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Evidence in the social sciences
Federica Russo
Philosophy | Humanities | Amsterdam
russofederica.wordpress.com | @federicarusso
Flash back
WHAT MAKES A CAUSAL MODEL
CAUSAL
3
A question of validity
A methodological concern thoroughly analysed
since the late ‘60s
Cook & Campbell
Decide whether a model is valid
Successful inferences
Correlations, causal relations, prediction, control, …
4
Types of validity are about whether:
Statistical conclusion validity
The correlation (covariation) between treatment and
outcome is validly inferred
Internal
Observed covariation between treatment and outcome
reflects a causal relationship, as those variables are
manipulated or measured
Construct and External
The cause-effect relationship holds over variation in
persons, settings, treatment variables, and measurement
variables
5
Validity is also about evidence
Beyond the ‘Cook & Campbell Tradition’, i.e.:
Representativeness of sample and possibility to
replicate studies
Evidential pluralism
To establish causal claims, we need multiple sources
of evidence
Difference-making and mechanisms
6
EVIDENCE AND
CAUSAL ASSESSMENT
7
Causal claims
C causes E
Red meat consumption causes cancer
Poverty causes delinquency
Education lowers child mortality
Exercising reduces cardiovascular disease
…
How do we know that?
What makes a causal claim true /versus/
What evidence supports a causal claim
8
AN INFERENTIALIST APPROACH
J. Reiss, Causation, Evidence, and Inference, Routledge 2015
9
Causal claims and evidence
The meaning of causal claims
What can we legitimately infer
Causal claims are inferentially related to evidence claims
Flexibility of causal language and nature of causal relata
Freed-up from the ‘straight jacket’
10
EVIDENTIAL PLURALISM
Russo & Williamson; Clarke et al; Moneta & Russo
11
Evidential pluralism
To establish a causal claim we need multiple sources
of evidence:
That C makes a difference to E
Correlations, counterfactuals, …
That C produces E
Mechanisms, processes, …
Russo and Williamson,
Interpreting causality in the health sciences, ISPS 2007
Epistemic causality and evidence-based medicine. HPLS 2011
Clarke et al,
The evidence that evidence-based medicine omits, Preventive Medicine 2013
Mechanisms and the evidence hierarchy, Topoi 2014
Moneta and Russo
Causal models and evidential pluralism in econometrics, Journal of economic
methodology, 2014
12
EVIDENCE OF DIFFERENCE-MAKING
13
… that C makes a difference to E
Qualitative differences
Counterfactual reasoning
Hypothesis generation
…
Quantitative differences
Statistical information
Dependencies in the form of statistical correlations
Results of interventions
14
Correlations, counterfactuals, etc support the
claim that E (causally) depends on C
Needs to be complemented with story about how
15
EVIDENCE OF PRODUCTION
16
… how C produces E
A story about how C causes E. Candidates:
Processes
Describing of continuous ‘world lines’ in physics (and possibly social
science)
Mechanisms
Account for organization of parts of a system and of their
interactions
Powers
Postulating (discovering?) modal properties of some parts of
mechanisms
17
EVIDENCE OF SOCIAL MECHANISMS
18
WHY MECHANISMS?
19
Physical (causal) connections
Process theories of causality
Salmon-Dowe approach
A development of Russell-Reichenbach
(world-lines, at-at theory)
Salmon: ‘put the cause into because’
The because is given by the physical, causal process
(Ontic explanation)
20
Processes in biology?
Machamer, Darden, Craver (2000, p. 7):
Although we acknowledge the possibility that Salmon’s
analysis may be all there is to certain fundamental types
of interactions in physics, his analysis is silent as to the
character of the productivity in the activities investigated
by many other sciences. Mere talk of transmission of a
mark or exchange of a conserved quantity does
not exhaust what these scientists know about
productive activities and about how activities effect
regular changes in mechanisms.
21
Processes in social science?
Russo (2009, p.26):
The need to look directly at social scientists’ work was motivated by a
possible difference between causal claims that involve reasonably clear
causal mechanisms and causal claims that do not. I went through five case
studies, and it turned out that none of them contains concepts typical of
aleatory causality in order to get an understanding of causal relations—to
borrow Salmon’s terminology again. Instead, statistical causality is
definitively preferred. However, to prefer statistical causality does not ipso
facto rule out mechanisms from the causal talk. […] the question is not
whether or not we aim at identifying causal mechanisms, rather, how do
we come to identify them. Causal mechanisms are not identified
through causal processes and interactions, but, according to
the social scientists’ practice, they are statistically modelled.
22
THE RECENT DEBATE
23
Machamer, Darden and Craver:
‘Mechanisms are entities and activities organized such that they are
productive of regular changes from start or set-up to finish or
termination conditions.’ (Machamer, Darden and Craver 2000 p3.)
Glennan:
‘A mechanism for a behavior is a complex system that produces that
behavior by the interaction of a number of parts, where the
interactions between parts can be characterized by direct, invariant,
change-relating generalizations.’ (Glennan 2002b pS344.)
Bechtel and Abrahamsen:
‘A mechanism is a structure performing a function in virtue of its
component parts, component operations, and their organization.
The orchestrated functioning of the mechanism is responsible for
one or more phenomena.’ (Bechtel and Abrahamsen 2005 p423.)
24
A CONSENSUS
25
Illari & Williamson:
A mechanism for a phenomenon is composed of entities
and activities organized so that they are responsible for the
phenomenon.
Illari & Williamson give up on:
Regularity
Start up, finishing conditions
Complex system
Mechanistic explanation:
Identification of the phenomenon
Identification of entities and activities involved
Identification of the organisation
26
Modelling mechanisms
Quantitative causal models model mechanisms
via the ‘recursive decomposition’
27
What are the causes of self-rated health in
the Baltic countries in the ‘90s?
X Y
Joint probability distribution
P(Ed, Soc, Phy, Loc, Psy, Alc, Self)
Recursive decomposition:
P(Self|Alc, Psy, Loc, Phy)
P(Alc|Ed, Psy, Phy)
P(Psy|Loc, Soc, Phy)
P(Loc|Ed)
P(Phy) P(Soc) P(Ed)
Health survey in the Baltic countries
Why does this represent
a mechanism?
28
What mechanism?
‘Modelling mechanisms’ is not tantamount to proving a
‘metaphysical account’ of mechanism
No ontological commitment to the (degree of) physical
existence of (social) mechanisms
Mechanisms are primarily epistemic
They have explanatory power
They track something real: making sense of what actually
happens
29
Difference-making, mechanisms
and validity
To decide whether correlations (=evidence of difference-making)
are causal we have to decide about the
validity of the whole model
That is, whether the mechanism provides
a good enough explanation of the correlations.
30
The causal interpretation is
model-dependent
Causal conclusions depend on the whole
‘model set up’ from which they are inferred
Statistical information + background knowledge +
causal information
Not a bad thing after all
Causation is not a ‘all or nothing’ affair
Nor a ‘once and for all’ affair
31
TO SUM UP AND CONCLUDE
32
Evidence, causal language,
and causal modelling
What is evidence in social science?
Evidence is couched in causal language
And embedded into causal modelling practice
33
Evidential pluralism
From the health sciences to the social sciences
Not what constitute causation, but what
supports causal claims
That and How are complementary evidential
components
34
OPEN ISSUES
35
And social causation?
One word, many things
Causal pluralism, but that’s for next talk!
36

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Evidence in the social sciences - Series of lectures on causal modelling in the social sciences

  • 1. Evidence in the social sciences Federica Russo Philosophy | Humanities | Amsterdam russofederica.wordpress.com | @federicarusso
  • 3. WHAT MAKES A CAUSAL MODEL CAUSAL 3
  • 4. A question of validity A methodological concern thoroughly analysed since the late ‘60s Cook & Campbell Decide whether a model is valid Successful inferences Correlations, causal relations, prediction, control, … 4
  • 5. Types of validity are about whether: Statistical conclusion validity The correlation (covariation) between treatment and outcome is validly inferred Internal Observed covariation between treatment and outcome reflects a causal relationship, as those variables are manipulated or measured Construct and External The cause-effect relationship holds over variation in persons, settings, treatment variables, and measurement variables 5
  • 6. Validity is also about evidence Beyond the ‘Cook & Campbell Tradition’, i.e.: Representativeness of sample and possibility to replicate studies Evidential pluralism To establish causal claims, we need multiple sources of evidence Difference-making and mechanisms 6
  • 8. Causal claims C causes E Red meat consumption causes cancer Poverty causes delinquency Education lowers child mortality Exercising reduces cardiovascular disease … How do we know that? What makes a causal claim true /versus/ What evidence supports a causal claim 8
  • 9. AN INFERENTIALIST APPROACH J. Reiss, Causation, Evidence, and Inference, Routledge 2015 9
  • 10. Causal claims and evidence The meaning of causal claims What can we legitimately infer Causal claims are inferentially related to evidence claims Flexibility of causal language and nature of causal relata Freed-up from the ‘straight jacket’ 10
  • 11. EVIDENTIAL PLURALISM Russo & Williamson; Clarke et al; Moneta & Russo 11
  • 12. Evidential pluralism To establish a causal claim we need multiple sources of evidence: That C makes a difference to E Correlations, counterfactuals, … That C produces E Mechanisms, processes, … Russo and Williamson, Interpreting causality in the health sciences, ISPS 2007 Epistemic causality and evidence-based medicine. HPLS 2011 Clarke et al, The evidence that evidence-based medicine omits, Preventive Medicine 2013 Mechanisms and the evidence hierarchy, Topoi 2014 Moneta and Russo Causal models and evidential pluralism in econometrics, Journal of economic methodology, 2014 12
  • 14. … that C makes a difference to E Qualitative differences Counterfactual reasoning Hypothesis generation … Quantitative differences Statistical information Dependencies in the form of statistical correlations Results of interventions 14
  • 15. Correlations, counterfactuals, etc support the claim that E (causally) depends on C Needs to be complemented with story about how 15
  • 17. … how C produces E A story about how C causes E. Candidates: Processes Describing of continuous ‘world lines’ in physics (and possibly social science) Mechanisms Account for organization of parts of a system and of their interactions Powers Postulating (discovering?) modal properties of some parts of mechanisms 17
  • 18. EVIDENCE OF SOCIAL MECHANISMS 18
  • 20. Physical (causal) connections Process theories of causality Salmon-Dowe approach A development of Russell-Reichenbach (world-lines, at-at theory) Salmon: ‘put the cause into because’ The because is given by the physical, causal process (Ontic explanation) 20
  • 21. Processes in biology? Machamer, Darden, Craver (2000, p. 7): Although we acknowledge the possibility that Salmon’s analysis may be all there is to certain fundamental types of interactions in physics, his analysis is silent as to the character of the productivity in the activities investigated by many other sciences. Mere talk of transmission of a mark or exchange of a conserved quantity does not exhaust what these scientists know about productive activities and about how activities effect regular changes in mechanisms. 21
  • 22. Processes in social science? Russo (2009, p.26): The need to look directly at social scientists’ work was motivated by a possible difference between causal claims that involve reasonably clear causal mechanisms and causal claims that do not. I went through five case studies, and it turned out that none of them contains concepts typical of aleatory causality in order to get an understanding of causal relations—to borrow Salmon’s terminology again. Instead, statistical causality is definitively preferred. However, to prefer statistical causality does not ipso facto rule out mechanisms from the causal talk. […] the question is not whether or not we aim at identifying causal mechanisms, rather, how do we come to identify them. Causal mechanisms are not identified through causal processes and interactions, but, according to the social scientists’ practice, they are statistically modelled. 22
  • 24. Machamer, Darden and Craver: ‘Mechanisms are entities and activities organized such that they are productive of regular changes from start or set-up to finish or termination conditions.’ (Machamer, Darden and Craver 2000 p3.) Glennan: ‘A mechanism for a behavior is a complex system that produces that behavior by the interaction of a number of parts, where the interactions between parts can be characterized by direct, invariant, change-relating generalizations.’ (Glennan 2002b pS344.) Bechtel and Abrahamsen: ‘A mechanism is a structure performing a function in virtue of its component parts, component operations, and their organization. The orchestrated functioning of the mechanism is responsible for one or more phenomena.’ (Bechtel and Abrahamsen 2005 p423.) 24
  • 26. Illari & Williamson: A mechanism for a phenomenon is composed of entities and activities organized so that they are responsible for the phenomenon. Illari & Williamson give up on: Regularity Start up, finishing conditions Complex system Mechanistic explanation: Identification of the phenomenon Identification of entities and activities involved Identification of the organisation 26
  • 27. Modelling mechanisms Quantitative causal models model mechanisms via the ‘recursive decomposition’ 27
  • 28. What are the causes of self-rated health in the Baltic countries in the ‘90s? X Y Joint probability distribution P(Ed, Soc, Phy, Loc, Psy, Alc, Self) Recursive decomposition: P(Self|Alc, Psy, Loc, Phy) P(Alc|Ed, Psy, Phy) P(Psy|Loc, Soc, Phy) P(Loc|Ed) P(Phy) P(Soc) P(Ed) Health survey in the Baltic countries Why does this represent a mechanism? 28
  • 29. What mechanism? ‘Modelling mechanisms’ is not tantamount to proving a ‘metaphysical account’ of mechanism No ontological commitment to the (degree of) physical existence of (social) mechanisms Mechanisms are primarily epistemic They have explanatory power They track something real: making sense of what actually happens 29
  • 30. Difference-making, mechanisms and validity To decide whether correlations (=evidence of difference-making) are causal we have to decide about the validity of the whole model That is, whether the mechanism provides a good enough explanation of the correlations. 30
  • 31. The causal interpretation is model-dependent Causal conclusions depend on the whole ‘model set up’ from which they are inferred Statistical information + background knowledge + causal information Not a bad thing after all Causation is not a ‘all or nothing’ affair Nor a ‘once and for all’ affair 31
  • 32. TO SUM UP AND CONCLUDE 32
  • 33. Evidence, causal language, and causal modelling What is evidence in social science? Evidence is couched in causal language And embedded into causal modelling practice 33
  • 34. Evidential pluralism From the health sciences to the social sciences Not what constitute causation, but what supports causal claims That and How are complementary evidential components 34
  • 36. And social causation? One word, many things Causal pluralism, but that’s for next talk! 36

Editor's Notes

  1. Recall the context: when I started, Salmon was still the dominant paradigm