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Kavan vyas
203
 Anterior portion of the pharynx is more
enlarged
 Constrioctor region is more stabble
 Pharyngeal tonsils are more developed and
enlarged….but swelling of it can obstruct
airway
 By the age of 6 all the pharyngeal structures are
almost 80 % developes
 more sensitive and softer …so there is risk of
inflammation
 It is funnle shaped in children while in adult it
has cylindrical shape
 Vocal cords have concave shape while in adult
they are flat
 It is located bit superiorly in children near C2
while in adults around C6.
 Normal length of trachea is around 57 mm
 While normal width is around 20mm
 More sensitive to inflammation
 C shaped cartilagenous rings are more softer
compared to adult but still it is developed so
that it can make airway open
 Obstruction or edema can easily occure
 Lungs have less tidal volume and alveolies are
less in number so it decrease space for gaseous
exchange
 Number of alveoli is almist around 2 millions
 Deadspace is higher in infant { 3ml/kg}
 Deadspace is the volume of air which is
inhaled that does not take part in the gas
exchange
Adult
Child
 Bluish body (cynosis)
 High breathing speed or low
 Different sounds during respiration
 Common cough
 Crying or problem during swallowing
( in tonsillitis)
 fever
 headache
1- otitis media
2- tonsillitis
3- apnea
4-croup vs epiglottises
5- bronchitis
6-bronchiolitis
7-RAD (asthama)
8- cystic fibrosis
 Note the ear on the left with clear
tympanic membrane (drum); ear on
the R the drum is bulging and filled
with pus

Acute Otitis Media
characterized by abrupt onset, pain, middle ear effusion, and
inflammation.
Note the injected
vessels and altered
shape of cone of
light.
Treatment – through
surgery
 May be bacterial or viral
 Most common bacterial agent: Group A beta-
hemolytic strep
 Throat cultures must be done to determine
origin
 Older child may develop peritonsillar abscess
 Treatment is symptomatic
 Antibiotics restricted to those with bacterial
infection
 Drug of choice: amoxicillin
 Surgery (with recurrent infections)
 Defined as delay of breathing over
20 seconds
 Manifestations
 Diagnostic tests
 Therapeutic Interventions and
Nursing Care
 Prematurity: most common and may vary
among neonates
 Infant apnea: no known cause; r/o seizures,
GERD, hypoglycemia
 Apnea:
 Cessation > 20
seconds
 S/S to assess:
 Cyanosis
 Marked pallor
 Hypotonia
 bradycardia
 Periodic
breathing
 Normal
breathing
pattern of NB
but never > 10-
15 seconds
 Even though
normal, all
parents are
taught CPR for
their NB
 Pneumocardiography
 CXR
 Blood chemistry studies
 ECG
 EEG
 Nurse sets parameters for HR according to age
 Gentle stimulation of infant
 Maintaining a neutral environment
 Instruct family with apnea monitors at home
CroupCroup
 Croup
 Usual age range: 1-3 yrs
 Inspiratory stridor
 Harsh cough (barking)
 Viral infection; afebrile
 Gradual onset, usually at night
 Improved with humidity; may
need racemic epi
 Treatable at home
 Resolves spontaneously
 Epiglottitis
 Usual age range 3-7 yrs
 May have stridor
 Caused by **H.influenzae, but
may staph and strep as well
 Sudden onset
 Sore throat and difficulty
swallowing
 May be an emergent situation
 Lateral soft tissue of neck xray
 Have equipment at bedside
 Drooling
 Dysphagia
 Dysphonia
 Distressed inspiratory efforts
 Observe for s/s respiratory distress
 Assess respiratory rates: >60
 Elevated temp ) 101º
 The child must NEVER be left alone
 NOTHING should be placed in the mouth
(laryngeal spasms could result)
 Croup
 Racemic epi nebulization
 Oral dexamethosone in a
single dose
 Acetaminophen
 Humidified O2 and IVs for
more severe cases
 Sedatives are
contraindicated
 Epiglottitis
 Child kept NPO
 IV antibiotics
 Antipyretics for fever
 Emergency hospitalization
 Etiology
 Inflammation of trachea and major bronchi
 Usually viral (Rhino and RSV)
 Occur with other conditions; may be confused with
RAD (asthma)
 Cough major symptom
 Gradual onset of rhinitis
 Productive cough (may be purulent) with  mucus
 Crackles, rhonchi
 Increase fluids
 Assess VS, secretions, respiratory effort
 S/S sleep deprivation from cough
 Antipyretics for fever
 Quiet activities for diversion
 Etiology
 RSV most common pathogen
 May acquire from older siblings
 Peak incidence @ 6 months
 Mild upper respiratory incident precedes
 Hyperinflation of the lungs on xray
 If mild, treated at home
 Humified O2 if hospitalized
 HOB elevated
 Abx not given unless secondary bacterial
intection
 RSV prevention most important
 Chronic inflammatory disorder
affecting mast cells, eosinophils,
and T lymphocytes
 Inflammation causes increase in
bronchial hyper-responsiveness to
variety of stimuli (dander, dust,
pollen, etc.)
 Most common chronic disease of
childhood; primary cause of school
absences
 Pathophysiology
 Increased airway resistance, decreased
flow rate; bronchospasm
 Increased work of breathing
 Progressive decrease in tidal volume
 Arterial pH changes: respiratory
alkalosis, metabolic acidosis
 Characterized by
 Mucosal edema,non productive cough
 Wheezing (r/t bronchospasm)
 Mucus plugging
 Combination of bronchodilators
and antiinflammatories
 Inhaled steroids first-line tx
 Regimen depends on classification
of child’s asthma
 “Rescue”: short-acting beta agonists (Ventolin,
Proventil)
 Anticholinergics
 Mast cell inhibitors (Intal)
 Systemic corticosteroids (for short course
management)
 Shake vigorously prior to use
 Exhale slowly and completely
 Place mouthpiece in mouth, closing
lips around it
 Press and release the med while
inhaling deeply and slowly
 Hold breath for 10 seconds and
exhale
 Repeat x1
 Exercise
 Infections
 Allergens
 Weather
changes
 .
 Factor responsible for manifestations
of the disease is mechanical
obstruction caused by increased
viscosity of mucous gland secretions
 Mucous glands produce a thick
protein that accumulates and dilates
the glands
 Passages in organs such as the
PANCREAS become obstructed
 First manifestation is meconium ileus
in NB
 Sweat chloride test
 Systems affected:
 Respiratory: thick mucus, inflammation,
wheezing, pneumonia, cough, CHF in latter
stage
 Pancreas: obstructed pancreatic ducts by
mucus and pancreatic enzymes (trypsin,
lipase, amylase) to duodenum
 GI: decrease in absorption of nutrients, fatty
stools (steatorrhea), flatus, usually thin
 Reproductive: 99% of males are sterile
 Frequently admitted with FTT
 Clubbing of the fingers
 Barrel chest
 Increased respirations, cyanosis
 Productive cough
 Positive sweat test (pilocarpine iontophoresis)
 72 hr. fecal fat determination
 Fasting blood sugar
 Liver function studies
 Sputum culture (to ID infective organisms)
 CXR
 Respiratory goal:
 Nutritional:
Fat soluble vitamins ADKE
High calorie, high protein, low fat
 Maintain Na balance (when
sweating and ill)
 Thairapy vest
By
Kavan vyas
203

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Pharyngeal and laryngeal structures in children vs adults

  • 2.  Anterior portion of the pharynx is more enlarged  Constrioctor region is more stabble  Pharyngeal tonsils are more developed and enlarged….but swelling of it can obstruct airway  By the age of 6 all the pharyngeal structures are almost 80 % developes
  • 3.  more sensitive and softer …so there is risk of inflammation  It is funnle shaped in children while in adult it has cylindrical shape  Vocal cords have concave shape while in adult they are flat  It is located bit superiorly in children near C2 while in adults around C6.
  • 4.  Normal length of trachea is around 57 mm  While normal width is around 20mm  More sensitive to inflammation  C shaped cartilagenous rings are more softer compared to adult but still it is developed so that it can make airway open  Obstruction or edema can easily occure
  • 5.  Lungs have less tidal volume and alveolies are less in number so it decrease space for gaseous exchange  Number of alveoli is almist around 2 millions  Deadspace is higher in infant { 3ml/kg}  Deadspace is the volume of air which is inhaled that does not take part in the gas exchange
  • 7.  Bluish body (cynosis)  High breathing speed or low  Different sounds during respiration  Common cough  Crying or problem during swallowing ( in tonsillitis)  fever  headache
  • 8. 1- otitis media 2- tonsillitis 3- apnea 4-croup vs epiglottises 5- bronchitis 6-bronchiolitis 7-RAD (asthama) 8- cystic fibrosis
  • 9.  Note the ear on the left with clear tympanic membrane (drum); ear on the R the drum is bulging and filled with pus
  • 10.
  • 11. Acute Otitis Media characterized by abrupt onset, pain, middle ear effusion, and inflammation. Note the injected vessels and altered shape of cone of light. Treatment – through surgery
  • 12.
  • 13.  May be bacterial or viral  Most common bacterial agent: Group A beta- hemolytic strep  Throat cultures must be done to determine origin  Older child may develop peritonsillar abscess
  • 14.  Treatment is symptomatic  Antibiotics restricted to those with bacterial infection  Drug of choice: amoxicillin  Surgery (with recurrent infections)
  • 15.  Defined as delay of breathing over 20 seconds  Manifestations  Diagnostic tests  Therapeutic Interventions and Nursing Care
  • 16.  Prematurity: most common and may vary among neonates  Infant apnea: no known cause; r/o seizures, GERD, hypoglycemia
  • 17.  Apnea:  Cessation > 20 seconds  S/S to assess:  Cyanosis  Marked pallor  Hypotonia  bradycardia  Periodic breathing  Normal breathing pattern of NB but never > 10- 15 seconds  Even though normal, all parents are taught CPR for their NB
  • 18.  Pneumocardiography  CXR  Blood chemistry studies  ECG  EEG
  • 19.  Nurse sets parameters for HR according to age  Gentle stimulation of infant  Maintaining a neutral environment  Instruct family with apnea monitors at home
  • 21.  Croup  Usual age range: 1-3 yrs  Inspiratory stridor  Harsh cough (barking)  Viral infection; afebrile  Gradual onset, usually at night  Improved with humidity; may need racemic epi  Treatable at home  Resolves spontaneously  Epiglottitis  Usual age range 3-7 yrs  May have stridor  Caused by **H.influenzae, but may staph and strep as well  Sudden onset  Sore throat and difficulty swallowing  May be an emergent situation  Lateral soft tissue of neck xray  Have equipment at bedside
  • 22.  Drooling  Dysphagia  Dysphonia  Distressed inspiratory efforts
  • 23.  Observe for s/s respiratory distress  Assess respiratory rates: >60  Elevated temp ) 101º  The child must NEVER be left alone  NOTHING should be placed in the mouth (laryngeal spasms could result)
  • 24.  Croup  Racemic epi nebulization  Oral dexamethosone in a single dose  Acetaminophen  Humidified O2 and IVs for more severe cases  Sedatives are contraindicated  Epiglottitis  Child kept NPO  IV antibiotics  Antipyretics for fever  Emergency hospitalization
  • 25.  Etiology  Inflammation of trachea and major bronchi  Usually viral (Rhino and RSV)  Occur with other conditions; may be confused with RAD (asthma)  Cough major symptom  Gradual onset of rhinitis  Productive cough (may be purulent) with  mucus  Crackles, rhonchi
  • 26.  Increase fluids  Assess VS, secretions, respiratory effort  S/S sleep deprivation from cough  Antipyretics for fever  Quiet activities for diversion
  • 27.  Etiology  RSV most common pathogen  May acquire from older siblings  Peak incidence @ 6 months  Mild upper respiratory incident precedes  Hyperinflation of the lungs on xray
  • 28.  If mild, treated at home  Humified O2 if hospitalized  HOB elevated  Abx not given unless secondary bacterial intection  RSV prevention most important
  • 29.  Chronic inflammatory disorder affecting mast cells, eosinophils, and T lymphocytes  Inflammation causes increase in bronchial hyper-responsiveness to variety of stimuli (dander, dust, pollen, etc.)  Most common chronic disease of childhood; primary cause of school absences
  • 30.  Pathophysiology  Increased airway resistance, decreased flow rate; bronchospasm  Increased work of breathing  Progressive decrease in tidal volume  Arterial pH changes: respiratory alkalosis, metabolic acidosis  Characterized by  Mucosal edema,non productive cough  Wheezing (r/t bronchospasm)  Mucus plugging
  • 31.  Combination of bronchodilators and antiinflammatories  Inhaled steroids first-line tx  Regimen depends on classification of child’s asthma
  • 32.  “Rescue”: short-acting beta agonists (Ventolin, Proventil)  Anticholinergics  Mast cell inhibitors (Intal)  Systemic corticosteroids (for short course management)
  • 33.  Shake vigorously prior to use  Exhale slowly and completely  Place mouthpiece in mouth, closing lips around it  Press and release the med while inhaling deeply and slowly  Hold breath for 10 seconds and exhale  Repeat x1
  • 34.  Exercise  Infections  Allergens  Weather changes
  • 35.  .
  • 36.
  • 37.
  • 38.  Factor responsible for manifestations of the disease is mechanical obstruction caused by increased viscosity of mucous gland secretions  Mucous glands produce a thick protein that accumulates and dilates the glands  Passages in organs such as the PANCREAS become obstructed  First manifestation is meconium ileus in NB  Sweat chloride test
  • 39.  Systems affected:  Respiratory: thick mucus, inflammation, wheezing, pneumonia, cough, CHF in latter stage  Pancreas: obstructed pancreatic ducts by mucus and pancreatic enzymes (trypsin, lipase, amylase) to duodenum  GI: decrease in absorption of nutrients, fatty stools (steatorrhea), flatus, usually thin  Reproductive: 99% of males are sterile
  • 40.  Frequently admitted with FTT  Clubbing of the fingers  Barrel chest  Increased respirations, cyanosis  Productive cough
  • 41.  Positive sweat test (pilocarpine iontophoresis)  72 hr. fecal fat determination  Fasting blood sugar  Liver function studies  Sputum culture (to ID infective organisms)  CXR
  • 42.  Respiratory goal:  Nutritional: Fat soluble vitamins ADKE High calorie, high protein, low fat  Maintain Na balance (when sweating and ill)  Thairapy vest

Editor's Notes

  1. Pediatric respiratory conditions may occur as a primary problem or as a complication of nonrespiratory conditions and may be life threatening or have long-term implications. Nurses must learn to assess the child’s current respiratory status quickly, monitor progress, and anticipate potential complications. Neurologic and immune processes may be linked to repiratory conditions as well.
  2. Tongue is larger in proportion to mouth Airway has larger amt of soft tissue than adult Cricoid cartilage encircles airway until middle school age Larynx is 2-3 cervical vertebrae higher Lungs have fewer alveoli at birth than at one year Mucous membranes lining are more loosely attached Chest wall is less rigid and more soft
  3. One of the most prevalent diseases of early childhood; highest in winter bec. Many cases of bacterial OM are preceded by a viral respiratory infection. The most common virus infections are RSV and influenza Most occur in the first 24 months of life but it may occur up to 7 years of age Children living in households with smokers have increased risk to have OM. Also those living in households with many members are more likely to have OM.
  4. The underlying cause of OM is the malfunctiioning eustachian tubes. This tube, which connects the middle ear to the nasopharynx is normally closed and flat, preventing organisms in the pharyngeal cavity from entering the middle ear. This tube opens to allow drainage of secretions produced by the middle ear mucosa and to equalize air pressure between the middle ear and the outside environment. Impaired drainage of the eustachian tube causes retention of secretions in the middle ear. Air is unable to escape through the obstructed tubes, is absorbed into the circulation, and causes negative pressure within the middle ear. If the tube opens, a difference in pressure causes bacteria to be swept into the middle ear where the organisms quickly prolifereate and invade the mucosa.
  5. Note the pus pockets and exudate Tonsils are lymph tissue that guards the entrance to the rezpiratory and GI systems Tonsils should not be removed unless they occlude the airway Can be treated with abx at home
  6. Begins on p. 1181 Manifestations: sore throat, tonsils enlarged and bright red, difficulty swallowing, nasal quality of speech, mouth breathing, hearing difficulty, snoring
  7. Clinical manifestations: cessation of breathing; cyanosis, marked pallor, hypotonia, bradycardia Diagnostic tests: r/o seizures with Eeg, r/o GERD, R/O RSV Therapeutic interventions and Nursing Care: apnea monitor if documented apnea, teaching CPR to parents prior to discharge
  8. Gentle cutaneous stimulation is used for neonates with mild apnea; drug therapy may include caffeine, oral theophylline or IV aminophylline to increase central respiratoyr drive and improve CO2 sensitivit Infants: if no underlying disorder is identified, home monitoring with a respiratory stimulant (caffeine, theophylline)
  9. Pneumocardiography, p. 1199 specifically tests for apnea by redording the HR and chest wall movements
  10. See p. 1200
  11. Croup: 3months-8yr; slowly progressive; attacks at night, barking cough mild elevation of temp; VIRAL; inspiratory stridor Epiglottitis: Onset 2yrs-8yrs; stridor, cough, BACTERIAL
  12. Croup: crying aggravates the condition; oral dexamethosone in a single dose decreases airway inflammation. Abx not indicated; sedatives are contraindicated bec. they depress respirations
  13. Do not examine or obtain material for culture from a child’ throat if epiglottitis is suspected because any stimulation with a tongue depressor or culture swab could trigger complete airway obstruction. Do not leave child unattended!
  14. Mucosal edema, increased airway irritation, mast cells release substances that act upon airways, bronchospasm, mucus plugging, increased work of breathing, gas exchange and tissue oxygenation is diminished,
  15. See pp. 1202-1214 Arterial pH abnormalities include respiratory alkalosis (early) or acidosis (late); metabolic acidosis (from hypoxemia, and the work of breathing. Airway inflammation causes smooth muscle construction in large airways. This occurs rapidly and improves significantly with bronchodilators. There is mucus plugging and mucosal edema that does respond to steroid. Young children are more likely to have hospitalization for asthma attacks since they have such small airways. PREVENTERS: allergy injections, decrease the allergens (carpets, pillows) CONTROLLERS: Cromolyn, steroids (inhaled), leukotrienes (Singular) RESCUERS: bronchodilators (beta agonists), steroids IV, IV fluids
  16. Mast cell inhibitors: Intal, an inhaled nonsteroidal antiinflammatory drug, prevents asthma sx by blocking the release of mast cell mediators. Given 30 before exposure to triggers
  17. A spacer may be used to help children who cannot coordinate inspiration with medication release. The space captures the medicine in a reservoir for the child to breathe in over a couple of minutes
  18. In the school age child, stress may also play a role. Explain the role of emotions and stress in the development of asthma symptoms, p. 1209
  19. A spacer, shown right, may be used with small chldren
  20. Inherited as an autosomal recessive trait; the affected child inherits the defective gene from both aprents, with an overal incidence of 1:4. The mutated gene responsible for CF is located on the long arm of chromosome 7, along with its protein product, cystic fibrosis transmembrane regulator. Characterized by several clinical features; increased viscosity of mucous gland secretions, a striking elevation of sweat electrolytes, an increase in several organic and enzymatic consitituents of saliva, and abnormalities in ANS function. Although both sodium and chloride are affected, the defect appears to be primarily a result of abnormal chloride movements. Children with DF demonstrate decreased pancreatic secretion of bicarbonate and chloride and an increase in sodium and chloride in both saliva and sweat. This characteristic is the basis for the sweat chloride diagnostic test.
  21. Here is the genetic ratio of parents with the gene; autosomal recessive trait, which means that both parents must carry the gene for the child to be affected. Of all patients in the US, 70% are diagnosed before the age of 2 years.
  22. Sweat test done for accurate diagnosis: measure amt of Cl after patch is applied. A negative test, however, does not necessarily eliminate the possibility of being affected by the disease. Genetic testing needs to be done if suspicious of CF with negative sweat test. Some babies with FTT may have the disease. Level < 40 for both Na and Cl; patients with CF have > 60 for both Na and Cl
  23. Steatorrhea (frothy, foul-smelling stools 2-3 times bulkier than normal) and flatus
  24. Show class how to measure this
  25. p. 1218
  26. Respiratory goal: removal of secretions (chest physiotherapy with Thairapy vest) by vibrations loosen mucus Nutritional goal:inc. weight, enzymes with all food (Creon, Pancrease, Ultrace) dosage is regulated by evaluation of the stool Infants may sometimes be given a predigested formula (Pregestimil, Nutramigen) which is more easily absorbed Enzyme regulation: dosage adjusted according to stool formation: less enzyme with constipation; more enzyme with loose, fatty stools. Only brand-name enzymes should be sued because generic enzymes are bioquivalent.