The document provides details about conducting a neurological assessment, including subjective and objective components. The subjective assessment involves collecting information from the patient about their medical history, complaints, and social history. The objective assessment consists of observing the patient, palpating areas, and performing examinations of things like cranial nerves, motor function, reflexes, coordination, and other body systems. The assessment is used to formulate a problem list and provisional diagnosis to guide the patient's treatment plan.
Mental function examination is a part of Neurologic and Psychiatric examination as an emergency and as an outpatient clinic.
Detail Mental examination is required for cases of Dementia in various neurological diseases.
This set of slides are not for Psychiatric patients with disturbance of thought and mood.
NDT, BOBATH TECHNIQUE, BASIC IDEA OF BOBATH, CONCEPT OF BOBATH, NEUROPHYSIOLOGY OF NDT, ICF MODEL, PRINCIPLES OF TREATMENT OF NDT IN STROKE AND CP, AUTOMATIC AND EQUILIBRIUM REACTIONS, KEY POINTS OF CONTROL, FACILITATION, INHIBITION AND HANDLING IN NDT
This is the presentation which was delivered to third year Bachelor of Physiotherapy students at Kathmandu University School of Medical Sciences (KUSMS), Dhulikhel, Nepal. Different schools of thoughts in manual therapy are the part of curriculum for the undergraduate students at KUSMS.
This scale is used to categrise spinal cord injury patients. it helps prognosticate the spinal cord injuires. it also helps define the treatment protocols for spinal cord injury patients. American Spinal Cord Injury Association made this scale so as to make a standardization in assesemnent technique in acute spinal cord injury patients.
Physiotherapy in MND
Dr. Quazi Ibtesaam Huma (MPT)
Dr. Suvarna Ganvir (Phd, Prof & HOD)
Dept. of Neurophysiotherapy
DVVPF’s College of Physiotherapy
Content
Introduction
Types of MND
Clinical Features of MND
Diagnostic Procedure
Management: 1) Pharmaceutical
2) Physiotherapy
Motor Neuron Disease
Motor Neuron Disease are a group of neurodegenerative disorders that affects the nerves in the spine and brain to progressively lose its function.
Motor neuron diseases (MND) include a heterogeneous spectrum of inherited and sporadic (no family history) clinical disorders of the upper motor neurons (UMNs), lower motor neurons (LMNs), or a combination of both.
Types of MND
Amyotrophic Lateral Sclerosis
Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disease, characterized by progressive degeneration of motor neurons in the spinal cord, brain stem, and motor cortex, leading to progressive muscle atrophy and weakness.
Clinical Features
UPPER MOTOR NEURON
Loss of Dexterity
Muscle Weakness
Spasticity
Hyperreflexia
Pathological reflexes
LOWER MOTOR NEURON
Muscle Weakness
Muscle Atrophy
Hypotonicity
Hyporeflexia
Fasciculation
Muscle Cramp
Impairment related to LMN
Other clinical features
Diagnostic Criteria
Diagnostic Procedure
EMG-
It include signs of active denervation, such as fibrillation potentials and positive sharp waves;
Signs of chronic denervation, such as large motor unit potentials (increased duration, increased proportion of polyphasic potentials, increased amplitude)
Unstable motor unit potential
Nerve Conduction Velocity Studies,
Muscle And Nerve Biopsies,
Neuroimaging Studies - MRI
Management- Multidisciplinary Approach
Physical Therapy Examination
Cognition
Pain
Psychosocial Function
Joint integrity, ROM and Muscle strength.
Motor Function: Gross motor and Fine motor
Muscle tone and reflexes
Cranial nerve integrity
Sensations
Gait
Respiratory Function
Physiotherapy goals in MND treatment.
Pain reduction
Prevention for contractures
Maintenance of joint mobility
Regular review of posture
Positioning to relieve discomfort
House Modification and ergonomic advice.
Management of Sialorrhea and Pseudobulbar Affect
Management for Dysphagia
PEG procedure.
A PEG may be recommended as the disease progresses.
A PEG is a type of gastrostomy tube inserted via endoscopic surgery that creates a permanent opening into the stomach for the introduction of food.
Studies have found that PEG insertion may prolong survival. Patients with PEG were found to live 1 to 4 months longer than those individuals who refused it.
Management of Dysphagia
A palatal lift prosthesis may be prescribed for individuals with good articulation but who have a breathy voice quality or decreased loudness because of excessive air loss through the nose.
The device, a dental appliance designed to attach to the existing teeth and to elevate the soft palate, is custom-made by a prosthodontist.
Mental function examination is a part of Neurologic and Psychiatric examination as an emergency and as an outpatient clinic.
Detail Mental examination is required for cases of Dementia in various neurological diseases.
This set of slides are not for Psychiatric patients with disturbance of thought and mood.
NDT, BOBATH TECHNIQUE, BASIC IDEA OF BOBATH, CONCEPT OF BOBATH, NEUROPHYSIOLOGY OF NDT, ICF MODEL, PRINCIPLES OF TREATMENT OF NDT IN STROKE AND CP, AUTOMATIC AND EQUILIBRIUM REACTIONS, KEY POINTS OF CONTROL, FACILITATION, INHIBITION AND HANDLING IN NDT
This is the presentation which was delivered to third year Bachelor of Physiotherapy students at Kathmandu University School of Medical Sciences (KUSMS), Dhulikhel, Nepal. Different schools of thoughts in manual therapy are the part of curriculum for the undergraduate students at KUSMS.
This scale is used to categrise spinal cord injury patients. it helps prognosticate the spinal cord injuires. it also helps define the treatment protocols for spinal cord injury patients. American Spinal Cord Injury Association made this scale so as to make a standardization in assesemnent technique in acute spinal cord injury patients.
Physiotherapy in MND
Dr. Quazi Ibtesaam Huma (MPT)
Dr. Suvarna Ganvir (Phd, Prof & HOD)
Dept. of Neurophysiotherapy
DVVPF’s College of Physiotherapy
Content
Introduction
Types of MND
Clinical Features of MND
Diagnostic Procedure
Management: 1) Pharmaceutical
2) Physiotherapy
Motor Neuron Disease
Motor Neuron Disease are a group of neurodegenerative disorders that affects the nerves in the spine and brain to progressively lose its function.
Motor neuron diseases (MND) include a heterogeneous spectrum of inherited and sporadic (no family history) clinical disorders of the upper motor neurons (UMNs), lower motor neurons (LMNs), or a combination of both.
Types of MND
Amyotrophic Lateral Sclerosis
Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disease, characterized by progressive degeneration of motor neurons in the spinal cord, brain stem, and motor cortex, leading to progressive muscle atrophy and weakness.
Clinical Features
UPPER MOTOR NEURON
Loss of Dexterity
Muscle Weakness
Spasticity
Hyperreflexia
Pathological reflexes
LOWER MOTOR NEURON
Muscle Weakness
Muscle Atrophy
Hypotonicity
Hyporeflexia
Fasciculation
Muscle Cramp
Impairment related to LMN
Other clinical features
Diagnostic Criteria
Diagnostic Procedure
EMG-
It include signs of active denervation, such as fibrillation potentials and positive sharp waves;
Signs of chronic denervation, such as large motor unit potentials (increased duration, increased proportion of polyphasic potentials, increased amplitude)
Unstable motor unit potential
Nerve Conduction Velocity Studies,
Muscle And Nerve Biopsies,
Neuroimaging Studies - MRI
Management- Multidisciplinary Approach
Physical Therapy Examination
Cognition
Pain
Psychosocial Function
Joint integrity, ROM and Muscle strength.
Motor Function: Gross motor and Fine motor
Muscle tone and reflexes
Cranial nerve integrity
Sensations
Gait
Respiratory Function
Physiotherapy goals in MND treatment.
Pain reduction
Prevention for contractures
Maintenance of joint mobility
Regular review of posture
Positioning to relieve discomfort
House Modification and ergonomic advice.
Management of Sialorrhea and Pseudobulbar Affect
Management for Dysphagia
PEG procedure.
A PEG may be recommended as the disease progresses.
A PEG is a type of gastrostomy tube inserted via endoscopic surgery that creates a permanent opening into the stomach for the introduction of food.
Studies have found that PEG insertion may prolong survival. Patients with PEG were found to live 1 to 4 months longer than those individuals who refused it.
Management of Dysphagia
A palatal lift prosthesis may be prescribed for individuals with good articulation but who have a breathy voice quality or decreased loudness because of excessive air loss through the nose.
The device, a dental appliance designed to attach to the existing teeth and to elevate the soft palate, is custom-made by a prosthodontist.
Diagnosis and Treatment of Psychosomatic Disorder (Educational Slides)Andri Andri
This is a standard presentation for teaching medical students and colleagues about psychosomatic disorder, its diagnosis and therapy. We hope by reading this slides, you will understand the nature of psychosomatic disorder and its current approach in therapy
1General status, vital signs, pain and nutrition Subjective d.docxfelicidaddinwoodie
1
General status, vital signs, pain and nutrition Subjective data
Student Name________________
(No patient names or initials allowed).
Submit using Word, with a .doc or .dox suffix; do not use .odt because the forms cannot be graded in that format—this goes for the assignments in all the upcoming weeks for this class.
NOTE: YOU MAY NOT USE A PATIENT FROM YOUR WORKPLACE FOR THIS ASSESSMENT. WE DO NOT WANT YOU TO VIOLATE HIPAA!
Questions
Findings
Current Status
1. Allergies
2. Present health concerns
3. Current medications (prescribed and over-the-counter)
4. Immunizations
Past History
5. Medical
6. Surgical
7. Hospitalizations
8. Injuries
Family History
9. List family medical concerns for 3 generations
Pain
(Everyone has had pain at some time or other-if your patient is healthy and currently pain-free, you may need to use a past instance of pain.)
10. Pain (using COLDSPA)
Character: how does it feel—what sort of pain is it?
11. Onset:
12. Location:
13. Duration:
14. Severity (scale of 1 – 10):
15. Pattern—what makes it better or worse:
16. Associated factors—does it cause you to have other symptoms too?
18. How does pain impact the other areas of life?
2. What are your concerns about the pain’s effect on
a. general activity?
b. mood/emotions?
c. concentration?
d. physical ability?
e. work?
f. relations with other people?
g. sleep?
h. appetite?
i. enjoyment of life?
Lifestyle and Health Practices
What types of recreation or physical exercise?
Duration of exercise periods, how many times per week?
Stress: Rate overall life stress on a scale of 1 – 10 (1 being least, 10 most). What are the greatest sources of stress?
Methods of coping with stress?
Use of tobacco, alcohol, recreational drugs
Sleep—typical hours per night
Objective data (General status and vital signs, pain and nutrition)
Adapted from Weber, Kelly & Sprengel, 2014: Lippincott, used with permission.
Questions
Findings
Current Status
1. Observe physical development (i.e., appears to be chronologic age).
2. Observe skin (i.e., general overall color, color variation, and condition).
3. Observe dress (occasion and weather appropriate).
4. Observe hygiene (cleanliness, odor, grooming).
5. Observe posture (i.e., erect and comfortable) and gait (i.e.,rhythmic and coordinated).
6. Observe general body build (muscle mass and fat distribution).
7. Observe consciousness level (alertness, orientation, appropriateness).
8. Observe comfort level-does patient exhibit visible signs of pain?
9. Observe behavior (body movements, affect, cooperativeness, purposefulness, and appropriateness).
10. Observe facial expression (culture-appropriate eye contact and facial expression).
11. Observe speech (pattern and style).
Vital Signs
12. Temperature (document route)
13. Heart rate (pulse-- rhythm, amplitude)
(Document units—beats per minute)
14. Respirations (rate, rhythm, and depth).
(Document units—breaths pe ...
Hemiparesis is a condition characterized by weakness or paralysis on one side of the body, typically resulting from damage to the brain or spinal cord. In a case presentation, it is essential to provide a comprehensive overview of the patient's history, including any relevant medical conditions or events such as stroke, traumatic brain injury, or tumor. Additionally, outlining the physical examination findings, such as decreased strength, altered reflexes, and possible sensory deficits on the affected side, aids in diagnosing and assessing the severity of hemiparesis. Diagnostic tests like brain imaging studies (CT or MRI) and electrophysiological evaluations may also be included to confirm the underlying cause and guide treatment strategies, which often involve a multidisciplinary approach focusing on rehabilitation, medication, and supportive care to improve functionality and quality of life for the patient.
Anti ulcer drugs and their Advance pharmacology ||
Anti-ulcer drugs are medications used to prevent and treat ulcers in the stomach and upper part of the small intestine (duodenal ulcers). These ulcers are often caused by an imbalance between stomach acid and the mucosal lining, which protects the stomach lining.
||Scope: Overview of various classes of anti-ulcer drugs, their mechanisms of action, indications, side effects, and clinical considerations.
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Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
2. INTRODUCTION:
Assessment:
“The art of evaluation,judgement and gauging
a given situation in the most efficient way
possible.
In the medical scenario, the above means the same, with
respect to a patient’s medical condition and its
subsequent treatment plan.
3. Medical assessment is divided
into 2 parts:
A.Subjective Assessment: Information from client’s point
of view, including concerns, feelings, thoughts and
perceptions, by the form of a 2-way interview.
B.Objective Assessment: A set of facts, representing
patient’s current health status based upon various forms
of observation, examination and carefully drafted
techniques that help the medical professional in getting
details about the patient.
4. A.Subjective assessment:
Demographic data:
Name:
Age/Gender:
Address:
Contact Information:
Patient Status: Conscious/Semi-Conscious/Unconscious.
(Semi-Conscious states include: Pseudocoma (Locked-in
Syndrome), Catatonia, Delirium, Dementia, Amnesia.)
Chief Complaints: As narrated by the patients, according to
his/her vocabulary, comfort and time.
5. These include:
Inability to- move one/both, UL or LLof one/both sides.
Inability to- move eyes, mouth, eat, chew, hear.
Problems with- balance, coordination, equilibrium,
headache Convulsions, spells or any other sensory
disturbances.
Weakness/pain in one/both UL or LL.
Synergy Patterns. (Primitive movements that dominate reflex and
voluntary effort when spasticity is present following a
cerebrovascular accident.)
6. History
:
H/o present illness:Allowed to narrate according to him/her.
But following pointers should be noted:
Onset/Duration
. Mental State.
Speech
Disturbances.
Consciousness.
Special senses.
Headache
mentions. Sleep
Pattern..
Movement
.
7. Past Medical History:
History of the following:
llnesses: Hereditary, Congenital, Infectious, Rheumatic
etc. Surgery, Trauma.
Travel History.
Vaccination Status.
History of Blood
Transfusion. History of
Childbirth.
History of past/ongoing
medications. History of any
Allergies.
8. Personal History:
Residence: Type of house patient is living
in. Marital History.
Occupation and Occupational
Hazards. Educational Status.
Habits/Addictions (Tea,Coffee, Tobacco, Alcohol, Marijuana, Narcotics
and other Psychoactive drugs).
Hours of Sleep and Exercise.
9. Family History:
Type of Family
(Nuclear/Joint). No. of
Members.
Consanguinity.
Other diseases running in the family
(HTN,DM,IHD,Anxiety, Schizophrenia)
10. Socioeconomic History:
Economic Status. (Income Information, about if the patient
is above/below B.P.L.)
Housing Status. (Hut, Makeshift house, Single/Double storey
house, Apartments, mansion etc.)
Nutritional Status.(Helps in assessment of nutrition deficiency
disorders) Other Social Problems.
Type of locality the Patient resides in. (Slums, Gated
colonies, Apartment complex etc.)
11. H/o Pain: SOCRATES mnemonic.
Site and Side– Where is the pain? Or the maximal site of the pain.
Onset – When did the pain start, and was it sudden or gradual?
Include also whether it is progressive or regressive.
Character – What is the pain like? An ache?
Stabbing? Radiation – Does the pain radiate
anywhere?
Associations – Any other signs or symptoms associated with the
pain? Time course – Does the pain follow any pattern?
Exacerbating/relieving factors – Does anything change the
pain? Severity – How bad is the pain?
12. Birth History: Natural Labour/C-section/TOLAC
Developmental History: Proper appearance and disappearance
of appropriate developmental milestones.
16. -ON OBSERVATION: Following pointers are seen;
● Ambulatory status
(Independent/Dependent/using appliances)
● Ventilatory status (Independent/Dependent)
● General
condition(Normal/Distressed/Indifferent/Belligerent
)
● Gait (Normal/Abnormal)
● Facial Symmetry
● Built (Endomorphic/Mesomorphic/Ectomorphic)
● Postural Assessment (In all views, Ant., Post.,
Lateral.)
17.
18. ● External Appliances (Sensory Aids, Mobility
Aids, Ventilator, Catheter)
● Attitude of the limb (In all positions)
● Presence of :
Edema, Rashes, Any deformity, Scars,Bandages,Open
wounds, Abnormal Bony Contours,Abnormal Skin
Colour.
● Speech (Normal/Abnormal)
● Sores (Bed/Decubitus/Vascular/Diabetic)
● Movements (Presence of any abnormal movements).
19. -ON PALPATION:
● Muscle Tone (Normal/Abnormal).
● Swelling (Blood, Synovial Fluid, Callus,
Acute, Chronic, Edematous).
● Skin:
Trophic changes (Dry, Moist, Oily,
Scaly) Temperature
Scar
● Tenderness (By Grading).
● Pain (By VAS or NPRS)
22. -ON EXAMINATION: Consists of the
thorough examination of the following:
● General Examination.
● Higher Mental Functions.
● Sensorium.
● Motor part.
● Reflexes.
● Coordination.
● Equilibrium.
● Balance.
● Functional.
● Other Systems of the Body.
30. Sensory
assessment:
● Superficial – touch, pain, temperature, pressure
● Deep – joint movement sense, joint position sense, vibration
● Combined – tactile localization, 2 point discrimination
stereognosis, barognosis, graphesthesia
31.
32.
33.
34.
35. Sensory grading
1-intact – normal accurate
response. 2-decreased – delayed
response.
3exaggerated – increased sensitivity or awareness of stimulus
after it has ceased.
4inaccurate – inappropriate perception to given
stimulus. 5-absent – no response.
6-inconsistent or ambiguous – response inadequate to assess.
36.
37.
38. ● Motor Assessment:
-Movement Evaluation: ROM (AROM & PROM)
End Feel
Capsular
Patterns MMT
Limb Length Measurement
Joint Position (Open&Close
Packed)
-Functional Evaluation: Postural Changes
ADL
Gait
LOOK FOR SYNERGYPATTERNS(Primitive movements
that dominate reflex and voluntary effort when spasticity
is present following a cerebrovascular accident)
41. ● Reflex Examination:
a. Primitive Reflexes: Reflex actions originating in the central
nervous system that are exhibited by normal infants, but
not neurologically intact adults, in response to particular
stimuli. These reflexes are suppressed by the
development of the frontal lobes as a child transitions
normally into child development.
Older children and adults with atypical neurology (e.g.,
people with cerebral palsy) may retain these reflexes and
primitive reflexes may reappear in adults. Reappearance may
be attributed to certain neurological conditions including
dementia (especially in a rare set of diseases called
frontotemporal degenerations), traumatic lesions, and strokes.
44. c. Deep Tendon Reflexes
Biceps (C5,C6)
Triceps (C7,C8)
Patellar (L2,L3,L4)
Hamstrings (L5,S1,S2)
Ankle (S1,S2)
d. Also check: Babinski sign, for ruling out UMN
lesion.
45.
46. ● Coordination assessment:
a. Non equilibrium:
Finger to nose
Finger to therapist
finger Finger to finger
Alternate nose to finger
Finger opposition
Mass grasp Pronation / Supination
Rebound Tapping (hand & foot)
Pointing and past pointing
Alternate heel to knee & heel to
toe Toe to examiner’s finger
Heel on shin
Drawing a circle (hand & Foot)
Fixation / position holding (UL&
LL)
47. Grading
5- Normal performance
4- minimal impairment – able to accomplish activity with
slightly less than normal speed and skill
3- moderate impairment – able to accomplish activity but
coordination deficits very noticeable movements are
slow, awkward and unsteady
2 – severe impairment – able only to initiate activity
without completion
1-Activity impossible
48. ● Equilibrium:
Standing – normal
posture Standing – vision
occluded Standing – feet
together Standing on one
foot
Standing – forward trunk flexion and return back to
neutral
Standing – lateral trunk
flexion Walk – tandem
walking
Walk – along a straight line
Walk – place feet on foot
marks Walk – sideways
Walk – backward
49. Grading
4- able to accomplish activity
3 – can complete activity, needs minor help to
maintain balance
2- Can complete activity with moderate to maximal
help 1-Activity impossible
51. Apart from Berg Balance scale, Functional balance grading
can be used. The grading is as follows:
Normal – able to maintain balance without support.
Accepts maximal challenge and can shift weight in all
directions.
Good – able to maintain balance without support. Accepts
moderate challenge and can shift weight although limitations
are evident
Fair – able to maintain balance without support cannot tolerate
challenge cannot maintain balance while shuffling weight Poor
– patient requires support to maintain balance
52. ● Functional Exam:
Done by FIM (Functional Independence Measure) scale, which is
a tool that explores an individual's physical, psychological and
social function. The tool is used to assess a patient's level of
disability as well as change in patient status in response to
rehabilitation or medical intervention.
53.
54.
55. ● Other body systems that should be examined are as
follows:
1. Integumentary system
2. Musculoskeletal system
3. Bowel and Bladder activity
4. Autonomic Changes
56. After following all above procedures, a PROBLEM LISTis
formulated, with patient’s inputs.
This is followed by formation of a PROVISIONALDIAGNOSIS
with DIFFERENTIAL DIAGNOSIS.
Post this the patient is referred for higher centres for:
-Biochemical/Hematological Investigations.
-Radiological Investigations.
Then, a FINAL DIAGNOSIS is made.