2. A 65y old man presented to the
A&E with
Attacks of colicky abdominal pain associated with
repeated vomiting and absolute constipation
On examination, the abdomen is hugely distended
showing visible peristalsis with a long midline scar of
previous laparotomy
The patient is tachycardic and dehydrated with
disturbed electrolytes
AXR showed dilated small bowel loops
5. Classifications of IO:
Type of obstruction
Mechanical (dynamic)
Functional (adynamic)
Time of presentation
Acute
Chronic
Extent of obstruction
Partial
Complete
Pathological nature of
obstruction
Simple mechanical
Strangulation
Paralytic ileus
6. Mechanical (dynamic)
Peristalsis is working
against a mechanical
obstruction
Extramural Intramural Intraluminal
• Adhesions
• Hernias
• Volvulus
• Tumours
• Congenital
anomalies
atresia
• Intussuscepti
on
• Neoplasm
• Stricture
• Gallstone
ileus
• Fecal
impaction
• FB bezor
• Traumatic
intraluminal
hematoma
7. Functional (adynamic)
Atony of the intestine
with loss of normal
peristalsis in the absence
of mechanical
obstruction e.g. ileus
Peristalsis present in a
non-propulsive form e.g.
MVO or pseudo-
obstruction
8. Commonest causes
Strangulated hernias
most common cause of
SBO in children
Adhesions most
common cause of SBO in
adults
Cancer colon most
common cause of LBO
9. Cardinal clinical features of acute
obstruction
Abdominal pain
Distension
Vomiting
Absolute constipation
10. Features according to level of obstruction
High small bowel obstruction
Vomiting early & profuse
rapid dehydration
Distension minimal
Few fluid levels on AXR
Low small bowel obstruction
Pain predominant
Central distension.
Vomiting delayed.
Multiple central fluid levels on
AXR
Large bowel obstruction
Distension early + marked
Pain mild
Vomiting & dehydration late
The proximal colon and
caecum are distended on AXR
15. Radiological features of obstruction
SBO
Straight segments that are generally
central and lie transversely
No gas is seen in the colon
Jejunum Valvulae conniventes
Pass across the width of the bowel
Regularly spaced, giving a
‘concertina’ or ladder effect
Ileum featureless
Caecum – a distended caecum a
rounded gas shadow in the RIF
Large bowel, except for the caecum
haustral folds (unlike VC)
Spaced irregularly
Do not cross the whole diameter of
the bowel
Do not have indentations placed
opposite one another
16. MCQ
The following features are present in SBO except
Distension
Vomiting
Dehydration
Diarrhoea
Hyperkalemia
Dilated bowel loops on AXR
17. Preoperative management (drip & suck)
NGT
Free drainage + 4-hourly
aspiration /continuous
or intermittent suction.
Decompression
proximal to the
obstruction
Reduce risk of
aspiration
IV fluids
Antibiotics
Urinary catheter
18. Surgery
Indications for early surgical
intervention
Principles of surgical
intervention for obstruction
Obstructed or strangulated external
hernia
Internal intestinal strangulation
Acute obstruction
Management of:
The segment at the site of
obstruction
The distended proximal bowel
The underlying cause of obstruction
19. Differentiation between viable and non-viable
intestine
Viable Non-
viable
Circulation • Dark color
becomes
lighter
• Mesentery
bleeds if
pricked
• Dark colour
remains
• No bleeding
if mesentery
is pricked
Peritoneum Shiny Dull & lustreless
Intestinal
musculature
• Firm
• Pressure
rings may or
may not
disappear
• Peristalsis
may be
observed
• Flabby, thin
& friable
• Pressure
rings persist
• No
peristalsis
21. MCQ
In the treatment of dynamic bowel obstruction
Conservative management is the only option
Non-viable bowel should be resected during the
operation
Volvulus can be treated conservatively
Drip and suck is tried in adhesive intestinal
obstruction
Fecal impaction should be treated operatively
Intussusception should be treated operatively
22. A 10 months old child presented to
the A&E with
Episodes of screaming and drawing up of the legs lasting
for 1-2min and recurs every 15-20min
The condition is associated with Pallor + sweating +
retching
Vomiting which is bile-stained
Initially, the passage of stool was normal, but now the
patient is passing blood and mucus PR
Hx of recent respiratory tract infection
On examination, the right iliac fossa felt empty with
Sausage-shaped mass palpated in the right hypochondrium
24. Definition
Segment of the gut
(intussusceptun) becomes
invaginated within an
immediately adjacent
segment (intussuscepiens)
Usually proximal into
distal
An example of a
strangulating obstruction
as the blood supply of the
inner layer is usually
impaired
25. Aetiology
Peak between 5-10 months
~90% idiopathic ?
• Upper respiratory tract
infection
• gastroenteritis
>2 years a pathological
lead point in 1/3
Adult lead point usually
a polyp (e.g. Peutz–Jeghers
syndrome), a submucosal
lipoma or other tumour.
27. Symptoms
Early
Intermittent colicky pain
Screaming and drawing up
of the legs
Pallor + sweating +
retching
Vomiting
Episodes 1-2min every 15-
20min
Redcurrant jelly stool
Late
Symptoms of peritonitis &
IO
28. Signs
Examination between episodes of
colic
Abdomen is not initially distended
Emptiness in the right iliac fossa
(sign of Dance)
Sausage-shaped mass
PR
Blood-stained mucus
Apex palpable / protrude from the
anus in extensive ileocolic or
colocolic
Unrelieved
Progressive dehydration
Abdominal distension
Gangrene peritonitis
29. Imaging in intussusception
AXR
IO
Absent caecal gas in
ileocolic
Soft tissue opacity in
children
Barium enema the claw
sign in ileocolic I
US high sensitivity in
children typical
doughnut appearance of
concentric rings in
transverse section
CT in equivocal cases
30. Pneumatic or hydrostatic reduction
Successful
free reflux of air or barium into
the small bowel
resolution of symptoms &
signs
Contraindicated if
Signs of peritonitis or
perforation
Known pathological lead point
Profound shock
>70% can be reduced non-
operatively
10% recurrence
31. Surgery
Radiological reduction failed
or contraindicated
Transverse right-sided
abdominal incision
Gently compress the most distal
part of the intussusception
toward its origin
The last part of the reduction is
the most difficult
Resection & 1ry anastomosis
Irreducible intussusception
Infarction
Lead point
32. MCQ
Intussusception
Most common in children
Always secondary to intestinal pathology, e.g. polyp,
Meckel’s diverticulum in infants
Ileo-colic is the least common
Can lead to an ischaemic segment
Pneumatic reduction is usually successful in children
Laparotomy is always required
33. A 70y old black male present to
the A&E with
Recurrent attacks of abdominal pain associated with
absolute constipation
The patient complained of nausea but no vomiting is
experienced
On examination, the abdomen is distended hugely,
tympanitic on percussion with exaggerated bowel
sounds
AXR showed the coffee-bean sign
34.
35. Definition
Twisting or axial rotation of a
portion of bowel around its
mesentery
When complete it forms a closed
loop of obstruction with resultant
ischaemia secondary to vascular
occlusion
May involve
Sigmoid colon commonest in
adults
Small intestine
Caecum
Stomach
Neonatal midgut volvulus
secondary to midgut malrotation is
life-threatening
36. Sigmoid volvulus
Predisposing factors
Symptoms
Large bowel obstruction
initially intermittent
Abdominal distension
early & progressive sign
Hiccough & retching
Vomiting occurs late
Constipation is absolute
In elderly chronic form
40. Treatment
Flexible sigmoidoscopy or
rigid sigmoidoscopy +
flatus tube
Laparotomy
Untwisting of the loop +
per anum decompression
Viable fixation of the
sigmoid colon to the
posterior abdominal wall
inexperienced hands
Resection preferable
Hartmann’s procedure +
subsequent re-
anastomosis
41. MCQ
Volvulus
May involve the sigmoid colon only
Neonatal midgut volvulus secondary to midgut
malrotation is life-threatening
The commonest spontaneous type in adults is caecal
volvulus
Sigmoid volvulus can be relieved by decompression per
anum
Surgery is required to prevent or relieve ischaemia
42. A 50y old lady presented to the
A&E with
Abdominal colicky pain
Nausea and vomiting
Absolute constipation
Distension
Tachycardia
Dry tongue
History of repeated laparotomies, the last one was rwo
years ago for perforated DU
44. Common causes of intra-abdominal
adhesions
1. Ischaemic areas • Sites of
anastomoses
• Vascular occlusion
2. Foreign material Talc, gauze, silk
3. Infection Peritonitis, T.B.
4. Inflammatory
conditions
Crohn’s disease
5. Radiation
enteritis
45. Bands
Congenital, e.g.
obliterated vitello-
intestinal duct
String band following
previous bacterial
peritonitis
Portion of greater
omentum, usually
adherent to the parietes
46. Prevention of adhesions
Factors that may limit
adhesion formation include:
Good surgical technique
Washing of the peritoneal
cavity with saline to
remove clots, etc.
Minimising contact with
gauze
Covering anastomosis and
raw peritoneal surfaces
47. Treatment of adhesive obstruction
Initially treat conservatively
provided there are no signs of
strangulation; should rarely
continue conservative
treatment for longer than 72
hours
At operation, divide only the
causative adhesion(s) and
limit dissection
Cover serosal tears
Invaginate (or resect) areas of
doubtful viability
Laparoscopic adhesiolysis
in chronic cases
48. MCQ
Prevention of intraoperative adhesions can be
done by
Good surgical technique
Copious irrigation with saline
Curetting the bowel with gauze
Excising omentum
Avoid using powdered gloves
49. The ward nurse asked you to review a
postoperative patient complaining of
Abdominal distension and absolute constipation
The patient vomited twice
No abdominal pain is felt
On examination, abdominal tenderness is mild and no
bowel sounds are heard on auscultation
AXR showed dilated small bowel loops with
multiple fluid levels
50.
51. Definition
Failure of transmission of
peristaltic waves secondary
to neuromuscular failure
i.e. in the myenteric
(Auerbach’s) and
submucous (Meissner’s)
plexuses
accumulation of fluid
and gas distension,
vomiting, absence of bowel
sounds & absolute
constipation
52. Causes
Postoperative: a variable duration
of 24–72
Intra-abdominal sepsis: localised
or generalised ileus
Reflex ileus: following
# Spine or ribs
Retroperitoneal hge
Plaster jacket
Metabolic:
Uraemia
Hypokalaemia
Diabetic ketoacidosis
Drugs in high doses:
Anticholinergics e.g. probanthine
Tricyclic antidepressants
53. Clinical features
72 hours after laparotomy:
no bowel sounds
flatus.
Abdominal distension
more marked &
tympanitic.
Pain is not a feature
Vomiting if no NGT
AXR gas-filled loops of
intestine + multiple fluid
levels
54. Prevention
Correction of biochemical
disturbances
Gentle handling of bowel
Nasogastric suction
Restriction of oral intake
Electrolyte balance
Enhanced recovery
programme with early
introduction of fluids and
solids
55. Treatment
1ry cause must be
removed
Drip & Suck
Rarely, in resistant
cases:
Adrenergic blocking
agent + cholinergic
stimulation, e.g.
neostigmine (the
Catchpole regimen)
Laparotomy to
exclude a hidden cause
& facilitate bowel
decompression
56. MCQ
Paralytic ileus
Is functional and not mechanical
Usually postoperative
Pain is predominant
Distension is present
Bowel sounds are present
Is due to neuromuscular failure
Usually treated surgically
57. You was asked to review a patient with
known retroperitoneal sarcoma
showing
Massive abdominal distension and respiratory distress
Abdominal pain, nausea and vomiting
Tympanitic abdomen and sluggish intestinal sounds
AXR showed dilated colon from the caecum to the
splenic flexure
Single-contrast water-soluble barium enema showed no
mechanical cause of LBO
61. Colonic pseudo-obstruction
Acute (Ogilvie’s syndrome) or
chronic
AXR colonic obstruction +
marked caecal distension
??Caecal perforation
Absence of a mechanical cause
requires urgent confirmation by
Colonoscopy
single-contrast water-soluble
barium enema
CT
Colonoscopic decompression
recur in 25% further colonoscopy
+ flatus tube
Colonoscopy fails or unavailable
Tube caecostomy
Subtotal colectomy and ileorectal
anastomosis.
62. MCQ
Colonic pseudo-obstruction
Is called Ogilvie’s syndrome in chronic cases
AXR shows dilated large bowel
Single-contrast water-soluble barium enema is used to
exclude mechanical cause
Colonoscopy is used for colonic decompression
Surgery is not an option for treatment
Editor's Notes
IntussusceptionMay be an intrinsic lesion serving as a lead point
It is vital to distinguish strangulating from non-strangulating intestinal obstruction because the former is a surgical emergency
Symptoms usually commence suddenly and recur regularly
Generalised tenderness and the presence of rigidity are indicative of the need for early laparotomy.
In cases of intestinal obstruction in which pain persists despite conservative management, even in the absence of the above signs, strangulation should be diagnosed
When strangulation occurs in an external hernia, the lump is tense, tender and irreducible, there is no expansile cough impulse and it has recently increased in size
US intususception
CT Sensitivity 80-90% IO
The degree of ischaemia is dependent on the tightness of the invagination, which is usually greatest as it passes through the ileocaecal valve.
Peak between 5-10 months ~90% idiopathic Upper respiratory tract infection or gastroenteritis may precede the condition Hyperplasia of Peyer’s patches in the terminal ileum
Episodes of screaming and drawing up of the legs in a previously well male infant.
The attacks last for a few minutes and recur repeatedly.
During attacks the child appears pale, whereas between episodes he may be listless.
Vomiting may or may not occur at the outset but becomes conspicuous and bile-stained with time.
Initially, the passage of stool may be normal, whereas, later, blood and mucus are evacuated – the ‘redcurrant jelly’ stool.
Signe de Dance
Rarely, natural cure may occur as a result of sloughing of the intussusception
After reduction, the terminal part of the small bowel and the appendix will be seen to be bruised and oedematous. The viability of the whole bowel should be checked carefully
intra-abdominal sepsis may give rise to localised or generalised ileus. Resultant adhesions may contribute a mechanical element to the initial neurogenic aetiology
Caecal perforation is a well-recognised complication