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CONGESTIVE HEART FAILURE
By: Rhoda Layugan
CONGESTIVE HEART FAILURE
• Heart failure is the inability of the heart to
maintain adequate cardiac output to meet
the metabolic needs of the body because
of impaired pumping ability.
• Diminished cardiac output results in
inadequate peripheral tissue perfusion.
• Congestion of the lungs and periphery
may occur; the client can develop acute
pulmonary edema.
RESEARCH
CLASSIFICATION
• Acute heart failure occurs suddenly.
• Chronic heart failure develops over time;
however, a client with chronic heart failure
can develop an acute episode.
TYPES OF HEART FAILURE
Right ventricular failure, left ventricular failure
• Because the 2 ventricles of the heart represent 2 separate pumping systems, it is
possible for
• 1 to fail alone for a short period.
• Most heart failure begins with left ventricular failure and progresses to failure of
both
• ventricles.
• Acute pulmonary edema, a medical emergency, results from left ventricular
failure.
• If pulmonary edema is not treated, death will occur from suffocation because the
client literally drowns in his or her own fluids.
• Forward failure, backward failure
• In forward failure, an inadequate output of
• the affected ventricle causes decreased perfusion to vital organs.
• In backward failure, blood backs up behind the affected ventricle,
causing increased pressure in the atrium behind the affected
ventricle.
Low output, high output
•In low-output failure, not enough cardiac output is
available to meet the demands of the body.
•High-output failure occurs when a condition causes the
heart to work harder to meet the demands of the body.
Systolic failure, diastolic failure
•Systolic failure leads to problems with con-
•traction and ejection of blood.
•Diastolic failure leads to problems with the
•heart relaxing and filling with blood.
COMPENSATORY
MECHANISM
• Compensatory mechanisms act to restore
cardiac output to near-normal levels.
• Initially, these mechanisms increase
cardiac output; however, they eventually
have a damaging effect on pump action.
• Compensatory mechanisms contribute to
an increase in myocardial oxygen
consumption; when this occurs,
myocardial reserve is exhausted and
clinical manifestations of heart failure
develop.
• Compensatory mechanisms include
increased heart rate, improved stroke
volume, arterial vasoconstriction, sodium
and water retention, and myocardial
hypertrophy.
PULMONARY EDEMA
INTERVENTIONS
• Assist the client to identify precipitating risk factors of heart failure
and methods of eliminating these risk factors.
• Encourage the client to verbalize feelings about the lifestyle
changes required as a result of the heart failure.
• Instruct the client in the prescribed medication regimen, which may
include digoxin, a diuretic, ACE inhibitors, low-dose beta blockers,
and vasodilators.
• Advise the client to notify the HCP if side effects occur from the
medications.
• Advise the client to avoid over-the-counter medications.
• Instruct the client to contact the HCP if he or she is unable to take
medications because of illness.
• Instruct the client to avoid large amounts of caffeine, found in coffee,
tea, cocoa, chocolate, and some carbonated beverages.
• Instruct the client about the prescribed low-sodium, low-fat, and low-
cholesterol diet.
• Provide the client with a list of potassium-rich foods because
diuretics can cause hypokalemia (except for potassium-retaining
diuretics).
• Instruct the client regarding fluid restriction, if prescribed, advising
the client to spread the fluid out during the day and to suck on hard
candy to reduce thirst.
• Instruct the client to balance periods of activity
• and rest.
• Advise the client to avoid isometric activities,
• which increase pressure in the heart.
• Instruct the client to monitor daily weight.
• Instruct the client to report signs of fluid retention such as edema or
weight gain.
THANK YOU!
Reference:
• Med-Surg 2017
• NCLEX 2016
• Udan Med-Surg

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CHF (Congestive Heart Failure)

  • 2. CONGESTIVE HEART FAILURE • Heart failure is the inability of the heart to maintain adequate cardiac output to meet the metabolic needs of the body because of impaired pumping ability.
  • 3. • Diminished cardiac output results in inadequate peripheral tissue perfusion. • Congestion of the lungs and periphery may occur; the client can develop acute pulmonary edema.
  • 5. CLASSIFICATION • Acute heart failure occurs suddenly. • Chronic heart failure develops over time; however, a client with chronic heart failure can develop an acute episode.
  • 6. TYPES OF HEART FAILURE Right ventricular failure, left ventricular failure • Because the 2 ventricles of the heart represent 2 separate pumping systems, it is possible for • 1 to fail alone for a short period. • Most heart failure begins with left ventricular failure and progresses to failure of both • ventricles. • Acute pulmonary edema, a medical emergency, results from left ventricular failure. • If pulmonary edema is not treated, death will occur from suffocation because the client literally drowns in his or her own fluids.
  • 7. • Forward failure, backward failure • In forward failure, an inadequate output of • the affected ventricle causes decreased perfusion to vital organs. • In backward failure, blood backs up behind the affected ventricle, causing increased pressure in the atrium behind the affected ventricle.
  • 8. Low output, high output •In low-output failure, not enough cardiac output is available to meet the demands of the body. •High-output failure occurs when a condition causes the heart to work harder to meet the demands of the body.
  • 9. Systolic failure, diastolic failure •Systolic failure leads to problems with con- •traction and ejection of blood. •Diastolic failure leads to problems with the •heart relaxing and filling with blood.
  • 10. COMPENSATORY MECHANISM • Compensatory mechanisms act to restore cardiac output to near-normal levels. • Initially, these mechanisms increase cardiac output; however, they eventually have a damaging effect on pump action.
  • 11.
  • 12. • Compensatory mechanisms contribute to an increase in myocardial oxygen consumption; when this occurs, myocardial reserve is exhausted and clinical manifestations of heart failure develop.
  • 13. • Compensatory mechanisms include increased heart rate, improved stroke volume, arterial vasoconstriction, sodium and water retention, and myocardial hypertrophy.
  • 14.
  • 16.
  • 17.
  • 18. INTERVENTIONS • Assist the client to identify precipitating risk factors of heart failure and methods of eliminating these risk factors. • Encourage the client to verbalize feelings about the lifestyle changes required as a result of the heart failure. • Instruct the client in the prescribed medication regimen, which may include digoxin, a diuretic, ACE inhibitors, low-dose beta blockers, and vasodilators.
  • 19. • Advise the client to notify the HCP if side effects occur from the medications. • Advise the client to avoid over-the-counter medications. • Instruct the client to contact the HCP if he or she is unable to take medications because of illness. • Instruct the client to avoid large amounts of caffeine, found in coffee, tea, cocoa, chocolate, and some carbonated beverages.
  • 20. • Instruct the client about the prescribed low-sodium, low-fat, and low- cholesterol diet. • Provide the client with a list of potassium-rich foods because diuretics can cause hypokalemia (except for potassium-retaining diuretics). • Instruct the client regarding fluid restriction, if prescribed, advising the client to spread the fluid out during the day and to suck on hard candy to reduce thirst.
  • 21. • Instruct the client to balance periods of activity • and rest. • Advise the client to avoid isometric activities, • which increase pressure in the heart. • Instruct the client to monitor daily weight. • Instruct the client to report signs of fluid retention such as edema or weight gain.
  • 23. Reference: • Med-Surg 2017 • NCLEX 2016 • Udan Med-Surg