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Brigadier Dr.
Khaled Elkholy
MD, MRCS, MSc
A 65y old man presented to the
A&E with
 Attacks of colicky abdominal pain associated with
repeated vomiting and absolute constipation
 On examination, the abdomen is hugely distended
showing visible peristalsis with a long midline scar of
previous laparotomy
 The patient is tachycardic and dehydrated with
disturbed electrolytes
 AXR showed dilated small bowel loops
Definition
 The normal flow of
intestinal contents is
interrupted
Classifications of IO:
 Type of obstruction
 Mechanical (dynamic)
 Functional (adynamic)
 Time of presentation
 Acute
 Chronic
 Extent of obstruction
 Partial
 Complete
 Pathological nature of
obstruction
 Simple mechanical
 Strangulation
 Paralytic ileus
Mechanical (dynamic)
 Peristalsis is working
against a mechanical
obstruction
Extramural Intramural Intraluminal
• Adhesions
• Hernias
• Volvulus
• Tumours
• Congenital
anomalies 
atresia
• Intussuscepti
on
• Neoplasm
• Stricture
• Gallstone
ileus
• Fecal
impaction
• FB  bezor
• Traumatic
intraluminal
hematoma
Functional (adynamic)
 Atony of the intestine
with loss of normal
peristalsis in the absence
of mechanical
obstruction e.g. ileus
 Peristalsis present in a
non-propulsive form e.g.
MVO or pseudo-
obstruction
Commonest causes
 Strangulated hernias 
most common cause of
SBO in children
 Adhesions  most
common cause of SBO in
adults
 Cancer colon  most
common cause of LBO
Cardinal clinical features of acute
obstruction
 Abdominal pain
 Distension
 Vomiting
 Absolute constipation
Features according to level of obstruction
 High small bowel obstruction
 Vomiting  early & profuse 
rapid dehydration
 Distension  minimal
 Few fluid levels on AXR
 Low small bowel obstruction
 Pain  predominant
 Central distension.
 Vomiting  delayed.
 Multiple central fluid levels on
AXR
 Large bowel obstruction
 Distension  early + marked
 Pain  mild
 Vomiting & dehydration  late
 The proximal colon and
caecum are distended on AXR
General examination
Signs of dehydration
 Tachycardia
 Oliguria
 Dry tongue
 Hypotension
Abdominal examination
 Inspection
 Distension
 Scars
 Visible peristalsis
 Hernia
 Palpation
 Mass
 Tenderness
 Percussion
 Tenderness
 Hyper-resonance
 Auscultation
 ↑Bowel sounds
 Silent
 Rectal examination
 Empty
 Blood
 Fecal mass
Clinical features of strangulation
 Constant pain
 Tenderness + rebound
tenderness
 Rigidity
 Shock
 Toxicity
Investigations
 CBC
 Urea & creatinine
 S. electrolytes
 AXR
 Ultrasound
 CT scan
Radiological features of obstruction
 SBO
 Straight segments that are generally
central and lie transversely
 No gas is seen in the colon
 Jejunum  Valvulae conniventes
 Pass across the width of the bowel
 Regularly spaced, giving a
‘concertina’ or ladder effect
 Ileum  featureless
 Caecum – a distended caecum  a
rounded gas shadow in the RIF
 Large bowel, except for the caecum 
haustral folds (unlike VC)
 Spaced irregularly
 Do not cross the whole diameter of
the bowel
 Do not have indentations placed
opposite one another
MCQ
The following features are present in SBO except
 Distension
 Vomiting
 Dehydration
 Diarrhoea
 Hyperkalemia
 Dilated bowel loops on AXR
Preoperative management (drip & suck)
 NGT
 Free drainage + 4-hourly
aspiration /continuous
or intermittent suction.
 Decompression
proximal to the
obstruction
 Reduce risk of
aspiration
 IV fluids
 Antibiotics
 Urinary catheter
Surgery
Indications for early surgical
intervention
Principles of surgical
intervention for obstruction
 Obstructed or strangulated external
hernia
 Internal intestinal strangulation
 Acute obstruction
Management of:
 The segment at the site of
obstruction
 The distended proximal bowel
 The underlying cause of obstruction
Differentiation between viable and non-viable
intestine
Viable Non-
viable
Circulation • Dark color
becomes
lighter
• Mesentery
bleeds if
pricked
• Dark colour
remains
• No bleeding
if mesentery
is pricked
Peritoneum Shiny Dull & lustreless
Intestinal
musculature
• Firm
• Pressure
rings may or
may not
disappear
• Peristalsis
may be
observed
• Flabby, thin
& friable
• Pressure
rings persist
• No
peristalsis
Conservative treatment
 Adhesions  drip & suck
 Ileo-caecal
intussusception 
hydrostatic effect of
barium enema
 Sigmoid volvulus 
rectal tube
 Fecal impaction 
enema
MCQ
In the treatment of dynamic bowel obstruction
 Conservative management is the only option
 Non-viable bowel should be resected during the
operation
 Volvulus can be treated conservatively
 Drip and suck is tried in adhesive intestinal
obstruction
 Fecal impaction should be treated operatively
 Intussusception should be treated operatively
A 10 months old child presented to
the A&E with
 Episodes of screaming and drawing up of the legs lasting
for 1-2min and recurs every 15-20min
 The condition is associated with Pallor + sweating +
retching
 Vomiting which is bile-stained
 Initially, the passage of stool was normal, but now the
patient is passing blood and mucus PR
 Hx of recent respiratory tract infection
 On examination, the right iliac fossa felt empty with
Sausage-shaped mass palpated in the right hypochondrium
Most commonly in children
Definition
 Segment of the gut
(intussusceptun) becomes
invaginated within an
immediately adjacent
segment (intussuscepiens)
 Usually proximal into
distal
 An example of a
strangulating obstruction
as the blood supply of the
inner layer is usually
impaired
Aetiology
 Peak between 5-10 months
 ~90% idiopathic  ?
• Upper respiratory tract
infection
• gastroenteritis
 >2 years  a pathological
lead point in 1/3
 Adult  lead point usually
a polyp (e.g. Peutz–Jeghers
syndrome), a submucosal
lipoma or other tumour.
Types
 Ileo-ileal
 Ileo-colic (ileo-caecal)
In most children
 Ileo-ileo-colic
 Colo-colic  common in
adults
Symptoms
Early
 Intermittent colicky pain
 Screaming and drawing up
of the legs
 Pallor + sweating +
retching
 Vomiting
 Episodes 1-2min every 15-
20min
 Redcurrant jelly stool
Late
 Symptoms of peritonitis &
IO
Signs
 Examination between episodes of
colic
 Abdomen is not initially distended
 Emptiness in the right iliac fossa
(sign of Dance)
 Sausage-shaped mass
 PR
 Blood-stained mucus
 Apex palpable / protrude from the
anus  in extensive ileocolic or
colocolic
 Unrelieved 
 Progressive dehydration
 Abdominal distension
 Gangrene  peritonitis
Imaging in intussusception
 AXR 
 IO
 Absent caecal gas in
ileocolic
 Soft tissue opacity in
children
 Barium enema  the claw
sign in ileocolic I
 US  high sensitivity in
children  typical
doughnut appearance of
concentric rings in
transverse section
 CT  in equivocal cases
Pneumatic or hydrostatic reduction
 Successful 
 free reflux of air or barium into
the small bowel
 resolution of symptoms &
signs
 Contraindicated if
 Signs of peritonitis or
perforation
 Known pathological lead point
 Profound shock
 >70% can be reduced non-
operatively
 10% recurrence
Surgery
 Radiological reduction failed
or contraindicated
 Transverse right-sided
abdominal incision
 Gently compress the most distal
part of the intussusception
toward its origin
 The last part of the reduction is
the most difficult
 Resection & 1ry anastomosis 
 Irreducible intussusception
 Infarction
 Lead point
MCQ
Intussusception
 Most common in children
 Always secondary to intestinal pathology, e.g. polyp,
Meckel’s diverticulum in infants
 Ileo-colic is the least common
 Can lead to an ischaemic segment
 Pneumatic reduction is usually successful in children
 Laparotomy is always required
A 70y old black male present to
the A&E with
 Recurrent attacks of abdominal pain associated with
absolute constipation
 The patient complained of nausea but no vomiting is
experienced
 On examination, the abdomen is distended hugely,
tympanitic on percussion with exaggerated bowel
sounds
 AXR showed the coffee-bean sign
Definition
 Twisting or axial rotation of a
portion of bowel around its
mesentery
 When complete it forms a closed
loop of obstruction with resultant
ischaemia secondary to vascular
occlusion
 May involve
 Sigmoid colon  commonest in
adults
 Small intestine
 Caecum
 Stomach
 Neonatal midgut volvulus
secondary to midgut malrotation is
life-threatening
Sigmoid volvulus
 Predisposing factors 
 Symptoms
 Large bowel obstruction
 initially intermittent
 Abdominal distension 
early & progressive sign
 Hiccough & retching
 Vomiting occurs late
 Constipation is absolute
 In elderly  chronic form
AXR  Coffee-bean sign or Omega sign Ὠ
Barium study
CT Scan
Treatment
 Flexible sigmoidoscopy or
rigid sigmoidoscopy +
flatus tube
 Laparotomy
 Untwisting of the loop +
per anum decompression
 Viable  fixation of the
sigmoid colon to the
posterior abdominal wall
 inexperienced hands
 Resection  preferable
Hartmann’s procedure +
subsequent re-
anastomosis
MCQ
Volvulus
 May involve the sigmoid colon only
 Neonatal midgut volvulus secondary to midgut
malrotation is life-threatening
 The commonest spontaneous type in adults is caecal
volvulus
 Sigmoid volvulus can be relieved by decompression per
anum
 Surgery is required to prevent or relieve ischaemia
A 50y old lady presented to the
A&E with
 Abdominal colicky pain
 Nausea and vomiting
 Absolute constipation
 Distension
 Tachycardia
 Dry tongue
 History of repeated laparotomies, the last one was rwo
years ago for perforated DU
Most common cause of intestinal obstruction in developed
countries
Common causes of intra-abdominal
adhesions
1. Ischaemic areas • Sites of
anastomoses
• Vascular occlusion
2. Foreign material Talc, gauze, silk
3. Infection Peritonitis, T.B.
4. Inflammatory
conditions
Crohn’s disease
5. Radiation
enteritis
Bands
 Congenital, e.g.
obliterated vitello-
intestinal duct
 String band following
previous bacterial
peritonitis
 Portion of greater
omentum, usually
adherent to the parietes
Prevention of adhesions
Factors that may limit
adhesion formation include:
 Good surgical technique
 Washing of the peritoneal
cavity with saline to
remove clots, etc.
 Minimising contact with
gauze
 Covering anastomosis and
raw peritoneal surfaces
Treatment of adhesive obstruction
 Initially treat conservatively
provided there are no signs of
strangulation; should rarely
continue conservative
treatment for longer than 72
hours
 At operation, divide only the
causative adhesion(s) and
limit dissection
 Cover serosal tears
 Invaginate (or resect) areas of
doubtful viability
 Laparoscopic adhesiolysis 
in chronic cases
MCQ
Prevention of intraoperative adhesions can be
done by
 Good surgical technique
 Copious irrigation with saline
 Curetting the bowel with gauze
 Excising omentum
 Avoid using powdered gloves
The ward nurse asked you to review a
postoperative patient complaining of
 Abdominal distension and absolute constipation
 The patient vomited twice
 No abdominal pain is felt
 On examination, abdominal tenderness is mild and no
bowel sounds are heard on auscultation
 AXR  showed dilated small bowel loops with
multiple fluid levels
Definition
 Failure of transmission of
peristaltic waves secondary
to neuromuscular failure
i.e. in the myenteric
(Auerbach’s) and
submucous (Meissner’s)
plexuses
  accumulation of fluid
and gas  distension,
vomiting, absence of bowel
sounds & absolute
constipation
Causes
 Postoperative: a variable duration
of 24–72
 Intra-abdominal sepsis: localised
or generalised ileus
 Reflex ileus: following
 # Spine or ribs
 Retroperitoneal hge
 Plaster jacket
 Metabolic:
 Uraemia
 Hypokalaemia
 Diabetic ketoacidosis
 Drugs in high doses:
 Anticholinergics e.g. probanthine
 Tricyclic antidepressants
Clinical features
 72 hours after laparotomy:
 no bowel sounds
 flatus.
 Abdominal distension 
more marked &
tympanitic.
 Pain is not a feature
 Vomiting  if no NGT
 AXR  gas-filled loops of
intestine + multiple fluid
levels
Prevention
 Correction of biochemical
disturbances
 Gentle handling of bowel
 Nasogastric suction
 Restriction of oral intake
 Electrolyte balance
 Enhanced recovery
programme with early
introduction of fluids and
solids
Treatment
 1ry cause must be
removed
 Drip & Suck
 Rarely, in resistant
cases:
 Adrenergic blocking
agent + cholinergic
stimulation, e.g.
neostigmine (the
Catchpole regimen)
 Laparotomy  to
exclude a hidden cause
& facilitate bowel
decompression
MCQ
Paralytic ileus
 Is functional and not mechanical
 Usually postoperative
 Pain is predominant
 Distension is present
 Bowel sounds are present
 Is due to neuromuscular failure
 Usually treated surgically
You was asked to review a patient with
known retroperitoneal sarcoma
showing
 Massive abdominal distension and respiratory distress
 Abdominal pain, nausea and vomiting
 Tympanitic abdomen and sluggish intestinal sounds
 AXR showed dilated colon from the caecum to the
splenic flexure
 Single-contrast water-soluble barium enema showed no
mechanical cause of LBO
Definition
 Obstruction, usually of
the colon, that occurs in
the absence of a
mechanical cause or
acute intra-abdominal
disease
Factors associated with pseudo-
obstruction
Idiopathic
 Metabolic
 Diabetes: intermittent porphyria
 Acute hypokalaemia
 Uraemia
 Myxodoema
 Severe trauma (especially to the lumbar spine and
pelvis)
 Shock
 Burns
 Myocardial infarction
 Stroke
Septicaemia
 Retroperitoneal irritation
 Blood
 Urine
 Enzymes (pancreatitis)
 Tumour
 Drugs
 Tricyclic antidepressants
 Phenothiazines
 Laxatives
 Secondary gastrointestinal involvement
 Scleroderma
 Chagas’ disease
Colonic pseudo-obstruction
 Acute (Ogilvie’s syndrome) or
chronic
 AXR  colonic obstruction +
marked caecal distension 
??Caecal perforation
 Absence of a mechanical cause
requires urgent confirmation by
 Colonoscopy
 single-contrast water-soluble
barium enema
 CT
 Colonoscopic decompression 
recur in 25%  further colonoscopy
+ flatus tube
 Colonoscopy fails or unavailable 
 Tube caecostomy
 Subtotal colectomy and ileorectal
anastomosis.
MCQ
Colonic pseudo-obstruction
 Is called Ogilvie’s syndrome in chronic cases
 AXR shows dilated large bowel
 Single-contrast water-soluble barium enema is used to
exclude mechanical cause
 Colonoscopy is used for colonic decompression
 Surgery is not an option for treatment
01.abdominal swelling 1

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01.abdominal swelling 1

  • 2. A 65y old man presented to the A&E with  Attacks of colicky abdominal pain associated with repeated vomiting and absolute constipation  On examination, the abdomen is hugely distended showing visible peristalsis with a long midline scar of previous laparotomy  The patient is tachycardic and dehydrated with disturbed electrolytes  AXR showed dilated small bowel loops
  • 3.
  • 4. Definition  The normal flow of intestinal contents is interrupted
  • 5. Classifications of IO:  Type of obstruction  Mechanical (dynamic)  Functional (adynamic)  Time of presentation  Acute  Chronic  Extent of obstruction  Partial  Complete  Pathological nature of obstruction  Simple mechanical  Strangulation  Paralytic ileus
  • 6. Mechanical (dynamic)  Peristalsis is working against a mechanical obstruction Extramural Intramural Intraluminal • Adhesions • Hernias • Volvulus • Tumours • Congenital anomalies  atresia • Intussuscepti on • Neoplasm • Stricture • Gallstone ileus • Fecal impaction • FB  bezor • Traumatic intraluminal hematoma
  • 7. Functional (adynamic)  Atony of the intestine with loss of normal peristalsis in the absence of mechanical obstruction e.g. ileus  Peristalsis present in a non-propulsive form e.g. MVO or pseudo- obstruction
  • 8. Commonest causes  Strangulated hernias  most common cause of SBO in children  Adhesions  most common cause of SBO in adults  Cancer colon  most common cause of LBO
  • 9. Cardinal clinical features of acute obstruction  Abdominal pain  Distension  Vomiting  Absolute constipation
  • 10. Features according to level of obstruction  High small bowel obstruction  Vomiting  early & profuse  rapid dehydration  Distension  minimal  Few fluid levels on AXR  Low small bowel obstruction  Pain  predominant  Central distension.  Vomiting  delayed.  Multiple central fluid levels on AXR  Large bowel obstruction  Distension  early + marked  Pain  mild  Vomiting & dehydration  late  The proximal colon and caecum are distended on AXR
  • 11. General examination Signs of dehydration  Tachycardia  Oliguria  Dry tongue  Hypotension
  • 12. Abdominal examination  Inspection  Distension  Scars  Visible peristalsis  Hernia  Palpation  Mass  Tenderness  Percussion  Tenderness  Hyper-resonance  Auscultation  ↑Bowel sounds  Silent  Rectal examination  Empty  Blood  Fecal mass
  • 13. Clinical features of strangulation  Constant pain  Tenderness + rebound tenderness  Rigidity  Shock  Toxicity
  • 14. Investigations  CBC  Urea & creatinine  S. electrolytes  AXR  Ultrasound  CT scan
  • 15. Radiological features of obstruction  SBO  Straight segments that are generally central and lie transversely  No gas is seen in the colon  Jejunum  Valvulae conniventes  Pass across the width of the bowel  Regularly spaced, giving a ‘concertina’ or ladder effect  Ileum  featureless  Caecum – a distended caecum  a rounded gas shadow in the RIF  Large bowel, except for the caecum  haustral folds (unlike VC)  Spaced irregularly  Do not cross the whole diameter of the bowel  Do not have indentations placed opposite one another
  • 16. MCQ The following features are present in SBO except  Distension  Vomiting  Dehydration  Diarrhoea  Hyperkalemia  Dilated bowel loops on AXR
  • 17. Preoperative management (drip & suck)  NGT  Free drainage + 4-hourly aspiration /continuous or intermittent suction.  Decompression proximal to the obstruction  Reduce risk of aspiration  IV fluids  Antibiotics  Urinary catheter
  • 18. Surgery Indications for early surgical intervention Principles of surgical intervention for obstruction  Obstructed or strangulated external hernia  Internal intestinal strangulation  Acute obstruction Management of:  The segment at the site of obstruction  The distended proximal bowel  The underlying cause of obstruction
  • 19. Differentiation between viable and non-viable intestine Viable Non- viable Circulation • Dark color becomes lighter • Mesentery bleeds if pricked • Dark colour remains • No bleeding if mesentery is pricked Peritoneum Shiny Dull & lustreless Intestinal musculature • Firm • Pressure rings may or may not disappear • Peristalsis may be observed • Flabby, thin & friable • Pressure rings persist • No peristalsis
  • 20. Conservative treatment  Adhesions  drip & suck  Ileo-caecal intussusception  hydrostatic effect of barium enema  Sigmoid volvulus  rectal tube  Fecal impaction  enema
  • 21. MCQ In the treatment of dynamic bowel obstruction  Conservative management is the only option  Non-viable bowel should be resected during the operation  Volvulus can be treated conservatively  Drip and suck is tried in adhesive intestinal obstruction  Fecal impaction should be treated operatively  Intussusception should be treated operatively
  • 22. A 10 months old child presented to the A&E with  Episodes of screaming and drawing up of the legs lasting for 1-2min and recurs every 15-20min  The condition is associated with Pallor + sweating + retching  Vomiting which is bile-stained  Initially, the passage of stool was normal, but now the patient is passing blood and mucus PR  Hx of recent respiratory tract infection  On examination, the right iliac fossa felt empty with Sausage-shaped mass palpated in the right hypochondrium
  • 23. Most commonly in children
  • 24. Definition  Segment of the gut (intussusceptun) becomes invaginated within an immediately adjacent segment (intussuscepiens)  Usually proximal into distal  An example of a strangulating obstruction as the blood supply of the inner layer is usually impaired
  • 25. Aetiology  Peak between 5-10 months  ~90% idiopathic  ? • Upper respiratory tract infection • gastroenteritis  >2 years  a pathological lead point in 1/3  Adult  lead point usually a polyp (e.g. Peutz–Jeghers syndrome), a submucosal lipoma or other tumour.
  • 26. Types  Ileo-ileal  Ileo-colic (ileo-caecal) In most children  Ileo-ileo-colic  Colo-colic  common in adults
  • 27. Symptoms Early  Intermittent colicky pain  Screaming and drawing up of the legs  Pallor + sweating + retching  Vomiting  Episodes 1-2min every 15- 20min  Redcurrant jelly stool Late  Symptoms of peritonitis & IO
  • 28. Signs  Examination between episodes of colic  Abdomen is not initially distended  Emptiness in the right iliac fossa (sign of Dance)  Sausage-shaped mass  PR  Blood-stained mucus  Apex palpable / protrude from the anus  in extensive ileocolic or colocolic  Unrelieved   Progressive dehydration  Abdominal distension  Gangrene  peritonitis
  • 29. Imaging in intussusception  AXR   IO  Absent caecal gas in ileocolic  Soft tissue opacity in children  Barium enema  the claw sign in ileocolic I  US  high sensitivity in children  typical doughnut appearance of concentric rings in transverse section  CT  in equivocal cases
  • 30. Pneumatic or hydrostatic reduction  Successful   free reflux of air or barium into the small bowel  resolution of symptoms & signs  Contraindicated if  Signs of peritonitis or perforation  Known pathological lead point  Profound shock  >70% can be reduced non- operatively  10% recurrence
  • 31. Surgery  Radiological reduction failed or contraindicated  Transverse right-sided abdominal incision  Gently compress the most distal part of the intussusception toward its origin  The last part of the reduction is the most difficult  Resection & 1ry anastomosis   Irreducible intussusception  Infarction  Lead point
  • 32. MCQ Intussusception  Most common in children  Always secondary to intestinal pathology, e.g. polyp, Meckel’s diverticulum in infants  Ileo-colic is the least common  Can lead to an ischaemic segment  Pneumatic reduction is usually successful in children  Laparotomy is always required
  • 33. A 70y old black male present to the A&E with  Recurrent attacks of abdominal pain associated with absolute constipation  The patient complained of nausea but no vomiting is experienced  On examination, the abdomen is distended hugely, tympanitic on percussion with exaggerated bowel sounds  AXR showed the coffee-bean sign
  • 34.
  • 35. Definition  Twisting or axial rotation of a portion of bowel around its mesentery  When complete it forms a closed loop of obstruction with resultant ischaemia secondary to vascular occlusion  May involve  Sigmoid colon  commonest in adults  Small intestine  Caecum  Stomach  Neonatal midgut volvulus secondary to midgut malrotation is life-threatening
  • 36. Sigmoid volvulus  Predisposing factors   Symptoms  Large bowel obstruction  initially intermittent  Abdominal distension  early & progressive sign  Hiccough & retching  Vomiting occurs late  Constipation is absolute  In elderly  chronic form
  • 37. AXR  Coffee-bean sign or Omega sign Ὠ
  • 40. Treatment  Flexible sigmoidoscopy or rigid sigmoidoscopy + flatus tube  Laparotomy  Untwisting of the loop + per anum decompression  Viable  fixation of the sigmoid colon to the posterior abdominal wall  inexperienced hands  Resection  preferable Hartmann’s procedure + subsequent re- anastomosis
  • 41. MCQ Volvulus  May involve the sigmoid colon only  Neonatal midgut volvulus secondary to midgut malrotation is life-threatening  The commonest spontaneous type in adults is caecal volvulus  Sigmoid volvulus can be relieved by decompression per anum  Surgery is required to prevent or relieve ischaemia
  • 42. A 50y old lady presented to the A&E with  Abdominal colicky pain  Nausea and vomiting  Absolute constipation  Distension  Tachycardia  Dry tongue  History of repeated laparotomies, the last one was rwo years ago for perforated DU
  • 43. Most common cause of intestinal obstruction in developed countries
  • 44. Common causes of intra-abdominal adhesions 1. Ischaemic areas • Sites of anastomoses • Vascular occlusion 2. Foreign material Talc, gauze, silk 3. Infection Peritonitis, T.B. 4. Inflammatory conditions Crohn’s disease 5. Radiation enteritis
  • 45. Bands  Congenital, e.g. obliterated vitello- intestinal duct  String band following previous bacterial peritonitis  Portion of greater omentum, usually adherent to the parietes
  • 46. Prevention of adhesions Factors that may limit adhesion formation include:  Good surgical technique  Washing of the peritoneal cavity with saline to remove clots, etc.  Minimising contact with gauze  Covering anastomosis and raw peritoneal surfaces
  • 47. Treatment of adhesive obstruction  Initially treat conservatively provided there are no signs of strangulation; should rarely continue conservative treatment for longer than 72 hours  At operation, divide only the causative adhesion(s) and limit dissection  Cover serosal tears  Invaginate (or resect) areas of doubtful viability  Laparoscopic adhesiolysis  in chronic cases
  • 48. MCQ Prevention of intraoperative adhesions can be done by  Good surgical technique  Copious irrigation with saline  Curetting the bowel with gauze  Excising omentum  Avoid using powdered gloves
  • 49. The ward nurse asked you to review a postoperative patient complaining of  Abdominal distension and absolute constipation  The patient vomited twice  No abdominal pain is felt  On examination, abdominal tenderness is mild and no bowel sounds are heard on auscultation  AXR  showed dilated small bowel loops with multiple fluid levels
  • 50.
  • 51. Definition  Failure of transmission of peristaltic waves secondary to neuromuscular failure i.e. in the myenteric (Auerbach’s) and submucous (Meissner’s) plexuses   accumulation of fluid and gas  distension, vomiting, absence of bowel sounds & absolute constipation
  • 52. Causes  Postoperative: a variable duration of 24–72  Intra-abdominal sepsis: localised or generalised ileus  Reflex ileus: following  # Spine or ribs  Retroperitoneal hge  Plaster jacket  Metabolic:  Uraemia  Hypokalaemia  Diabetic ketoacidosis  Drugs in high doses:  Anticholinergics e.g. probanthine  Tricyclic antidepressants
  • 53. Clinical features  72 hours after laparotomy:  no bowel sounds  flatus.  Abdominal distension  more marked & tympanitic.  Pain is not a feature  Vomiting  if no NGT  AXR  gas-filled loops of intestine + multiple fluid levels
  • 54. Prevention  Correction of biochemical disturbances  Gentle handling of bowel  Nasogastric suction  Restriction of oral intake  Electrolyte balance  Enhanced recovery programme with early introduction of fluids and solids
  • 55. Treatment  1ry cause must be removed  Drip & Suck  Rarely, in resistant cases:  Adrenergic blocking agent + cholinergic stimulation, e.g. neostigmine (the Catchpole regimen)  Laparotomy  to exclude a hidden cause & facilitate bowel decompression
  • 56. MCQ Paralytic ileus  Is functional and not mechanical  Usually postoperative  Pain is predominant  Distension is present  Bowel sounds are present  Is due to neuromuscular failure  Usually treated surgically
  • 57. You was asked to review a patient with known retroperitoneal sarcoma showing  Massive abdominal distension and respiratory distress  Abdominal pain, nausea and vomiting  Tympanitic abdomen and sluggish intestinal sounds  AXR showed dilated colon from the caecum to the splenic flexure  Single-contrast water-soluble barium enema showed no mechanical cause of LBO
  • 58.
  • 59. Definition  Obstruction, usually of the colon, that occurs in the absence of a mechanical cause or acute intra-abdominal disease
  • 60. Factors associated with pseudo- obstruction Idiopathic  Metabolic  Diabetes: intermittent porphyria  Acute hypokalaemia  Uraemia  Myxodoema  Severe trauma (especially to the lumbar spine and pelvis)  Shock  Burns  Myocardial infarction  Stroke Septicaemia  Retroperitoneal irritation  Blood  Urine  Enzymes (pancreatitis)  Tumour  Drugs  Tricyclic antidepressants  Phenothiazines  Laxatives  Secondary gastrointestinal involvement  Scleroderma  Chagas’ disease
  • 61. Colonic pseudo-obstruction  Acute (Ogilvie’s syndrome) or chronic  AXR  colonic obstruction + marked caecal distension  ??Caecal perforation  Absence of a mechanical cause requires urgent confirmation by  Colonoscopy  single-contrast water-soluble barium enema  CT  Colonoscopic decompression  recur in 25%  further colonoscopy + flatus tube  Colonoscopy fails or unavailable   Tube caecostomy  Subtotal colectomy and ileorectal anastomosis.
  • 62. MCQ Colonic pseudo-obstruction  Is called Ogilvie’s syndrome in chronic cases  AXR shows dilated large bowel  Single-contrast water-soluble barium enema is used to exclude mechanical cause  Colonoscopy is used for colonic decompression  Surgery is not an option for treatment

Editor's Notes

  1. IntussusceptionMay be an intrinsic lesion serving as a lead point
  2. It is vital to distinguish strangulating from non-strangulating intestinal obstruction because the former is a surgical emergency Symptoms usually commence suddenly and recur regularly Generalised tenderness and the presence of rigidity are indicative of the need for early laparotomy. In cases of intestinal obstruction in which pain persists despite conservative management, even in the absence of the above signs, strangulation should be diagnosed When strangulation occurs in an external hernia, the lump is tense, tender and irreducible, there is no expansile cough impulse and it has recently increased in size
  3. US  intususception CT  Sensitivity 80-90% IO
  4. The degree of ischaemia is dependent on the tightness of the invagination, which is usually greatest as it passes through the ileocaecal valve.
  5. Peak between 5-10 months ~90% idiopathic  Upper respiratory tract infection or gastroenteritis may precede the condition  Hyperplasia of Peyer’s patches in the terminal ileum
  6. Episodes of screaming and drawing up of the legs in a previously well male infant. The attacks last for a few minutes and recur repeatedly. During attacks the child appears pale, whereas between episodes he may be listless. Vomiting may or may not occur at the outset but becomes conspicuous and bile-stained with time. Initially, the passage of stool may be normal, whereas, later, blood and mucus are evacuated – the ‘redcurrant jelly’ stool.
  7. Signe de Dance Rarely, natural cure may occur as a result of sloughing of the intussusception
  8. After reduction, the terminal part of the small bowel and the appendix will be seen to be bruised and oedematous. The viability of the whole bowel should be checked carefully
  9. intra-abdominal sepsis may give rise to localised or generalised ileus. Resultant adhesions may contribute a mechanical element to the initial neurogenic aetiology
  10. Caecal perforation is a well-recognised complication