2. Anterior portion of the pharynx is more
enlarged
Constrioctor region is more stabble
Pharyngeal tonsils are more developed and
enlarged….but swelling of it can obstruct
airway
By the age of 6 all the pharyngeal structures are
almost 80 % developes
3. more sensitive and softer …so there is risk of
inflammation
It is funnle shaped in children while in adult it
has cylindrical shape
Vocal cords have concave shape while in adult
they are flat
It is located bit superiorly in children near C2
while in adults around C6.
4. Normal length of trachea is around 57 mm
While normal width is around 20mm
More sensitive to inflammation
C shaped cartilagenous rings are more softer
compared to adult but still it is developed so
that it can make airway open
Obstruction or edema can easily occure
5. Lungs have less tidal volume and alveolies are
less in number so it decrease space for gaseous
exchange
Number of alveoli is almist around 2 millions
Deadspace is higher in infant { 3ml/kg}
Deadspace is the volume of air which is
inhaled that does not take part in the gas
exchange
7. Bluish body (cynosis)
High breathing speed or low
Different sounds during respiration
Common cough
Crying or problem during swallowing
( in tonsillitis)
fever
headache
8. 1- otitis media
2- tonsillitis
3- apnea
4-croup vs epiglottises
5- bronchitis
6-bronchiolitis
7-RAD (asthama)
8- cystic fibrosis
9. Note the ear on the left with clear
tympanic membrane (drum); ear on
the R the drum is bulging and filled
with pus
11. Acute Otitis Media
characterized by abrupt onset, pain, middle ear effusion, and
inflammation.
Note the injected
vessels and altered
shape of cone of
light.
Treatment – through
surgery
12.
13. May be bacterial or viral
Most common bacterial agent: Group A beta-
hemolytic strep
Throat cultures must be done to determine
origin
Older child may develop peritonsillar abscess
14. Treatment is symptomatic
Antibiotics restricted to those with bacterial
infection
Drug of choice: amoxicillin
Surgery (with recurrent infections)
15. Defined as delay of breathing over
20 seconds
Manifestations
Diagnostic tests
Therapeutic Interventions and
Nursing Care
16. Prematurity: most common and may vary
among neonates
Infant apnea: no known cause; r/o seizures,
GERD, hypoglycemia
17. Apnea:
Cessation > 20
seconds
S/S to assess:
Cyanosis
Marked pallor
Hypotonia
bradycardia
Periodic
breathing
Normal
breathing
pattern of NB
but never > 10-
15 seconds
Even though
normal, all
parents are
taught CPR for
their NB
19. Nurse sets parameters for HR according to age
Gentle stimulation of infant
Maintaining a neutral environment
Instruct family with apnea monitors at home
21. Croup
Usual age range: 1-3 yrs
Inspiratory stridor
Harsh cough (barking)
Viral infection; afebrile
Gradual onset, usually at night
Improved with humidity; may
need racemic epi
Treatable at home
Resolves spontaneously
Epiglottitis
Usual age range 3-7 yrs
May have stridor
Caused by **H.influenzae, but
may staph and strep as well
Sudden onset
Sore throat and difficulty
swallowing
May be an emergent situation
Lateral soft tissue of neck xray
Have equipment at bedside
23. Observe for s/s respiratory distress
Assess respiratory rates: >60
Elevated temp ) 101º
The child must NEVER be left alone
NOTHING should be placed in the mouth
(laryngeal spasms could result)
24. Croup
Racemic epi nebulization
Oral dexamethosone in a
single dose
Acetaminophen
Humidified O2 and IVs for
more severe cases
Sedatives are
contraindicated
Epiglottitis
Child kept NPO
IV antibiotics
Antipyretics for fever
Emergency hospitalization
25. Etiology
Inflammation of trachea and major bronchi
Usually viral (Rhino and RSV)
Occur with other conditions; may be confused with
RAD (asthma)
Cough major symptom
Gradual onset of rhinitis
Productive cough (may be purulent) with mucus
Crackles, rhonchi
26. Increase fluids
Assess VS, secretions, respiratory effort
S/S sleep deprivation from cough
Antipyretics for fever
Quiet activities for diversion
27. Etiology
RSV most common pathogen
May acquire from older siblings
Peak incidence @ 6 months
Mild upper respiratory incident precedes
Hyperinflation of the lungs on xray
28. If mild, treated at home
Humified O2 if hospitalized
HOB elevated
Abx not given unless secondary bacterial
intection
RSV prevention most important
29. Chronic inflammatory disorder
affecting mast cells, eosinophils,
and T lymphocytes
Inflammation causes increase in
bronchial hyper-responsiveness to
variety of stimuli (dander, dust,
pollen, etc.)
Most common chronic disease of
childhood; primary cause of school
absences
33. Shake vigorously prior to use
Exhale slowly and completely
Place mouthpiece in mouth, closing
lips around it
Press and release the med while
inhaling deeply and slowly
Hold breath for 10 seconds and
exhale
Repeat x1
38. Factor responsible for manifestations
of the disease is mechanical
obstruction caused by increased
viscosity of mucous gland secretions
Mucous glands produce a thick
protein that accumulates and dilates
the glands
Passages in organs such as the
PANCREAS become obstructed
First manifestation is meconium ileus
in NB
Sweat chloride test
39. Systems affected:
Respiratory: thick mucus, inflammation,
wheezing, pneumonia, cough, CHF in latter
stage
Pancreas: obstructed pancreatic ducts by
mucus and pancreatic enzymes (trypsin,
lipase, amylase) to duodenum
GI: decrease in absorption of nutrients, fatty
stools (steatorrhea), flatus, usually thin
Reproductive: 99% of males are sterile
40. Frequently admitted with FTT
Clubbing of the fingers
Barrel chest
Increased respirations, cyanosis
Productive cough
41. Positive sweat test (pilocarpine iontophoresis)
72 hr. fecal fat determination
Fasting blood sugar
Liver function studies
Sputum culture (to ID infective organisms)
CXR
42. Respiratory goal:
Nutritional:
Fat soluble vitamins ADKE
High calorie, high protein, low fat
Maintain Na balance (when
sweating and ill)
Thairapy vest
Pediatric respiratory conditions may occur as a primary problem or as a complication of nonrespiratory conditions and may be life threatening or have long-term implications.
Nurses must learn to assess the child’s current respiratory status quickly, monitor progress, and anticipate potential complications. Neurologic and immune processes may be linked to repiratory conditions as well.
Tongue is larger in proportion to mouth
Airway has larger amt of soft tissue than adult
Cricoid cartilage encircles airway until middle school age
Larynx is 2-3 cervical vertebrae higher
Lungs have fewer alveoli at birth than at one year
Mucous membranes lining are more loosely attached
Chest wall is less rigid and more soft
One of the most prevalent diseases of early childhood; highest in winter bec. Many cases of bacterial OM are preceded by a viral respiratory infection. The most common virus infections are RSV and influenza
Most occur in the first 24 months of life but it may occur up to 7 years of age
Children living in households with smokers have increased risk to have OM. Also those living in households with many members are more likely to have OM.
The underlying cause of OM is the malfunctiioning eustachian tubes. This tube, which connects the middle ear to the nasopharynx is normally closed and flat, preventing organisms in the pharyngeal cavity from entering the middle ear. This tube opens to allow drainage of secretions produced by the middle ear mucosa and to equalize air pressure between the middle ear and the outside environment. Impaired drainage of the eustachian tube causes retention of secretions in the middle ear. Air is unable to escape through the obstructed tubes, is absorbed into the circulation, and causes negative pressure within the middle ear. If the tube opens, a difference in pressure causes bacteria to be swept into the middle ear where the organisms quickly prolifereate and invade the mucosa.
Note the pus pockets and exudate
Tonsils are lymph tissue that guards the entrance to the rezpiratory and GI systems
Tonsils should not be removed unless they occlude the airway
Can be treated with abx at home
Begins on p. 1181
Manifestations: sore throat, tonsils enlarged and bright red, difficulty swallowing, nasal quality of speech, mouth breathing, hearing difficulty, snoring
Clinical manifestations: cessation of breathing; cyanosis, marked pallor, hypotonia, bradycardia
Diagnostic tests: r/o seizures with Eeg, r/o GERD, R/O RSV
Therapeutic interventions and Nursing Care: apnea monitor if documented apnea, teaching CPR to parents prior to discharge
Gentle cutaneous stimulation is used for neonates with mild apnea; drug therapy may include caffeine, oral theophylline or IV aminophylline to increase central respiratoyr drive and improve CO2 sensitivit
Infants: if no underlying disorder is identified, home monitoring with a respiratory stimulant (caffeine, theophylline)
Pneumocardiography, p. 1199 specifically tests for apnea by redording the HR and chest wall movements
Croup: crying aggravates the condition; oral dexamethosone in a single dose decreases airway inflammation. Abx not indicated; sedatives are contraindicated bec. they depress respirations
Do not examine or obtain material for culture from a child’ throat if epiglottitis is suspected because any stimulation with a tongue depressor or culture swab could trigger complete airway obstruction.
Do not leave child unattended!
Mucosal edema, increased airway irritation, mast cells release substances that act upon airways, bronchospasm, mucus plugging, increased work of breathing, gas exchange and tissue oxygenation is diminished,
See pp. 1202-1214
Arterial pH abnormalities include respiratory alkalosis (early) or acidosis (late); metabolic acidosis (from hypoxemia, and the work of breathing. Airway inflammation causes smooth muscle construction in large airways. This occurs rapidly and improves significantly with bronchodilators. There is mucus plugging and mucosal edema that does respond to steroid. Young children are more likely to have hospitalization for asthma attacks since they have such small airways.
PREVENTERS: allergy injections, decrease the allergens (carpets, pillows)
CONTROLLERS: Cromolyn, steroids (inhaled), leukotrienes (Singular)
RESCUERS: bronchodilators (beta agonists), steroids IV, IV fluids
Mast cell inhibitors: Intal, an inhaled nonsteroidal antiinflammatory drug, prevents asthma sx by blocking the release of mast cell mediators. Given 30 before exposure to triggers
A spacer may be used to help children who cannot coordinate inspiration with medication release. The space captures the medicine in a reservoir for the child to breathe in over a couple of minutes
In the school age child, stress may also play a role. Explain the role of emotions and stress in the development of asthma symptoms, p. 1209
A spacer, shown right, may be used with small chldren
Inherited as an autosomal recessive trait; the affected child inherits the defective gene from both aprents, with an overal incidence of 1:4. The mutated gene responsible for CF is located on the long arm of chromosome 7, along with its protein product, cystic fibrosis transmembrane regulator.
Characterized by several clinical features; increased viscosity of mucous gland secretions, a striking elevation of sweat electrolytes, an increase in several organic and enzymatic consitituents of saliva, and abnormalities in ANS function. Although both sodium and chloride are affected, the defect appears to be primarily a result of abnormal chloride movements. Children with DF demonstrate decreased pancreatic secretion of bicarbonate and chloride and an increase in sodium and chloride in both saliva and sweat. This characteristic is the basis for the sweat chloride diagnostic test.
Here is the genetic ratio of parents with the gene; autosomal recessive trait, which means that both parents must carry the gene for the child to be affected. Of all patients in the US, 70% are diagnosed before the age of 2 years.
Sweat test done for accurate diagnosis: measure amt of Cl after patch is applied. A negative test, however, does not necessarily eliminate the possibility of being affected by the disease.
Genetic testing needs to be done if suspicious of CF with negative sweat test. Some babies with FTT may have the disease.
Level < 40 for both Na and Cl; patients with CF have > 60 for both Na and Cl
Steatorrhea (frothy, foul-smelling stools 2-3 times bulkier than normal) and flatus
Show class how to measure this
p. 1218
Respiratory goal: removal of secretions (chest physiotherapy with Thairapy vest) by vibrations loosen mucus
Nutritional goal:inc. weight, enzymes with all food (Creon, Pancrease, Ultrace) dosage is regulated by evaluation of the stool
Infants may sometimes be given a predigested formula (Pregestimil, Nutramigen) which is more easily absorbed
Enzyme regulation: dosage adjusted according to stool formation: less enzyme with constipation; more enzyme with loose, fatty stools. Only brand-name enzymes should be sued because generic enzymes are bioquivalent.