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Basics of ECG
Interpretation
The Normal Conduction System
EKG Leads
The standard EKG has 12 leads: 3 Standard Limb Leads
3 Augmented Limb Leads
6 Precordial Leads
The axis of a particular lead represents the viewpoint from
which it looks at the heart.
All Limb Leads
Precordial Leads
Precordial Leads
Anatomic Groups
(Summary)
Rate
►Rule of 300
►10 Second Rule
Rule of 300
Take the number of “big boxes” between neig
hboring QRS complexes, and divide this into 3
00. The result will be approximately equal to
the rate
Although fast, this method only works for reg
ular rhythms.
What is the heart rate?
(300 / 6) = 50 bpm
www.uptodate.com
What is the heart rate?
(300 / ~ 4) = ~ 75 bpm
www.uptodate.com
What is the heart rate?
(300 / 1.5) = 200 bpm
The Rule of 300
It may be easiest to memorize the following table:
# of big boxes Rate
1 300
2 150
3 100
4 75
5 60
6 50
10 Second Rule
As most EKGs record 10 seconds of rhythm per page
, one can simply count the number of beats present
on the EKG and multiply by 6 to get the number of b
eats per 60 seconds.
This method works well for irregular rhythms.
What is the heart rate?
33 x 6 = 198 bpm
The Alan E. Lindsay ECG Learning Center ; http://medstat.med.utah.edu/kw/ecg/
The QRS Axis
By near-consensus, the norm
al QRS axis is defined as ran
ging from -30° to +90°.
-30° to -90° is referred to as a
left axis deviation (LAD)
+90° to +180° is referred to as
a right axis deviation (RAD)
Determining the Axis
►The Quadrant Approach
►The Equiphasic Approach
Determining the Axis
Predominantly
Positive
Predominantly
Negative
Equiphasic
The Quadrant Approach
1. Examine the QRS complex in leads I and aVF to determine
if they are predominantly positive or predominantly negativ
e. The combination should place the axis into one of the 4
quadrants below.
The Quadrant Approach
2. In the event that LAD is present, examine lead II to determi
ne if this deviation is pathologic. If the QRS in II is predomi
nantly positive, the LAD is non-pathologic (in other words, th
e axis is normal). If it is predominantly negative, it is pathol
ogic.
Quadrant Approach: Example 1
Negative in I, positive in aVF  RAD
The Alan E. Lindsay EC
G Learning Center http:/
/medstat.med.utah.edu/
kw/ecg/
Quadrant Approach: Example 2
Positive in I, negative in aVF  Predominantly positive in II 
Normal Axis (non-pathologic LAD)
The Alan E. Lindsay EC
G Learning Center http:/
/medstat.med.utah.edu/
kw/ecg/
The Equiphasic Approach
1. Determine which lead contains the most equiphasic QRS co
mplex. The fact that the QRS complex in this lead is equall
y positive and negative indicates that the net electrical vect
or (i.e. overall QRS axis) is perpendicular to the axis of this
particular lead.
2. Examine the QRS complex in whichever lead lies 90° away
from the lead identified in step 1. If the QRS complex in th
is second lead is predominantly positive, than the axis of th
is lead is approximately the same as the net QRS axis. If t
he QRS complex is predominantly negative, than the net Q
RS axis lies 180° from the axis of this lead.
Equiphasic Approach: Example 1
Equiphasic in aVF  Predominantly positive in I  QRS axis ≈ 0°
The Alan E. Lindsay ECG Learning Center ; http://medstat.med.utah.edu/kw/ecg/
Equiphasic Approach: Example 2
Equiphasic in II  Predominantly negative in aVL  QRS axis ≈ +150°
The Alan E. Lindsay ECG Learning Center ; http://medstat.med.utah.edu/kw/ecg/
Systematic Approach
► Rate
► Rhythm
► Axis
► Wave Morphology
 P, T, and U waves and QRS compl
ex
► Intervals
 PR, QRS, QT
► ST Segment
Rhythms/Arrhythmias
►Sinus
►Atrial
►Junctional
►Ventricular
Sinus Rhythms: Criteria/Types
► P waves upright in I, II, aVF
► Constant P-P/R-R interval
► Rate
► Narrow QRS complex
► P:QRS ratio 1:1
► P-R interval is normal and constant
Sinus Arrhythmias: Criteria/Types
►Normal Sinus Rhythm
►Sinus Bradycardia
►Sinus Tachycardia
►Sinus Arrhythmia
Normal Sinus Rhythm
• Rate is 60 to 100
Sinus Bradycardia
• Can be normal variant
• Can result from medication
• Look for underlying cause
Sinus Tachycardia
• May be caused by exercise, fever,
hyperthyroidism
• Look for underlying cause, slow the rate
Sinus Arrhythmia
• Seen in young patients
• Secondary to breathing
• Heart beats faster
Atrial Arrhythmias: Criteria/Types
►P waves inverted in I, II and aVF
►Abnormal shape
 Notched
 Flattened
 Diphasic
►Narrow QRS complex
Atrial Arrhythmias: Criteria/Types
►Premature Atrial Contractions
►Multifocal Atrial Tachycardia
►Atrial Flutter
►Atrial Fibrillation
Premature Atrial Contraction
• QRS complex narrow
• RR interval shorter than sinus QRS
complexes
• P wave shows different morphology
than sinus P wave
Ectopic Atrial Rhythm
• Narrow QRS complex
• P wave inverted
Wandering Atrial Pacemaker
• 3 different P wave morphologies
possible with ventricular rate < 100 bpm
Multifocal Atrial Tachycardia
• 3 different P wave morphologies
with ventricular rate> 100 bpm
Atrial Flutter
• Regular ventricular rate 150 bpm
• Varying ratios of F waves to QRS
complexes, most common is 4:1
• Tracing shows 2:1 conduction
Atrial Flutter
• Tracing shows 6:1 conduction
Atrial Fibrillation
• Tracing shows irregularly irregular
rhythm with no P waves
• Ventricular rate usually > 100 bpm
Atrial Fibrillation
• Tracing shows irregularly irregular
rhythm with no P waves
• Ventricular rate is 40
►P wave
 May be absent
►Buried in QRS
 If present
► inverted in leads I, II, and aVF
►Inverted after QRS
Junctional Arrhythmias: Criteria
►PR interval < 0.12 secs
►Rate: Varies
►Narrow QRS complex
Junctional Arrhythmias: Criteria
Junctional Arrhythmias: Types
► Premature Junctional Contractions
► Junctional Escape Rhythm
► Accelerated Junctional Tachycardia
► Junctional Tachycardia
► Reentrant Tachycardia
► AVNRT
Junctional Escape Rhythm
• Junctional origin
• Rate is 40 to 60
Junctional Tachycardia
• Junctional origin
• Rate is > 100
• Secondary to bypass tract within AV node
• Premature Atrial Contraction (PAC)
depolarizes
AV Nodal Reentrant Tachycardia (A
VNRT)
Rate Summary
► Sinus Tachycardia - 100-160 BPM
► Atrial Tachycardia - 150-250 BPM
► Atrial Flutter - 150-250 BPM
► Junctional Tachycardia - 100-180 BPM
AV Nodal Blocks
• Delay conduction of impulses from
sinus node
• If AV node does not let impulse
through, no QRS complex is seen
• AV nodal block classes:
1st, 2nd, 3rd degree
1st Degree AV Block
• PR interval constant
• >.2 sec
• All impulses conducted
2nd Degree AV Block Type 1
• AV node conducted each impulse
slower and finally no impulse is
conducted
• Longer PR interval, finally no QRS
complex
2nd Degree AV Block Type 2
• Constant PR interval
• AV node intermittently conducts
no impulse
• AV node conducts no impulse
• Atria and ventricles beat at intrinsic
rate (80 and 40 respectively)
• No association between P waves and
QRS complexes
3rd Degree AV Block
• Caused by bypass tra
ct
• AV node is bypassed,
delay
• EKG shows short PR
interval <.11 sec
• Upsloping to QRS
complex (delta wave)
Another Consideration:
Wolfe-Parkinson-White (WPW)
• Delta wave, short PR interval
WPW
Ventricular Arrhythmias: Criter
ia/Types
► Wide QRS
complex
► Rate :
variable
► No P waves
► Premature Ventricular Contracti
ons
► Idioventricular Rhythm
► Accelerated IVR
► Ventricular Tachycardia
► Ventricular Fibrillation
• Occurs earlier than sinus beat
• Wide, no P wave
Premature Ventricular Contraction
• Escape rhythm
• Rate is 20 to 40 bpm
Idioventricular Rhythm
• Rate is 40 to 100 bpm
Accelerated Idioventricular Rhythm
• Rate is > than 100 bpm
Ventricular Tachycardia
Torsades de Pointes
• Occurs secondary to prolonged
QT interval
• Unorganized activity of ventricle
Ventricular Tachycardia/Fibrillation
Ventricular Fibrillation
Chamber Enlargements
EKG Criteria
► -S V1, V2 + R V5,V6 > 35 42.5% 95% (Sokolow-Lyon)
► -R V5 orV6 > 27 25% 98%
► -R V5 or V6 > 25 Framingham 14% 86%
► -R plus S > 45 any lead 45% 93%
► Limb leads
► -R in L + S in V3 >25 in men,
► and >20 in women. 16% 97%
► (Cornell Voltage)
► -R in I + S in III > 25 10.6% 100%
► -R in L > 11 man, 9 wom 10.6% 100%
► -R in avF > 20 1.3% 99.5%
► -S in avR > 14
LVH with Strain
Right Ventricular Hypertrophy
►Reversal of precordial pattern
 R waves prominent in V1 and V2
 S waves smaller in V1 and V2
 S waves become prominent in V5 and V
6
Right Ventricular Hypertrophy
Left Atrial Enlargement: Criteria
► P wave
► Notch in P wave
 Any lead
 Peaks > 0.04 secs
► V1
 Terminal portion of P wave > 1mm deep and >
0.04 sec wide
P Wave: Left Atrial Enlargement
Left Atrial Enlargement
Lead V1
►Tall, peaked P wave
> 2.5 mm in any lead
►Most prominent P waves in leads I, II and aV
F
Right Atrial Enlargement: Criteria
Right Atrial Enlargement
Bundle Branch Blocks
Bundle Branch Blocks
►Complete
 QRS > .12 secs
►Incomplete
 QRS .10 - .12 secs
►Left
 Complete
 Incomplete
►Right
 Complete
 Incomplete
Criteria for Left Bundle Branch Bl
ock (LBBB)
► Bizarre QRS Morphology
 High voltage S wave in V1, V2 & V3
 Tall R wave in leads I, aVL and V5-6
► Often LAD
► QRS Interval
► ST depression in leads I, aVL, & V5-V6
► T wave inversion in I, aVL, & V5-V6
Left Bundle Branch Block
Criteria for Right Bundle Branc
h Block (RBBB)
► QRS morphology
 Wide S wave in leads I and V4-V6
 RSR’ pattern in leads V1, V2 and V3
► QRS duration
► ST depression in leads V1 and V2
► T wave inversion in leads V1 and V2
Right Bundle Branch Block
Ischemia and Infarction
Normal Complexes and Segments
J Point
Ischemia
•T wave inversion, ST segment depression
•Acute injury: ST segment elevation
•Dead tissue: Q wave
Measurements
ST Segment Depression
Can be characterised as:-
►Downsloping
►Upsloping
►Horizontal
EKG Changes: Ischemia →
Acute Injury→ Infarction
Evolution of Transmural Infarctio
n
Hyperacute T waves
Q Waves
Non Pathological Q waves
Q waves of less than 2mm are normal
Pathological Q waves
Q waves of more than 2mm
indicate full thickness myocardial
damage from an infarct
Late sign of MI (evolved)
Look for Grouped Patterns (Footp
rints)
►ST Depressions = Ischemia
►ST Elevations = injury
►Q Waves & T Wave Inversion = Infarction
ST-T Wave Changes
T waves
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