that affect the bones can increase the risk of early
failure of implants
Remember 2 mm as a key figure. An implant needs about 2 mm of bone between it and the neighboring tooth to allow normal bone metabolism. This space allows for normal bone metabolism to maintain the periodontium around the tooth, and enough space for force-adaptation of the osseointegrating column of bone around the implant. 2 mm is also minimal safe distance to plan from the tip of the implant to any anatomical limitation. For me, staying a little further away from limiting anatomy is more comfortable, but 2 mm is a minimum even for those with cajones grandes.
The second important number is 3 mm. This refers to the minimum distance between implants. Since there is no periodontal ligament around implants, there is inherently less blood supply around implants than around a natural tooth, which receives a rich blood supply from the PDL. The effects of a lower blood supply has manifested over time clinically and in the literature as more bone loss between implants that are closer than 3 mm. Cleansability is even more ideal with closer to 4 mm between implants, but many times with two adjacent implants we are closer to the 3 mm minimum. A tantalizing finding in recent literature is the preservation of tissue when platform-switching aka medialized abutments aka coronal beveling is present in the implant design. It is clear that there is a biologic volume that can be accommodated around the abutment junction by creating more space vs. that same biologic adaptation running down the implant body to the first thread of the olden days.
Remember 2 mm as a key figure. An implant needs about 2 mm of bone between it and the neighboring tooth to allow normal bone metabolism. This space allows for normal bone metabolism to maintain the periodontium around the tooth, and enough space for force-adaptation of the osseointegrating column of bone around the implant. 2 mm is also minimal safe distance to plan from the tip of the implant to any anatomical limitation. For me, staying a little further away from limiting anatomy is more comfortable, but 2 mm is a minimum even for those with cajones grandes.
The second important number is 3 mm. This refers to the minimum distance between implants. Since there is no periodontal ligament around implants, there is inherently less blood supply around implants than around a natural tooth, which receives a rich blood supply from the PDL. The effects of a lower blood supply has manifested over time clinically and in the literature as more bone loss between implants that are closer than 3 mm. Cleansability is even more ideal with closer to 4 mm between implants, but many times with two adjacent implants we are closer to the 3 mm minimum. A tantalizing finding in recent literature is the preservation of tissue when platform-switching aka medialized abutments aka coronal beveling is present in the implant design. It is clear that there is a biologic volume that can be accommodated around the abutment junction by creating more space vs. that same biologic adaptation running down the implant body to the first thread of the olden days.
Remember 2 mm as a key figure. An implant needs about 2 mm of bone between it and the neighboring tooth to allow normal bone metabolism. This space allows for normal bone metabolism to maintain the periodontium around the tooth, and enough space for force-adaptation of the osseointegrating column of bone around the implant. 2 mm is also minimal safe distance to plan from the tip of the implant to any anatomical limitation. For me, staying a little further away from limiting anatomy is more comfortable, but 2 mm is a minimum even for those with cajones grandes.
The second important number is 3 mm. This refers to the minimum distance between implants. Since there is no periodontal ligament around implants, there is inherently less blood supply around implants than around a natural tooth, which receives a rich blood supply from the PDL. The effects of a lower blood supply has manifested over time clinically and in the literature as more bone loss between implants that are closer than 3 mm. Cleansability is even more ideal with closer to 4 mm between implants, but many times with two adjacent implants we are closer to the 3 mm minimum. A tantalizing finding in recent literature is the preservation of tissue when platform-switching aka medialized abutments aka coronal beveling is present in the implant design. It is clear that there is a biologic volume that can be accommodated around the abutment junction by creating more space vs. that same biologic adaptation running down the implant body to the first thread of the olden days.