A detailed description of benign paroxysmal positional vertigo (BPPV): the symptoms, causes, diagnosis, and treatment methods.For more information, please visit www.everydayhearing.com
Provides information concerning gravity, rotation and acceleration
Serves as a reference for the somatosensory & visual systems
Contributes to integration of arousal, conscious awareness of the body via connections with vestibular cortex, thalamus and reticular formation
Learn more about the types, symptoms and causes of balance disorders. Diagnostic and treatment options such as vestibular rehabilitation and cognitive behavioral therapy will be discussed.
A detailed description of benign paroxysmal positional vertigo (BPPV): the symptoms, causes, diagnosis, and treatment methods.For more information, please visit www.everydayhearing.com
Provides information concerning gravity, rotation and acceleration
Serves as a reference for the somatosensory & visual systems
Contributes to integration of arousal, conscious awareness of the body via connections with vestibular cortex, thalamus and reticular formation
Learn more about the types, symptoms and causes of balance disorders. Diagnostic and treatment options such as vestibular rehabilitation and cognitive behavioral therapy will be discussed.
As part of a class presentation, we attempted to make this to briefly explain what Torticollis meas, the Types of presentation of Torticollis, and Management strategies for a Physiotherapist for Congenital Torticollis especially.
I hope this helps. :)
The pictures and information had been taken from internet, complied to make a brief presentation for the purpose of class presentation.
I do not own any content.
As part of a class presentation, we attempted to make this to briefly explain what Torticollis meas, the Types of presentation of Torticollis, and Management strategies for a Physiotherapist for Congenital Torticollis especially.
I hope this helps. :)
The pictures and information had been taken from internet, complied to make a brief presentation for the purpose of class presentation.
I do not own any content.
Talk on Ophthalmic causes of head ache.
Dr Ashish Mahobia M.S.,F.R.F.,F.N.B.
Consultant Eye surgeon and Vitreo retinal specialist
Sai baba Eye hospital & retina centre.,Near chhoti line ,Fafadih , Raipur , Chattisgarh,India.PIN 492001 .Phone 0771 - 4037979 ,4025063,Mobile:+91-9329117979
In-service project for clinical affiliation with Hingham PT, Inc. (Januay 2014-April 2014)
Review of vestibular system, common diagnosis and how to examine, evaluate and treat.
I also reviewed and supplied the clinic with the Four Step Square Test and Dynamic Gait Index in order to allow them to implement these outcome assessments into their clinic for individuals with balance/vestibular deficits
Theoretical framework of infant physiotherapyAnwesh Pradhan
MPT class- Theoretical framework of infant physiotherapy. Require 3 class. Help us to decide the paediatric physiotherapy approach for paediatric patient.
Anatomy of Cranial Nerve for BPT class.
Require 10 classes. Require help of brain specimen during the class. Testing of the nerves can also be taken together.
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
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Acute scrotum is a general term referring to an emergency condition affecting the contents or the wall of the scrotum.
There are a number of conditions that present acutely, predominantly with pain and/or swelling
A careful and detailed history and examination, and in some cases, investigations allow differentiation between these diagnoses. A prompt diagnosis is essential as the patient may require urgent surgical intervention
Testicular torsion refers to twisting of the spermatic cord, causing ischaemia of the testicle.
Testicular torsion results from inadequate fixation of the testis to the tunica vaginalis producing ischemia from reduced arterial inflow and venous outflow obstruction.
The prevalence of testicular torsion in adult patients hospitalized with acute scrotal pain is approximately 25 to 50 percent
1. Anwesh Pradhan, MPT
(Neurological & Psychosomatic Disorders)
Assistant Professor, Nopany Institute of Healthcare Studies, Kolkata
Benign Paroxysmal Positional
Vertigo
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
2. Vertigo
Vertigo is the illusion of movement of the self or the
environment
Causes:
Due to mechanical problems of the inner ear (e.g.,
BPPV)
Due to lesions that cause loss of function of vestibular
pathways on one side (e.g., vestibular neuritis)
Sudden imbalance of tonic neural activity in the
vestibulocortical pathway (labyrinth–VIIIth nerve–
vestibular nucleus–vestibular thalamus–vestibular
cortex)
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
3. Benign Paroxysmal Positional
Vertigo (BPPV)
BPPV is the most common cause of vertigo
BPPV was first described by Barany (1921)
Problem of inner ear
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
4. Benign Paroxysmal Positional Vertigo
(BPPV)
Typically, a patient with BPPV will complain of brief
episodes of vertigo precipitated by rapid change of
head positions or postures.
Sometimes symptoms are brought about by assuming
very specific head positions like-
Head positions involve rapid extension of the neck,
often with the head turned to one side (as when
looking up to a high shelf or backing a car out of a
garage)
Lateral head tilts toward the affected ear.
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
5. Benign Paroxysmal Positional Vertigo
(BPPV)
The symptoms often appear when a patient rolls
from side to side in bed.
Patients can usually identify the offending head
position, which they often studiously avoid.
Many patients also complain of mild postural
instability between attacks.
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
6. Benign Paroxysmal Positional
Vertigo (BPPV)
The vertigo lasts only 30 seconds to 2 minutes (usually
less than 1 minute) and disappears even if the
precipitating position is maintained.
Hearing loss, aural fullness, and tinnitus are not seen in
this condition, which most commonly occurs
spontaneously in the elderly population but can be
seen in any age group after even mild head trauma.
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
7. Benign Paroxysmal Positional
Vertigo (BPPV)
Women are more commonly affected than men.
Bilateral involvement can be found in 10% of the
spontaneous cases and 20% of the traumatic cases.
Spontaneous remissions are common, but recurrences
can occur, and the condition may trouble the patient
intermittently for years.
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
8. Cupulolithiasis Theory
Described by Schuknecht (1969)- found basophilic
deposits on the cupula of the posterior canal of BPPV
patients
In cupulolithiasis, the debris (probably fragments of
otoconia- calcium carbonate crystals, from the utricle)
adhere to the cupula of the semicircular canals making the
ampulla gravity sensitive
With movement into the head hanging position, gravity
displaces the weighted cupula, resulting in an abnormal
signal from that canal - Vertigo
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
11. Canalithiasis Theory
Proposed by Hall, Ruby and McClure (1979)- debris is
not adherent to cupula
The debris (calcium carbonate crystals) will float freely in
the endolymph in the long arm of the Semicircular canals
When the head is moved into the head-hanging position,
the debris moves to the most dependent portion of the
canal
The movement of the debris causes the endolymph to
move, in turn overcoming the inertia of the cupula, and an
abnormal signal is sent to the central nervous system-
Vertigo
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
15. Dix-Hallpike test [anterior or posterior canal
benign paroxysmal positional vertigo]
1. The patient sits on the bed or examination table, and the head is
turned
2. 45 degrees horizontally toward the labyrinth to be tested
(position 1).
3. The examiner quickly brings the head and trunk straight back
“en bloc,” so that the head is hanging over the edge of the
examination table by 20 to 30 degrees (position 2).
4. The examiner asks whether the patient has vertigo and observes
for nystagmus.
5. The patient’s upper body is then brought up slowly to a sitting
position with the head still turned 45 degrees, and nystagmus is
sought again.
6. This test then is repeated with the head turned 45 degrees in the
other direction.
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
16. • The figure below also shows the right labyrinth with
free-floating otoconia in the right posterior
semicircular canals (large black arrows). During the
Hallpike- Dix test, this debris would move, resulting in
nystagmus andvertigo when the test is performed to
the right side but notwhen the test is performed to the
left side. (Modified fromTusa and Herdman, 1998.4)
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
17. Side-lying test [for anterior or posterior canal
benign paroxysmal positional vertigo]
1. The patient sits on the bed or examination table with the legs
over the side, and the head is rotated 45 degrees horizontally
away from the labyrinth to be tested (position 1).
2. The examiner then quickly brings the patient’s head and trunk
down on the side opposite to the direction the head is turned
(position 2).
3. The patient is asked to report any vertigo and is observed for
nystagmus.
4. The patient is then brought to a sitting position with the head still
turned 45 degrees, and the examiner rechecks for nystagmus and
vertigo.
5. The test is repeated with head turned 45 degrees horizontally to
the other side.
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
18. During the Side-lying test, the debris would move,
resulting in nystagmus and vertigo when the test is
performed to the affected side but not when it is is
performed to the unaffected side.
This test is also useful for anterior canal BPPV,
because debris in this canal would move when the test
is done on the affected side. (Modified from Tusa and
Herdman, 1998.4)
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
19. Roll test [for horizontal canal benign paroxysmal
positional vertigo]
1. The patient is laid supine with the head flexed 20 degrees.
2. The head is quickly rolled to one side, nystagmus is looked
for and the patient is asked to report any vertigo.
3. The head is then slowly rolled back to a supine position.
4. The head is then quickly rolled to the other side,
nystagmus is looked for, and the patient is asked to report
any vertigo. (Modified from Tusa and Herdman, 1998.4)
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
20. Test Series [vertigo when lying on the right
side]
The series is as follows:
1. Perform the Dix-Hallpike test on the left side.
2. Perform the Dix-Hallpike test on the right side.
3. If the patient has no vertigo: Before sitting the patient
up from the right side, perform a roll test by having the
patient turn the head quickly to the left.
4. After 30 seconds, have the patient quickly turn the
head back to the right.
5. After 30 seconds, have the patient sit up.
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
22. Treatment of BPPV
Canalithiasis: The Canalith Repositioning
Procedure- Modified by Epley
Cupulolithiasis: The Liberatory Maneuver-Modified
by Semonts
Bar-B-Que roll or Canalith repositioning treatment
for horizontal SCC BPPV
Brandt-Daroff Habituation Exercises
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
23. Procedure of Epley’s Canalith Repositioning
Maneuver (Posterior SSC)
1. Patient is made to sit in the treatment table comfortably.
2. Starting position is sitting on the treatment table with
patient’s head turned 45º to the affected side.
3. A pillow is placed behind the patient so that on lying
back it will be under patient’s shoulder.
4. The patient is made to lie back quickly with shoulders on
the pillow, neck extended, and head resting on the edge of
the treatment table. In this position, the affected ear is
underneath. This position is maintained for 3 min.
5. Then the patient’s head is turned 90º to the opposite side
(without raising it) and the body rolled such that the
shoulders are aligned perpendicularly to the floor
(affected ear up) and maintained again for 4 min.
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
24. 6. Patient’s body and head is turned another 90º towards the
opposite side i.e. the nose points 30º below the plane of the
treatment table and maintained for another 4 mins.
7. Then sit up on the treatment table upright quickly.
8. Then the patient is asked to maintain this position of head
for 48 hours and avoid provoking position for one week.
Idea is based on that the debris is free-floating in the
posterior canal, and the position changes are designed
to move the debris out of the posterior canal and into
common crus
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
25. Patient is treated with Epley’s canalith repositioning maneuver.
Diagrammatic representation of the movement of the loose otoconia inside the
posterior semicircular canal during Epley’s canalith repositioning maneuver.
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
26. Canalith Repositioning Treatment
for Anterior SCC BPPV
Anterior SCC BPPV is treated the same way as
posterior SCC BPPV. The difficulty lies in deciding
which side to treat. The best way to decide is based on
the direction of the nystagmus rather than on the side
of the dependent labyrinth (ear) during the Dix-
Hallpike test.
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
27. Procedure of Semont’s Liberatory
Maneuver (Posterior SCC)
1. Patient is made to sit in the treatment table
comfortably.
2. Therapist will hold the head of the patient with both
hands.
3. Then the patient is swinged to the affected side
quickly with the head hanging down. If nystagmus
comes or patient speak about the vertigo then
maintain the position for 2-3 mins, if no nystagmus
comes or the patient does not speak about the vertigo
then turn the head in 45º face up, nystagmus or
vertigo will come now and then maintain the same
position for 2-3 mins
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
28. 4. Therapist has to hold the patient’s head and neck with
both hands and swing the patient quickly to the
opposite side. The speed of the head must be 0 at the
moment it touches the treatment table. These position
is maintained for 5 mins. Then quickly the patient is
brought to sitting position. And then the patient is
asked to maintain this upright position of head for 48
hours and avoid provoking position for 1 week.
The approach works by floating the debris through
the canal system to the common crus
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
29. Patient is treated with Semont’s liberatory maneuver
Diagrammatic representation of various position of the patient in
Semont’s liberatory maneuver.NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
30. Liberatory Maneuver (Semont or
Brisk Treatment) for Anterior SCC BPPV
For anterior SCC BPPV, the procedure must be modified in
order to move the head in the plane of the anterior SCC.
For sake of discussion, assume that the patient has debris in
the right anterior SCC. The procedure is as follows:
1. The patient sits on the examination table sideways, but the
head is rotated 45 degrees toward the right side.
2. The patient is then moved rapidly onto the right side
(parallel to the plane of the affected anterior SCC).
3. After 1 minute, the patient is rapidly moved through the
initial sitting position to the left side with the head still
positioned 45 degrees toward the right side (nose is now
angled 45 degrees up toward the ceiling).
4. The patient holds this position for 1 minute and then moves
slowly to a sitting position.
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
31. Bar-B-Que roll or Canalith repositioning
treatment for horizontal SCC BPPV
To dislodge otoconia attached to the cupula of the
horizontal SCC (cupulolithiasis) and treatment of
horizontal SCC canalithiasis
1. The patient’s head slowly rolls away from the affected ear
until the face is pointed up; this position is held for about
15 seconds, or until the dizziness stops.
2. The patient continues to roll the head in the same direction
until the affected ear is up; this position is held for about 15
seconds, or until the dizziness stops.
3. The patient rolls the head and body in the same direction
until the face is down.
4. After 15 seconds, the patient slowly sits up, keeping the
head level or pitched down 30 degrees.
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
32. These two variations of the CRT for horizontal SCC
BPPV are referred to as the 270-degree roll and the
360- degree roll, respectively
Patients can be taught to perform this treatment at
home. Patients repeat this treatment once each morning
until they experience no symptoms during the
treatment
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
33. Brandt-Daroff treatment [for treatment of
posterior semicircular canal benign paroxysmal positional vertigo]
The patient is moved quickly into the side-lying
position on the affected side and stays in that position
until 30 seconds after the vertigo has stopped.
The patient then sits up and again waits for the vertigo
to stop. The patient then repeats the movement to the
opposite side, stays there for 30 seconds after vertigo
stops, and sits up.
The entire treatment is repeated 10 to 20 times, three
times a day, until the patient has no vertigo for 2 days
in a row. AC anterior canal; PC posterior canal. Black
arrows indicate position and movement of debris.
(Modified from Tusa and Herdman, 1998.4)
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
35. Brandt-Daroff Habituation Exercises
for Anterior SCC BPPV
Brandt-Daroff exercises can be used to treat anterior
SCC BPPV, because the head is moved in the plane of
the posterior SCC on one side and of the anterior SCC
on the other side.
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
36. Other Habituation exercises
Tangeman & Wheeler: 3 Phases
Phase I: similar to Brandt & Daroff protocal and
consist of having the patient move repeatedly to
Hallpike-Dix position
Phase II & III: Wide varity of balance exercises that
that incorporate eye and head movt. – similar to
Sawthorne-Cooksey exercise for vestibular
hypofunction.
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
38. Sources
Shepard NT, Solomon D. Functional Operation of the Balance System
in Daily Activities. Otolaryngologic Clinics of North America
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Minor LB. Physiological principles of vestibular function on earth and
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2):S5-S15.
Abdel Razek OA. Anatomy of the Vestibular System.
www.emedicine.com
Hoffman R, Strunk C. Vestibular Anatomy and Physiology.
Department of Otolaryngology Grand Rounds University of Texas
Medical Branch December 9, 1992.
Baloh RW. Dizziness, Hearing Loss, and Tinnitus. Philadephia, F.A.
Davis Company, 1998.
Jahn AF, Santos-Sacchi J. Physiology of the Ear. Second edition. San
Diego, Singular, 2001.
Friedman I, Ballantyne J. Ultrastructural Atlas of the Inner Ear.
London, Butterworth & Co., 1984.
NIHS, Kol:Mobility P&R,MDN:IAP-WB,2015
39. Wall C, Vrabec JT. Vestibular Function and Anatomy. In: Head & Neck Surgery
Otolaryngolog. Philadelphia, Lippincott Williams & Wilkins, 2001:1641-1650.
Herdman SJ, Wolf SL, Vestibular Rehabilitation. Philadephia, F.A. Davis
Company,2007
Barany, R. (1921). Diagnose von Krankeiterschernumgenin Berieche des
Otolithenappataten. Acta Otolaryngol; 2:434-437.
Hecker HC, Haug CO, Herndon J: Treatment of the vertiginous patient with
Cawthornes vestibular exercises. Laryngoscope 84 (11):2065-2072, 1974.
Brandt T, Daroff RB. Physical therapy for benign paroxysmal positional vertigo.
Arch Otolaryngol. 1980; 106: 484–485.
Tangeman PT, Wheeler J: Inner ear concussion syndrome vestibular
implications and PT treatment. Topics in Acute Care and Trauma Rehab 1
(1):72-83, 1986.
Semont A, Freyss G, Vitte E. Curing the BPLS with a liberatory maneuver. Adv
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