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PRESENTED BY
Mrs. SOUMYA SUBRAMANI, M.Sc.(N)
LECTURER, MSN DEPARTMENT
CON- SRIPMS, COIMBATORE.
Mrs. Soumya.M
1
Mrs. Soumya.M 2
Burns is defined as a traumatic injury to
the skin or other organic tissue primarily
caused by thermal or other acute
exposures. Burn occurs when some or all of
the cells in the skin or other tissues are
destroyed by heat, cold, electricity,
radiation or causative chemicals.
-WHO DEFINITION
Mrs. Soumya.M 3
ETIOLOGY OF BURNS
THERMAL CHEMICAL ELECTRICAL RADIATION INHALATION
Mrs. Soumya.M 4
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 ANATOMIC CHANGES
 PHYSIOLOGICAL CHANGE
 Protective function
 Excretory function
 Regulation of water and electrolyte imbalances
 Sensory function
 Synthesis function
 Temperature regulation
Mrs. Soumya.M 15
Macrophages damages within the tissues
Release of chemical mediators
Constriction of blood vessels
Blood vessels thrombosis
Necrosis
Mrs. Soumya.M 16
FLUID SHIFT
After initial vasoconstriction blood vessel near the burns dilate
Fluid leak into the interstitial space (THIRD SPACING OR CAPILLARY LEAK SYNDROME)
Decreases the blood volume and BP Extensive edema Electrolyte imbalances
Hypovolemia Vascular dehydration - hyperkalaemia
Hemoconcentration -hyponatremia
Increasing blood viscosity
Reduced small vessel blood flowMrs. Soumya.M 17
FLUID REMOBILIZATION
Mrs. Soumya.M 18
Initial fluid shift and hypovolemia causes decreased CO until 36
hours, increases with fluid resuscitation
Mrs. Soumya.M 19
Mrs. Soumya.M 20
GASTEROINTESTINAL CHANGES
 Decreased gastric perfusion Impaired
gastric mucosal integrity and motility
 Sympathetic nervous system stress response
increases secretion of epinephrine and
norepinephrine inhibit GI motility
Decreased peristalsis PARALYTIC
ILEUS &CURLING’S ULCER
Mrs. Soumya.M 21
RENAL ALTERATION
 Myoglobin and destructed RBCs are
excreted by kidneys
 Inadequate blood supply causes occlusion
of renal tubules resulting in acute tubular
necrosis and & RF
Mrs. Soumya.M 22
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FOURTH DEGREE BURN
Mrs. Soumya.M 31
RULE OF NINE
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WOUND CLEANSING
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Burns