PMS and PMDD are two conditions that affect women's mental and physical health. Solh Wellness explains symptoms, causes, and treatment options for both the conditions.
Management of hyperkalemia dakahlia medical syndicate 2o18FAARRAG
This document discusses hyperkalemia (high potassium levels), including its definition, classification, causes, and approach to assessment and management. Some key points:
- Hyperkalemia is defined as a serum potassium level above 5.0-5.5 mEq/L in adults and can be mild (5.5-6.0 mEq/L), moderate (6.1-7.0 mEq/L), or severe (≥7.0 mEq/L). Levels over 7-8.5 mEq/L can cause cardiac issues.
- Causes include reduced renal excretion, shifts of potassium from cells to blood, and excessive potassium intake. Patients with kidney disease or
- Primary immune thrombocytopenia (ITP) is an autoimmune disorder characterized by isolated thrombocytopenia caused by autoantibodies against platelets.
- ITP is mediated by antiplatelet autoantibodies which coat platelets, leading to their phagocytosis by macrophages and accelerated platelet clearance.
- Diagnosis of ITP involves excluding other causes of thrombocytopenia based on a complete history, physical exam, blood tests and blood smear. Bone marrow testing is usually not required for initial diagnosis.
this power point descripe diabetic ketoacidosis in pediatric age group .. we talk about the risk of it .. management specially (fluid management) as case study .. complications and the treatment of brain oedema .. i hope to be auseful one .. enjoy
Heparin-induced thrombocytopenia (HIT) is defined as a decrease in platelet count occurring 5-10 days after starting heparin treatment along with hypercoagulability and the presence of heparin-dependent antibodies. There are two main types of HIT - type 1 is a mild transient decrease while type 2 is an antibody-mediated thrombocytopenia associated with high thrombosis risk. Diagnosis involves clinical features and platelet factor 4 antibody testing, with a strongly positive test supporting HIT. Treatment involves stopping heparin
Chronic kidney disease and esrd(end stage renal diseaseZeelNaik2
CKD and ESRD.
Chronic Kidney Disease.
End-Stage Renal Disease.
CKD is a progressive loss of function over several months to years, characterized by gradual replacement of normal kidney architecture with interstitial fibrosis.
CKD is defined as either of the following conditions for a minimum of 3 months: GFR less than 60 ml/min/1.73 m2, or old damage to the kidneys with or without a decrease in GFR.
The prevalence of CKD increases with age and is greater in females.
CKD is a disease when GFR falls below 60 ml/min/1.73 m2 over at least 3 months.
CKD is a broad term that includes subtle decreases in kidney function that develop over a minimum of 3 months.
In contrast acute kidney injury refers to any deterioration in kidney function that happens in less than 3 months.
This document provides a summary of guidelines for the management of patients with non-ST-elevation acute coronary syndromes (NSTE-ACS). It discusses definitions, pathophysiology, initial evaluation and management including risk stratification using ECG, biomarkers and risk scores. It provides recommendations on standard medical therapies in the early hospital care including oxygen, nitrates, analgesics, beta-blockers and calcium channel blockers. It emphasizes the importance of risk stratification, biomarker testing, and initiation of anti-ischemic therapies in the first 24 hours for management of NSTE-ACS.
by the renowned pediatrician, Dr Satish Deopujari,
National Chairperson (Ex)
Intensive Care Chapter I A P
Founder Chairman.....
National conference on pediatric critical care
Professor of pediatrics ( Hon ) JNMC:Wardha
Nagpur : INDIA
The document provides an overview of the management of sepsis and septic shock. It discusses that early goal-directed therapy within the first 6 hours including antibiotics, fluids, vasopressors and inotropes if needed can significantly improve outcomes. Other key points covered include the definitions and diagnostic criteria for sepsis; appropriate antibiotic therapy and vasopressor use; importance of lung-protective ventilation; role for activated protein C, steroids, tight glucose control and renal replacement therapy. Prognosis depends on early recognition and treatment as mortality increases significantly with delayed or inadequate care.
Management of hyperkalemia dakahlia medical syndicate 2o18FAARRAG
This document discusses hyperkalemia (high potassium levels), including its definition, classification, causes, and approach to assessment and management. Some key points:
- Hyperkalemia is defined as a serum potassium level above 5.0-5.5 mEq/L in adults and can be mild (5.5-6.0 mEq/L), moderate (6.1-7.0 mEq/L), or severe (≥7.0 mEq/L). Levels over 7-8.5 mEq/L can cause cardiac issues.
- Causes include reduced renal excretion, shifts of potassium from cells to blood, and excessive potassium intake. Patients with kidney disease or
- Primary immune thrombocytopenia (ITP) is an autoimmune disorder characterized by isolated thrombocytopenia caused by autoantibodies against platelets.
- ITP is mediated by antiplatelet autoantibodies which coat platelets, leading to their phagocytosis by macrophages and accelerated platelet clearance.
- Diagnosis of ITP involves excluding other causes of thrombocytopenia based on a complete history, physical exam, blood tests and blood smear. Bone marrow testing is usually not required for initial diagnosis.
this power point descripe diabetic ketoacidosis in pediatric age group .. we talk about the risk of it .. management specially (fluid management) as case study .. complications and the treatment of brain oedema .. i hope to be auseful one .. enjoy
Heparin-induced thrombocytopenia (HIT) is defined as a decrease in platelet count occurring 5-10 days after starting heparin treatment along with hypercoagulability and the presence of heparin-dependent antibodies. There are two main types of HIT - type 1 is a mild transient decrease while type 2 is an antibody-mediated thrombocytopenia associated with high thrombosis risk. Diagnosis involves clinical features and platelet factor 4 antibody testing, with a strongly positive test supporting HIT. Treatment involves stopping heparin
Chronic kidney disease and esrd(end stage renal diseaseZeelNaik2
CKD and ESRD.
Chronic Kidney Disease.
End-Stage Renal Disease.
CKD is a progressive loss of function over several months to years, characterized by gradual replacement of normal kidney architecture with interstitial fibrosis.
CKD is defined as either of the following conditions for a minimum of 3 months: GFR less than 60 ml/min/1.73 m2, or old damage to the kidneys with or without a decrease in GFR.
The prevalence of CKD increases with age and is greater in females.
CKD is a disease when GFR falls below 60 ml/min/1.73 m2 over at least 3 months.
CKD is a broad term that includes subtle decreases in kidney function that develop over a minimum of 3 months.
In contrast acute kidney injury refers to any deterioration in kidney function that happens in less than 3 months.
This document provides a summary of guidelines for the management of patients with non-ST-elevation acute coronary syndromes (NSTE-ACS). It discusses definitions, pathophysiology, initial evaluation and management including risk stratification using ECG, biomarkers and risk scores. It provides recommendations on standard medical therapies in the early hospital care including oxygen, nitrates, analgesics, beta-blockers and calcium channel blockers. It emphasizes the importance of risk stratification, biomarker testing, and initiation of anti-ischemic therapies in the first 24 hours for management of NSTE-ACS.
by the renowned pediatrician, Dr Satish Deopujari,
National Chairperson (Ex)
Intensive Care Chapter I A P
Founder Chairman.....
National conference on pediatric critical care
Professor of pediatrics ( Hon ) JNMC:Wardha
Nagpur : INDIA
The document provides an overview of the management of sepsis and septic shock. It discusses that early goal-directed therapy within the first 6 hours including antibiotics, fluids, vasopressors and inotropes if needed can significantly improve outcomes. Other key points covered include the definitions and diagnostic criteria for sepsis; appropriate antibiotic therapy and vasopressor use; importance of lung-protective ventilation; role for activated protein C, steroids, tight glucose control and renal replacement therapy. Prognosis depends on early recognition and treatment as mortality increases significantly with delayed or inadequate care.
Kidney transplantation is the most effective therapy for end-stage renal disease. The transplanted organ can come from a live or deceased donor. Immunosuppressive medications are used to prevent rejection and include corticosteroids, calcineurin inhibitors, mTOR inhibitors, and antimetabolites. Common post-transplant complications include acute rejection, infections like cytomegalovirus, and chronic allograft dysfunction.
This document discusses deep vein thrombosis (DVT) prophylaxis in the intensive care unit (ICU). It defines DVT and pulmonary embolism, noting that VTE is a common cause of death in hospitalized patients. It discusses populations at high risk for VTE, including ICU patients, and risk factors like immobilization. Methods of prophylaxis include mechanical methods like compression devices and pharmacological methods like low molecular weight heparins and unfractionated heparin. Risk assessment tools can evaluate risk of both thrombosis and bleeding to guide selection of prophylaxis methods. The document provides dosing and safety guidelines for different prophylaxis options and outlines a protocol for prophylaxis in neurocritical ICU patients and those
Blood transfusion & its component therapykitubhaimbbs
This document discusses blood coagulation and blood transfusions. It covers several key points:
1) Only 30% of countries have nationwide transfusion services, and 80% of the world's population only has access to 20% of safely collected and tested blood.
2) The main indications for blood transfusions are to increase oxygen-carrying capacity and intravascular volume when a patient is hemorrhaging.
3) There are several potential complications from blood transfusions like changes in oxygen transport, coagulopathy, allergic reactions, and hemolytic transfusion reactions. Proper testing and protocols are important to minimize risks.
This document discusses bacterial infection of tunneled hemodialysis catheters. It notes that while fistulas are preferred, many patients still initiate dialysis with catheters which have a high risk of infection. Catheter-related bloodstream infections can lead to serious complications and increased mortality. Common pathogens involved include Staphylococcus, Pseudomonas, and Candida. Prevention strategies include proper insertion technique, exit site care, and use of antimicrobial locks. Treatment of infections involves antibiotics tailored to the pathogen as well as potentially removing the catheter. Duration of treatment depends on the severity and type of infection.
This document provides guidelines for the management of febrile neutropenia. It defines neutropenia and its levels of severity. It describes risk factors for infection and common pathogens. It outlines the evaluation, including diagnostic tests and imaging. It provides recommendations for empiric antibiotic therapy based on risk level. It also covers antifungal therapy, management of specific infections like typhlitis, and use of colony-stimulating factors. The goal is to guide clinicians in promptly diagnosing and treating potential infections in immunocompromised patients with febrile neutropenia.
Hepatopulmonary syndrome (HPS) is defined by the triad of liver disease, impaired oxygenation, and intrapulmonary vascular dilatations. It occurs in 4-47% of patients with chronic liver disease. Clinical manifestations include dyspnea, cyanosis, clubbing, and platypnea. Diagnosis involves demonstrating an increased alveolar-arterial oxygen gradient and contrast echocardiography showing intrapulmonary shunting. Treatment options include oxygen supplementation and liver transplantation, which can resolve HPS by correcting the underlying liver disease.
Chronic Lypmhocytic leukemia/SLL/B-PLL/T-PLL/ATLL By SOLOMON SUasb by SOLOMON SUASB
the following presenation include
Introduction/Background
• Etiology of CLL
• Symptoms
• Test and Diagnosis
• Staging
• Prognosis
• Treatment
• B Cell diseases BPLL
• T cell diseases
T-PLL
ATLL
LGLL
The document discusses hyponatremia, defining it as a low serum sodium concentration and describing the physiology and pathophysiology of sodium regulation in the body. It examines the epidemiology, classification, clinical manifestations, diagnosis, and treatment of hyponatremia, providing details on evaluating volume status, calculating sodium deficits, and correcting sodium levels based on chronicity and symptoms.
Transfusion-related acute lung injury (TRALI) is a potentially fatal syndrome characterized by acute respiratory distress within 6 hours of blood transfusion. It is believed to be caused by anti-leukocyte antibodies in plasma products that cause leukocyte aggregation in the lungs, inflammatory injury, and non-cardiogenic pulmonary edema. TRALI has an incidence of 1 in 5000 transfusions and mortality rate of 5-25%. Risk factors include plasma-rich products, multiparous donors, and underlying patient conditions. Diagnosis involves new pulmonary edema within 6 hours of transfusion in the absence of circulatory overload. Treatment focuses on supportive care, with most patients recovering within 72 hours. Prevention strategies include leukoreduction of blood products
Renal regulation of potassium balance is critical for maintaining normal potassium levels. The kidneys excrete most of the daily potassium intake and reabsorb potassium in the proximal tubule. In the distal tubule and collecting duct, aldosterone stimulates potassium secretion. Hyperkalemia occurs when potassium levels shift from cells to extracellular fluid or potassium excretion is decreased. Hypokalemia is usually caused by increased potassium excretion due to mineralocorticoid excess or drugs like diuretics. Both conditions can cause cardiac arrhythmias.
This document discusses heparin-induced thrombocytopenia (HIT). HIT is an immune-mediated reaction to heparin that results in platelet activation and thrombocytopenia. It can lead to thrombotic complications in 20-50% of patients. The document reviews the pathophysiology of HIT, defines its criteria, discusses diagnostic assays and algorithms, and outlines treatment and management approaches including alternative anticoagulants like lepirudin, argatroban, and danaparoid. Early recognition and treatment are important to prevent life-threatening thrombotic events associated with HIT.
Role of transfusion medicine in hematopoietic stem cellFigo Khan
The role of transfusion medicine in hematopoietic stem cell transplantation involves donor evaluation and stem cell collection, processing, cryopreservation, thawing, and infusion. Transfusion medicine specialists ensure proper HLA typing and immunohematologic compatibility between donors and recipients. They collect stem cells via bone marrow aspiration, peripheral blood apheresis, or umbilical cord blood collection. Collected stem cells are processed, cryopreserved, thawed as needed, and infused into recipients. Transfusion medicine specialists also provide transfusion support and monitor for engraftment and complications related to ABO blood group compatibility.
This document provides information about platelet transfusion. It discusses what platelets are, their role in hemostasis, normal platelet counts, causes of thrombocytopenia, indications for platelet transfusion, contraindications, donor criteria, preparation of platelet concentrates, dosing, response to transfusion, complications including immunological and non-immunological issues, and methods to reduce complications like use of leukoreduced products. The document contains detailed information about platelet immunology, causes of refractoriness, its management, and methods to improve safety and availability of platelet transfusion like use of matched donors and crossmatching.
This document provides an overview of diabetic ketoacidosis (DKA), including its definition, epidemiology, pathophysiology, etiology, clinical manifestations, investigations, differential diagnosis, prevention, treatment, and pitfalls. DKA is characterized by hyperglycemia, ketonemia, and metabolic acidosis. It is caused by insulin deficiency and excess counterregulatory hormones. Treatment involves fluid and electrolyte replacement, insulin therapy, and glucose administration to prevent hypoglycemia. Complications include cerebral edema, infection, and pulmonary edema.
The document discusses evaluation and treatment of hypokalemia. It states that the urine potassium-to-creatinine ratio is usually less than 13 mEq/g creatinine with hypokalemia caused by transcellular shifts, GI losses, diuretic use, or poor diet, and is higher with renal potassium wasting. Oral or IV potassium replacement should be based on severity, with mild cases receiving tablets and moderate cases receiving higher doses orally or IV. Severe hypokalemia under 2.5 mEq/L or with symptoms requires IV replacement at 10-20 mmol/hour depending on the route and ECG monitoring. Guidelines recommend KCl infusion in non-dextrose solutions under
This document provides information on diabetic ketoacidosis (DKA). It begins with an introduction stating that DKA is a life-threatening complication of diabetes mellitus that predominantly occurs in type 1 diabetes but can also occur in 10-30% of newly diagnosed type 2 diabetes cases. It then discusses the pathophysiology of DKA involving a complex relationship between insulin and counterregulatory hormones resulting in hyperglycemia, ketone formation, and metabolic acidosis. Clinical findings are related to hyperglycemia, volume depletion, and acidosis. Treatment goals are volume replacement, correction of hyperglycemia and electrolyte/acid-base imbalances, and treatment of underlying causes. A timeline is provided outlining management from initial presentation
Acute hemolytic transfusion reaction (AHTR) is an immune or non-immune mediated reaction that occurs soon after a blood transfusion. Symptoms include fever, hemoglobinemia, hemoglobinuria, and hypotension. Management involves stopping the transfusion, monitoring vital signs, and administering medications like dopamine for hypotension. Complications can include acute kidney injury, anemia, and shock. Prevention methods aim to avoid mislabeled blood products and ensure proper patient identification and blood product compatibility.
Aplastic Anemia is a rare condition in which the body stops producing enough blood cells as a result of bone marrow damage. In aplastic anemia, the stem cells in the bone marrow are damaged. The bone marrow is either empty (aplastic) or contains few blood cells (hypoplastic).
Premenstrual Dysphoric Disorder (PMDD)-a brief medical study.. martinshaji
Premenstrual syndrome (PMS) is a medical condition that affects some women of childbearing age. More than one in three women suffer from PMS. One in 20 suffer so severely that their lives are seriously affected. PMS is related to a variety of physical and psychological symptoms that occur just before the menstrual period.
please comment
thank you ...
Premenstrual Dysphoric Disorder (PMDD) is a severe form of Premenstrual Syndrome that affects around 8% of women. The exact causes are unknown but hormones are thought to play a role. Symptoms include depression, tension, irritability and occur during the week before a woman's period. Risk factors include family history, anxiety, depression and lifestyle factors. Diagnosis involves tracking symptoms and ruling out other conditions. Treatment focuses on lifestyle changes and may include birth control, antidepressants or therapy. With proper treatment, most women find their symptoms improve or become tolerable.
Kidney transplantation is the most effective therapy for end-stage renal disease. The transplanted organ can come from a live or deceased donor. Immunosuppressive medications are used to prevent rejection and include corticosteroids, calcineurin inhibitors, mTOR inhibitors, and antimetabolites. Common post-transplant complications include acute rejection, infections like cytomegalovirus, and chronic allograft dysfunction.
This document discusses deep vein thrombosis (DVT) prophylaxis in the intensive care unit (ICU). It defines DVT and pulmonary embolism, noting that VTE is a common cause of death in hospitalized patients. It discusses populations at high risk for VTE, including ICU patients, and risk factors like immobilization. Methods of prophylaxis include mechanical methods like compression devices and pharmacological methods like low molecular weight heparins and unfractionated heparin. Risk assessment tools can evaluate risk of both thrombosis and bleeding to guide selection of prophylaxis methods. The document provides dosing and safety guidelines for different prophylaxis options and outlines a protocol for prophylaxis in neurocritical ICU patients and those
Blood transfusion & its component therapykitubhaimbbs
This document discusses blood coagulation and blood transfusions. It covers several key points:
1) Only 30% of countries have nationwide transfusion services, and 80% of the world's population only has access to 20% of safely collected and tested blood.
2) The main indications for blood transfusions are to increase oxygen-carrying capacity and intravascular volume when a patient is hemorrhaging.
3) There are several potential complications from blood transfusions like changes in oxygen transport, coagulopathy, allergic reactions, and hemolytic transfusion reactions. Proper testing and protocols are important to minimize risks.
This document discusses bacterial infection of tunneled hemodialysis catheters. It notes that while fistulas are preferred, many patients still initiate dialysis with catheters which have a high risk of infection. Catheter-related bloodstream infections can lead to serious complications and increased mortality. Common pathogens involved include Staphylococcus, Pseudomonas, and Candida. Prevention strategies include proper insertion technique, exit site care, and use of antimicrobial locks. Treatment of infections involves antibiotics tailored to the pathogen as well as potentially removing the catheter. Duration of treatment depends on the severity and type of infection.
This document provides guidelines for the management of febrile neutropenia. It defines neutropenia and its levels of severity. It describes risk factors for infection and common pathogens. It outlines the evaluation, including diagnostic tests and imaging. It provides recommendations for empiric antibiotic therapy based on risk level. It also covers antifungal therapy, management of specific infections like typhlitis, and use of colony-stimulating factors. The goal is to guide clinicians in promptly diagnosing and treating potential infections in immunocompromised patients with febrile neutropenia.
Hepatopulmonary syndrome (HPS) is defined by the triad of liver disease, impaired oxygenation, and intrapulmonary vascular dilatations. It occurs in 4-47% of patients with chronic liver disease. Clinical manifestations include dyspnea, cyanosis, clubbing, and platypnea. Diagnosis involves demonstrating an increased alveolar-arterial oxygen gradient and contrast echocardiography showing intrapulmonary shunting. Treatment options include oxygen supplementation and liver transplantation, which can resolve HPS by correcting the underlying liver disease.
Chronic Lypmhocytic leukemia/SLL/B-PLL/T-PLL/ATLL By SOLOMON SUasb by SOLOMON SUASB
the following presenation include
Introduction/Background
• Etiology of CLL
• Symptoms
• Test and Diagnosis
• Staging
• Prognosis
• Treatment
• B Cell diseases BPLL
• T cell diseases
T-PLL
ATLL
LGLL
The document discusses hyponatremia, defining it as a low serum sodium concentration and describing the physiology and pathophysiology of sodium regulation in the body. It examines the epidemiology, classification, clinical manifestations, diagnosis, and treatment of hyponatremia, providing details on evaluating volume status, calculating sodium deficits, and correcting sodium levels based on chronicity and symptoms.
Transfusion-related acute lung injury (TRALI) is a potentially fatal syndrome characterized by acute respiratory distress within 6 hours of blood transfusion. It is believed to be caused by anti-leukocyte antibodies in plasma products that cause leukocyte aggregation in the lungs, inflammatory injury, and non-cardiogenic pulmonary edema. TRALI has an incidence of 1 in 5000 transfusions and mortality rate of 5-25%. Risk factors include plasma-rich products, multiparous donors, and underlying patient conditions. Diagnosis involves new pulmonary edema within 6 hours of transfusion in the absence of circulatory overload. Treatment focuses on supportive care, with most patients recovering within 72 hours. Prevention strategies include leukoreduction of blood products
Renal regulation of potassium balance is critical for maintaining normal potassium levels. The kidneys excrete most of the daily potassium intake and reabsorb potassium in the proximal tubule. In the distal tubule and collecting duct, aldosterone stimulates potassium secretion. Hyperkalemia occurs when potassium levels shift from cells to extracellular fluid or potassium excretion is decreased. Hypokalemia is usually caused by increased potassium excretion due to mineralocorticoid excess or drugs like diuretics. Both conditions can cause cardiac arrhythmias.
This document discusses heparin-induced thrombocytopenia (HIT). HIT is an immune-mediated reaction to heparin that results in platelet activation and thrombocytopenia. It can lead to thrombotic complications in 20-50% of patients. The document reviews the pathophysiology of HIT, defines its criteria, discusses diagnostic assays and algorithms, and outlines treatment and management approaches including alternative anticoagulants like lepirudin, argatroban, and danaparoid. Early recognition and treatment are important to prevent life-threatening thrombotic events associated with HIT.
Role of transfusion medicine in hematopoietic stem cellFigo Khan
The role of transfusion medicine in hematopoietic stem cell transplantation involves donor evaluation and stem cell collection, processing, cryopreservation, thawing, and infusion. Transfusion medicine specialists ensure proper HLA typing and immunohematologic compatibility between donors and recipients. They collect stem cells via bone marrow aspiration, peripheral blood apheresis, or umbilical cord blood collection. Collected stem cells are processed, cryopreserved, thawed as needed, and infused into recipients. Transfusion medicine specialists also provide transfusion support and monitor for engraftment and complications related to ABO blood group compatibility.
This document provides information about platelet transfusion. It discusses what platelets are, their role in hemostasis, normal platelet counts, causes of thrombocytopenia, indications for platelet transfusion, contraindications, donor criteria, preparation of platelet concentrates, dosing, response to transfusion, complications including immunological and non-immunological issues, and methods to reduce complications like use of leukoreduced products. The document contains detailed information about platelet immunology, causes of refractoriness, its management, and methods to improve safety and availability of platelet transfusion like use of matched donors and crossmatching.
This document provides an overview of diabetic ketoacidosis (DKA), including its definition, epidemiology, pathophysiology, etiology, clinical manifestations, investigations, differential diagnosis, prevention, treatment, and pitfalls. DKA is characterized by hyperglycemia, ketonemia, and metabolic acidosis. It is caused by insulin deficiency and excess counterregulatory hormones. Treatment involves fluid and electrolyte replacement, insulin therapy, and glucose administration to prevent hypoglycemia. Complications include cerebral edema, infection, and pulmonary edema.
The document discusses evaluation and treatment of hypokalemia. It states that the urine potassium-to-creatinine ratio is usually less than 13 mEq/g creatinine with hypokalemia caused by transcellular shifts, GI losses, diuretic use, or poor diet, and is higher with renal potassium wasting. Oral or IV potassium replacement should be based on severity, with mild cases receiving tablets and moderate cases receiving higher doses orally or IV. Severe hypokalemia under 2.5 mEq/L or with symptoms requires IV replacement at 10-20 mmol/hour depending on the route and ECG monitoring. Guidelines recommend KCl infusion in non-dextrose solutions under
This document provides information on diabetic ketoacidosis (DKA). It begins with an introduction stating that DKA is a life-threatening complication of diabetes mellitus that predominantly occurs in type 1 diabetes but can also occur in 10-30% of newly diagnosed type 2 diabetes cases. It then discusses the pathophysiology of DKA involving a complex relationship between insulin and counterregulatory hormones resulting in hyperglycemia, ketone formation, and metabolic acidosis. Clinical findings are related to hyperglycemia, volume depletion, and acidosis. Treatment goals are volume replacement, correction of hyperglycemia and electrolyte/acid-base imbalances, and treatment of underlying causes. A timeline is provided outlining management from initial presentation
Acute hemolytic transfusion reaction (AHTR) is an immune or non-immune mediated reaction that occurs soon after a blood transfusion. Symptoms include fever, hemoglobinemia, hemoglobinuria, and hypotension. Management involves stopping the transfusion, monitoring vital signs, and administering medications like dopamine for hypotension. Complications can include acute kidney injury, anemia, and shock. Prevention methods aim to avoid mislabeled blood products and ensure proper patient identification and blood product compatibility.
Aplastic Anemia is a rare condition in which the body stops producing enough blood cells as a result of bone marrow damage. In aplastic anemia, the stem cells in the bone marrow are damaged. The bone marrow is either empty (aplastic) or contains few blood cells (hypoplastic).
Premenstrual Dysphoric Disorder (PMDD)-a brief medical study.. martinshaji
Premenstrual syndrome (PMS) is a medical condition that affects some women of childbearing age. More than one in three women suffer from PMS. One in 20 suffer so severely that their lives are seriously affected. PMS is related to a variety of physical and psychological symptoms that occur just before the menstrual period.
please comment
thank you ...
Premenstrual Dysphoric Disorder (PMDD) is a severe form of Premenstrual Syndrome that affects around 8% of women. The exact causes are unknown but hormones are thought to play a role. Symptoms include depression, tension, irritability and occur during the week before a woman's period. Risk factors include family history, anxiety, depression and lifestyle factors. Diagnosis involves tracking symptoms and ruling out other conditions. Treatment focuses on lifestyle changes and may include birth control, antidepressants or therapy. With proper treatment, most women find their symptoms improve or become tolerable.
You don't have to live with the emotional turmoil that PMS mood swings can bring. Instead, find relief through certain lifestyle changes and medications.
The document describes a 31-year-old woman who presents with premenstrual syndrome (PMS) characterized by emotional and physical symptoms in the week before her period, including mood swings, hot flashes, breast tenderness, and weight gain. Her symptoms resolve after the start of her menstrual bleeding. The document discusses diagnostic criteria and treatments for PMS and premenstrual dysphoric disorder, including lifestyle changes, cognitive behavioral therapy, and antidepressants like SSRIs.
Some women get through their monthly periods easily with few or no concerns. Their periods come like clockwork, starting and stopping at nearly the same time every month, causing little more than a minor inconvenience.
This document discusses premenstrual syndrome (PMS) and its management. PMS refers to physical, psychological, and emotional symptoms related to a woman's menstrual cycle that usually disappear shortly before or after the start of menstrual flow. Common symptoms include irritability, lower back pain, tension, and dysphoria. While the exact causes are unclear, hormone changes are believed to play a role. Management includes lifestyle changes, dietary modifications, medications, and therapy. A severe form is premenstrual dysphoric disorder (PMDD), which causes debilitating emotional symptoms. The best approach for partners is to be understanding, avoid bringing up her condition, give massages, and not take things personally during this time.
The document discusses Premenstrual Syndrome (PMS), a group of symptoms linked to a woman's menstrual cycle that commonly occurs 1-2 weeks before her period. PMS can range from mild to severe and symptoms vary between women but may include physical symptoms like breast tenderness as well as psychological symptoms like depression or irritability. Diagnosis involves patient history and ruling out other conditions, while treatment focuses on lifestyle changes like exercise and diet as well as potential drug therapies to manage symptoms.
The document provides information about women's mental health and how it relates to various life stages and health conditions. It discusses how hormones and life events like menstruation, menopause, pregnancy, and postpartum periods can impact mental health. Conditions like PMS, PMDD, depression, anxiety, and PTSD are more common in women and often relate to biological and hormonal factors. Mental health challenges may also arise from health issues like polycystic ovarian syndrome, infertility, or intimate partner violence. The document aims to educate about understanding women's mental health in the context of biological, psychological, and social factors.
1. Premenstrual syndrome (PMS) is characterized by mild to moderate physical and mental symptoms that occur in the days or weeks before menstruation and disappear with the onset of menstruation.
2. The causes of PMS are unclear but likely involve hormonal changes during the menstrual cycle. Symptoms vary greatly between women and can include mood changes, breast tenderness, bloating, and cramps.
3. Treatment options aim to relieve symptoms and may include over-the-counter pain relievers, dietary changes, exercise, contraceptives, and consulting a doctor or gynecologist. Managing PMS requires finding what works best for each individual woman.
1. Premenstrual syndrome (PMS) is characterized by mild to moderate physical and mental symptoms that occur in the days or weeks before menstruation and disappear with the onset of menstruation.
2. The causes of PMS are unclear but likely involve hormonal changes during the menstrual cycle. Symptoms vary greatly between women and can include mood changes, breast tenderness, bloating, and cramps.
3. Treatment options aim to relieve symptoms and may include over-the-counter pain relievers, dietary changes, exercise, contraceptives, and consulting a doctor or gynecologist. Managing PMS requires finding what works best for each individual woman.
Jindal Naturopathy: PMS or Premenstrual Syndrome is a condition that occurs right before the menstrual cycle. It includes various physical as well as psychological symptoms. It typically occurs during the last 2 weeks of the menstrual cycle. Some common symptoms include cramps, headaches, and bloating. Some women and girls may also experience Premenstrual Dysphoric Disorder (PMDD) – anxiety and depression. Even though there can be medicines, not all options are effective in this situation. Hence, naturopathy treatment is the right fit option that can ease and reduce discomfort. You can count on a well-known Jindal Naturopathy for the right solution and read this post for more details.
Major depression (MD) is an illness that affects mood, body, and thoughts. It impacts sleep, appetite, feelings of self-worth, and how one thinks. MD cannot be willed away and without treatment, symptoms can last for weeks, months or years. Treatment such as antidepressants and psychotherapy can help most people with MD. Physical and genetic factors along with life stressors can contribute to the development of MD.
Depression is a serious medical condition that affects mood and behaviors. It is more common in women than men, affecting about 1 in 8 women at some point in their lifetime. While the core symptoms are the same, women often experience depression differently than men and are more susceptible to factors like hormonal changes, stress, and rumination. Effective treatment for women may require consideration of biological differences and adjusting medication doses or monitoring side effects more closely.
Dr. Keith Reisler practices as an OBGYN in Plano, Texas. In addition to providing routine care for patients, Keith Reisler, MD, sees patients with reproductive-health-related issues, such as premenstrual syndrome (PMS).
Period is one of the important thing of in women's life. It is not just a monthly cycle but much more than this. It is also states about your health. So if you are not getting your period or getting irregular period then it may be a symptom of PCOS. So lets clear your doubts and book a pcos profile test at https://fmdiagnostics.com/product/fm-pcos-profile/
Julie, a 25-year-old woman who recently gave birth to her second child, is experiencing symptoms of depression including crying, irritability, sleep issues, loss of appetite, and feelings of guilt. The document discusses depression in women, noting it is twice as common in women and can occur during times of hormonal changes like premenstrual, postpartum, and perimenopause. Treatment options include counseling, medication, lifestyle changes, and addressing any nutrient deficiencies.
The document discusses mood disorders like depression and the criteria for major depressive episodes according to the DSM-IV-TR. It then covers treatment options like pharmacotherapy, noting that SSRIs are usually the first choice medication due to their safety profile. Finally, it discusses risk factors for suicide like mental illness, depression, and substance abuse issues.
The document discusses mood disorders like depression and the criteria for diagnosing major depressive episodes. It then covers treatment options like pharmacotherapy, focusing on selective serotonin reuptake inhibitors (SSRIs) as a first-line treatment. SSRIs are generally well-tolerated but can cause side effects like sexual dysfunction, nausea, headaches, and insomnia. Precautions are discussed around pregnancy, the elderly, children, and drug interactions. Risk factors for suicide are also outlined.
Menstrual cycle disorders, Menopause and Drugs in Pregnancy and Lactationgoogle
The document discusses menstrual cycle disorders, menopause, and drugs in pregnancy and lactation. It covers topics like the menstrual cycle, premenstrual syndrome (PMS), diagnostic criteria for PMS and premenstrual dysphoric disorder (PDD), hormones that contribute to PMS symptoms, vitamins and minerals that can reduce PMS symptoms, and treatments for PMS. It also discusses dysmenorrhea, the difference between primary and secondary dysmenorrhea, and treatments for dysmenorrhea that include NSAIDs and combined oral contraceptives.
The document discusses menstrual cycle disorders, menopause, and drugs in pregnancy and lactation. It covers topics like the menstrual cycle, premenstrual syndrome (PMS), diagnostic criteria for PMS and premenstrual dysphoric disorder (PDD), hormones that contribute to PMS symptoms, vitamins and minerals that can reduce PMS symptoms, and treatments for PMS. It also discusses dysmenorrhea, the difference between primary and secondary dysmenorrhea, and treatments for dysmenorrhea that include NSAIDs and combined oral contraceptives.
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2. PMS and PMDD are two conditions that
affect women's mental and physical health
in the days before menstruation. Despite
some similarities in symptoms, there are
various conditions with distinct diagnostic
criteria and treatment approaches. In this
blog, we'll look at the differences between
PMS and PMDD, as well as the symptoms
and treatments available.
Introduction
3. WhatisPMS?
Premenstrual syndrome (PMS) is a disorder that affects women in
the days or weeks preceding their menstrual cycle. PMS symptoms
vary by woman, but they commonly include mood swings, irritability,
fatigue, bloating, and breast tenderness. Headaches, backaches,
and digestive problems are also common in women.
PMS is a common condition affecting 3 of every 4 (estimated)
menstruating women. These symptoms are typically mild and do not
disrupt daily activities. However, PMS can be severe for some
women and affect their quality of lifestyle.
4. Premenstrual dysphoric disorder (PMDD) is a
more severe form of PMS that affects up to 8%
of women. The symptoms of PMDD are similar
to those of PMS but are more intense and can
significantly impact a woman's daily life.
PMDD is a severe condition that can interfere
with a woman's ability to function at work,
school, or home. It's essential to seek treatment
if you are experiencing PMDD.
WhatisPMDD?
5. The main difference between PMS and PMDD is the
symptoms' severity and impact on daily life. While
PMS can cause discomfort and mild mood changes,
PMDD can significantly affect a woman's ability to
function in everyday life, work, or relationships.
Additionally, the diagnostic criteria for PMDD are
more specific than those for PMS. PMDD requires at
least five symptoms to be present, while PMS does not
have an exact number of symptoms.
WhataretheKeydifferencesbetweenPMSandPMDD?
6. Tension or anxiety
Mood swings and irritability or
anger
Appetite changes and food
cravings
Insomnia
Social withdrawal
Change in libido
Emotional and behavioral signs
& symptoms
CommonPremenstrualSyndromeSymptoms
Joint or muscle pain
Headache & fatigue
Weight gain related to fluid
retention
Abdominal bloating
Breast tenderness
Acne flare-ups
Constipation or diarrhea
Physical signs and symptoms
7. Feelings of sadness or hopelessness or suicidal
thoughts
Severe stress, tension, or anxiety
Panic attacks
Inappropriate mood swings and bouts of crying
Constant irritability or anger that affects other people
Loss of interest in normal daily activities and
relationships
While a person going through PMD shows the above
symptoms in a severe state, here are some common
Premenstrual Dysphoric Disorder symptoms.
PremenstrualDysphoricDisorderSymptoms:
8. Cyclic changes in hormones. Premenstrual syndrome symptoms
change with hormonal fluctuations and disappear during pregnancy
and menopause.
Chemical changes in the brain. Serotonin fluctuations, a brain
chemical (neurotransmitter) thought to play an essential role in mood
states, could trigger PMS symptoms. Serotonin deficiency may
contribute to premenstrual depression, fatigue, food cravings, and
sleep issues.
The exact causes of PMS are still unknown; however, the factors which
can majorly contribute towards development:
WhatCausesPMS?
9. PMDD has no known cause, but it is thought to be an
abnormal response to hormonal fluctuations during the
menstrual cycle. PMDD and low serotonin levels are also
often linked together, and changes in estrogen and
progesterone levels in the two weeks preceding
menstruation are thought to affect serotonin levels.
Serotonin-mediated brain cells regulate mood, attention,
sleep, and pain. As a result, long-term changes in serotonin
levels can cause PMDD symptoms.
WhatCausesPMDD?
10. There are ways through which PMS can be managed.
Women can benefit from supplements or OTC (over-the-
counter) therapies, while some may require prescription
medications. Specific changes in lifestyle approaches can
also prove to be beneficial.
Whether you require treatment depends on the severity of
your symptoms and their effect on your life. You can discuss
your symptoms with your healthcare provider, who can
recommend the best treatment.
HowDoITreatPMS?
11. Your medical professional will decide the best course of
treatment for your PMDD symptoms. Many women with
PMDD take an antidepressant known as selective
serotonin reuptake inhibitors (SSRIs) consistently
throughout the month or an increased dose for two
weeks before their periods.
PMDD is also treatable with hormones. Many women
find that stopping ovulation with medication can
eliminate the hormone fluctuations that cause symptoms.
Your doctor may also advise you to use progesterone or
estrogen-containing medications or creams.
HowDoITreatPMDD?
12. In conclusion, PMS and PMDD are two conditions that can impact
women's mental and physical health before menstruation. While
they share some symptoms, PMDD is a more severe form of PMS
that must be diagnosed using specific criteria. Both conditions can
be treated with lifestyle changes, medications, or a combination. If
you have PMS or PMDD symptoms, speak with your healthcare
provider, who can help you decide on the best action.
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Conclusion
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