2. Why do we care...?Why do we care...?
AHA 2016AHA 2016
OOH - >350K - 12% survivalOOH - >350K - 12% survival
IH - 209K - 24.8% survivalIH - 209K - 24.8% survival
#1 Cause of death - heart disease#1 Cause of death - heart disease
VF, VT, high degree AV BlockVF, VT, high degree AV Block
3. Back toBack to
the Basicsthe Basics
Coronary muscleCoronary muscle
cells got the beatcells got the beat
13. Right Coronary Artery MIRight Coronary Artery MI
HypotensionHypotension
after NTGafter NTG
BradycardiaBradycardia
Nausea/vomitingNausea/vomiting
HiccoughingHiccoughing
Urge to defecateUrge to defecate
17. 3rd Degree Heart Block3rd Degree Heart Block
No conduction through the AV nodeNo conduction through the AV node
18. Mobitz II - 2nd DegreeMobitz II - 2nd Degree
Heart BlockHeart Block
Problem in His-Purkinje SystemProblem in His-Purkinje System
19. Overdrive PacingOverdrive Pacing
Tachydysrhythmia not responsive to drugTachydysrhythmia not responsive to drug
therapy or electrocardioversiontherapy or electrocardioversion
Usually only good HR <180Usually only good HR <180
Goal to break re-entry circuitGoal to break re-entry circuit
AV node able to take controlAV node able to take control
Can cause VFibCan cause VFib
20.
21. AsystoleAsystole
2010 AHA recommendations2010 AHA recommendations
No - out of hospital cardiac arrestNo - out of hospital cardiac arrest
YesYes
Conduction system problem, P waves presentConduction system problem, P waves present
Drug-induced - Digoxin, Procainamide,Drug-induced - Digoxin, Procainamide, ΒΒ--
blockersblockers
Unexpected arrest - anesthesia, surgery, cath,Unexpected arrest - anesthesia, surgery, cath,
etcetc
Following defibrilliationFollowing defibrilliation
22. Pediatric PacingPediatric Pacing
#1 cause of bradycardia - hypoxia#1 cause of bradycardia - hypoxia
Bradycardia from surgically-induced heart blockBradycardia from surgically-induced heart block
Congenital AV blockCongenital AV block
Viral MyocarditisViral Myocarditis
Bradycardia - toxin, drug overdoseBradycardia - toxin, drug overdose
PPM failurePPM failure
23. Stand-by PlacementStand-by Placement
AMI showing signs of heart blockAMI showing signs of heart block
Awaiting cardiac surgeryAwaiting cardiac surgery
Awaiting PPM, lead wire change, generatorAwaiting PPM, lead wire change, generator
changechange
Cardiac cath patientsCardiac cath patients
Post cardioversion bradycardiaPost cardioversion bradycardia
24. Temporary IndicationsTemporary Indications
Situation where permanent pacing not neededSituation where permanent pacing not needed
Bradycardia secondary to hypothermiaBradycardia secondary to hypothermia
In severe, heart won’t respondIn severe, heart won’t respond
PEA from: drug overdose, electrolytePEA from: drug overdose, electrolyte
abnormality, toxic exposureabnormality, toxic exposure
Trauma affecting cardiac functionTrauma affecting cardiac function
26. Temporary IndicationsTemporary Indications
When unable to place transvenous pacerWhen unable to place transvenous pacer
Non-native Tricuspid valveNon-native Tricuspid valve
Potential for bleeding - anticoagulantsPotential for bleeding - anticoagulants
Potential for infection - immunocompromisedPotential for infection - immunocompromised
27. Transcutaneous PacingTranscutaneous Pacing
GoalsGoals
Keep patient hemodynamically stableKeep patient hemodynamically stable
Until underlying problem resolvedUntil underlying problem resolved
Until permanent pacing strategy appliedUntil permanent pacing strategy applied
30. MethodsMethods
Place padsPlace pads
Connect EKG leadsConnect EKG leads
Pacemaker to demandPacemaker to demand
Pace rate >30bpm above intrinsic rhythmPace rate >30bpm above intrinsic rhythm
mA to 70mA to 70
Start and increase mA until capture (max of 120 -Start and increase mA until capture (max of 120 -
130)130)
Upon capture, set current 5 - 10 mA aboveUpon capture, set current 5 - 10 mA above
thresholdthreshold
32. MethodsMethods
Place padsPlace pads
Connect EKG leadsConnect EKG leads
Pacemaker to demandPacemaker to demand
Pace rate >30bpm above intrinsic rhythmPace rate >30bpm above intrinsic rhythm
mA to 70mA to 70
Start and increase mA until capture (max of 120 -Start and increase mA until capture (max of 120 -
130)130)
Upon capture, set current 5 - 10 mA aboveUpon capture, set current 5 - 10 mA above
thresholdthreshold
35. ResultsResults
Associated with reduced L ventricular systolicAssociated with reduced L ventricular systolic
pressurepressure
Lower stroke index 2nd to AV dyssynchronyLower stroke index 2nd to AV dyssynchrony
Because of muscle contractions, can provideBecause of muscle contractions, can provide
greater COgreater CO