The patient is unable to stand or walk because he is unable to move or bear weight on his right leg. On examination:
- Strength is normal in the left leg but there is weakness of dorsiflexion and plantarflexion in the right leg.
- Reflexes are normal in the left leg but hyperreflexic in the right leg.
- Sensation to light touch, pinprick and temperature are intact throughout.
- There is spasticity in the right leg.
Damage to what system(s) is causing this patient’s problems?
Lesion of the right lateral corticospinal tract at approximately L2.
The findings are consistent with an
here i am to explain the Anatomy and physiology of part of the Pyramidal tract, that is the corticospinal tract. I also added the clinical significance of corticospinal tract. The course of the corticospinal tract are well explained.
here i am to explain the Anatomy and physiology of part of the Pyramidal tract, that is the corticospinal tract. I also added the clinical significance of corticospinal tract. The course of the corticospinal tract are well explained.
BROADMANN AREA 1,2,3 Grouped as primary somatosensory cortex. Location – Post-Central gyrus on lateral surface of brain. Tactile representation is orderly arranged (in an inverted fashion) from the toe (at the top of the cerebral hemisphere) to mouth (at the bottom)
BROADMANN AREA 1,2,3 Grouped as primary somatosensory cortex. Location – Post-Central gyrus on lateral surface of brain. Tactile representation is orderly arranged (in an inverted fashion) from the toe (at the top of the cerebral hemisphere) to mouth (at the bottom)
Review on Case Hemisection of the Spinal Cord (Brown Sequard Syndrome)
This case is a trigger in Neuroscience Module in Medical Faculty University of Indonesia
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
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Spinal cord lesions module
1. Lesions of the Spinal Cord
Learning Module
Click to Begin
2. Main Menu
Overview describes the module content & learning objectives
Please complete this section first!
Contents houses the 9 interactive lesion lessons and directions
for completing them.
Patient Cases provides practice with feedback using patient
cases.
Exit
3. Overview
Introduction
Learning Objectives
Overview Menu Main Menu Exit
4. Introduction
• This module reviews lesions of the spinal cord
• Module organization consists of three components. Overview
consists of this Introduction and the Learning Objectives. Contents
consists of Navigation Instructions, a Legend, and 9 interactive
lesion lessons. Cases consists of Instructions and 3 interactive
patient cases with feedback.
• At the bottom of each page a navigation bar contains options to
move throughout the module.
• Material is presented at both the behavioral level and the
neuroanatomical level.
• The behavioral level is presented first and depicts a patient’s clinical
presentation.
• The neuroanatomical level depicts the detailed anatomy of first-
order, second-order and third-order neurons.
• The neuroanatomical level accounts for the patient’s behavioral
presentation on examination under normal and lesioned conditions.
Overview Menu Main Menu Exit
5. Learning Objectives
After completing this module you should be able to:
1. describe the signs and symptoms caused by a lesion
of the spinal cord (fasciculus gracilis and fasciculus
cuneatus, lateral corticospinal tract, and lateral
spinothalamic tract).
2. given a patient case (examination results and chief
complaint), identify the functional systems causing
the sensory and motor impairments.
3. correlate neurology information between the
behavioral and neuroanatomical levels.
Overview Menu Main Menu Exit
6. Contents
Read these Instructions!
Legend: symbols used throughout the module
Review of the Spinal Cord (Under Construction)
Lesion lessons
Dorsal column lesion Hemicord lesion
Fasciculus cuneatus lesion Central cord syndrome
Lateral corticospinal tract lesion Anterior cord syndrome
Lateral spinothalamic tract lesion Posterior cord syndrome
Transverse cord lesion
Main Menu Exit
7. Instructions
• This module contains 9 interactive lesion lessons with animation.
• Lesson lessons begin with a question about the symptoms
produced by that particular lesion.
• Clicking the answer button will reveal the answer to the question.
• Clicking the explanation button will lead to both behavioral and
neuroanatomical explanations of the lesion.
• Each presentation is launched by clicking the animation button.
The same button serves to replay the animation if desired.
• Any of the lessons may be accessed by simply clicking on the
lesion title on the Contents page.
• Please refer to the Legend that defines the symbols used
throughout the module.
Main Menu Content Menu Exit
8. Legend
Mechanism of injury First-order neuron
Lesion Second-order neuron
Pain stimulus
Third-order neuron
Light touch stimulus
Sensory impairment
Function intact
Function lost
Main Menu Content Menu Exit
9. R L
Lesion of the right dorsal
column at L1 produces what
impairment?
Click for answer
Damage to the right dorsal column at L1 causes the
absence of light touch, vibration, and position
sensation in the right leg. Only fasciculus gracilis
exists below T6.
Click for explanation
Main Menu Content Menu Legend Exit
10. Right Dorsal Column Lesion
Click to animate
DRG
R L
L1
Dorsal column lesion
Ipsilateral loss of light touch,
Common causes vibration, and position sense
include MS, generalized below the lesion level
penetrating injuries,
and compression Below T6 only the fasciculus gracilis
from tumors. is present.
Main Menu Content Menu Legend Exit
11. R L
Lesion of the right fasciculus
cuneatus at C3 produces what
impairment?
Click for answer
Damage to the right fasciculus cuneatus at C3
causes the absence of light touch, vibration, and
position sensation in the right arm and upper trunk.
Click for explanation
Main Menu Content Menu Legend Exit
12. Right Fasciculus Cuneatus Lesion
Click to animate
DRG
R L
C3
Fasciculus cuneatus lesion
Ipsilateral loss of light touch,
Common causes vibration, and position sense
include MS, In the right arm and upper trunk
penetrating injuries,
and compression
from tumors.
Main Menu Content Menu Legend Exit
13. R L
Lesion of the right lateral
corticospinal tract at L1
produces what impairment?
Click for answer
Damage to the right lateral corticospinal tract at L1
causes upper motor neurons signs (weakness or
paralysis, hyperreflexia, and hypertonia) in the right leg.
Click for explanation
Main Menu Content Menu Legend Exit
14. Right Lateral Corticospinal Tract Lesion
UMN
Click to animate
R L
L1
Lateral corticospinal tract lesion
Common causes Ipsilateral upper motor neurons signs
include penetrating generalized below the lesion level
injuries, lateral UMN signs
compression from Weakness (Spastic paralysis)
tumors, and MS. Hyperreflexia (+ Babinski, clonus)
Hypertonia
Main Menu Content Menu Legend Exit
15. R L
Lesion of the right lateral
spinothalamic tract at L1
produces what impairment?
Click for answer
Damage to the right lateral spinothalamic tract at L1
causes the absence of pain and temperature
sensation in the left leg.
Click for explanation
Main Menu Content Menu Legend Exit
16. Right Lateral Spinothalamic Tract Lesion
Click to animate
DRG
R L
L1
Lateral spinothalamic tract lesion
Common causes Contralateral loss of pain
include MS, and temperature sense
penetrating injuries,
and compression
from tumors.
Main Menu Content Menu Legend Exit
17. R L
Lesion of the anterior gray and
white commissures (central
cord syndrome) at C5-C6
produces what impairment?
Click for answer
Damage to the anterior gray and white commissures at
C5-C6 causes the absence of pain and temperature
sensation in the C5 and C6 dermatomes in both upper
extremities.
Click for explanation
Main Menu Content Menu Legend Exit
18. Central Cord Syndrome
Click to animate
C5-C6 DRG
R L DRG
Lateral
Spinothalamic
Tract
Common causes
include posttraumatic Impaired pain and temperature
contusion and sensation, C5-C6 dermatomes,
syringomyelia, and
bilaterally
intrinsic spinal cord
tumors.
Main Menu Content Menu Legend Exit
19. R L
Complete transection of the right
half the spinal cord (Hemicord or
Brown-Sequard syndrome) at L1
produces what impairments?
Click for answer
Damage to the right dorsal columns at L1 causes the
absence of light touch, vibration, and position sense in
the right leg. Damage to the lateral corticospinal tract
causes upper motor neuron signs in the right leg
(Monoplegia), and damage to the lateral spinothalamic
tract causes the absence of pain and temperature
sensation in the left leg.
Click for explanation
Main Menu Content Menu Legend Exit
20. Hemicord Lesion (Brown-Sequard Syndrome)
Click to animate
R L
L1
Hemicord lesion
Common causes
Dorsal column lesion
include penetrating
Ipsilateral loss of light touch,
injuries, lateral
vibration, and position sense
compression from
tumors, and MS. Lateral corticospinal tract lesion
Ipsilateral upper motor neurons signs
Build the lesion Lateral spinothalamic tract lesion
Contralateral loss of pain
and temperature sense
Main Menu Content Menu Legend Exit
21. Hemicord Lesion (Brown-Sequard Syndrome)
UMN
Click to animate
DRG
DRG
R L
L1
Dorsal column lesion
Ipsilateral loss of light touch,
vibration, and position sense
Lateral corticospinal tract lesion
Ipsilateral upper motor neurons signs
Lateral spinothalamic tract lesion
Contralateral loss of pain
and temperature sense
Hemicord lesion
Main Menu Content Menu Legend Exit
22. R L
Complete transection of the
spinal cord (Transverse cord
lesion) at L1 would produce
what impairments?
Click for answer
Damage to the dorsal columns, bilaterally, causes the
absence of light touch, vibration, and position sense in
the both legs. Damage to the lateral corticospinal tracts,
bilaterally, cause upper motor neuron signs in the both
legs (Paraplegia), and damage to the lateral
spinothalamic tracts, bilaterally, cause the absence of
pain and temperature sensation in the both legs.
Click for explanation
Main Menu Content Menu Legend Exit
23. Transverse Cord Lesion
Click to animate
R L
Transverse cord lesion
Common causes Dorsal column lesion
include trauma, Bilateral loss of light touch,
tumors, transverse vibration, and position sense
myelitis, and MS. Lateral corticospinal tract lesion
Bilateral upper motor neurons signs
Build the lesion Lateral spinothalamic tract lesion
Bilateral loss of pain and
temperature sense
Main Menu Content Menu Legend Exit
24. Transverse Cord Lesion UMN UMN
Click to animate
DRG
DRG
R L
Dorsal column lesion
Ipsilateral loss of light touch,
vibration, and position sense
Lateral corticospinal tract lesion
Ipsilateral upper motor neurons signs
Lateral spinothalamic tract lesion
Contralateral loss of pain
and temperature sense
Transverse cord lesion
Main Menu Content Menu Legend Exit
25. R L
Complete transection of the
dorsal columns, bilaterally,
(posterior cord syndrome) in
the cervical region would
produce what impairments?
Click for answer
Damage to the dorsal columns (fasciculus gracilis
and cuneatus), bilaterally, causes the absence of
light touch, vibration, and position sense, bilaterally,
from the neck down (below the lesion level).
Click for explanation
Main Menu Content Menu Legend Exit
26. Posterior Cord Syndrome
Click to animate
DRG
DRG
R L
Common causes Dorsal column lesion (bilateral)
include trauma, Bilateral loss of light touch,
compression from vibration, and position sense,
posteriorly located generalized below lesion level
tumors, and MS.
Main Menu Content Menu Legend Exit
27. Complete transection of the lateral R L
corticospinal and lateral spinothalamic
tracts with sparing of the dorsal
columns, bilaterally, (anterior cord
syndrome) in the cervical region would
produce what impairments?
Click for answer
Damage to the lateral corticospinal tracts cause upper motor
neuron signs, bilaterally, below the lesion level. Damage to
lower motor neurons in the ventral horns cause lower motor
neuron signs, bilaterally, at the lesion level. Damage to the
lateral spinothalamic tracts cause absence of pain and
temperature sensation, bilaterally, below the lesion level.
Sparing of the dorsal columns leaves light touch, vibration,
and position sense intact throughout.
Click for explanation
Main Menu Content Menu Legend Exit
28. Anterior Cord Syndrome UMN
UMN
Click to animate
DRG
DRG
R L
Anterior cord lesion
Common causes Lateral corticospinal tract lesion
include anterior Ipsilateral upper motor neurons signs
spinal artery
infarct, trauma, Lateral spinothalamic tract lesion
and MS. Contralateral loss of pain
and temperature sense
Main Menu Content Menu Legend Exit
30. Case Instructions
• These patient cases are intended to facilitate the
integration and clinical application of information
about lesions of the spinal cord by coupling the
findings on examination and patient interview with
their neuroanatomical correlates.
• Cases are presented from two perspectives. What
lesion would account for a given set of examination
results and patient history? For a given lesion, what
signs and symptoms would be expected on
examination?
• Click on a Case number to begin the exercise.
Main Menu Case Menu Exit
31. Review Questions: Case 1
The patient complains of “clumsiness” of her left leg due to uncertainty of the
limb’s position in space. Active and passive ROM and strength are within
normal limits (WNL) throughout. Light touch, two-point discrimination,
proprioception, and vibration sense are intact in the right lower extremity but
absent in all dermatomes below the umbilicus in the left lower extremity. She
is able to distinguish sharp from dull WNL in lower extremities, bilaterally.
Damage to what system(s) is causing this patient’s problems? Answer
Lesion of the left dorsal column (fasciculus gracilis) at approximately T10.
Lateral corticospinal tracts are intact, bilaterally: AROM and strength are WNL
Lateral spinothalamic tracts are intact, bilaterally: sharp/ dull is WNL
Dorsal column is intact on the right: light touch, two-point discrimination,
proprioception, and vibration are WNL
Dorsal column is absent on the left: light touch, two-point discrimination,
proprioception (limb position in space), and vibration are absent in all
dermatomes below the umbilicus
Lesion level, T10: the umbilicus is located in the T10 dermatome
Main Menu Case Menu Exit Show lesion
32. Left Dorsal Column Lesion
Click to animate
DRG
R L
T10
Dorsal column lesion
Ipsilateral loss of light touch,
vibration, and position sense
Main Menu Case Menu Exit
33. Review Questions: Case 2
After a fall from his horse, the patient was alert and oriented but unable to move
anything but his head. He was unable to sense light touch or pain from the neck
down. He could turn his head but shoulder shrug was weak. Speech was
normal but respiration was labored and required a respirator.
Damage to what system(s) is causing this patient’s problems? Answer
Complete transection of the spinal cord (transverse lesion ) at approximately C3
(Tetroplegia, Christopher Reeve)
Lateral corticospinal tracts absent, bilaterally, below C3: unable to move any
body part except head and shoulder shrug (C3-5)
Dorsal columns absent , bilaterally, below C3: unable to sense light touch below
neck
Lateral spinothalamic tracts absent, bilaterally, below C3: unable to sense pain
below neck
Lesion level, C3: patient was alert and oriented (cortex and reticular activating
system intact), he could turn his head (spinal accessory nerve), shoulder shrug
and respiration were weak (shoulder elevator and respiratory muscles C3-5)
Main Menu Case Menu Exit Show lesion
34. Transverse Cord Lesion UMN UMN
Click to animate
DRG
DRG
R L
C3
Dorsal column lesion
Ipsilateral loss of light touch,
vibration, and position sense
Lateral corticospinal tract lesion
Ipsilateral upper motor neurons signs
Lateral spinothalamic tract lesion
Contralateral loss of pain and
temperature sense
Transverse cord lesion
Main Menu Case Menu Exit
35. Review Questions: Case 3
Following surgical repair of a knife wound the patient is unable to stand or walk because
he is unable to move or bear weight on his right leg. Light touch, position and vibration
sense are WNL in the left lower extremity but absent in the right below the crest of the
ilium. Active range of motion and strength are normal in the left lower extremity but
absent in the right (hip, knee, and ankle). Pain and temperature sensation are intact in
the right lower extremity but absent in the left below T12.
Damage to what system(s) is causing this patient’s problems? Answer
Hemisection of the spinal cord on the right at approximately L1
Dorsal column is intact on the left but absent on the right: light touch, position
and vibration sense are WNL in the left lower extremity but absent in the right
Lateral corticospinal tract is intact on the left but absent on the right: active
range of motion and strength are normal in the left lower extremity but absent in
the right
Lateral spinothalamic tract is intact on the left but absent on the right: pain and
temperature sensation are intact in the right lower extremity but absent in the left
Lesion level, approximately L1: hip flexion absent on right (L2), pain and
temperature sense absent below T12
Main Menu Case Menu Exit Show lesion
36. Hemicord Lesion (Brown-Sequard Syndrome)
UMN
Click to animate
DRG
DRG
R L
T12
Dorsal column lesion
Ipsilateral loss of light touch,
vibration, and position sense
Lateral corticospinal tract lesion
Ipsilateral upper motor neurons signs
Lateral spinothalamic tract lesion
Contralateral loss of pain and
temperature sense
Hemicord lesion
Main Menu Case Menu Exit