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 ‫الرحيم‬ ‫الرحمن‬ ‫هللا‬ ‫بسم‬
 Department of surgery
 Unit:-
 Mr Dhirar
 Mr Alhassan
 Mr Haidar
 Tettorial tittle:
 Intestinal obstruction
 Presented by:
 Ola abdalsalam
CONTENT
 Surgical anatomy
 Classifications
 C/F
 Management
 Specific types of intestinal obstruction
SMALL BOWEL
 6ms length
 Duodenum 25 cm
 Jejunum :proximal 40%
 Ileum:distal 60%
 Blood supply: superior mesentric artery
 Venous drainage: superior mesenteric vein
 Nerve:sympT9-T11 parasymp:vagus
LARGE BOWEL
 1.5 meter
 Blood Supply
♦ Iliocolic, right colic, and middle colic
arteries branches of superior mesenteric
artery supply the colon from caecum to
splenic flexure.
♦ Left colic, sigmoid, superior rectal arteries
branches of inferior mesenteric artery supply
the descending and sigmoid colon
CLACCIFICATIONS
Class 1:
 Dynamic adynamic
 Class 2:
 Small bowel large bowel
 Class 3:
 Acute Chronic acute on chronic
Class 4:
 Aquired congenital
DYNAMIC
extramural mural intramural
adhesions T.B Gall stone
hernia Crohn disease Round worm
intusseception malignancy Inspissated feaces
volvulus Meconium ileus
ADYNAMIC
 Postoperative period
 Electrolyte imbalance
 Spinal injuries
 Uraemia paralytic ileus
 Diabetes mellitus
 Mesenteric ischaemia
 Pseudo-obstruction
Congenital causes:-
 Congenital megacolon
 Duodenal atresia
 Intestinal atresia
 Band and adhesions
 Malrotation
 Volvulus neonatorum
Dynamic intestinal obstruction
PATHOPHYIOLOGY
 Changes proximal to the bowel obstruction:
Intestinal obstruction
↓
Increased peristalsis
↓
Becomes vigorous
↓
Obstruction not relieved
↓
Peristalsis ceases.
↓
Flaccid, paralysed, dilated bowel
 Fluid collects just proximal to the obstruction
which is derived from saliva, stomach, pancreas
and intestine
 Defective absorption, decreased fluid intake,
loss of fluid by vomiting, sequestration of fluid
into the bowel lumen dehydration and
electrolyte disturbance
 Increased bacterial colony in the bowel due
to altered luminal content and environment
→ multiplication →toxins → further mucosa
damage → translocation of bacteria across
mucosa into submucosa and also absorption
of bacterial into the circulation→bacteraemia
 Changes at the site of the obstruction:
Initially venous return is impaired.
↓
Congestion, oedema of bowel wall occurs which turns
purple.
↓
Later this jeopardizes the arterial supply.
↓
Loss of shineness, blackish discolouration, loss of peristalsis.
↓
Gangrene.
↓
Perforation occurs.
↓
Bacteria and toxins migrate into the peritoneum.
↓
Peritonitis
 Bowel distal to the obstruction is inactive
and collapsed
CLOSED LOOP OBSTRUCTION
CARDINAL FEATURES
 Abdominal pain
 Vomiting
 Distension
 Absolute conistipation
Proximal s.bowel Distal s.bowel Large bowel
Severe vomiting,
dehydration, no or
less distension,
colicky pain
Central distension,
vomiting,
dehydration
central abdominal pain
Constipation,
distension—
early;
Late vomiting less
pain
Vitally:
 Tachycardia
 Tahypnea
 Febrile
 hypotensive
Abdominal examination:
 Tenderness
 Reboud tenderness,guardning,rigidity
 Bowel sound: exaggerated /abscent
 PR: empty dilated rectum
INVESTIGATIONS
 General:
 CBC
 RFT + electrolytes
 ABG
 Coagulation profile
INVESTIGATIONS FOR DIAGNOSIS:
 Erect +supine abdominal x ray:
 Multiple air fluid level
 Dilated loop:-
 Small bowel > 3 cm diameter
 proximal large bowel> 9 cm
 transverse colon > 5.5 cm
 sigmoid colon> 5 cm
 Specific features
 pneumobilia
 CT scan
HAUSTRATION
VALVULAE CONNIVENT
TREATMENT
 NPO
 Fluid and electrolyte management
 NGT
 Catheter
 laprotomy
 Postsurgery Complications:-
♦ Pelvic abscess.
♦ Subphrenic abscess.
♦ Biliary or faecal fistulas.
♦ Burst abdomen.
♦ Bands and adhesions.
♦ Incisional hernias
SPECIAL TYPES
DUODENAL ATRESIA
- It is the commonest site of intestinal atresia
- It is usually a complete stenosis of the second
part
- Defective fusion of foregut and midgut with
failure of recanalization
-Duodenal atresia may be preampullary
(nonbilious vomiting) or postampullary (bilious
vomiting)
 Associated with:
 Down syndrome 30%
 CHD 30%
 ARM 10%
 Polyhydramnios and prematurity 50%
C/F
♦ Jaundice.
♦ Bilious/nonbilious vomiting immediately after
birth
♦ Dehydration. Electrolyte changes are
common.
♦ Growth retardation of newborn due to
deprived nutrition
INVESTIGATIONS
 Plain X-ray shows classic double-bubble sign
with absence of air in the distal part
 U/S will show distended stomach and
proximal duodenum( rail road track)
TREATMENT
 duodenodoudenostomy
INTESTINAL ATRESIA
 Jejunoileal atresia
 Intrauterine mesenteric vascular occlusion
 Commonly:proximal jejunum and distal ileum
 X-ray : triple bubble
 Barium enema: microcolon
 Treatment:
 Resection and anastomosis
 Jejunoplasty(extensive length involved)
MALROTATION
 Stages of normal rotation:
 Stage 1: coelomic cavity cannot
accommodate midgut during this period
protrude into umblical cord as physiological
hernia
 Stage 2: midgut migrate into coelomic
cavity;small bowel in left side;it rotate 270
anticlockwise into rt iliac
fossa;duodenojejunal segment rotate 270
anticlock to reach left of SMA and behind the
colon
 Stage 3: fusion of different parts of mesentry
and posterior peritoneum
 Errors:
 Stage 1: gastroschisis
 Stage 2: non-rotation
 Incomplete rotation
 Reverse rotation
 Hyper-rotation
 Stage 3: mobile caecum and ascending
colon
 Dark blood per rectum
 Erythrema of anterior abdominal wall
 Teatment: ladd operation
MECONIUM ILEUS
 Commonly associated with cystic disease of
pancreas but not necessarily always
 Respiratory dysfunction
 Exocrine pancreatic insufficiency
 High salt in the sweat > 90 mmol/L
 Complications of meconium ileus:
 Intestinal bolus obstruction
 Perforation and peritonitis
 Gangrene, volvulus formation
INVESTIGATIONS
♦ Plain X-ray shows calcified meconium pellets with
multiple air fluid levels which appear as ‘soap-
bubbles’
♦ Vomitus of the patient which does not contain trypsin,
when poured on the exposed X-ray film will not digest
the gelatin of the film
♦ Pilocarpine is injected into skin to stimulate the
sweating and collected sweat (100 µg sweat)is
analysed for sodium and chloride.
♦ Elevated albumin level in meconium
TREATMENT
Nonoperative measures:-
Dissolution through enema can be tried
using gastrografin
Operative measures:-
Bishop-Koop operation in critical pt
 If fit: resection and anastmosis
INTUSSUSCEPTION (ISS)
 It is invagination of one portion (segment) of
bowel into the adjacent segment.
Types
1. Antegrade: Most common.
2. Retrograde
♦ It can be ileo-colic (most common type,
75%), colocolic, ileoiliocolic
♦ It is common in weaning period of a child
(common in male),between the period of 6-9
months
AETIOLOGY
 Change in diet during weaning
 Upper respiratory tract viral infection
 Intestinal polyps
 Submucous lipoma
 Leiomyoma of intestine
 Meckel’s diverticulum
 Carcinoma
♦ Apex is the one which advances;
♦ Intussuscipiens is the one which receives
♦ Intussusceptum
FEATURES
 on/off
 screaming
 Red currant jelly stool
 Palpable mass (85%)
– Sausage shaped smooth, firm mass
– Mass does not move with respiration
– Mobile in all directions
– Resonant
– Mass contracts under the palpating fingers
– Mass appears and disappears
Empty right iliac fossa
INVESTIGATIONS
♦ Barium enema shows typical claw sign
♦ Ultrasound shows target sign or
pseudokidney sign
TREATMENT
 Nonoperative measures:
 Reduction by hydrostatic pressure but
contraindicated in:
 1- doubtful diagnosis
 2-Late cases
 3-abdominal distension/rigidity
 Operative:
 Manual reduction
 Resection and anastamosis
Recurrence rate:
♦ In hydrostatic reduction—10%.
♦ In open manual reduction—2%.
♦ In resection—very less < 1%
VOLVULUS
 Definition
It is the twist (rotation) in the axis of the loop
of the bowel either clockwise or anticlockwise
♦ Sigmoid colon is the commonest site (anticlock wise)—
65%.
♦ Caecal volvulus
Caecum is the second common site(clockwise) 30%
 Caecum will be markedly distended and found in the
centre of the abdomen
 X-ray shows round gas shadow in right iliac region.
CT scan is very useful. Barium enema is also helpful.
Resection and anastomosis (surgery) is the only treatmen
SEGMOID VOLVULUS
 It is common in patients with chronic
constipation with laxative abuse
 Predisosing factors:
Adhesions
Peridiverticulitis
Overloaded redundant pelvic colon
Long pelvic mesocolon
Narrow attachment of sigmoid mesocolon
 More than 180 : luminal obsrtuction
 It requires one and half turn of rotation to
cause vascular obstruction
INVESTIGATIONS FOR DIAGNOSES
1. Plain X-ray:(diagnostic in 70-80%)
Ω sign (omega sign).
Coffee-bean sign.
2. Contrast enema:
Birds beak sign
3. CT scan
TREATMENT
 A flatus tube or Sigmoidoscope
 Laprotomy
PARALYTIC ILEUS
 It is a state in which intestines fail to transmit
peristalsis due to failure of neuromuscular
mechanism
C/F
♦ No passage of flatus.
♦ No bowel sounds.
♦ Marked abdominal distension.
♦ Vomiting of large volume of fluid.
♦ Tachycardia.
♦ Respiratory distress due to pressure over the
diaphragm.
♦ Dull abdominal pain (not colicky).
♦ Features of fluid/protein/electrolyte imbalance
TREATMENT
♦ Nasogastric aspiration.
♦ The primary cause is treated.
♦ IV fluids.
♦ Electrolyte management.
♦ Catheterisation and urine output
measurement
 Measurement of abdominal girth
Decompression of the large bowel can be
tried by inserting a flatus tube per anally
ADHESIONS AND BANDS
 Causes may be classified as:—
 Congenital adhesions
 Ischaemic
 Traumatic
 Irritants
 Inflammatory
TYPES
 Type I—Fibrinous adhesions occur during 5-
10th post-surgical period. It usually gets
resolved completely. It is avascular .
Type II—Fibrous adhesions. Due to lack/poor
blood supply It will persist and precipitate
intestinal obstruction
 1) Pain may get aggravated or relieved on
change ofposture.
 2) Pain in the region of old abdominal scar.
 3) Tenderness is elicited by pressure over the
scar
 Conservative(72 hrs)
 surgical
INTERNAL HERNIA
 Portion of bowel entrapped in one of the
retroperotineal fossae or congenital
mesenteric defect including:
 Foramen of winslow
 Defect in mesentry;transverse
mesocolon;broad ligament
 Diaphragmatic hernia
 Duodenal;ceacal or intersegmoid fossae
 Treatment:
 Division of constricting agent
PSEUDO-OBSTRUCTION
 Obstruction in absence of mechanical cause
or acute intra-abdominal disease
 Acute colonic: Ogilvie syndrome
 Marked ceacal distention/ceacal perforation
 Management:
 Exclude the mechanical causes
 Treat the underlying cause
 I.V neostigmine 1 mg
 decompression

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intestinal obstruction.pptx

  • 1.  ‫الرحيم‬ ‫الرحمن‬ ‫هللا‬ ‫بسم‬  Department of surgery  Unit:-  Mr Dhirar  Mr Alhassan  Mr Haidar  Tettorial tittle:  Intestinal obstruction  Presented by:  Ola abdalsalam
  • 2. CONTENT  Surgical anatomy  Classifications  C/F  Management  Specific types of intestinal obstruction
  • 3. SMALL BOWEL  6ms length  Duodenum 25 cm  Jejunum :proximal 40%  Ileum:distal 60%  Blood supply: superior mesentric artery  Venous drainage: superior mesenteric vein  Nerve:sympT9-T11 parasymp:vagus
  • 4.
  • 5. LARGE BOWEL  1.5 meter  Blood Supply ♦ Iliocolic, right colic, and middle colic arteries branches of superior mesenteric artery supply the colon from caecum to splenic flexure. ♦ Left colic, sigmoid, superior rectal arteries branches of inferior mesenteric artery supply the descending and sigmoid colon
  • 6. CLACCIFICATIONS Class 1:  Dynamic adynamic  Class 2:  Small bowel large bowel  Class 3:  Acute Chronic acute on chronic Class 4:  Aquired congenital
  • 7. DYNAMIC extramural mural intramural adhesions T.B Gall stone hernia Crohn disease Round worm intusseception malignancy Inspissated feaces volvulus Meconium ileus
  • 8. ADYNAMIC  Postoperative period  Electrolyte imbalance  Spinal injuries  Uraemia paralytic ileus  Diabetes mellitus  Mesenteric ischaemia  Pseudo-obstruction
  • 9. Congenital causes:-  Congenital megacolon  Duodenal atresia  Intestinal atresia  Band and adhesions  Malrotation  Volvulus neonatorum
  • 11. PATHOPHYIOLOGY  Changes proximal to the bowel obstruction: Intestinal obstruction ↓ Increased peristalsis ↓ Becomes vigorous ↓ Obstruction not relieved ↓ Peristalsis ceases. ↓ Flaccid, paralysed, dilated bowel
  • 12.  Fluid collects just proximal to the obstruction which is derived from saliva, stomach, pancreas and intestine  Defective absorption, decreased fluid intake, loss of fluid by vomiting, sequestration of fluid into the bowel lumen dehydration and electrolyte disturbance
  • 13.  Increased bacterial colony in the bowel due to altered luminal content and environment → multiplication →toxins → further mucosa damage → translocation of bacteria across mucosa into submucosa and also absorption of bacterial into the circulation→bacteraemia
  • 14.  Changes at the site of the obstruction: Initially venous return is impaired. ↓ Congestion, oedema of bowel wall occurs which turns purple. ↓ Later this jeopardizes the arterial supply. ↓ Loss of shineness, blackish discolouration, loss of peristalsis. ↓ Gangrene. ↓ Perforation occurs. ↓ Bacteria and toxins migrate into the peritoneum. ↓ Peritonitis
  • 15.  Bowel distal to the obstruction is inactive and collapsed
  • 17. CARDINAL FEATURES  Abdominal pain  Vomiting  Distension  Absolute conistipation
  • 18. Proximal s.bowel Distal s.bowel Large bowel Severe vomiting, dehydration, no or less distension, colicky pain Central distension, vomiting, dehydration central abdominal pain Constipation, distension— early; Late vomiting less pain
  • 20. Abdominal examination:  Tenderness  Reboud tenderness,guardning,rigidity  Bowel sound: exaggerated /abscent  PR: empty dilated rectum
  • 21. INVESTIGATIONS  General:  CBC  RFT + electrolytes  ABG  Coagulation profile
  • 22. INVESTIGATIONS FOR DIAGNOSIS:  Erect +supine abdominal x ray:  Multiple air fluid level  Dilated loop:-  Small bowel > 3 cm diameter  proximal large bowel> 9 cm  transverse colon > 5.5 cm  sigmoid colon> 5 cm  Specific features  pneumobilia  CT scan
  • 23.
  • 26. TREATMENT  NPO  Fluid and electrolyte management  NGT  Catheter  laprotomy
  • 27.  Postsurgery Complications:- ♦ Pelvic abscess. ♦ Subphrenic abscess. ♦ Biliary or faecal fistulas. ♦ Burst abdomen. ♦ Bands and adhesions. ♦ Incisional hernias
  • 29. DUODENAL ATRESIA - It is the commonest site of intestinal atresia - It is usually a complete stenosis of the second part - Defective fusion of foregut and midgut with failure of recanalization -Duodenal atresia may be preampullary (nonbilious vomiting) or postampullary (bilious vomiting)
  • 30.
  • 31.  Associated with:  Down syndrome 30%  CHD 30%  ARM 10%  Polyhydramnios and prematurity 50%
  • 32. C/F ♦ Jaundice. ♦ Bilious/nonbilious vomiting immediately after birth ♦ Dehydration. Electrolyte changes are common. ♦ Growth retardation of newborn due to deprived nutrition
  • 33. INVESTIGATIONS  Plain X-ray shows classic double-bubble sign with absence of air in the distal part  U/S will show distended stomach and proximal duodenum( rail road track)
  • 34.
  • 36. INTESTINAL ATRESIA  Jejunoileal atresia  Intrauterine mesenteric vascular occlusion  Commonly:proximal jejunum and distal ileum
  • 37.
  • 38.  X-ray : triple bubble  Barium enema: microcolon  Treatment:  Resection and anastomosis  Jejunoplasty(extensive length involved)
  • 39. MALROTATION  Stages of normal rotation:  Stage 1: coelomic cavity cannot accommodate midgut during this period protrude into umblical cord as physiological hernia  Stage 2: midgut migrate into coelomic cavity;small bowel in left side;it rotate 270 anticlockwise into rt iliac fossa;duodenojejunal segment rotate 270 anticlock to reach left of SMA and behind the colon
  • 40.  Stage 3: fusion of different parts of mesentry and posterior peritoneum
  • 41.  Errors:  Stage 1: gastroschisis  Stage 2: non-rotation  Incomplete rotation  Reverse rotation  Hyper-rotation  Stage 3: mobile caecum and ascending colon
  • 42.  Dark blood per rectum  Erythrema of anterior abdominal wall  Teatment: ladd operation
  • 43. MECONIUM ILEUS  Commonly associated with cystic disease of pancreas but not necessarily always  Respiratory dysfunction  Exocrine pancreatic insufficiency  High salt in the sweat > 90 mmol/L
  • 44.  Complications of meconium ileus:  Intestinal bolus obstruction  Perforation and peritonitis  Gangrene, volvulus formation
  • 45. INVESTIGATIONS ♦ Plain X-ray shows calcified meconium pellets with multiple air fluid levels which appear as ‘soap- bubbles’ ♦ Vomitus of the patient which does not contain trypsin, when poured on the exposed X-ray film will not digest the gelatin of the film ♦ Pilocarpine is injected into skin to stimulate the sweating and collected sweat (100 µg sweat)is analysed for sodium and chloride. ♦ Elevated albumin level in meconium
  • 46. TREATMENT Nonoperative measures:- Dissolution through enema can be tried using gastrografin Operative measures:- Bishop-Koop operation in critical pt  If fit: resection and anastmosis
  • 47. INTUSSUSCEPTION (ISS)  It is invagination of one portion (segment) of bowel into the adjacent segment. Types 1. Antegrade: Most common. 2. Retrograde
  • 48. ♦ It can be ileo-colic (most common type, 75%), colocolic, ileoiliocolic ♦ It is common in weaning period of a child (common in male),between the period of 6-9 months
  • 49. AETIOLOGY  Change in diet during weaning  Upper respiratory tract viral infection  Intestinal polyps  Submucous lipoma  Leiomyoma of intestine  Meckel’s diverticulum  Carcinoma
  • 50. ♦ Apex is the one which advances; ♦ Intussuscipiens is the one which receives ♦ Intussusceptum
  • 51.
  • 52. FEATURES  on/off  screaming  Red currant jelly stool  Palpable mass (85%) – Sausage shaped smooth, firm mass – Mass does not move with respiration – Mobile in all directions – Resonant – Mass contracts under the palpating fingers – Mass appears and disappears Empty right iliac fossa
  • 53. INVESTIGATIONS ♦ Barium enema shows typical claw sign ♦ Ultrasound shows target sign or pseudokidney sign
  • 54.
  • 55. TREATMENT  Nonoperative measures:  Reduction by hydrostatic pressure but contraindicated in:  1- doubtful diagnosis  2-Late cases  3-abdominal distension/rigidity  Operative:  Manual reduction  Resection and anastamosis
  • 56. Recurrence rate: ♦ In hydrostatic reduction—10%. ♦ In open manual reduction—2%. ♦ In resection—very less < 1%
  • 57. VOLVULUS  Definition It is the twist (rotation) in the axis of the loop of the bowel either clockwise or anticlockwise
  • 58. ♦ Sigmoid colon is the commonest site (anticlock wise)— 65%. ♦ Caecal volvulus Caecum is the second common site(clockwise) 30%  Caecum will be markedly distended and found in the centre of the abdomen  X-ray shows round gas shadow in right iliac region. CT scan is very useful. Barium enema is also helpful. Resection and anastomosis (surgery) is the only treatmen
  • 59. SEGMOID VOLVULUS  It is common in patients with chronic constipation with laxative abuse  Predisosing factors: Adhesions Peridiverticulitis Overloaded redundant pelvic colon Long pelvic mesocolon Narrow attachment of sigmoid mesocolon
  • 60.  More than 180 : luminal obsrtuction  It requires one and half turn of rotation to cause vascular obstruction
  • 61. INVESTIGATIONS FOR DIAGNOSES 1. Plain X-ray:(diagnostic in 70-80%) Ω sign (omega sign). Coffee-bean sign. 2. Contrast enema: Birds beak sign 3. CT scan
  • 62.
  • 63. TREATMENT  A flatus tube or Sigmoidoscope  Laprotomy
  • 64. PARALYTIC ILEUS  It is a state in which intestines fail to transmit peristalsis due to failure of neuromuscular mechanism
  • 65. C/F ♦ No passage of flatus. ♦ No bowel sounds. ♦ Marked abdominal distension. ♦ Vomiting of large volume of fluid. ♦ Tachycardia. ♦ Respiratory distress due to pressure over the diaphragm. ♦ Dull abdominal pain (not colicky). ♦ Features of fluid/protein/electrolyte imbalance
  • 66. TREATMENT ♦ Nasogastric aspiration. ♦ The primary cause is treated. ♦ IV fluids. ♦ Electrolyte management. ♦ Catheterisation and urine output measurement
  • 67.  Measurement of abdominal girth Decompression of the large bowel can be tried by inserting a flatus tube per anally
  • 68. ADHESIONS AND BANDS  Causes may be classified as:—  Congenital adhesions  Ischaemic  Traumatic  Irritants  Inflammatory
  • 69. TYPES  Type I—Fibrinous adhesions occur during 5- 10th post-surgical period. It usually gets resolved completely. It is avascular . Type II—Fibrous adhesions. Due to lack/poor blood supply It will persist and precipitate intestinal obstruction
  • 70.  1) Pain may get aggravated or relieved on change ofposture.  2) Pain in the region of old abdominal scar.  3) Tenderness is elicited by pressure over the scar
  • 72. INTERNAL HERNIA  Portion of bowel entrapped in one of the retroperotineal fossae or congenital mesenteric defect including:  Foramen of winslow  Defect in mesentry;transverse mesocolon;broad ligament  Diaphragmatic hernia  Duodenal;ceacal or intersegmoid fossae
  • 73.  Treatment:  Division of constricting agent
  • 74. PSEUDO-OBSTRUCTION  Obstruction in absence of mechanical cause or acute intra-abdominal disease  Acute colonic: Ogilvie syndrome  Marked ceacal distention/ceacal perforation
  • 75.
  • 76.  Management:  Exclude the mechanical causes  Treat the underlying cause  I.V neostigmine 1 mg  decompression