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B Y T I N A
THE CORNEA
OUTLINES
 INTRODUCTION
 FACTS
 LAYER OF THE CORNEA
 FUNCTION
INTRODUCTION
 It’s the outer most layer of the eye, domed shaped,
transparent tissue that covers the eyes.
 It has about three quarters of the optical power of
the eye
 its is inserted into the sclera at the limbus, its
about 540 micrometer thick centrally
FACTS!!!
 Did u know!!! the cornea is the mostly densely
innervated tissues in the body!!
 Supplied by the first branch of trigeminal nerve
 It has no blood vessels although its alive, it
receives all the nutrients from the aqueous humor
(posteriorly) and tears (anteriorly).
Cornea transparency is due to;
 Uniform structure
 A vascularity
 Deturgescence (relative dehydration)
 cornea has five layers ;
i) The epithelium;
 Is stratified squamous and non keratinized
 It has nerve endings
 acts as a barrier of the eye to the foreign material
eg dust.
 It absorb oxygen and nutrients form tears
ii) Bowman’s membrane
 Its formed by collagen fibres, when injured it forms
scar as it heals, if the scar will form centrally the
there will be visual loss.
iii) The stroma
 It contribute to about 90% of the corneal thickness.
 Its mainly water and collagens, collagens are the
ones which gives the cornea its structure and
elasticity.
 The stoma can scar but can not regenerate.
iv) Descement’s membrane
 It has a regenerative potentials
v) The endothelium
 It maintain corneal deturgescence by pumping
excess fluid out of the stoma, failure of
endothelium to do its work can lead to cornea
edema
 Its cells cannot regenerate.
 There is loss of cells in this layer as the age
increases
DIAGRAM
FUNCTION OF THE CORNEA
 It helps in focusing the light that come into the eye
so as it can fall on the retina.
 It’s a physical barrier together with tears and eyelid
against the dirty and germs that come into contact
with the eye.
 It also protect the eye against the UV light from the
sun.
 The cornea can be affected by different diseases,
keratoconus being among them
KERATOCONUS
 It’s a disease characterized by progressive thinning
of the cornea and takes a cone shape which results
to astigmatism (the cornea cannot focus the light
to the retina).
 Most of refraction of light occurs at the air –cornea
interface so change in the cornea shape can lead to
major impact on vision.
Signs
 Bulging of the lower eyelid on down
gazing(munson sign)
 Oil droplets reflex on direct ophthalmoscope
 Scissors reflex on retinoscopy
 Stromal scarring this occurs when descement’s
membrane has rupture
 Some research suggest that its due to imbalance of
enzymes in the cornea which leads to oxidative
damages leads to weakening and bulging of the
cornea.
 Overexposure to the ultraviolet light,
 Excessive rubbing of the eye ,
 Poor fitted contact lens
 Genetic predisposition.
CAUSES OF KERATOCONUS
Can glasses correct the keratoconus?
 Glasses can only correct the regular astigmatism but
as for keratoconus it is an irregular astigmatism.
 Contact lenses can be used to make the air –cornea
interface more spherical so as the light can focus to
the eye.
MANAGEMENT OPTIONS
 Contact lenses
 Collagens cross linking; it prevent the progression of
early form of the disease (riboflavin eye drops which
is then exposed to the sun light)
 Corneal graft
 Corneal ring segment implant
THANK YOU !!!!!!

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Cornea

  • 1. B Y T I N A THE CORNEA
  • 2. OUTLINES  INTRODUCTION  FACTS  LAYER OF THE CORNEA  FUNCTION
  • 3. INTRODUCTION  It’s the outer most layer of the eye, domed shaped, transparent tissue that covers the eyes.  It has about three quarters of the optical power of the eye  its is inserted into the sclera at the limbus, its about 540 micrometer thick centrally
  • 4.
  • 5. FACTS!!!  Did u know!!! the cornea is the mostly densely innervated tissues in the body!!  Supplied by the first branch of trigeminal nerve  It has no blood vessels although its alive, it receives all the nutrients from the aqueous humor (posteriorly) and tears (anteriorly).
  • 6. Cornea transparency is due to;  Uniform structure  A vascularity  Deturgescence (relative dehydration)
  • 7.  cornea has five layers ; i) The epithelium;  Is stratified squamous and non keratinized  It has nerve endings  acts as a barrier of the eye to the foreign material eg dust.  It absorb oxygen and nutrients form tears
  • 8. ii) Bowman’s membrane  Its formed by collagen fibres, when injured it forms scar as it heals, if the scar will form centrally the there will be visual loss.
  • 9. iii) The stroma  It contribute to about 90% of the corneal thickness.  Its mainly water and collagens, collagens are the ones which gives the cornea its structure and elasticity.  The stoma can scar but can not regenerate.
  • 10. iv) Descement’s membrane  It has a regenerative potentials
  • 11. v) The endothelium  It maintain corneal deturgescence by pumping excess fluid out of the stoma, failure of endothelium to do its work can lead to cornea edema  Its cells cannot regenerate.  There is loss of cells in this layer as the age increases
  • 13. FUNCTION OF THE CORNEA  It helps in focusing the light that come into the eye so as it can fall on the retina.  It’s a physical barrier together with tears and eyelid against the dirty and germs that come into contact with the eye.  It also protect the eye against the UV light from the sun.
  • 14.  The cornea can be affected by different diseases, keratoconus being among them
  • 15. KERATOCONUS  It’s a disease characterized by progressive thinning of the cornea and takes a cone shape which results to astigmatism (the cornea cannot focus the light to the retina).  Most of refraction of light occurs at the air –cornea interface so change in the cornea shape can lead to major impact on vision.
  • 16. Signs  Bulging of the lower eyelid on down gazing(munson sign)  Oil droplets reflex on direct ophthalmoscope  Scissors reflex on retinoscopy  Stromal scarring this occurs when descement’s membrane has rupture
  • 17.  Some research suggest that its due to imbalance of enzymes in the cornea which leads to oxidative damages leads to weakening and bulging of the cornea.  Overexposure to the ultraviolet light,  Excessive rubbing of the eye ,  Poor fitted contact lens  Genetic predisposition. CAUSES OF KERATOCONUS
  • 18. Can glasses correct the keratoconus?  Glasses can only correct the regular astigmatism but as for keratoconus it is an irregular astigmatism.  Contact lenses can be used to make the air –cornea interface more spherical so as the light can focus to the eye.
  • 19. MANAGEMENT OPTIONS  Contact lenses  Collagens cross linking; it prevent the progression of early form of the disease (riboflavin eye drops which is then exposed to the sun light)  Corneal graft  Corneal ring segment implant