This document provides tips and instructions for using a PowerPoint presentation on vascular anomalies. It recommends freely editing and modifying the slides. It advises showing blank slides first to elicit what students already know before presenting content. This active learning approach should be repeated in three revisions. Good for self-study as well. Notes include a bibliography. The presentation then provides information on vascular anomalies, including classification, hemangiomas, vascular malformations, etiology, clinical features, management, and associated syndromes such as Klippel-Trenaunay-Weber and Sturge-Weber. QR codes and links are given to access the presentation on mobile devices and for the presenter's slide collection.
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Vasculr malformations.pptx
1. Tips on using my ppt.
1. You can freely download, edit, modify and put your
name etc.
2. Don’t be concerned about number of slides. Half the
slides are blanks except for the title.
3. First show the blank slides (eg. Aetiology ) > Ask
students what they already know about ethology of
today's topic. > Then show next slide which enumerates
aetiologies.
4. At the end rerun the show – show blank> ask questions >
show next slide.
5. This will be an ACTIVE LEARNING SESSION x
three revisions.
6. Good for self study also.
7. See notes for bibliography.
7. Hemangioma
• Vascular tumor-like structure
• Present at or around birth or appeared later
in life.
• Self-involuting
• Growing faster than body of patient
• Eventually disappear
17. Symptoms: site
• Head and neck (59%)
• Trunk (24%)
• Lower extremities (10%)
• Upper extremities (7%)
• Liver and other viscra
18. Symptoms: Appearance
• Colored congenital lesion
• Vary from a hypopigmented macule to a
bruiselike macule.
• Lymphangioma-Soft doughy masses that
are located in the head and neck region,
19. Symptoms:Natural history
• Proliferative postnatal growth phase that
lasts for 3-9 months
• Gradual involution that occurs over 2-6
years.
• Involution is usually complete by age 7-10
years.
20. Symptoms:Complications
• In some cases, hemangiomas can be life-
threatening or severely problematic,
interfering with eating, breathing, seeing,
hearing, and speaking.
29. Get this ppt in mobile
1. Download Microsoft
PowerPoint from play
store.
2. Open Google assistant
3. Open Google lens.
4. Scan qr code from
next slide.
31. Get my ppt collection
• https://www.slideshare.net/drpradeeppande/
edit_my_uploads
• https://www.dropbox.com/sh/x600md3cvj8
5woy/AACVMHuQtvHvl_K8ehc3ltkEa?dl
=0
• https://www.facebook.com/doctorpradeeppa
nde/?ref=pages_you_manage
Editor's Notes
drpradeeppande@gmail.com
7697305442
The terminology used to define, describe and categorize vascular anomalies, abnormal lumps made up of blood vessels, has changed. The term hemangioma originally described any vascular tumor-like structure, whether it was present at or around birth or appeared later in life. Mulliken et al. categorized these conditions into two families: one of self-involuting tumors, growing lesions that eventually disappear, and another of malformations, enlarged or abnormal vessels present at birth and essentially permanent. The importance of this distinction is that it makes it possible for early-in-life differentiation between lesions that will resolve versus those that are permanent. Examples of permanent malformations include port-wine stains (capillary vascular malformation) and masses of abnormal swollen veins(venous malformations).[3] The Mulliken categorisation has received major confirmation following discovery of the Glut-1 marker.[citation needed]
The terminology used to define, describe and categorize vascular anomalies, abnormal lumps made up of blood vessels, has changed. The term hemangioma originally described any vascular tumor-like structure, whether it was present at or around birth or appeared later in life. Mulliken et al. categorized these conditions into two families: one of self-involuting tumors, growing lesions that eventually disappear, and another of malformations, enlarged or abnormal vessels present at birth and essentially permanent. The importance of this distinction is that it makes it possible for early-in-life differentiation between lesions that will resolve versus those that are permanent. Examples of permanent malformations include port-wine stains (capillary vascular malformation) and masses of abnormal swollen veins(venous malformations).[3] The Mulliken categorisation has received major confirmation following discovery of the Glut-1 marker.[citation needed]
Vascular malformations - These are subdivided into high-flow (arterial, arteriovenous) malformations and slow-flow (venous, capillary, lymphatic) malformations;
Vascular malformations - These are subdivided into high-flow (arterial, arteriovenous) malformations and slow-flow (venous, capillary, lymphatic) malformations;