This document describes the key features to note when describing basic skin lesions, including size, shape, color, and distribution. It categorizes lesions as primary, secondary, or special and defines common types such as macules, papules, vesicles, bullae, and pustules. Secondary lesions include scales, crusts, erosions, and fissures. The document provides examples of different lesions and explores their characteristic arrangements, colors, and predilection for specific body sites.
Ocular involvement in HIV could be caused by opportunistic infections, vascular abnormalities, neoplasms, neuro-ophthalmic conditions, and adverse effects of medications.
Ocular involvement in HIV infection occurs most commonly due to opportunistic infections and neoplasms. But also can be due to drug related and direct infections.
Opportunistic infections like CMV retinitis occur with a significantly reduced CD4 T-cell count and are one of the common causes of blindness in HIV patients.
Unlike other diseases, ocular infection in these immunosuppressed patients is associated with minimal inflammatory signs.
HIV has been isolated from tears, cornea, vitreous, and chorioretinal tissue in affected persons.
The ocular structures affected by HIV include the adnexa, anterior segment, posterior segment, and orbit.
Neuro ophthalmological manifestations also may be seen.
The institution of highly active antiretroviral therapy (HAART) has caused a dramatic improvement in the immune status of HIV-infected individuals and a change in the clinical presentation and course of opportunistic infections.
Ocular involvement in HIV could be caused by opportunistic infections, vascular abnormalities, neoplasms, neuro-ophthalmic conditions, and adverse effects of medications.
Ocular involvement in HIV infection occurs most commonly due to opportunistic infections and neoplasms. But also can be due to drug related and direct infections.
Opportunistic infections like CMV retinitis occur with a significantly reduced CD4 T-cell count and are one of the common causes of blindness in HIV patients.
Unlike other diseases, ocular infection in these immunosuppressed patients is associated with minimal inflammatory signs.
HIV has been isolated from tears, cornea, vitreous, and chorioretinal tissue in affected persons.
The ocular structures affected by HIV include the adnexa, anterior segment, posterior segment, and orbit.
Neuro ophthalmological manifestations also may be seen.
The institution of highly active antiretroviral therapy (HAART) has caused a dramatic improvement in the immune status of HIV-infected individuals and a change in the clinical presentation and course of opportunistic infections.
Other cutaneous problems associated with viral infectionsdr maria saeed
This ppt include Pityriasis rosea,Papular pruritic gloves and socks syndrome,Torch infection,gianotti crosti syndrome,Measles from text book of Rook's dermatology
263778731218 Abortion Clinic /Pills In Harare ,sisternakatoto
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micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
2. ● Size
● Types of Lesions
● Shape of lesions
● Pattern
● Color and pigmentation
● Distribution over the body surface
● Distribution of Lesions
WHEN DESCRIBING A SKIN
LESION,IT IS IMPORTANT TO NOTE
THE FOLLOWING FEATURES:-
3. TYPES OF LESION
Basic skin lesions are broadly categorized as :
1. Primary
2. Secondary
3. special
4. ● Primary lesions :- Basic reaction patterns of skin
with a definite morphology.
● Secondary lesion :- Develop during the evolutionary
process of skin disease or are created by scratching or
infection.
● Special skin lesion :- Specific for certain disease.
6. MACULE
● A Flat lesion with well
circumscribed change in skin
colour.Macules are non-
palpable.
● They are 0.5cm-1cm in size.
● Discoloration may be brown,
blue ,red and hypopigmented
or hyperpigmented
7. PATCH
● A macule is called a
patch usually larger than
1cm in size.
● Eg :- Vitiligo, melasma,
Congenital birth mark
etc.
8. PAPULE
● Elevated solid lesion less
than 1 cm.
● Papules may be of
various colors.
● E.g. Mole, Acne etc.
9. PLAQUE
● It is an indurated area of skin larger than 1 cm in
diameter which may be raised or depressed from
skin surface.
17. NODULE
● A Nodule is a large ( 0.5 – 5.0 cm ) firm lesion
● Could be warm, soft,fluctuant,movable,fixed or
painful.
● Surface-smooth,keratotic,ulcerated or fungating.
19. ABSCESS
● An Abscess is a collection of
pus usually caused by
bacterial infection.
● We usually find two types of
abscess (I) Skin Abscess (II)
Internal Abscess.
20. WHEAL
● It is a transient swelling
of skin disappearing
within 24 hrs.
● It is formed due to
sudden extravasation of
fluid in the dermis.
● Eg: Urticaria(hives)
21. CYST
● It is a spherical or oval
sac or an encapsulated
cavity containing fluid
or semi solid material.
● It is lined with true
epithelium.
23. SCALE
● Excess dead epidermal
cells that are produced
by abnormal
keratinization and
shedding
● Eg: Psoriasis, Icthyosis
24. CRUST
● Dried exudate of body
fluids (blood / serous
fluid).
● Which might be either
yellow / red.
● E.g. :- Tinea Capitis,
Impetigo.
25. EROSION
● A focal loss of epidermis.
● Erosions do not
penetrate below the
dermoepidermal
junction and therefore
heal without scarring.
● Eg:- tinea pedis,
candidiasis, eczematous
disease, herpes simplex.
26. ULCER
● A focal loss of epidermis
and/or dermis
● Scarring depends on the
depth of the ulcer
● Eg-
Pyoderma
gangrenosum,decubitus
etc.
28. FISSURE
● It is a linear loss of
continuity of skin due to
excessive tension.
● Eg:- eczema(fingertips),
intertrigo.
29. SCAR
● It is replacement of
normal skin by fibrous
tissue in the process of
healing of damaged skin.
● Scars are of two types-
hypertrophic and
atrophic.
● Eg:- acne, burns, herpes
zoster, keloid.
30. ● An area of overgrowth of
fibrous tissue that usually
develops after healing of skin
injury & extends beyond the
original defect.
Keloid Scar
31. ATROPHY
● Atrophy is reduction in size
and number of skin cells.
● It may be limited to epidermis,
dermis, or subcutaneous tissue.
● Eg:- leprosy,
atrophoderma,
lipoatrophy.
32. LICHENIFICATION
● Repeated rubbing of
skin results in
thickening and
hyperpigmentation of
skin.
● The skin markings
become prominent.
● Eg:- Lichen simplex
chronicus, Atopic
dermatitis.
34. BURROW
● It is a serpentine tunnel
made by scabies mite in
stratum corneum.
● The open end of the
tunnel has a papule.
35. COMEDONE
● It is a tiny plug present at
opening of hair follicle
formed by keratin and
sebum.
● It is of two types: Open
comedone (black head)
and Closed comedone
(white head).
36. MILIA
● It is a tiny superficial
cyst with epidermal
lining. Milia are seen on
face at periorbital region.
37. TELENGIECTASIA
● It is visible dilataion of
capillaries of skin which
blanch on pressure.
● Eg:- Dermatomyositis,
Systemic sclerosis.
38. POIKILODERMA
● It is a combination of
reticulate telengiectasia,
pigmentary change and
atrophy.
● Eg:- Dermatomyositis,
poikilo derma of civatte
48. COLOUR OF SKIN LESIONS
Colour Examples
Black Melanin e.g. melanoma.
Brown ( Dark, Pale
& Muddy)
Freckles, Beckers nevus.
Blue-Purple Angiomas ( Vascular Lesions), Blue nevus.
Red- Brown Secondary syphilis, Drug Eruptions.
Scarlet Red Pyogenic granuloma
Yellow- white Xanthomas, Molluscum contagiosum, Skin tags.
White-Pale Vitiligo, Tinea versicolor
Normal skin Colour is due to melanin, haemoglobin,
oxy-haemoglobin, carotenoid.
55. DISTRIBUTION OF LESIONS
● Dermatomal/
zosteriform.
● Blaschkoid- following lines of
skin cell migration during
embryogenesis.
Longitudinal on limbs
Circumferential on trunk.
56. ● Symmetrical – Classical Psoriasis
(equally on extensor surfaces.)
● Asymmetrical – Cutaneous T-Cell
Lymphoma
● Photo Distributed – Dermatoses
caused by light – Chronic Actinic
Dermatitis.
● Airborne – It occurs due to allergens
on light spared areas.
57. DISORDERS HAVING PREDILECTION FOR
SPECIFIC BODY SITES
Body
Site
Type of Disorder Example
Scalp Hair Disorder, Dermatosis. Alopecia Areata,
Androgenic Areata.
Eye Lids Inflammatory Dermatosis,
Localized Lesion.
Contact Allergy, Atopic
Eczema.
Ears Inflammatory Dermatosis,
Localized Lesion.
Seborrhoeic Dermatitis,
Psoriasis.
Face Inflammatory Dermatosis,
Localized Lesion, Infection.
Acne, Seborrhoeic
Dermatitis, Lupus Rash.
Lips Inflammatory Dermatosis,
Localized Lesion, Infection.
Cheilitis, Viral warts
Cont…
58. Body
Site
Type of Disorder Example
Hands Inflammatory Dermatosis,
Localized Lesion, Infection,
Nail Disorder.
Erythema Multiform,
Photo-sensitivity,
Onychomycosis, Scabies
Limbs Inflammatory Dermatosis,
Localized Lesion, Infection.
Psoriasis, Contact
Dermatitis.
Feet Inflammatory Dermatosis,
Localized Lesion, Infection.
Psoriasis, Fungal
Infections, Corns and
Callus
Axillae Inflammatory Dermatosis,
Localized Lesion, Infection.
Psoriasis, Contact
Dermatitis.
Genitals Inflammatory Dermatosis,
Localized Lesion, Infection.
Genital Warts, STD.