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ATELECTASIS
(COLLAPSE)
RESPIRATORY SYSTEM
ATELECTASIS
• Defined as the
– “Collapse of pulmonary parenchyma”
– Loss of lung volume
• Caused by inadequate expansion of airspaces.
• Results in shunting of inadequately oxygenated
blood from pulmonary arteries into veins
• Giving rise to a ventilation-perfusion imbalance
and hypoxia.
ATELECTASIS
• Could be due to
– Incomplete expansion of a lung or part of a lung
in the newborn
• Primary atelectasis OR Neonatal atelectasis
– The collapse of previously inflated lung
• Secondary atelectasis OR Acquired
atelectasis
ATELECTASIS
Secondary atelectasis / Acquired atelectasis
According to the underlying mechanism
Three forms
1. Resorption Atelectasis
2. Compression Atelectasis
3. Contraction Atelectasis
ATELECTASIS
• Resorption Atelectasis
RESORPTION ATELECTASIS
• Due to obstruction that prevents air from
reaching distal airways.
• Air already present gradually absorbed-
alveolar collapse follows.
• Depending on the level of obstruction,
–an entire lung,
–a complete lobe,
–or one or more segments may be involved.
RESORPTION ATELECTASIS
• Common cause; obstruction of a
bronchus by a mucous or mucopurulent
plug in;
–Postoperative states
–Bronchial asthma
–Bronchiectasis
–Chronic bronchitis
–Or the aspiration of foreign bodies,
particularly in children.
COMPRESSION ATELECTASIS
COMPRESSION ATELECTASIS; Also c/a
passive or relaxation atelectasis
Pleural effusions:
–congestive heart failure (CHF), neoplastic
effusions, tuberculosis
• Basal atelectasis; resulting from the
elevated position of the diaphragm in;
• bedridden patients, patients with ascites,
and patients during and after surgery.
CONTRACTION ATELECTASIS
CONTRACTION ATELECTASIS
–Local or generalized fibrotic
changes in the lung or pleura may
prevent full expansion
ATELECTASIS
• Atelectasis (except that caused by
contraction)
–is potentially reversible due to collapse
lung parenchyma can be re-expanded
–and should be treated promptly to prevent
hypoxemia and superimposed infection of
the collapsed lung.
Pulmonary Edema
Left Heart Failure and
Pulmonary Edema
• LVF occurs when the left ventricle fails to function as
an effective forward pump, causing a back-pressure
of blood into the pulmonary circulation
• May be caused by a variety of forms of heart disease
including ischemic, valvular, and hypertensive heart
disease
• Untreated, significant LVF culminates in pulmonary
edema
Left Heart Failure and
Pulmonary Edema
• Signs and symptoms
– Severe respiratory distress
– Severe apprehension, agitation, confusion
– Cyanosis (if severe)
– Diaphoresis
– Adventitious lung sounds
– JVD
– Abnormal vital signs
Left Heart Failure and
Pulmonary Edema
• Signs and symptoms
– Severe respiratory distress
– Severe apprehension, agitation, confusion
– Cyanosis (if severe)
– Diaphoresis
– Adventitious lung sounds
– JVD
– Abnormal vital signs
Acute Pulmonary Edema
• May be CARDIAC or NON-CARDIAC in origin.
• Results from conditions such as:
– Increased pulmonary capillary pressure
– Increased pulmonary capillary permeability
– Decreased oncotic pressure
– Lymphatic insufficiency
– mixed or unknown mechanisms

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Atelectasis (collapse)

  • 2. ATELECTASIS • Defined as the – “Collapse of pulmonary parenchyma” – Loss of lung volume • Caused by inadequate expansion of airspaces. • Results in shunting of inadequately oxygenated blood from pulmonary arteries into veins • Giving rise to a ventilation-perfusion imbalance and hypoxia.
  • 3. ATELECTASIS • Could be due to – Incomplete expansion of a lung or part of a lung in the newborn • Primary atelectasis OR Neonatal atelectasis – The collapse of previously inflated lung • Secondary atelectasis OR Acquired atelectasis
  • 4. ATELECTASIS Secondary atelectasis / Acquired atelectasis According to the underlying mechanism Three forms 1. Resorption Atelectasis 2. Compression Atelectasis 3. Contraction Atelectasis
  • 6. RESORPTION ATELECTASIS • Due to obstruction that prevents air from reaching distal airways. • Air already present gradually absorbed- alveolar collapse follows. • Depending on the level of obstruction, –an entire lung, –a complete lobe, –or one or more segments may be involved.
  • 7. RESORPTION ATELECTASIS • Common cause; obstruction of a bronchus by a mucous or mucopurulent plug in; –Postoperative states –Bronchial asthma –Bronchiectasis –Chronic bronchitis –Or the aspiration of foreign bodies, particularly in children.
  • 9. COMPRESSION ATELECTASIS; Also c/a passive or relaxation atelectasis Pleural effusions: –congestive heart failure (CHF), neoplastic effusions, tuberculosis • Basal atelectasis; resulting from the elevated position of the diaphragm in; • bedridden patients, patients with ascites, and patients during and after surgery.
  • 11. CONTRACTION ATELECTASIS –Local or generalized fibrotic changes in the lung or pleura may prevent full expansion
  • 12. ATELECTASIS • Atelectasis (except that caused by contraction) –is potentially reversible due to collapse lung parenchyma can be re-expanded –and should be treated promptly to prevent hypoxemia and superimposed infection of the collapsed lung.
  • 14. Left Heart Failure and Pulmonary Edema • LVF occurs when the left ventricle fails to function as an effective forward pump, causing a back-pressure of blood into the pulmonary circulation • May be caused by a variety of forms of heart disease including ischemic, valvular, and hypertensive heart disease • Untreated, significant LVF culminates in pulmonary edema
  • 15. Left Heart Failure and Pulmonary Edema • Signs and symptoms – Severe respiratory distress – Severe apprehension, agitation, confusion – Cyanosis (if severe) – Diaphoresis – Adventitious lung sounds – JVD – Abnormal vital signs
  • 16. Left Heart Failure and Pulmonary Edema • Signs and symptoms – Severe respiratory distress – Severe apprehension, agitation, confusion – Cyanosis (if severe) – Diaphoresis – Adventitious lung sounds – JVD – Abnormal vital signs
  • 17. Acute Pulmonary Edema • May be CARDIAC or NON-CARDIAC in origin. • Results from conditions such as: – Increased pulmonary capillary pressure – Increased pulmonary capillary permeability – Decreased oncotic pressure – Lymphatic insufficiency – mixed or unknown mechanisms