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11Hyperintense Basal Ganglia
on T1-Weighted Magnetic
Resonance Imaging
CLINICAL IMAGAGING
AN ATLAS OF DIFFERENTIAL DAIGNOSIS
EISENBERG
DR. Muhammad Bin Zulfiqar
PGR-FCPS III SIMS/SHL
• Fig SK 11-1 Intracranial hemorrhage. (A)
Increased signal in the left basal ganglion. (B)
Unenhanced CT shows slightly increased
attenuation in the same region.23
• Fig SK 11-2 Petechial hemorrhagic infarct.
Increased signal in the right basal ganglion
(straight arrows) and temporal area (curved
arrows).23
• Fig SK 11-3 Parenteral nutrition. Increased signal intensity in the globus pallidus
(arrows).23
• Fig SK 11-4 Calcification (hypoparathyroidism). (A)
Unenhanced CT scan shows extensive calcification in
the basal ganglia (arrows) and subcortical white matter
(arrowheads) bilaterally. (B) T1-weighted MR image
shows increased signal intensity in the basal ganglia.23
• Fig SK 11-5 Neurofibromatosis. (A) T1-weighted image
shows hyperintensity involving the globus pallidus and
internal capsule (arrowheads) bilaterally. (B) On the
corresponding T2-weighted image, there are small
nodular foci of increased intensity in the globus
pallidus and internal capsule bilaterally, but the extent
and morphology of the signal abnormality differ from
those on the T1-weighted image.23
• Fig SK 11-6 Hypoxic ischemic encephalopathy.
Hyperintense foci in the basal ganglia bilaterally
and laminar hyperintensity along the cerebral
cortex (arrows) that is more prominent in the
parieto-occipital region.23
• Fig SK 11-7 Chronic hepatic failure. Increased
signal intensity in the globus pallidus
(arrows).23
• Fig SK 11-8 Diabetic hyperglycemia. Increased
signal intensity in the left putamen (arrow). The
patient had an abrupt onset of involuntary
movements involving the right limbs.23
• Fig SK 11-9 Japanese encephalitis. (A) Axial T1-weighted image
shows increased signal intensity suggestive of subacute
hemorrhage in the thalamus (solid arrows) and corpus striatum
(open arrows) bilaterally. (B) On a coronal T1-weighted image, there
is increased signal intensity in the corpus striatum (open arrows),
thalamus (short arrows), and cerebral peduncle (long arrows)
bilaterally, as well as in the right hippocampus (arrowhead).23
11 hyperintense basal ganglia on t1 weighted magnetic resonance
11 hyperintense basal ganglia on t1 weighted magnetic resonance

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11 hyperintense basal ganglia on t1 weighted magnetic resonance

  • 1. 11Hyperintense Basal Ganglia on T1-Weighted Magnetic Resonance Imaging
  • 2. CLINICAL IMAGAGING AN ATLAS OF DIFFERENTIAL DAIGNOSIS EISENBERG DR. Muhammad Bin Zulfiqar PGR-FCPS III SIMS/SHL
  • 3. • Fig SK 11-1 Intracranial hemorrhage. (A) Increased signal in the left basal ganglion. (B) Unenhanced CT shows slightly increased attenuation in the same region.23
  • 4. • Fig SK 11-2 Petechial hemorrhagic infarct. Increased signal in the right basal ganglion (straight arrows) and temporal area (curved arrows).23
  • 5. • Fig SK 11-3 Parenteral nutrition. Increased signal intensity in the globus pallidus (arrows).23
  • 6. • Fig SK 11-4 Calcification (hypoparathyroidism). (A) Unenhanced CT scan shows extensive calcification in the basal ganglia (arrows) and subcortical white matter (arrowheads) bilaterally. (B) T1-weighted MR image shows increased signal intensity in the basal ganglia.23
  • 7. • Fig SK 11-5 Neurofibromatosis. (A) T1-weighted image shows hyperintensity involving the globus pallidus and internal capsule (arrowheads) bilaterally. (B) On the corresponding T2-weighted image, there are small nodular foci of increased intensity in the globus pallidus and internal capsule bilaterally, but the extent and morphology of the signal abnormality differ from those on the T1-weighted image.23
  • 8. • Fig SK 11-6 Hypoxic ischemic encephalopathy. Hyperintense foci in the basal ganglia bilaterally and laminar hyperintensity along the cerebral cortex (arrows) that is more prominent in the parieto-occipital region.23
  • 9. • Fig SK 11-7 Chronic hepatic failure. Increased signal intensity in the globus pallidus (arrows).23
  • 10. • Fig SK 11-8 Diabetic hyperglycemia. Increased signal intensity in the left putamen (arrow). The patient had an abrupt onset of involuntary movements involving the right limbs.23
  • 11. • Fig SK 11-9 Japanese encephalitis. (A) Axial T1-weighted image shows increased signal intensity suggestive of subacute hemorrhage in the thalamus (solid arrows) and corpus striatum (open arrows) bilaterally. (B) On a coronal T1-weighted image, there is increased signal intensity in the corpus striatum (open arrows), thalamus (short arrows), and cerebral peduncle (long arrows) bilaterally, as well as in the right hippocampus (arrowhead).23