Muscles of mastication /certified fixed orthodontic courses by Indian dental ...Indian dental academy
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The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
Indian dental academy provides dental crown & Bridge,rotary endodontics,fixed orthodontics,
Dental implants courses.for details pls visit www.indiandentalacademy.com ,or call
0091-9248678078
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
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Growth and development of mandible in childrenDr. Harsh Shah
a brief idea about the development of mandible for indian students looking for a quick review from dentistry department
all the best to students
Presented by : Harsh SHah
Dept. of Orthodontics
SDDCH PBN
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
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ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
New Drug Discovery and Development .....NEHA GUPTA
The "New Drug Discovery and Development" process involves the identification, design, testing, and manufacturing of novel pharmaceutical compounds with the aim of introducing new and improved treatments for various medical conditions. This comprehensive endeavor encompasses various stages, including target identification, preclinical studies, clinical trials, regulatory approval, and post-market surveillance. It involves multidisciplinary collaboration among scientists, researchers, clinicians, regulatory experts, and pharmaceutical companies to bring innovative therapies to market and address unmet medical needs.
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
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- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
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Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
2. GROWTH
Growth, according to Todd, it’s the increase in size
According to Krogmann, it’s the increase in size, change
in proportion, & progressive complexity
2
GROWTH & DEVELOPMENT
3. According to Todd, Development is the progress
towards maturity.
According to Moyers ,all naturally occurring
progressive, unidirectional sequential change occurring
in the life of an individual as its existence as single cell
to its elaboration as a multifactorial unit , terminating
towards death
3
DEVELOPMENT
4. Bones of the base of the skull /cranial base are formed
initially in cartilage &are later transformed by
endochondral ossification to bone.
Early in embryonic life , centers of ossification appears in
chondrocranium , indicating the eventual location of
basioccipital, sphenoid , & ethmoid bones that form cranial
base
4
7. ossification proceeds bands of cartillage called
Synchondroses remain b/w the centers of
ossification.
as
7
Remains of primary cartilaginous skeleton of cranial
base
Bands of cartillage present b/w sphenoid, ethmoid &
occipital bones
Form important growth sites at the base of the skull
8. Types of Synchondroses
Intersphenoidal Fuses at birth
Intraoccipital Fuses at 3-5 yrs
Spheno-occipital Fuses at 20 yrs
Spheno-ethmoidal exactly not known
8
Cranial base grows by cartilaginous growth in Synchondroses
which later get calcified
10. Fig. showing growth at the synchondrosis. A band of immature
proliferating cartilage cells is located at the center of synchondrosis,
while the band of mature cartilage cells proliferate on both sides, away
from the center & endochondral ossification takes place on both
margins. Growth at synchondrosis lengthens this area of synchondrosis10
11. Spheno occipital Synchondroses are responsible
for most of the lengthening of the cranial base
b/w foramen magnum & Sella turcica postnatal
which in turn helps in the lengthening /growth
of the Naso maxillary complex
11
12. Morphologically, a synchondrosis is similar to the long
bone growth plate, except that growth at the
synchondrosis is not unipolar , but bipolar.
The synchondrosis can be regarded as two growth
plates positioned back to back so that they share a
common zone of actively proliferating chondroblasts,
or the “rest zone”.
12
HISTOLOGY OF SYNCHONDROSES
13. The different zones of the synchondrosis mirror each other
such that there is cartilage in the centre and bone at each end.
Synchondroses is an area of cellular hyperplasia &
hypertrophy in the center with bands of maturing cartilage
cells extending in both directions which eventually being
replaced by bone.
13
14. 14
Fig. shows histologic appearance of
synchondrosis with ossification taking place
on both sides of primary cartillage
15. Cranial base flexion is a unique cranial feature of
modern human beings and also a reflection of brain
evolution
Fusion along the Spheno-occipital Synchondroses is
believed to be responsible for cranial base flexion,
which develops in concert with the development of the
upper airway and the ability to vocalize.
15
CRANIAL BASE ANGLE
16. During development, the anterior and posterior cranial base
flexes at the sella turcica in the middle sagittal plane and thus
constitutes an angle in the cranial base, termed the cranial
base angle or saddle angle
16
17. Abnormal growth of cranial base can result in
severe dentofacial deformity eg – Craniofacial
Dysostosis
An obtuse cranial base angle increases the
depth of maxilla & causes Mandibular
retrognathism & vice versa
17
Clinical implications of growth of cranial base
18. 18
An Obtuse cranial Base
angle causing Maxillary
Prognathism
An Acute Cranial Base
angle causing Mandibular
Prognathism
20. Enlow’s Counterpart Principle
Counterpart principle of craniofacial growth states
that the growth of any given facial/cranial part
relates specifically to other structural counterparts
in face & cranium.
cranial base growth have effect on maxillary &
mandibular growth .
Maxillary growth is based on growth of Anterior
Cranial Fossa
20
21. Similarly width of pharyngeal cavity depends on
width of middle cranial fossa
Width of Mid-cranial fossa is same as that ramus
width
ANS- Posterior Nasal Spine length of maxilla
determine the length of Corpus of mandible
21
22. Maxillary tuberosity determining the width of lingual
tuberosity
Posterior cranial Fossa determine the mandibular
position
Amount , direction & magnitude of cranial base
determine the amount , direction & magnitude of
maxilla & mandible
22
23. POSTNATAL DEVELOPMENT OF MAXILLA
1. Zygomatico-
maxillary
2. Zygomatico-
frontal
3. Intermaxillary
4. Frontomaxillary
Connective tissue growth “Bone fill at the space”
23
RemodelingGrowth at sutures Translation
Active growth at
tuberosity
Periosteal
matrix
funtion
Deposition/
resorption
Passive
Forward &
downward
Capsular
matrix
Nasal
septum
25. Internal displacement of Nasomaxillary complex
itself due to growth .
It’s the movement of bone surface caused by
deposition & resorption towards the depository
surface .Its otherwise called Transformation
25
DRIFT
26. Displacement is the growth of bone as a whole unit
so that the bone is taken away from its articulation
with other bones.
Also called as Translation
26
DISPLACEMENT
27. Maxilla articulate with the surrounding bone with
help of sutures.
Sutures incl. Zygomaticomaxillary,
Frontomaxillary, Pterygopalatine ,
Zygomaticotemporal etc..
According to sicher
Growth in sutures Move
maxilla downwards & forwards
27
CONNECTIVE TISSUE PROLIFERATION
28. But it’s only a secondary & not a primary mechanism
28
30. Translation/Displacement is process by which
specific local areas come to occupy new actual
positions in succession as entire bone enlarges.
2 types
30
Active/Primary
Passive/Secon
dary
31. Active displacement takes place when the growth at
the tuberosity of the maxilla pushes the maxilla
forward.
Passive displacement takes place when maxilla grows
downward & forward by the growth of the Spheno-
Occipital Synchondrosis of the cranial base /growth of
nasal septum.
31
Active/Primary Displacement
Passive/Secondary Displacement
32. Passive displacement also takes place when the
maxillary bone is translated in space by the growth
of corresponding capsular matrices
Three main capsules w.r.t Nasomaxillary complex
32
ORBITA
L
NASAL ORAL
33. In Remodeling simultaneous resorption and
deposition of the maxilla while maintaining the
integrity & shape of bone.
Maxillary growth matures first in Width followed
by Length & Height
Width across the 2nd molar & 3rd molar increases
until the end of growth in len`gth
33
REMODELLING
34. Midpalatal suture is active up to 15 yrs. There is
bone fill in the midpalatal area due to sutural growth
resorption in lateral aspect.
In case of Maxillary sinus sinus
enlarges Resorption on inner aspect
& deposition on outer aspect
34
MAXILLARY WIDTH
35. There is removal of bone from periosteum, lining
the inner aspect of the nasal cavity & deposition
takes place in the endosteal surface , allowing the
expansion of nasal cavity
35
NASAL CAVITY
36. In the antero-posterior direction there is growth by
apposition in the posterior tuberosity area so that
there is increased space for permanent teeth.
As the maxilla moves forward , there is resorption
of the anterior surface of the periosteum from ANS
to alveolar margin incisors, result
Concave anterior margin
Deposition occurs in the ANS to make it make
prominent
36
ANTERO-POSTERIOR DEPTH
37. Resorption anterior region &
Apposition posterior region
of zygomatic bone –result Translation
of zygomatic bone posteriorly
37
39. In vertical direction maxillary bones increase in height by
apposition along the alveolar process
This increase is seen as long as the teeth erupt
This contributes early increase in height
of maxilla
39
MAXILLARY HEIGHT
40. 40% of the maxillary height is achieved by this.
Resorption Palatal surfaces &
Deposition Palatal roof
End result Downward shift of palate
40
41. Follows the concept of expanding ‘V’ by Enlow
41
POSTNATAL GROWTH OF PALATE
Bone deposition inner aspect of ‘V’
Direction of growth Wide end of ‘V’
Periosteal surface lined by Osteoclasts
Endosteal surface lined with Osteoblasts
43. Growth in width is completed first then length &
height
Growth in width of Jaws & Dental arches completed
before adolescent growth spurt
43
TIMING OF GROWTH IN WIDTH,
LENGTH & HEIGHT
44. As the jaws grow in length posteriorly, they also
increase in width
for the mandible, both molar & bicondylar widths
shows small increase until end of growth in length
Growth in length & height of jaws continues through
the puberty .
44
45. In both sexes , growth in vertical height of face
continues longer than growth in length, with the
late vertical growth in mandible.
Increase in facial height & concomitant eruption
of teeth continue throughout the life, but decline
to adult life
45
46. POSTNATAL GROWTH OF
MANDIBLE
Mandible at birth is much smaller in size & there is
slight variation in shape from the adult form
Infant mandible has a short more or less horizontal
ramus with Obtuse Gonial angle
Mandibular growth continues at relatively steady
rate before puberty.
46
47. The condyles are low & at the position along the
occlusal plane .
Symphyseal suture has not yet ossified
47
49. o Growth in the first year involves growth at the
symphyseal suture & lateral expansion in the
anterior region to accommodate the erupting the
teeth .
o Mental foramen is directed at right angles to the
surface of the corpus.
o There is increased deposition in the posterior
surface of ramus of mandible.
49
GROWTH IN THE FIRST YEAR
50. MANDIBLE IN THE ADULT
Mandible in the adult is different from the mandible
of an infant.
Ramus is longer & gonial angle is less obtuse.
50
52. All those changes taking place with the growth of
mandible is in the form of expanding V.
It’s easier to visualize mandible as V-shaped bone
than maxilla because of it’s horseshoe shape.
52
V- PRINCIPLE OF GROWTH
55. Lateral of ramus Deposition &
Lingual surface Resorption
of mylohyoid ridge
Coronoid process Apposition
its Lateral surface Resorption
Condyle Resorption at
lateral aspect
55
GROWTH IN WIDTH
56. Thus Inter-ramal distance is efficiently increased by
the growth of mandible following the V- Principle
The growth of mandible in length A-P is by the
deposition of bone at the posterior surface of the
Ramus &
Resorption at the Anterior surface
56
GROWTH IN LENGTH
57. This helps lengthen the mandible
anterior part of the ramus is occupied by posterior
part of the body in the future to Accommodate
permanent molars
57
Deposition - +
Resorption - -
58. Alveolar process height correlates well with the
eruption of teeth
Bone deposition taking place in the lower
border of mandible also contributes to increase
in height of the mandible
58
GROWTH IN HEIGHT
59. Arne Bjork et.al , Dept. Of Orthodontics, Royal
dental college , Copenhagen, Denmark performed
longitudinal Radiographic study by Implant method
for studying Jaw rotations
Longitudinal study involved about 110 Danish
children of 7 yrs. to 18 yrs old.
59
GROWTH ROTATIONS
61. 61
3 types of metallic
implants tested
A)
Kirschner
wire
B)
Cr-Co
Alloy
C)
Tantalum
Wire
62. 62
For the radiographic profile analysis of mandibular growth, one implant was
inserted in the mid-line of the symphysis, and three on the right side nearest the
film: under the first and second premolars, and in the external aspect of the
ramus on a level with the occlusal lines.
63. condition Bjork Proffit
Rotation of
mandibular core
relative to
Cranial Base
Total rotation Internal rotation
Rotation of
Mandibular
plane relative to
Cranial base
Matrix rotation Total rotation
Rotation of
Mandibular
plane relative to
Intramatrix
rotation
External
rotation63
TERMINOLOGY OF ROTATIONAL CHANGES OF JAWS
64.
Total Rotation = Internal Rotation - External Rotation
Relationship b/w Matrix, Total & Intramatrix
Rotation (Bjork)
Matrix Rotation = Total Rotation – Intramatrix
Rotation
64
Relationship b/w Total , Internal &
External rotation (Proffit)
65. Bone that surrounds the inferior alveolar nerve
& the rest of the mandible consists of its
functional processes
Functional processes incl. muscular processes
, the condylar process, functions incase being
the articulation of the jaw with the skull.
65
CORE OF THE MANDIBLE
67. If implants are placed in areas of stable bone
away from the functional processes, it can be
observed that In most individuals , the core of
the mandible rotates during growth in a way
that tend to decrease the mandibular plane
angle
(i.e up anteriorly & down posteriorly)
67
68. Bjork & Skieller distinguished 2 contributions to Internal rotation(
Total rotation) of the mandible
Matrix Rotation/rotn around Condyle
Intramatrix Rotation/rotn centered
within the body of the mandible
68
Total
rotation
69. Variation of internal rotation of mandible b/w
individuals, ranging up to 10 to 15 degrees.
For an average individual with normal vertical
facial height there is about -15 degrees internal
rotation from age 4 – adult life
25% - Matrix rotation &
75% - Intramatrix rotation69
70. When the core of the mandible rotates forward an average
of 15degrees, orientation of jaw from outside
decreases only 2 – 4 degrees(av..)
Internal rotation is not expressed in jaw orientation , surface
changes tends to compensate i.e. ,posterior part of lower
border of mandible may be the area of resorption, while
anterior aspect of lower border is unchanged / little
apposition
70
Reason
72. SHORT FACED INDIVIDUALS/
FORWARD ROTATORS
These individuals are characterized by short anterior
lower facial height
Excessive forward rotation of mandible, due to an
increase in normal internal rotation & a decrease in
external compensation
72
73. Square type jaw + Low mandibular plane
angle+ Square Gonial angle +skeletal Deep bite
malocclusion + crowded Incisors
Muscles much stronger they mature early
Space closure is very difficult .
73
75. Characterized by excessive lower anterior face height.
Palatal plane rotates down posteriorly.
Creates a negative rather than the normal
positive inclination to the true horizontal
75
76. Mandible shows an opposite ,backward rotation
with an increase in mandibular plane angle
Weak musculature & mature late , so avoid mechanics
which increase vertical height of patient
facial height ratio (upper: lower) is exaggerated
76
77. Avoid bite planes
Avoid anchor bends
Avoid class II elastics
77
In these patients we should avoid :-
78. This type of rotation is normally associated
with Skeletal Anterior Open Bite
malocclusion(because chin rotates back well as
down)
Backward rotation of mandible also occurs in
patients with abnormalities/pathological changes
affecting the TMJ
In TMJ patients growth of condyle is restricted
78
80. Rotation pattern of jaw growth obviously influences
tooth eruption
It can also influence the direction of eruption &
ultimate antero-posterior position of incisor teeth .
80
INTERACTION BETWEEN JAW ROTATION
& TOOTH ERUPTION
81. Path of eruption of Maxillary teeth is downward
& forward
Normally maxilla rotates slightly few degrees
forward & frequently backward
Forward Rotation tends to tip
incisors forwards & increasing their prominence.
Backward Rotation directs ant.
teeth more posteriorly than normal, up righting them
& decreasing their prominence
81
82. Movement of teeth relative to cranial base obviously
could be produced by
Translocation bring about ½ of the total maxillary
tooth movement during adolescent growth
82
83. Eruption path of mandibular teeth is upward &
forward.
Normal Internal rotation of the mandible carries
the jaw upward in front
This rotation alters the eruption path of incisors,
tending to direct them more posteriorly than
would other wise have been the case.
83
84. When excessive rotation occurs in short face type of
development, the incisors tend to carried into an
overlapping position even if they erupt very little;
hence the tendency for Deep Bite Malocclusion
The rotation also progressively upright’s incisors,
displacing them lingually & causing a tendency
towards Crowding
84
85. In long face growth pattern anterior open bite will
develop as the anterior face height increases unless
incisor erupt for an extreme distance
Rotation of jaw also carries the incisor forward ,
creating a dental protrusion
85
86. Change in
soft tissues
like Nose&
Lips
Change in
Eruption –
Active &
Passive
Alignment
changes &
changes in
Occlusion
86
AGE CHANGES IN GROWTH
PATTERN
87. Changes in soft tissue not only continues with
aging, they are much larger in magnitude than
changes in hard tissue .
The Lips &other soft tissues of face , sag downward
with aging.
The result is a decrease in exposure of upper
incisors, & an increase in exposure of lower
incisors , both at rest & on smile
87
CHANGES IN FACIAL SOFT
TISSUE
88. With aging , Lips also become progressively thinner,
less vermillion display.
88
90. Active Eruption
Active eruption has been described as the eruption
process of a tooth and their alveoli through the
gingival tissues (Moshrefi 2000). This phase ends
when the tooth makes contact with the opposing
dentition but may continue with occlusal wear or loss
of opposing teeth (Dolt 1997).
90
CHANGES IN ERUPTION
91. Passive eruption begins once active eruption has
completed. This takes place as the dentogingival unit
migrates in the apical direction until it is adjacent to
the cemento-enamel junction (CEJ) (Evian et al. 1993).
In contrast to active eruption, passive eruption is the
apparent lengthening of the crown due to the loss of
attachment, or recession of the gingiva, also due to
inflammation.
91
PASSIVE ERUPTION
93. Its due to
93
CHANGES IN ALIGNMENT &
OCCLUSION
Lack of
Attrition
Pressure
from 3rd
Molars
Late
mandibul
ar Growth
94. Raymond Begg ,a pioneer Australian orthodontist noted his
studies of Australian aborigines that malocclusion is
uncommon but large amounts of interproximal & occlusal
attrition occurred
He concluded that in modern populations the teeth became
crowded when attrition didn’t occur with soft diets, &
advocated wide spread extraction of premolar teeth to
provide equivalent of the attrition he saw in aborgines
94
LACK OF ATTRITION
95. a) Late incisor crowding coincides with the time of
eruption of 3rd molars
b) So one school of thought says that the pressure
from the erupting 3rd molars, causes mesial
migration of teeth,which is the reason for late
incisor crowding
c) But the amount of pressure from 3rd molars is not
sufficient to cause pressure effect & changes in
lower incisors
95
PRESSURE FROM THIRD MOLARS
96. Mandibular growth continues even after the
cessation of the maxillary growth in late teens
When mandible grows forward relative to maxilla,
in late teens mandibular incisors tends to move
lingually, particularly if any excess rotation is
present.
Due to the mandibular growth, if there where any
tight anterior occlusion before the late mandibular
growth occurs one of the 3 of the following can
occur 96
LATE MANDIBULAR GROWTH
97. Mandible is displaced distally & can cause TMJ
distortion & displacement of Articular disc
Upper incisors may flare forward, opening
space b/w the teeth
Lower Incisors may displace distally & become
crowded
97
98. Malocclusions & Dentofacial deformity arises through variations
in normal developmental process
A thorough background in craniofacial growth & development is
necessary for every dentist
A thorough knowledge is also necessary because orthodontic
treatment involves the manipulation of skeletal growth & dental
growth .
So once alteration/modification is been done, its done for ever
98
CONCLUSION
99. Contemporary Orthodontics - William R Proffit
Relationship b/w - Open Anatomy Journal
synchondrosis & craniofacial gth 2010/ Vol 2
Journal of Dental Research - http//jdr.Sagepub.com
Sutural Growth by Implant - http//ejo.oxfordjournals.org
method
Morphogenic analysis of facial
growth - Enlow
Orthodontics (Art & Science) - S I Balaji
Orthodontics - Sridhar Premkumar
Textbook of orthodontics - Gurkeerat Singh
99
BIBILIOGRAPHY