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PREPARED BY:
JISA SHEHZAAN ALTHAF
I.
DISORDERS OF SKIN
TYPES OF ECZEMA
Atopic Dermatitis
Genetically influenced, chronic, relapsing disease
associated with immunologic irregularity involving
inflammatory mediators, exaggerated by a cutaneous
response to environmental allergens; associated with
allergic rhinitis and asthma; most severe in childhood.
Multiple presentations include :-
1) Acute-bright erythema, oozing vesicles, with extreme pruritus
2) Subacute-scaly, light red to red-brown plaques
3) Chronic stage-thickened skin with accentuation of skin markings
(lichenification), possible hyperpigmentation; dry skin; common
in antecubital and popliteal space in adults;
• Lubrication of dry (xerotic) skin
• Topical immunomodulators (pimecrolimus [Elidel]; Tacrolimus
[Protopic])
• Topical corticosteroids, systemic corticosteroids if severe,
• Phototherapy for relapses
• Reduction of stress reduces flares
• Antibiotics for secondary infection as needed
Manifestation of delayed hypersensitivity
Absorbed agent acting as antigen
Sensitization after several exposures
Appearance of lesions 2-7 days after contact with allergen
Red papules and plaques
Sharply circumscribed with occasional vesicles
Usually pruritic
Area of dermatitis frequently takes shape of causative
agent (e.g., metal allergy and band-like dermatitis on
ring finger)
• Topical corticosteroids, antihistamines
• Skin lubrication
• Elimination of contact allergen
• Avoidance of irritating affected area
• Systemic corticosteroids if sensitivity severe
2) SKIN DISORDERS.pptx
2) SKIN DISORDERS.pptx
2) SKIN DISORDERS.pptx
2) SKIN DISORDERS.pptx
2) SKIN DISORDERS.pptx
2) SKIN DISORDERS.pptx
2) SKIN DISORDERS.pptx

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2) SKIN DISORDERS.pptx

  • 3.
  • 4.
  • 5.
  • 6.
  • 7.
  • 8. TYPES OF ECZEMA Atopic Dermatitis Genetically influenced, chronic, relapsing disease associated with immunologic irregularity involving inflammatory mediators, exaggerated by a cutaneous response to environmental allergens; associated with allergic rhinitis and asthma; most severe in childhood.
  • 9.
  • 10. Multiple presentations include :- 1) Acute-bright erythema, oozing vesicles, with extreme pruritus 2) Subacute-scaly, light red to red-brown plaques 3) Chronic stage-thickened skin with accentuation of skin markings (lichenification), possible hyperpigmentation; dry skin; common in antecubital and popliteal space in adults;
  • 11. • Lubrication of dry (xerotic) skin • Topical immunomodulators (pimecrolimus [Elidel]; Tacrolimus [Protopic]) • Topical corticosteroids, systemic corticosteroids if severe, • Phototherapy for relapses • Reduction of stress reduces flares • Antibiotics for secondary infection as needed
  • 12. Manifestation of delayed hypersensitivity Absorbed agent acting as antigen Sensitization after several exposures Appearance of lesions 2-7 days after contact with allergen
  • 13.
  • 14. Red papules and plaques Sharply circumscribed with occasional vesicles Usually pruritic Area of dermatitis frequently takes shape of causative agent (e.g., metal allergy and band-like dermatitis on ring finger)
  • 15. • Topical corticosteroids, antihistamines • Skin lubrication • Elimination of contact allergen • Avoidance of irritating affected area • Systemic corticosteroids if sensitivity severe