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104676280 case-study-brain-tumor-final
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Guagua National Colleges
Sta. Filomena, Guagua, Pampanga
College of Nursing
Case Study of Brain
Tumor
Prepared By: 2nd
Year
Miranda, Justin Kier D.
2. Cruz, Anzelyn B.
David, Stephanie G.
Miranda, Jazmin Gail D.
Prepared To:
Mrs. Ma. Jaemee Anne B. Lopez
I. Introduction
“Human existence is always associated with complexities. Man itself is a structured
compound. It is with system and subsystems that connect its function to enable to
breath, to move and to think.”
- Tolstoy
The main switch in man’s anatomical and physiological function is his brain. The brain
consists of a huge network of neurons that control the body’s vital functions. So far, this
system is vulnerable, and its optimal function depends on several key factors. Therefore
any alteration to this system and function greatly affects the body as a whole.
The Brain Tumor is a mass of cells that have grown and multiplied uncontrollably. There
are two types of brain tumor the benign tumor and malignant brain tumor
A benign tumor does not contain cancer cells and usually, once removed, does not
recur. Most benign brain tumors have clear borders, meaning they do not invade
surrounding tissue. These tumors can, cause symptoms similar to cancerous tumors
because of their size and location in the brain.
Malignant brain tumors contain cancer cells. Malignant brain tumors are usually fast
growing and invade surrounding tissue. Malignant brain tumors very rarely spread to
other areas of the body, but may recur after treatment. Sometimes, brain tumors that
are not cancer are called malignant because of their size and location, and the damage
they can do to vital functions of the brain.
Brain tumors can occur at any age. Brain tumors that occur in infants and children are
very different from adult brain tumors, both in terms of the type of cells and the
responsiveness to treatment.
3. This case study which primarily talks about brain tumor is directed towards presenting
the disease, the management and intervention and the other vital facts that remain in
oblivion to the great number of population of this country.
Considering that the brain tumor truly and evidently has a devastating impact of our
nation’s health our Group BSN-II of GNC has regarded this study significant to the fields
of nursing education practice and research because the completion of this study does
not only meet the terms for dissemination information purposes, but for sensible
learning as well.
OBJECTIVES:
A. General objective:
To be able to choose a case study that will contribute and expand our
knowledge and improve our skills on specific procedures this is BRAIN
TUMOR.
Our group has formulated the following specific objectives to guide us
toward the completion of this case study. That we may be able to:
B. Specific Objective(s):
1. Established good intrapersonal and professional relationship with our
patient and her accompanying family members
2. Share our knowledge and skills to each other
3. Work together with the health care team
4. Provide significant health teaching that would promote our patient
health and wellness
5. Formulate effectiveness nursing care plan
6. Formulate specific, measurable, attainable, realistic and time bounded
objectives that will serve a guide for the accomplishment of the study
(SMART)
7. List the actual and possible symptoms that our patient may manifest
8. Research the drug study of the given medication to our patient
9. List all the references used in the study
C. Current Trends
4. This article is about children born with birth defects or to mothers together with a
history of multiple stillbirths that may have a higher-than-normal risk of brain cancer.
Since these sometimes involve some type of genetic abnormalities, they can increase
the risk of having a brain tumor. Some preliminary evidence, Dr. Partap said, suggests
that “defects of the heart and brain may be particularly linked to childhood cancer.”
Symptoms of brain tumors are also not clear to Pediatricians. So, researchers are
doing their best to find the solution to their problems.
We think that having some type of genetic abnormalities can increase the case of
having a brain tumor, because we know it is connected to the brain. Having brain tumor
can be frustrating to both the patient with brain tumor and his/her family, which is why
we concur about people needing to know the symptoms of brain tumor. So, as early as
possible, we can detect if there is brain tumor or not and we can treat it right away.
5. From Reuters Health Information
Birth Defects Tied to Pediatric Brain Tumors
By Amy Norton
NEW YORK (Reuters Health) Aug 10 - Children born with birth defects or to mothers with a history of multiple stillbirths may have a higher-than-normal risk of
brain cancer, a new study suggests.
The risks are still small, researchers say, as children only rarely develop brain cancer. Each year, about 4,000 U.S. children and teenagers are diagnosed with a
tumor of the central nervous system (brainor spinal cord), according to the American Cancer Society.
Small portions are caused by specific, inherited genetic syndromes, but otherwise little is known about why childrendevelop brain and spinal cancers.
The new f indings, publishedonline August 8th in Pediatrics, highlight the potential importance of genetic factors, the researchers say.
Using a Californiadatabase on cancer cases in the state, the researchers found 3,733 cases of brain or spinal tumors diagnosed among children younger than 15
between 1988 and 2006.
Ov erall, 1.2% of those children had been bornwith a birth defect -- vs. 0.6% of 15,000 cancer-freeCalifornia children studied for comparison.
And children with a birth defect hadincreasedrisks of certain tumors.
They were nearly four times as likely as children without birth defects to develop a primitiveneuroectodermal tumor.
Similarly , theirrisk of germ cell tumors was elevatedmore than six-fold.
Children with birth defects were not, however, at higher risk for themost commontype of braincancer in the study group -- gliomas, which accounted for 57%of
cases.
The study alsofoundheightened tumorrisks among children whose mothers hadhad at least two late pregnancy losses in thepast -- meaning the fetus died after
the 20th week of pregnancy.
These children were about threetimes as likely as otherkids to develop sometype of brainor spinal tumor.
Since both birth defects andpregnancy losses often involvesome type of genetic abnormality, it's possiblethat explains the higher cancer risks, according to the
researchers.
"Genetics may play a larger role in central nervous system cancer than previously believed," saidlead researcher Dr. Sonia Partap, of Stanford University and
Lucile Packard Children's Hospital in Palo Alto, California.
Early miscarriages were not linked to cancer risks in a woman's other children. So it's possiblethat the genetic abnormalities that cause early pregnancy loss are
not connectedto cancer, while gene defects that are "compatiblewith life tosome degree" do contribute to cancer risk, Dr. Partaptold Reuters Health in an email.
As f or birth defects, past studies have connected them tohigher risks of childhood cancers in general.
But researchers are still trying tofigureout whether it's only certain birth defects that come with a higher risk. Some preliminary evidence, Dr. Partap said,
suggests that defects of the heart and brain may be particularly linked to childhood cancer.
But Dr. Partap also stressedthat even witha relatively increased risk of brainor spinal cancer, the absolute risk to any one child is small.
"Parents should know that there is still a very low risk of central nervous system cancer," she said.
At the same time, she added, pediatricians should be aware that there is a slightly higher chance of the tumors in certain children.
Sy mptoms of brain tumors may be vague and vary from child tochild. But some possible signs include morning headaches; mental changes like memory and
concentration problems; unusual sleepiness; changes in vision, hearing or speech; and balanceor coordination problems.
SOURCE: http://bit.ly/oWBZpY
6. II. Demographic Data
A. Personal information:
Ms. H.A is 2 year old patient, confined at DPMMH, residence of Del
Carmen, Lubao Pampanga. Her birthday is on March 26, 2009. She has a
twin sister. She is the youngest among the 3 siblings. Her religion is Roman
Catholic. According to her mother, H.A loves to sing and dance.
B. History:
Ms. H.A was admitted to the hospital last January 01, 2012 with a chief
complaint of headache, vomiting, high fever and seizures.
Present history:
Last November 27, 2011 the pt. complains of headache, so the S.O brought her
to the clinic for check-up. The doctor prescribes medications for the headaches
but it did not worked. So the pt .was brought to PMSH (Pampanga Medical
Specialist Hospital) because of the headache and seizures and the doctor
ordered for EEG, but the result is normal. The pt. was admitted again to MMH
(Macabali Memorial Hospital) but has been transferred to Mother Theresa of
Calcuta for a CT scan and been diagnosis of BRAIN TUMOR. Because of
financial support,they transferred her to DPMMH (Diosdado P. Macapagal
Memorial Hospital)
Family History
No history of diseases.
Past History
According to her mother, Ms. H.A didn’t have any past illness or disease.
7. III. Physical Assessment:
General Appearance:
Received a patient who is a 2 year old girl, lying on bed unconsciously with an
IVF of D5 0.3 NaCl 500cc @ 350cc level, regulated @ 4-5mgtts/min infusing well on her
L hand and also hooked with an O² of 3L/min via nasal cannula.
Normal Vital Signs:
T: 36-37.5 oC
RR: 25-50 bpm
CR: 80-150 bpm
Vital signs:
T:40.0 oC
RR: 30 bpm
8. CR: 160 bpm
Organ/Body Parts Normal Findings Significant Findings
Skin Fair in complexion
With good skin turgor
Oily Skin
Cold clammy skin
Nails No evidence of clubbing of
fingernails
Capillary refill: within 2-3
seconds
Head Skull:
Hair texture: black and oily
curly hair strands
Scalp: fair in complexion
(-) lesions
Asymmetrical frontal lobe
Hair partially distributed
Eyes
Peri-orbital area
Eyelashes
Eyelids
Conjunctiva
Pupils
Cornea
Sclera
Thin eyebrows, black in
color
Equally distributed, curled
slightly outward
Skin intact, (-) discharge
Shiny and smooth
Pink palpebral conjunctiva
PERLA
(Pupils Equal and Reactive
to Light and
Accommodation)
Clear
(-) lesions
Appeared convex
White and buff
9. (-)
Ears
Auricles Fair in complexion,
symmetrical elastic, and
mobile when pinch, and
aligned with the outer cantus
of the eyes
(+) wet cerumen
Nose
External nose
Nasal septum
Nasal cavity
Symmetrical and not tender
Intact and in midline
Pink colored mucosa,
(+) black and white cilia
Mouth
Teeth
Tongue
Lips
White in color
Pinkish in color
Pink in color (+) cheilosis
Neck
Thyroid gland
Lymph nodes
(-) Bulging mass
Normal
(-) Bruits are palpated
(-) Swelling
(-) Enlargement
(-) Tenderness
10. Chest
Respiratory rate
Breathing pattern
Heart sounds
Normally fast
Normal Breathing Pattern
normal: no murmur
(-) chest pain
(-) palpitation
Abdomen
Color
Contour
Palpation
Fair in complexion
Normal bowel sounds
Palpation: soft, non-tender
Musculoskeletal
Upper extremities
Pulses Radial and brachial pulse is
normal and palpable
Lower extremities
Legs Long and thin legs
IV. Laboratory and Diagnostic Result
Lab Test Patients Results Normal Value Interpretation
Complete
Blood Count
(CBC)
Hemoglobin: 136
Erythrocytes: 4.78
Hematocrit: 0.41
Leucocytes: 8.9
Lymphocytes: 0.60
Platelet Count: 492
120 – 170 g/L
4.0 – 5.0 x 10
0.36 – 0.46
4.5 – 11
0.20 – 0.40
150 – 450
Normal
Normal
Normal
Normal
There is abnormal cell mutation
There is abnormal cell mutation
Blood
Chemistry
RBS: 150 80 – 115 It
11. Cranial CT-Scan
There is a 3.3 x 6.1 x 4.9 cm (LxWxAP) lobulated, heterogeneous mass with cystic and
homogeneously enhancing solid components, involving the right thalamus, right side of
the pons, medical aspect of the right temporal lobe and inferoposterior aspect of the
frontal lobe. Associated perifocal edema, contralateralshift if the midline structures,
lateral displacement of the dorsal horn of the right lateral ventricle and compression of
the third and right lateral ventricles. Resultant moderate dilatation of the lateral
ventricles with subependymal seepage is seen.
The posterior fossas are unremarkable.
There is no intracranial hemorrhage.
The rest of the cisterns and sulci are not widened.
The visualized paranasal sinuses and mastoids are well aerated. The cranium is intact.
Impression:
Complex mass with cystic and solid components as described involving the right
thalamus, right side of the pons, medical aspect of the right temporal lobe and
inferoposterior aspect of the frontal lobe with associated perifocal edems, mild mass
effect and secondary obstructive hydrocephalus. Primary consideration is a neoplastic
process such as glioblastoma multiforme.
V. Review of system
CENTRAL NERVOUS SYSTEM
Nervous System
12. The nervous system is broken down into two major parts: the central nervous system,
which includes the brain and spinal cord, and the peripheral nervous system, which
includes all nerves, which carry impulses to and from the brain and spinal cord. These
include our sense organs, the eyes, the ears, our sense of taste, smell and touch, as
well as our ability to feel pain.
Spinal Cord
The spinal cord is a long bundle of neural tissue continuous with the brain that occupies
the interior canal of the spinal column and functions as the primary communication link
between the brain and the rest of the body. The spinal cord receives signals from the
peripheral senses and relays them to the brain.
13. Brain
The brain is the largest and most complex part of the nervous system. It is compose of
more than 100 billion neurons and associated fibers. The brain tissues have a gelatin
like consistency. The semi-solid organ weighs about 1400g (approximately 3 pounds) in
the adult human.
1. The frontal lobes (motor complex) controls voluntary motor activity.
2. The parietal areas these same areas are thought to contribute to reasoning,
problem solving activities and emotional stability.
14. 3. The occipital lobe contains a primary visual receptive (interpretation) area and
visual association areas.
4. The temporal lobe is located under (inferior to) the lateral sulcus. It contains
primary auditory receptive area and secondary auditory association areas.
Brain Stem
The brain stem is the part of the brain that connects the cerebrum and diencephalons
with the spinal cord.
Medulla Oblongata
The medulla oblongata is located just above the spinal cord. This part of the brain is
responsible for several vital autonomic centers including
The respiratory center, which regulates breathing.
The cardiac center that regulates the rate and force of the heartbeat.
The vasomotor center, which regulates the contraction of smooth muscle in the
blood vessel, thus controlling blood pressure.
The medulla also controls other reflex actions including vomiting, sneezing, coughing
and swallowing.
Pons
Continuing up the brain stem, it reaches the Pons. The pons lay just above the medulla
and acts as a link between various parts of the brain. The pons connects the two halves
of the cerebellum with the brainstem, as well as the cerebrum with the spinal cord. The
pons, like the medulla oblongata, contains certain reflex actions, such as some of the
respiratory responses.
Midbrain
15. The midbrain extends from the pons to the diencephalon. The midbrain acts as a relay
center for certain head and eye reflexes in response to visual stimuli. The midbrain is
also a major relay center for auditory information.
Diencephalon
The diencephalons are located between the cerebrum and the mid brain. The
diencephalons houses important structures including the thalamus, the hypothalamus
and the pineal gland.
Thalamus
The thalamus is responsible for "sorting out" sensory impulses and directing them to a
particular area of the brain. Nearly all sensory impulses travel through the thalamus.
Hypothalamus
The hypothalamus is the great controller of body regulation and plays an important role
in the connection between mind and body, where it serves as the primary link between
the nervous and endocrine systems. The hypothalamus produces hormones that
regulate the secretion of specific hormones from the pituitary. The hypothalamus also
maintains water balance, appetite, sexual behavior, and some emotions, including fear,
pleasure and pain.
Limbic System
The limbic system, often referred to as the "emotional brain", is found buried within the
cerebrum. Like the cerebellum, evolutionarily the structure is rather old.
Cerebellum (little brain)
16. The functions of the cerebellum include the coordination of voluntary muscles, the
maintenance of balance when standing, walking and sitting, and the maintenance of
muscle tone ensuring that the body can adapt to changes in position quickly.
Cerebrum
The largest and most prominent part of the brain, the cerebrum governs higher mental
processes including intellect, reason, memory and language skills. The cerebrum can
be divided into 3 major functions:
Sensory Functions - the cerebrum receives information from a sense organ; i.e.,
eyes, ears, taste, smell, feelings, and translates this information into a form that
can be understood.
Motor Functions - all voluntary movement and some involuntary movement.
Intellectual Functions - responsible for learning, memory and recall.
Meninges
The meninges are made up of three layers of connective tissue that surround and
protect both the brain and spinal cord. The layers include the Dura mater, the arachnoid
and the pia matter.
Pia mater is a vascular layer of connective tissue that is so closely connected to
the brain and spinal cord that is follows every sulcus and fissures.
Dura mater is a tough non-stretchable vascular membrane with 2 layers the
outer and inner layer.
Reflex Mechanism
Our conscious autonomic responses to internal and external stimuli known as reflex
responses provide many homeostatic functions. Although the spinal cord is often
thought of as the reflex center, it is not the only site for regulation .Many of the complex
reflexes controlling the heart rate, breathing, blood pressure, swallowing, coughing, and
vomiting are found in the brain stem.
Cerebrospinal Fluid
17. The cerebrospinal fluid is a clear liquid that circulates in and around the brain and spinal
cord. Its function is to cushion the brain and spinal cord, carry nutrients to the cells and
remove waste products from these tissues.
Neurons:
A neuronal cell body (soma) is like other cell in that it contains most of the organelles
seen in other cells.
There are several types of neurons - anaxonic neurons: small neurons where the
dendrites and axons are indistinguishable.
Bipolar neurons: small neurons with two distinct processes; a dendritic process
and an axon extending from the cell body.
Unipolar neurons: large neurons with the cell body lying to one side of the
continuous dendritic process and axon.
Multipolar neurons: large neurons with several dendrites and a single axon
extending from the cell body.
Bipolar neurons: Bipolar neurons are CNS neurons specific for transmitting
information from specialized sensory systems: sight, smell and hearing.
Grey and white matter: Grey matter consisting of unmyelinated neurons is the
processing area of the CNS. White matter – located in the inner cortex and surrounding
grey matter in the spinal cord - provide pathways of communication between grey
matters.
Glial Cells
CNS Glial Cell Types: There are 4 types of glial cells:
1. astrocytes - Regulates the chemical microenvironment surrounding neurons.
2. Oligodendrocytes - Myelinate central nervous system axons.
3. Microglia - Migrating phagocytic cells resembling immune cells that remove
waste, debris, and pathogens.
4. Ependymal cells - Columnar cells that line the ventricles of the brain and the
spinal canal in the spinal cord.
Peripheral Nervous System
18. The PNS includes all neurons other than those in the brain and spinal cord. It consists
of pathways of nerve fibers between the CNS and all outlying structures in the body.
Included in the PNS are 12 pairs of cranial nerves and 31 pairs of spinal nerves.
Nerves
Nerves are made up of specialized cells, which act as little wires, transmitting
information to and from the central nervous system and brain. Nerves form the network
of connections that receive signals (known as sensory input) from the environment and
within the body, and transmit the body's responses, or instructions for action, to the
muscles, organs, and glands. Nerve cells are located outside the central nervous
system or spinal cord.
Cranial Nerve
12 pairs of cranial nerves arise from the brain. Most of the cranial nerves are composed
of both motor and sensory neurons although a few cranial nerves carry only sensory
impulses. Except for the olfactory and optic nerves, whose nuclei lie just below the
cerebrum, all other cranial nerve nuclei lie within the brain stem
The Cranial Nerves
19. Nerves Type Function
I
Olfactory
sensory olfaction (smell)
II
Optic
sensory
vision
(Contain 38% of all the axons connecting to the brain.)
III
Oculomotor
motor* eyelid and eyeball muscles
IV
Trochlear
motor* eyeball muscles
V
Trigeminal
mixed
Sensory: facial and mouth sensation
Motor: chewing
VI
Abducens
motor* eyeball movement
VII
Facial
mixed
Sensory: taste
Motor: facial muscles and
salivary glands
VIII
Auditory
sensory hearing and balance
IX
Glossopharyngeal
mixed
Sensory: taste
Motor: swallowing
X
Vagus
mixed
main nerve of the
parasympathetic nervous system (PNS)
XI
Accessory
motor swallowing; moving head and shoulder
XII
Hypoglossal
motor* tongue muscles
20. VI. Pathophysiology
Risk Factors + normal cells
↓
Initiation
↓
Promotion
↓
Malignant conversion
↓
Progression
↓
Tumor occupy normal tissue spaces
↓
Destroy major function of the Thalamus
Sorting out sensory impulses
↓
No senses
↓
Cerebral edema
↓
Brain tumor
↓
Death
VII. Course in the Ward
21. Doctor’s Order
January 01, 2012
8:20 PM
Please admit the pt. to ROC For continued therapy
Secure consent For legal purposes
TPR q shift and recorded To obtain baseline data for
comparison
NPO temporarily To prevent aspiration
Lab result CBC typing To identify infection
IVF of D5 0.3 NaCl 500cc
KVO
For route of medication
Cefuroxime vial 400mg slow IV
push q6 NST
To treat bacterial infections
O2 inhalation 3L To help the patient to support
decreased perfusion
Continue high back rest To help improve venous drainage,
reduce arterial pressure, and may
improve cerebral perfusion
Refer to Dra. Balagtas For neuro evaluation
January 02, 2012
9:20 AM
Paracetamol 0.8mL
TID – now
Prn for T = 38.8 oC
To decrease hyperthermia
January 02, 2012
9:50 AM
T = 40 oC
Continue medications To continue the therapeutic regimen
DAT w/ aspiration precaution To prevent aspiration
TSB To evaporate heat in the body
Carry out orders of Dra. Balagtas For evaluation and management
January 02, 2012
CTScan To identify tumor, cerebral edema
or hydrocephalus
Give Dyphenhydramine TIV at
0.1mg/kg/dose now
To sedate the patient from having
seizures
Refer to Dr. For further evaluation and
management
Give Dexamethasone at 0.1 mg/kg
TIV now often q 12 hours
To decrease cerebral inflammation
and edema
Kindly IVF rate as replacement
May also be dehydrated
To hydrate the patient
22. January 02, 2012
4:45 PM Seizure
Dyphenhydramine 12.5mg IV now To sedate patient from seizure
January 03, 2012
7:05 AM
Continue medications To continue therapeutic regimen
Carry out referal to Dr. Rivera and
Dr. Beltran
For further evaluation and
management
TF D5 0.3 NaCl 500cc x SR To help in hemorrhagic shock
23. VIII. Nursing Care Process
ASSESSMENT NURSING
DIAGNOSIS
SPECIFIC
EXPLANATION
PLANING NURSING
INTERVENTION
RATIONALE EVALUATION
S:
O:
>Febrile, T=40°C
in both axilla;
warm to touch
with flushing
Hyperthermia r/t
increase
Intracranial
pressure
ENTRY OF PATHOGEN
IN THE SYSTEMIC
CIRCULATION
REGULATION OF
TOXIN IN THE BODY
RELEASE OF PYROGEN
STIMULATION OF THE
HYPOTHALAMUS
INCREASE OR
ALTERRATION OF
THERMOREGULATION
INCREASE BODY
TEMPERATURE
Short Term:
After 2-3 hours of
nursingintervention
the patient will be
ableto decrease
body temperature
from 40°C to 37°C.
Long Term:
After 2 days of
nursing intervention
the patient will be to
maintain normal
body temperature
Do/perform
tepid sponge
bath
Assess body
temperature
from time to
time
Do not apply
alcohol for TSB
Advise the so to
increaseoral
To help
decrease body
temperature
To know what
is the
responseof
clientto TSB
Alcohol
increases
peripheral
vascular
constriction
&CNS
depression
Additional
fluids help
Short Term:
The patient
shall
Demonstrated
temperature
within normal
range, from 40
°C to 37.5°C
Long Term:
The patient
shall have
demonstrated
behaviors to
monitor and
promote
normothemia
24. HYPERTHEMIA fluid intakeof
the patient
Remove excess
clothingand
covers
prevent
elevated
temperature
associated
with
dehydration
These
decrease
warmth and
increase
evaporative
cooling
26. S:
O:
>Unconscious
>febrile
Ineffective
cerebral
perfusion
relatedto
interruptionof
bloodflow
Intracranial
pressure
Pressure exerted
in the cranium by
its content
Brain, blood and
cerebrospinal fluid
Associated with
vasospasm or
obstruction in the
arteries supplying
the brain with
blood
Increase vascular
resistance can
result due to
increase ICP
Leading to
decrease and or
absence of blood
flow to the brain
cells
Short Term:
After2-3 hours of
nursing
interventionthe
SO will verbalized
understandingof
condition,therapy
regimenand
whentocontact
healthprovider
Long Term:
After2 days of
nursing
interventionthe
patientwill
demonstrate
behaviorsandlife
style changesto
improve
circulationsuchas
relaxation
techniques.
Independent:
Assesspatient
condition
.Positionhead
slightly
elevatedand
inneutral
position
Take patients
temperature
at least4
hours
Keeppatients
inneutral
alignment
Provide quite,
restful
To be able to
identifypresent
physiologic
disturbances
Reduces arterial
pressureby
promoting venous
drainageand may
improve cerebral
perfusion.
Hyperthermia
causes increased
ICP hypothermia
causes decrease
cerebral perfusion
pressure
To keep the
carotid flow
unobstructed
thereby
promoting
perfusion
Continual
stimulation can
increaseICP.
Short Term:
The So shall
have
verbalized
understanding
of condition,
therapy
regimenand
whento
contact health
care provider
Long Term:
The patient
shall have
Demonstrated
behaviorsand
life style
changesto
improve
circulation
such as
relaxation
techniques.
27. Because of this
there will be
decrease or
absence of oxygen
supplyto the brain
cells
So there is
ineffective
cerebral perfusion
environment.
Note history of
brief/intermitte
nt periods or
black out
Monitor
patients
behavior and
mental status
for onset of
restlessness,
agitation
confusion
Dependent:
Administer
supplemental
oxygen.
Because this
suggest transient
ischemic attacks
Changes in
behavior and
mental status are
sign of altered
cerebral perfusion
Reduces
hypoxemia, which
can cause
cerebral
vasodilatation
and increase
pressure/ edema
formation.
28. ASSESSMENT NURSING
DIAGNOSIS
Scientific
EXPLANATION
PLANING NURSING
INTERVENTION
RATIONALE EVALUATION
S:
O:
> Unconscious
>seizures
Risk for injury
related to
disruption in the
normal flow of
electricity in the
brain
Altered neuronal cells
Increased frequency
and amptitude
Neuronal firing
spreads
Seizures
Unpredictable
movement or
behavior
Risk for Injury
Short Term:
After 2-3 hours of
nursingintervention
the patient’s
seizures will be
lessen
Long Term:
After 2 days of
nursingintervention
the patients seizures
will beremove
Assess patient
condition
Keep padded
siderailsup
with bed in the
lowest position
Provide
information
regarding the
condition that
may resultin
risk for injury.
Assess muscle
strength gross
and fine motor
coordination
To be ableto
identify
present
physiologic
disturbances
Minimizes
injury should
seizureoccur
whilepatient
is in bed
to promote
awareness
to determine
the severity of
body
weakness and
to be ableto
perform
appropriate
intervention
Short Term:
The patient’s
seizures shall
be lessen
Long Term:
The patient’s
seizures shall
be removed
30. IX. Drug Study
Drug Name Classification Indications Mechanis
m of
Action
Adverse Effect Nursing
Considerations
Rationale
Generic
Name:
Diphenhydra
mine
Brand Name:
Oral: Allerdyl
(CAN),
AllerMax
Caplets,
Banophen,
Banophen
allergy,
Benaryl
allergy,
Diphen AF,
Diphenhist
Captabs,
Genahist,
Siladryl
Antihistamine,
Anti-motion-
sickness drug,
Antiparkinsoni
an,
Cough
Suppressant,
Sedative-
hypnotic
-> Relief of symptoms
associated with
perennial and seasonal
allergic rhinitis;
vasomotor rhinitis;
allergic conjunctivitis,
mild, uncomplicated
urticaria and
angioedema;
amelioration of allergic
reactions to blood or
plasma;
dermatographism;
adjunctive theraphy in
anaphylactic reactions.
-> Active and
prophylactic treatment
of motion sickness.
->Nighttime sleep aid
->Parkinsonism
(including drug induced
parkinsonism and
extrapyramidal
reactions), in the
elderly intolerant of
more potent drugs, for
milder forms of disorder
in the other age groups,
and in combination with
centrally acting
Competitiv
ely blocks
the effect of
histamine
at H1-
receptor
sites, has
antropine-
like,
antipruritic,
and
sedative
effects.
CNS: Drowsiness,
sedation, dizziness,
disturbed
coordination, fatigue,
confusion,
restlessness,
excitation,
nervousness, tremor,
headache, blurred
vision, diplopia
CV: Hypotension,
palpitations,
bradycardia,
tachycardia,
extrasystoles
stomatitis
G.I: Epigastric
distress, anorexia,
increased appétit and
weight gain, nausea,
vomiting, diarrhea r
constipation
G.U: Urinary
frequency, dysuria,
urinary retention, early
menses, decreased
libido, impotence
Hematologic:
Hemolytic anemia,
hypoplastic anemia,
-> Administer with
food.
->Avoid driving and
using Dangerous
machine.
-> Administer syrup
form for patient who
can’t take tablets.
->Advice patient to rise
slowly from lying or
sitting position.
->Monitor children
closely.
-> To
prevent GI
upset.
-> To avoid
accident
that may
cause by
the side
effects.
->To
prevent
aspiration.
->To
prevent
orthostatic
hypotensio
n
-> To
identify
paradoxica
l reaction.
31. anticholinergic
antiparkinsonian drugs.
->Syrup formulation:
Suppression of cough
due to colds or allergy.
thrombocytopenia,
leucopenia,
agranulocytosis,
pancytopenia.
Respiratory:
Thickening of
bronchial secretions,
chest tightness,
wheezing, nasal
stiffness, dry mouth,
dry nose, dry throat,
sore throat.
32. Drug Name Classification Indications Mechanis
m of
Action
Adverse Effect Nursing
Considerations
Rationale
Generic
Name:
Cefuroxime
Brand Name:
Ceftin
Zinacef
Antibiotics;
Cephalosporin
Oral(cefuroxime axetil)
-> Pharingitis, tonsillitis
caused by
streptococcus
pyogenes
->otitis media caused
by streptococcus
pneumonia, S.
pyogenes,
Haemophilus influenza,
Moraxella catarrhalis
NEW INDICATION
Acute bacterial
maxillary sinusitis
caused by S.
pneumonia, H.
influenza
-> lower respiratory
infections caused by S.
pneumonia,
Haemaphilus
parainfluenzae, H.
influenza
-> UTI caused by
E.Coli, klebsiella
pneumonia
-> Uncomplicated
gonorrhea (urethral and
endocervical)
Inhibits
synthesis
of bacterial
cell wal,
causing cell
death
CNS: Headache,
dizziness, lethargy,
paresthesias
GI: Nausea, vomiting,
diarrhea, anorexia,
abdominal pain,
flatulence,
pseudomembranous
colitis, heaptotoxicity
GU: Nephrotoxicity
Hematologic: Bone
marrow depression
Hypersensitivity:
Ranging from rash to
fever to anphylasis;
serum sickness
reaction
-> Avoid crushing
tablets.
-> Give PO drug with
meal.
-> Have vitamin K
available.
-> Take full course
therapy even if you are
feeling better.
->To
prevent
tasting the
bitter taste
of the drug.
-> To
decrease
GI upset
and
enhance
absorption.
-> In case
of
hypoprothr
ombinemia
occurs.
-> To
prevent
drug
tolerance.
33. -> skin and skin
structure infections,
including impetigo
caused by
streptococcus aureus,
S. pyogenes
-> Treatment of early
lyme disease
Parental(cefuroxime
sodium)
-> lower respiratory
infections caused by S.
pneumonia, S. aureus,
E. coli, Klebsiella
pneumonia, H.
Influenza, S. pyogenes
-> Dematologic
infections caused by S.
aureus, S. pyogenes,
E. coli, K. pneumonia,
Enterobacter
-> UTIs caused by E.
coli, K. pneumonia
-> Uncomplicated and
disseminated
gonorrhea caused by
N. gonorrhhoeae
-> Septicimia caused
by S. pneumonia, H.
influenzae, S. aureus,
N. mengingitidis.
-> Bone and joint
34. infections due to S.
aureus
-> Perioperative
prophylaxis
-> Treatment of acute
bacterial maxillary
sinusitis in patient 3
mo-12 yr
35. Drug Name Classification
s
Indications Mechanis
m of
Action
Adverse Effects Nursing
Considerations
Rationale
Generic
Name:
Acetaminoph
en
Brand Name:
Tempra;
Tylenol
Analgesic;
Antipyretic
-> Temporary reduction
of fever; temporary
relief of minor aches
and pains caused by
common cold and
influenza, headache,
sore throat, toothache,
menstrual cramps,
backache, minor
arthritis pain, and
muscles pains.
-> Unlabeled use:
Propylaxis in children
and patient at risk for
seizures who are
receiving DTP
vaccination to reduce
incidence of fever and
pain.
Antipyretic
s:
Reducing
fever by
acting
directly on
the
hypothalam
ic heat-
regulating
center to
cause
vasodilatio
n and
sweating,
which heals
to lessen
heat.
CNS: Headache
CV: Chest pain;
dyspnea; myocardial
damage when dose of
5-8g/day are ingested
daily for several
weeks or when
dosages of 4g/day are
ingested for 1year.
GI: Hepatic toxicity
and failure, jaundice
GU: Acute renal
failure, renal tubular
necrosis.
Hematologic:
methamoglobinemia-
-cyanosis; hemolytic
anemia; anuria;
neutropenia;
leukopenia;
pancytopenia;
thrombopenia;
hypoglycemia
Hypersensitivity:
Rash, Fever
-> Give pedia patient
on liquid form of
medication.
-> TSB.
-> Take medicine q4.
-> Give drug with food.
-> To avoid
splitting up
and easy
to swallow.
-> To
evaporate
heat of the
patient.
-> To
complete
therapeutic
regiments.
-> To
prevent GI
upset.
36.
37. Drug Study Classificati
on
Indications Mechanis
m of
Action
Adverse Effects Nursing
Considerations
Rationale
Generic
Name:
Dexamthason
e
Brand
Name/s:
Dexasone,
Dexone,
Hexadrol
Corticostero
id
Glucocortic
oid
Hormone
->Management of
cerebral edema
->Diagnostic agent
in adrenal disorders
->Relieves
inflammation
Dexameth
asone
suppresse
s
inflammati
on and the
normal
immune
response.
It prevents
the release
of
substances
in the body
that
causes
inflammati
on.
Systemic
Administration
CNS: Seizures,
vertigo, headaches,
pseudotumor cerebri,
euphoria, insomnia,
mood swings,
depression, psychosis,
intracerebral
hemorrhage, reversible
cerebral atrophy in
infants, caratacts, IOP,
glaucoma
CV: Hypertension,
Heart failure,
necrotizing angritis
Endocrine: Growth
retardation, decreased
carbohydrates
tolerance, DM,
cushingoid state,
secondary
adrenocortical and
pituitary
unresponsiveness
GI: Peptic or
esophageal ulcer,
pancreatitis, abdominal
distention
->Give drug with
food.
->
-> To
minimize
GI
irritation.
->
38. GU: Amenorrhea,
irregular menses
Hematologic: Fluid
and electrolytes
disturbance, negative
nitrogen balance,
increased blood sugar,
glycosuria, increased
serum cholesterol,
decreased serum T3
and T4 levels
Hypersensitivity:
Anaphylactoid or
hypersensitivity
reactions
Musculoskeletal:
Muscle weakness,
steroid myopathy, loss
of muscle mass,
osteoporosis,
spontaneous fractures
Other/s: Impaired
wound healing;
petechiae;
ecchymoses;
increased sweating;
thin and fragile skin,
acne;
immunosuppression;
and masking of signs
of infection; activation
of latent infections,
39. including TB, fungal ,
and viral eye
infections; pneumonia;
abscess; septic
infection; GI and GU
infections
40. X. Discharge Planning
M- Medicine
-Instructed patient to take the medications.
E-Exercise
-Instructed patient to do the ADL.
T-Treatment
-Continue medications and promote supportive treatment as PRN, such as TSB and Paracetamol.
H-Health Teaching
-Instruct SO to give nutritional foods like green leafy vegetables example (malungay, ampalaya and bitter melon).
-Instruct SO to give food rich in fiber.
-Instruct SO to avoid food rich in saturated fats and hydrogenated oils.
-Instruct SO to give foods rich in vitamin C.
41. O-Out patient
-instructed patient to return after one week @ OPD @ 8AM for follow-up checkup
D-Diet
-instructed patient to avoid or limit foods rich in saturated fats and hydrogenated oils
-DAT with aspiration diet
42. XI. Bibliography
Book(s):
Joyce M. Black and Jane Hokanson Hawks, Medical Durgical Nursing (7th Edition) 2004, EL SEVIER (Singapore) PTE
LTD.
Marilynn E. Doenges, Mary Frances Moorhouse, and Alice C. Murr, Nurse’s Pocket Guide (12th Edition) 2008, Nursing:
Joanne Patzek DaCunha, RN, MSN.
Amy M. Karch, 2011 LIPPINCOTT’S: Nursing Drug Guide, 2011, Chris Burghargt.
Website(s):
http://www.medscape.com/viewarticle/747859, 2012.
http://www.emedicinehealth.com/anatomy_of_the_central_nervous_system/article_em.htm
http://users.rcn.com/jkimball.ma.ultranet/BiologyPages/C/CNS.html, November 18, 2011.
http://serendip.brynmawr.edu/bb/kinser/Structure1.html, 10:45:07 EDT, June 3, 2005.
http://www.chw.org/display/router.asp?DocID=22484, 2012.
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