This document contains abstracts from the 2005 SWACSM Annual Meeting. The summaries are:
1) A study found that inhibiting ERK1/2 prevented the contraction-induced increase in muscle fatty acid metabolism and CD36 content in a dose-dependent manner, without affecting AMPK activity. ERK1/2 phosphorylation was positively correlated with fatty acid uptake and oxidation. This suggests ERK1/2 is required for contraction-induced increases in fatty acid metabolism.
2) A study found that inhibiting various potassium channels individually did not alter vascular tone in soleus feed arteries, but inhibiting inward rectifier potassium channels and the sodium-potassium pump together significantly increased vascular tone. This suggests these channels contribute to