Presentation about Knowledge and medical and nursing Management , Dm types , clinical features, complications of diabetes mellitus
. King Saud university
SIGNIFICANCE
OVERVIEW
WHAT IS DIABETES?
DEFINITION
MECHANISM
PREVELANCE
EPIDEMIOLOGY
CLASSIFICATION
GESTATIONAL DIABETES
RISK FACTORS
DIAGNOSIS
COMPLICATIONS
MEDICAL TEST
MEDICAL NUTRITIONAL THERAPY
HERBS FOR DIABETES
MYTHS AND FACTS
REFERENCES
SIGNIFICANCE
OVERVIEW
WHAT IS DIABETES?
DEFINITION
MECHANISM
PREVELANCE
EPIDEMIOLOGY
CLASSIFICATION
GESTATIONAL DIABETES
RISK FACTORS
DIAGNOSIS
COMPLICATIONS
MEDICAL TEST
MEDICAL NUTRITIONAL THERAPY
HERBS FOR DIABETES
MYTHS AND FACTS
REFERENCES
Type 1 Diabetes Mellitus is a form of diabetes mellitus that results from the autoimmune destruction of the insulin-producing beta cells in the pancreas. Genetic factors are believed to be a major com- ponent for the development of type 1diabetes, but the con- cordance rate for the development of diabetes in identical twins is only about 40%, suggesting that non-genetic factors play an important role in the expression of the disease. Viruses are one environmental factor that is implicated in the pathogenesis of type 1 diabetes mellitus.
Shashikiran Umakanth made this presentation at the "First Endocrine Update Program” – ENDO EGYPT 2015, from 17-20 December 2015 in the Historic City of Luxor, Egypt. This endocrine update was organised by the Egyptian Association of Endocrinology , Diabetes and Atherosclerosis (EAEDA) in collaboration with the Endocrine Society, USA.
Nursing Management · Monitor blood sugar and use a sliding scale to treat high levels of glucose · Educate patient about diabetes · Examine feet .
Diagnosis involves measuring blood glucose levels. Ongoing specialized assessment and evaluation for complications are essential for diabetes management.
Type 1 Diabetes Mellitus is a form of diabetes mellitus that results from the autoimmune destruction of the insulin-producing beta cells in the pancreas. Genetic factors are believed to be a major com- ponent for the development of type 1diabetes, but the con- cordance rate for the development of diabetes in identical twins is only about 40%, suggesting that non-genetic factors play an important role in the expression of the disease. Viruses are one environmental factor that is implicated in the pathogenesis of type 1 diabetes mellitus.
Shashikiran Umakanth made this presentation at the "First Endocrine Update Program” – ENDO EGYPT 2015, from 17-20 December 2015 in the Historic City of Luxor, Egypt. This endocrine update was organised by the Egyptian Association of Endocrinology , Diabetes and Atherosclerosis (EAEDA) in collaboration with the Endocrine Society, USA.
Nursing Management · Monitor blood sugar and use a sliding scale to treat high levels of glucose · Educate patient about diabetes · Examine feet .
Diagnosis involves measuring blood glucose levels. Ongoing specialized assessment and evaluation for complications are essential for diabetes management.
Diabetes is a disease that occurs when your blood glucose, also called blood sugar, is too high. Blood glucose is your main source of energy and comes from the food you eat. Insulin, a hormone made by the pancreas, helps glucose from food get into your cells to be used for energy. Sometimes your body doesn’t make enough—or any—insulin or doesn’t use insulin well. Glucose then stays in your blood and doesn’t reach your cells.
Advanced PharmacologyDiabetes is a group of metabolic diseasmilissaccm
Advanced Pharmacology
Diabetes is a group of metabolic diseases characterized by hyperglycemia resulting from defects in insulin secretion, insulin action, or both. ( McCance & Huether 2019). Per The American Diabetic Association (ADA) each year 1.5 million Americans are diagnosed with diabetes. There are different types of diabetes in this paper we will differentiate between the various types and focus on type 2 diabetes.
Comparison of different types of diabetes including drug treatment
Type 1 Diabetes: caused by Beta-cell destruction leading to absolute insulin deficiency ( pancreas stops producing insulin) . This is the most common form of diabetes. Patients with type 1 diabetes are insulin-dependent. About 75% of them develop before the age of 30. Patients are prone to diabetic ketoacidosis. Type 1 diabetes is secondary to other diseases such as pancreatitis, it is also said to be caused by genetic predisposition and environmental factors. It is most common in people of African and Asian descent. Clinical manifestations of type 1 diabetes include Polydipsia, polyuria, polyphagia, weight loss, and fatigue. Patients with type 1 diabetes take different types of insulin. Rapid-acting (Lispro ), short-acting ( regular insulin ) intermediate-acting ( NPH), long-acting (glargine).
Type 2 Diabetes; is caused by the progressive loss of b-cell insulin secretion frequency on the background of insulin resistance ( insulin becomes resistant, may be produced but does not do its job). Patients with type 2 diabetes are usually not insulin dependent but might be insulin requiring depending on the severity of hyperglycemia. Not ketosis-prone but may form ketones under stress. obesity in the abdominal region. Mostly occurs in those 40years of age and above . has a strong genetic predisposition and is often associated with dyslipidemia and hypertension. Prevalence is highest among American Indians and Alaska Natives and lowest among non-Hispanic whites. Clinical manifestations of type 2 diabetes are nonspecific but the most common ones are; recurrent infections ( boils and carbuncles, skin infections, and prolonged wound healing ), genital pruritus, visual changes, paresthesias, fatigue, and Acanthosis nigricans ( brown to black pigmentation in body folds associated with insulin resistance ). Treatment of diabetes type two involves lifestyle modifications like weight loss which results in improved insulin sensitivity, and glucose tolerance, and preserves beta-cell function. In cases where diet and exercise yield unsatisfactory results, first line of drugs for type 2 diabetes is Sulfonylurea, and in some cases insulin is added if the oral hypoglycemic agents do not work. they are nine classes of oral hypoglycemic agents with metformin being the drug of choice.
Gestational Diabetes Mellitus ( GDM); Any degree of glucose intolerance with onset or first recognition of symptoms during pregnancy. Caused by insulin resistance combined with inad ...
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Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
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O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
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Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
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- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
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2. Contents
Introduction
Types of diabetes mellitus
Signs and symptoms of diabetes mellitus
Pathophysiology of diabetes mellitus
Complications of diabetes mellitus
Diagnosis of diabetes mellitus
Medical Management of diabetes mellitus
Nursing Management of diabetic patients
Discharge plane
Reference
3. Introduction
Diabetes mellitus: is a chronic metabolic disorders
charaterised by high level of blood glucose
(hyperglycaemia).
Diabetes mellitus is now one of the most common
disease all over the world. This disease classified as
two types : DM type 1 which is due to insulin
insufficiency and DM type 2 wich is due to insulin
resistance and gestational DM wich is rare and come
withe some female during pregnancy period.
DM affect all body tissues and organs so diabetic
patients com with multiple clinical features involving
many systems.
5. DM Type 1
(Insulin dependent
diabetic)
results from failure
of pancreas to
produce insulin
hormone wich is
responsible for
blood glucose
regulation,
- this type onset
suddenly, most
commonly affect
children.
6. DM Type 1
- DM type 1 is
mandatory managed
by insulin therapy.
- The prevalence of
this type is 10% of
diabetic patient.
7. DM Type 2
( insulin independent
diabetes) results
from failure of body's
cells to respond to
insulin hormone
properly
(insulin resistance).
8. DM Type 2
DM Type 2: this type
onset gradully, most
commonly affect adult
person.
This is type managed by
oral drugs or nsulin
therapy when disease
uncontrolled with
previous drugs.
The prevalence of this
type is 90% of diabetic
patient.
10. Symptoms of DM
The classic symptoms of diabetes mellitus are as follows:
* Polyuria ( excessive urination)
* Polydipsia( excessive thrist)
* Polyphagia (strong desire to eat)
* Unexplained weight loss
* Other symptoms may include fatigue, nausea, and
blurred vision.
Symptoms of type 1 DM developed rapidly during weeks or
month, However, many patients with type 2 diabetes are
asymptomatic, and their disease remains undiagnosed for
many years.
11.
12. Pathophysiology of type 1 DM
The main isulin hormone function is to transport of
glucose from blood to body's tissues e.g: fat, muscles.
Insulin hormone produced by beta cells that lies in
langerhans's islets of pancrease and destruction of
insulin-secreting beta cells mass result in declines
insulin secretion until the available insulin is
unadequate to maintain normal blood glucose levels,
hyperglycemia develops and diabetes mellitus
diagnosed.
13. Pathophysiology of type 2 DM
Type 2 diabetes is characterized by peripheral
insulin resistance, which has been attributed
to elevated levels of free fatty acids and
proinflammatory cytokines in plasma, leads to
decreased glucose transport into muscle cells,
elevated hepatic glucose production, and
increased breakdown of fat.
14. Complications of DM
- Macrovascular complications including: increase
risks of cardiovascular diseases e.g:coronary artery
diseases and cerebrovasculare accident.
- Microvascular complications: diabeteic retinopathy,
nephropathy and neuropathy. a vasculopathy
and neuropathy is the leading cause of Diabeteic
Foot and occasionally requiring ambutation.
15.
16. Diagnosis of DM
Diagnostic criteria by the American Diabetes
Association (ADA) include the following:
* A fasting plasma glucose (FPG) level of
126
mg/dL
or higher.
*
2
-
hour plasma glucose level of
200
mg/dL or
higher during a
75
-
tset ecnarelot esoculg laro g
(TTGO)
,
* A random plasma glucose of
200
mg/dL
or
higher
in a patient with classic symptoms of hyperglycemia
or hyperglycemic crisis.
21. Medical management of
DM
- Goals of medical management
- Nutritional management
- pharmacological therapy
22. Goals of DM treatment
- Microvascular risk reduction through control
of glycemia and blood pressure
- Macrovascular risk reduction through control
of lipids and hypertension, smoking cessation
- Metabolic and neurologic risk reduction
through control of
hyper-glycemia
24. Pharmalogical therapy
* Exogenous insulin: In type 1 diabetes,
exogenous insulin must be administered for
life because the body loses the ability to
produce insulin.
* Oral antidiabetic agents may be effective for
patients who have type 2 diabetes.
26. Nursing Assessment
- Assess the patient’s history and physical condition,
assess the patient’s blood pressure 'Assess the body
mass index and visual acuity of the patient.
- Perform examination of foot, skin, mouth and
nervous system.
- Laboratory examinations: HgbA1C, fasting blood
glucose, lipid profile, microalbuminuria test, serum
creatinine level, urinalysis, and ECG must be
requested and performed.
27. Nursing Diagnoses
- Risk for unstable blood glucose level
- Risk for infection.
- Risk for disturbed sensory perception
- Impaired skin integrity related to delayed
wound healing.
- Ineffective peripheral tissue perfusion
28. Nursing Priorities
- Restore fluid/electrolyte and acid-base
balance.
- Correct metabolic abnormalities.
- Identify/assist with management of underlying
cause of disease process.
- Prevent complications.
- Provide information about disease process,
prognosis, self-care, and treatment needs.
29. Nursing Interventions
- Educate about home glucose monitoring.
- Discuss how client’s antidiabetic medications
work.
- Check viability of insulin.
- Review type of insulin used.
- Check injection sites periodically
30. Discharge and Home Care
Guidelines
- Simplify the treatment regimen if it is
difficult for the patient to follow.
- Adjust the treatment regimen to meet
patient requests.
- Educate client on wound care, insulin
preparation, and glucose monitoring
- Patient empowerment is the focus of
diabetes education.
31.
32. Reference
- [Guideline] Diagnosis and classification of diabetes
mellitus. Diabetes Care. 2010 Jan. 33 Suppl 1:S62-9.
-American Diabetes Association. Standards of medical
care in diabetes--2012. Diabetes Care
- Complete Nurse’s Guide to Diabetes Care
- Medical-Surgical Nursing: Assessment and Management
of Clinical Problems
- Brunner & Suddarth’s Textbook of Medical-Surgical
Nursing