This document summarizes a study investigating modulating neuronal transmission pathways in the amygdala to potentially reduce amygdala dysfunction. The study hypothesizes that controlling abnormal neuronal activity, like modifying signal transmission speeds between the amygdala and frontal cortex, could balance frontal cortex activity and reduce amygdala overactivation, similar to modulating cardiac signals to reduce arrhythmias. Specific diagnostic tests and pharmacological molecules may help evaluate physiological changes in neuronal transmission and modulate conditions. The communication intends to generate research hypotheses, not diagnose or treat.