The document summarizes the anatomy and embryology of the ear and related structures. It is divided into four main parts: outer, middle and inner ear, as well as pharynx. The outer ear collects and directs sound waves. The middle ear contains ossicles that amplify vibrations through the tympanic membrane. The inner ear converts these vibrations into neural signals via hair cells in the cochlea. The pharynx is divided into naso, oro, and hypopharynx and contains structures like the tonsils, epiglottis and vocal cords. Embryologically, the structures develop from pharyngeal arches which contribute muscles, nerves and skeletal components to the head
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Ear is composed of three parts: External ear, middle ear, and the Inner ear.
Hearing tests (Rinne's and Weber's tests).
Most important hearing and ear diseases are included.
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Human ear, organ of hearing and equilibrium that detects and analyzes sound by transduction (or the conversion of sound waves into electrochemical impulses) and maintains the sense of balance (equilibrium).
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As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
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These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
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i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
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STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
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anatomy and physiology of the ear.ppt
1. Main Components of the Hearing
Mechanism:
Divided into 4 parts (by function):
• Outer Ear
• Middle Ear
• Inner Ear
• Central Auditory Nervous System
2. Major Divisions of the Ear
Peripheral Mechanism Central Mechanism
Outer
Ear
Middle
Ear
Inner
Ear
VIII
Cranial
Nerve
Brain
3. Embryology.
• The most typical feature in development of
the head and neck is formed by the
pharyngeal or branchial arches.
• These arches appear in the fourth and fifth
weeks of development and contribute to the
characteristic external appearance of the
embryo
4. Embryology of the pharyngeal arches
- From the arches are derived muscle and
nerves of the head and neck which are
involved in speech.
• The first 8 weeks constitutes the period of
greatest embryonic development of the head
and neck. There are 5 arches named
pharyngeal or branchial arches.
5. • Between these arches are the grooves or
clefts externally and the pouches internally.
• The derivatives of arches are usually of
mesoderm origin. The original mesoderm of
the arches gives rise to the musculature of the
face and neck.
• The groove is lined by outside surface
ectoderm while the pouch is lined by inside
endoderm.
6.
7. • Each arch has an artery, nerve, and cartilage bar.
• The core of each arch receives substantial
numbers of neural crest cells, which migrate into
the arches to contribute to skeletal components
of the face.
• The muscular components of each arch have
their own cranial nerve, and wherever the
muscle cells migrate, they carry their nerve
component with them
8.
9. C L I N I C A L C O R R E L A T E S
Deafness and External Ear Abnormalities
Congenital deafness, may be caused by abnormal development of the
membranous and bony labyrinths or by malformations of the auditory
ossicles and eardrum. In the most extreme cases the tympanic cavity and
external meatus are absent.
Most forms of congenital deafness are caused by genetic factors, but
environmental Factors may also interfere with normal development of the
internal and middle ear. Rubella virus, affecting the embryo in the seventh
or 8th wk, may cause severe damage to the organ of Corti. It has also been
suggested that poliomyelitis, diabetes, hypothyroidism, and toxoplasmosis
can cause congenital deafness.
10. • External ear defects are common; they
include minor and severe abnormalities often
associated with other malformations.
• Congenital microtia occurs about 1:20.000
births.
11. • The auricle is formed early. Therefore,
malformation of the auricle implies
malformation of the middle ear, On the other
hand, a normal auricle with canal atresia
indicates development in the 28th week, by
which time ossicles and middle ear are
already formed.
• Improper fusion of the first and second
branchial arches results in a preauricular.
12. Malformation of first branchial arch and
groove results in:
a. Auricle abnormality (first and second arches)
b. Bony meatus atresia (first groove)
c. Abnormal incus and malleus (first and second
arches)
Anotia and microtia often combined with EAC
stenosis.
Rubella embrypopathy- middle ear dysplasia.
14. Structures of the Outer Ear
1. Auricle (Pinna)
Functions
- Collects sound
- Helps in sound
localization
-Directing sounds to
the
eardrum.
- Cosmesis
15. 2. External Auditory Canal
• Approx. 26 millimeters (mm) in length and 7
mm in diameter in adult ear. “S” shaped
• Size and shape vary among individuals.
• Outer 1/3 - cartilage; inner 2/3 - mastoid
bone
• Cerumenous glands moisten/soften skin
• Presence of some cerumen is normal
16. Functions
• Warms air before it reaches the TM.
• Protects TM from physical damage.
• Resonator so as to amplify sound.
17. Cerumen
• The purpose of wax:
– Repel water
– Trap dust, sand particles, micro-organisms, and
other debris
– Moisturize epithelium in ear canal
– Odor discourages insects
– Antibiotic, antibacterial, antifungal properties
– Cleanse ear canal
18. Outer Ear Hearing Disorders
Outer ear
CHARGE syndrome
Down Syndrome
◦ Ears small and low set
Fetal Alcohol
Syndrome
◦ Deformed ears
DiGeorge syndrome
◦ Low set ears
21. Function of Middle Ear
Conduction
◦ Conduct sound from the outer ear to the inner ear
Protection
◦ Creates a barrier that protects the middle and inner
areas from foreign objects
◦ Middle ear muscles may provide protection from
loud sounds
Transducer
◦ Converts acoustic energy to mechanical energy
◦ Converts mechanical energy to hydraulic energy
Amplifier
◦ Transformer action of the middle ear
22. Tympanic Membrane
• The eardrum separates the outer ear from the
middle ear
• Creates a barrier that protects the middle and
inner areas from foreign objects
• Cone-shaped in appearance
– about 17.5 mm in diameter
• The eardrum vibrates in response to sound
pressure waves.
• The membrane movement is incredibly small
– as little as one-billionth of a centimeter
24. Eustachian Tube
The eustachian tube connects the front wall of
the middle ear with the nasopharynx .
The eustachian tube also operates like a valve,
which opens during swallowing and yawning
◦ This equalizes the pressure on either side of the
eardrum, which is necessary for optimal hearing.
◦ Without this function, a difference between the
static pressure in the middle ear and the outside
pressure may develop, causing the eardrum to
displace inward or outward
This reduces the efficiency of the middle ear and less acoustic energy
will be transmitted to the inner ear.
25. Mastoid Process of Temporal Bone
Bony ridge behind the auricle
Hardest bone in body, protects cochlea and
vestibular system
Provides support to the external ear and
posterior wall of the middle ear cavity
Contains air cavities which can be reservoir for
infection
29. Function of Inner Ear
• Convert mechanical
sound waves to neural
impulses that can be
recognized by the brain
for Hearing
• Balance
– Linear motion (vestibule)
– Rotary motion (canals)
31. • 8th Cranial Nerve or “Auditory Nerve” carries
signals from cochlea to brain
• Fibers of the auditory nerve are present in the
hair cells of the inner ear
• Auditory Cortex: Temporal lobe of the brain
where sound is perceived and analyzed.
33. Nonorganic Hearing Loss
• Sometimes referred to as functional, feigning,
etc.
• No physical evidence of hearing loss
• Conscious and unconscious
• Adults: medical/legal reasons
• Children: attention, psychological, reward,
etc.
34. Summary of hearing physiology.
Acoustic energy, in the form of sound waves, is
channeled into the ear canal by the pinna. Sound waves
hit the tympanic membrane and cause it to vibrate, like
a drum, changing it into mechanical energy. The
malleus, which is attached to the tympanic membrane,
starts the ossicles into motion. The stapes moves in and
out of the oval window of the cochlea creating a fluid
motion, or hydraulic energy. The fluid movement
causes membranes in the Organ of Corti to shear
against the hair cells. This creates an electrical signal
which is sent up the Auditory Nerve to the brain. The
brain interprets it as sound!
39. Common problems.
• Wax impaction- soften then syringe 3-5 days.
• O. externa- topical antibiotic, antifungal,
steriod.
• O. Media- Rx URTI
• O. M with perforation, chronic- topical
opthalmic topical antibiotics.
For all water precaution.
40. Requires referal
• >7-10days.
• Persistence of symptoms/signs or progressing
• TM perforation.
• Mastoiditis
• Unilateral H.Loss, tinnitus
41. Nose
• Divided into external nose and nasal cavity
• External nose
– made up of bone above
– nasal bones, frontal processes of maxilla, and
nasal process of frontal bone
– cartilage below
– upper and lower lateral cartilages, septal cartilage
42.
43. Nasal cavity
• Extends from nares anteriorly (singular: naris) to
choanae posteriorly
• Medial wall - nasal septum (cartilage, perpendicular
plate ethmoid, vomer)
• Lateral wall - three turbinates (superior, middle,
inferior)
• Roof - cribriform plate - leads to anterior cranial fossa
• Floor - hard palate
44.
45. Lateral wall nose
• Middle meatus lies under middle turbinate -
opening called hiatus semilunaris into which
the maxillary, frontal, and ethmoid sinuses
drain
• Inferior meatus lies under inferior turbinate -
receives the opening of the nasolacrimal duct
46.
47. Roof nose
• The mucosa in the superior part of the nose
contains nerve endings from the olfactory
nerve which come through the cribriform
plate (called olfactory mucosa)
• Function of the nose:
– warm, humidify air
– filter out particulate matter from air
– mucociliary blanket
50. Maxillary sinuses
• Located in body of maxilla
• Roof is floor of orbit, medial wall is lateral wall
of nose, floor is hard palate (teeth can erupt
into sinus)
• Opens into middle meatus through hiatus
semilunaris
51.
52. Frontal sinus
• Contained in frontal bone, anterior to anterior
cranial foss (infections can thus spread into
brain and cause meningitis or abscess)
• Bony septum divides two sides
• Opens into middle meatus via frontonasal
duct
53.
54. Sphenoid sinus
• Lies within body of sphenoid bone
• Opens into sphenoethmoidal recess above
superior turbinate
• Septum separates into two sides
• Lateral wall contains cavernous sinus with
internal carotid artery and nerves
• Optic nerve runs along lateral roof
55.
56. Ethmoid sinuses
• Contained within ethmoid bone, between
nose and orbit
• Separated from orbit by thin layer of bone
(lamina papyracea, allows spread of sinus
infection into orbit)
• Drain mostly into middle meatus
• A series of small cells
57.
58.
59. Pharynx
• Divided into nasopharynx, oropharynx, and
hypopharynx
• Nasopharynx -
– behind nose
– Eustachian tube orifices
– adenoids
60.
61. Pharynx
• Oropharynx
– soft palate to upper border epiglottis
– base of tongue (with lingual tonsils)
– palatine tonsils
– median/lateral glossoepiglottic folds
– vallecula is area just lateral to median GEF
– two folds of mucus membrane near tonsil (anterior and
posterior tonsillar pillar aka palatoglossal and
palatopharyngeal arch)
62.
63. Hypopharynx
• Behind and lateral to larynx
• lower border is cricoid cartilage (opening into
esophagus)
• Epiglottis is anterior fold of mucosa and
cartilage, flops down over larynx to prevent
aspiration during swallowing
• Aryepiglottic folds, pharygoepiglottic folds
• Piriform sinuses are lateral to larynx
64.
65. Vocal cords
• True vocal cords - nonkeratinizing squamous
epithelium, over muscles that can move and
tense cord
• False vocal cords - aka vestibular folds - above
true vocal cords
• Ventricle is between the two
• Vocal cords attached to thyroid cartilage
anterior, arytenoid cartilage posterior