In these slides we will go through the surgical anatomy of the gut,pathophysiology of intestinal obstruction, clinical presentation and management. Also we will discuss specific types of intestinal obstruction.
3. SMALL BOWEL
6ms length
Duodenum 25 cm
Jejunum :proximal 40%
Ileum:distal 60%
Blood supply: superior mesentric artery
Venous drainage: superior mesenteric vein
Nerve:sympT9-T11 parasymp:vagus
4.
5. LARGE BOWEL
1.5 meter
Blood Supply
♦ Iliocolic, right colic, and middle colic
arteries branches of superior mesenteric
artery supply the colon from caecum to
splenic flexure.
♦ Left colic, sigmoid, superior rectal arteries
branches of inferior mesenteric artery supply
the descending and sigmoid colon
6. CLACCIFICATIONS
Class 1:
Dynamic adynamic
Class 2:
Small bowel large bowel
Class 3:
Acute Chronic acute on chronic
Class 4:
Aquired congenital
11. PATHOPHYIOLOGY
Changes proximal to the bowel obstruction:
Intestinal obstruction
↓
Increased peristalsis
↓
Becomes vigorous
↓
Obstruction not relieved
↓
Peristalsis ceases.
↓
Flaccid, paralysed, dilated bowel
12. Fluid collects just proximal to the obstruction
which is derived from saliva, stomach, pancreas
and intestine
Defective absorption, decreased fluid intake,
loss of fluid by vomiting, sequestration of fluid
into the bowel lumen dehydration and
electrolyte disturbance
13. Increased bacterial colony in the bowel due
to altered luminal content and environment
→ multiplication →toxins → further mucosa
damage → translocation of bacteria across
mucosa into submucosa and also absorption
of bacterial into the circulation→bacteraemia
14. Changes at the site of the obstruction:
Initially venous return is impaired.
↓
Congestion, oedema of bowel wall occurs which turns
purple.
↓
Later this jeopardizes the arterial supply.
↓
Loss of shineness, blackish discolouration, loss of peristalsis.
↓
Gangrene.
↓
Perforation occurs.
↓
Bacteria and toxins migrate into the peritoneum.
↓
Peritonitis
15. Bowel distal to the obstruction is inactive
and collapsed
18. Proximal s.bowel Distal s.bowel Large bowel
Severe vomiting,
dehydration, no or
less distension,
colicky pain
Central distension,
vomiting,
dehydration
central abdominal pain
Constipation,
distension—
early;
Late vomiting less
pain
22. INVESTIGATIONS FOR DIAGNOSIS:
Erect +supine abdominal x ray:
Multiple air fluid level
Dilated loop:-
Small bowel > 3 cm diameter
proximal large bowel> 9 cm
transverse colon > 5.5 cm
sigmoid colon> 5 cm
Specific features
pneumobilia
CT scan
29. DUODENAL ATRESIA
- It is the commonest site of intestinal atresia
- It is usually a complete stenosis of the second
part
- Defective fusion of foregut and midgut with
failure of recanalization
-Duodenal atresia may be preampullary
(nonbilious vomiting) or postampullary (bilious
vomiting)
30.
31. Associated with:
Down syndrome 30%
CHD 30%
ARM 10%
Polyhydramnios and prematurity 50%
32. C/F
♦ Jaundice.
♦ Bilious/nonbilious vomiting immediately after
birth
♦ Dehydration. Electrolyte changes are
common.
♦ Growth retardation of newborn due to
deprived nutrition
33. INVESTIGATIONS
Plain X-ray shows classic double-bubble sign
with absence of air in the distal part
U/S will show distended stomach and
proximal duodenum( rail road track)
39. MALROTATION
Stages of normal rotation:
Stage 1: coelomic cavity cannot
accommodate midgut during this period
protrude into umblical cord as physiological
hernia
Stage 2: midgut migrate into coelomic
cavity;small bowel in left side;it rotate 270
anticlockwise into rt iliac
fossa;duodenojejunal segment rotate 270
anticlock to reach left of SMA and behind the
colon
40. Stage 3: fusion of different parts of mesentry
and posterior peritoneum
42. Dark blood per rectum
Erythrema of anterior abdominal wall
Teatment: ladd operation
43. MECONIUM ILEUS
Commonly associated with cystic disease of
pancreas but not necessarily always
Respiratory dysfunction
Exocrine pancreatic insufficiency
High salt in the sweat > 90 mmol/L
44. Complications of meconium ileus:
Intestinal bolus obstruction
Perforation and peritonitis
Gangrene, volvulus formation
45. INVESTIGATIONS
♦ Plain X-ray shows calcified meconium pellets with
multiple air fluid levels which appear as ‘soap-
bubbles’
♦ Vomitus of the patient which does not contain trypsin,
when poured on the exposed X-ray film will not digest
the gelatin of the film
♦ Pilocarpine is injected into skin to stimulate the
sweating and collected sweat (100 µg sweat)is
analysed for sodium and chloride.
♦ Elevated albumin level in meconium
47. INTUSSUSCEPTION (ISS)
It is invagination of one portion (segment) of
bowel into the adjacent segment.
Types
1. Antegrade: Most common.
2. Retrograde
48. ♦ It can be ileo-colic (most common type,
75%), colocolic, ileoiliocolic
♦ It is common in weaning period of a child
(common in male),between the period of 6-9
months
49. AETIOLOGY
Change in diet during weaning
Upper respiratory tract viral infection
Intestinal polyps
Submucous lipoma
Leiomyoma of intestine
Meckel’s diverticulum
Carcinoma
50. ♦ Apex is the one which advances;
♦ Intussuscipiens is the one which receives
♦ Intussusceptum
51.
52. FEATURES
on/off
screaming
Red currant jelly stool
Palpable mass (85%)
– Sausage shaped smooth, firm mass
– Mass does not move with respiration
– Mobile in all directions
– Resonant
– Mass contracts under the palpating fingers
– Mass appears and disappears
Empty right iliac fossa
56. Recurrence rate:
♦ In hydrostatic reduction—10%.
♦ In open manual reduction—2%.
♦ In resection—very less < 1%
57. VOLVULUS
Definition
It is the twist (rotation) in the axis of the loop
of the bowel either clockwise or anticlockwise
58. ♦ Sigmoid colon is the commonest site (anticlock wise)—
65%.
♦ Caecal volvulus
Caecum is the second common site(clockwise) 30%
Caecum will be markedly distended and found in the
centre of the abdomen
X-ray shows round gas shadow in right iliac region.
CT scan is very useful. Barium enema is also helpful.
Resection and anastomosis (surgery) is the only treatmen
59. SEGMOID VOLVULUS
It is common in patients with chronic
constipation with laxative abuse
Predisosing factors:
Adhesions
Peridiverticulitis
Overloaded redundant pelvic colon
Long pelvic mesocolon
Narrow attachment of sigmoid mesocolon
60. More than 180 : luminal obsrtuction
It requires one and half turn of rotation to
cause vascular obstruction
64. PARALYTIC ILEUS
It is a state in which intestines fail to transmit
peristalsis due to failure of neuromuscular
mechanism
65. C/F
♦ No passage of flatus.
♦ No bowel sounds.
♦ Marked abdominal distension.
♦ Vomiting of large volume of fluid.
♦ Tachycardia.
♦ Respiratory distress due to pressure over the
diaphragm.
♦ Dull abdominal pain (not colicky).
♦ Features of fluid/protein/electrolyte imbalance
66. TREATMENT
♦ Nasogastric aspiration.
♦ The primary cause is treated.
♦ IV fluids.
♦ Electrolyte management.
♦ Catheterisation and urine output
measurement
67. Measurement of abdominal girth
Decompression of the large bowel can be
tried by inserting a flatus tube per anally
68. ADHESIONS AND BANDS
Causes may be classified as:—
Congenital adhesions
Ischaemic
Traumatic
Irritants
Inflammatory
69. TYPES
Type I—Fibrinous adhesions occur during 5-
10th post-surgical period. It usually gets
resolved completely. It is avascular .
Type II—Fibrous adhesions. Due to lack/poor
blood supply It will persist and precipitate
intestinal obstruction
70. 1) Pain may get aggravated or relieved on
change ofposture.
2) Pain in the region of old abdominal scar.
3) Tenderness is elicited by pressure over the
scar
72. INTERNAL HERNIA
Portion of bowel entrapped in one of the
retroperotineal fossae or congenital
mesenteric defect including:
Foramen of winslow
Defect in mesentry;transverse
mesocolon;broad ligament
Diaphragmatic hernia
Duodenal;ceacal or intersegmoid fossae