This document outlines the steps for a general physical examination of the respiratory system. It describes examining the patient's general appearance, vital signs, posture, nutrition, skin, eyes, neck, chest, hands, nails, lymph nodes and feet. Specific signs of conditions like tuberculosis, clubbing and cyanosis are highlighted. Breath sounds, respiratory rate and patterns are assessed. The pulse, blood pressure, JVP and oxygen saturation are evaluated. The nasal cavity, sinuses and oral cavity are examined. Other body systems are also assessed to identify related conditions. References for further information are provided.
a detailed study on pulmonary function testmartinshaji
this study details about all the aspects of pulmonary function test, lung volumes& capacities , tests such as spirometry , carbon monoxide diffusion capacity, chest x ray, body plethesmography , nitrogen washout etc
please comment
thank u
a detailed study on pulmonary function testmartinshaji
this study details about all the aspects of pulmonary function test, lung volumes& capacities , tests such as spirometry , carbon monoxide diffusion capacity, chest x ray, body plethesmography , nitrogen washout etc
please comment
thank u
Please find the power point on Brainsteam stroke. I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
Rheumatic fever (acute rheumatic fever) is a disease that can affect the heart, joints, brain, and skin. Rheumatic fever can develop if strep throat and scarlet fever infections are not treated properly. Early diagnosis of these infections and treatment with antibiotics are key to preventing rheumatic fever.
Rheumatic fever (RF) is an inflammatory disease that can involve the heart, joints, skin, and brain.[1] The disease typically develops two to four weeks after a streptococcal throat infection.[2] Signs and symptoms include fever, multiple painful joints, involuntary muscle movements, and occasionally a characteristic non-itchy rash known as erythema marginatum.[1] The heart is involved in about half of the cases.[1] Damage to the heart valves, known as rheumatic heart disease (RHD), usually occurs after repeated attacks but can sometimes occur after one. The damaged valves may result in heart failure, atrial fibrillation and infection of the valves.[1]
Case study- An 11 year old Polynesian male presents with fever up to 39 degrees (102 degrees F), joint pain and swelling, along with shortness of breath. The fever comes and goes at random times of the day. The symptoms have been present now for 4 days.
this ppt gives information about COPD , Asthma(the respiratory disease)As stated before, diseases of the heart affect the lungs and diseases of the lungs affect the heart.
This is because of the peculiar characteristics of pulmonary vasculature. The pressure in the pulmonary arteries is much lower than in the systemic arteries.
The pulmonary arterial system is466 SECTION III Systemic Pathology thinner than the systemic arterial system.
They are thin elastic vessels which can be easily distinguished from thick-walled bronchial arteries supplying the large airways and the pleura.
General diseases of vascular origin occurring in the lungs such as pulmonary oedema, pulmonary congestion, pulmonary embolism and pulmonary infarction, have all been already discussed.
What are the pulmonary function tests used?
What are the indications?
What are the contraindications?
How to perform each and prepare patients?
How to interpret and reach a diagnosis?
How to clean and calibrate devices?
Please find the power point on Brainsteam stroke. I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
Rheumatic fever (acute rheumatic fever) is a disease that can affect the heart, joints, brain, and skin. Rheumatic fever can develop if strep throat and scarlet fever infections are not treated properly. Early diagnosis of these infections and treatment with antibiotics are key to preventing rheumatic fever.
Rheumatic fever (RF) is an inflammatory disease that can involve the heart, joints, skin, and brain.[1] The disease typically develops two to four weeks after a streptococcal throat infection.[2] Signs and symptoms include fever, multiple painful joints, involuntary muscle movements, and occasionally a characteristic non-itchy rash known as erythema marginatum.[1] The heart is involved in about half of the cases.[1] Damage to the heart valves, known as rheumatic heart disease (RHD), usually occurs after repeated attacks but can sometimes occur after one. The damaged valves may result in heart failure, atrial fibrillation and infection of the valves.[1]
Case study- An 11 year old Polynesian male presents with fever up to 39 degrees (102 degrees F), joint pain and swelling, along with shortness of breath. The fever comes and goes at random times of the day. The symptoms have been present now for 4 days.
this ppt gives information about COPD , Asthma(the respiratory disease)As stated before, diseases of the heart affect the lungs and diseases of the lungs affect the heart.
This is because of the peculiar characteristics of pulmonary vasculature. The pressure in the pulmonary arteries is much lower than in the systemic arteries.
The pulmonary arterial system is466 SECTION III Systemic Pathology thinner than the systemic arterial system.
They are thin elastic vessels which can be easily distinguished from thick-walled bronchial arteries supplying the large airways and the pleura.
General diseases of vascular origin occurring in the lungs such as pulmonary oedema, pulmonary congestion, pulmonary embolism and pulmonary infarction, have all been already discussed.
What are the pulmonary function tests used?
What are the indications?
What are the contraindications?
How to perform each and prepare patients?
How to interpret and reach a diagnosis?
How to clean and calibrate devices?
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Muktapishti is a traditional Ayurvedic preparation made from Shoditha Mukta (Purified Pearl), is believed to help regulate thyroid function and reduce symptoms of hyperthyroidism due to its cooling and balancing properties. Clinical evidence on its efficacy remains limited, necessitating further research to validate its therapeutic benefits.
- Video recording of this lecture in English language: https://youtu.be/kqbnxVAZs-0
- Video recording of this lecture in Arabic language: https://youtu.be/SINlygW1Mpc
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
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Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Basavarajeeyam is a Sreshta Sangraha grantha (Compiled book ), written by Neelkanta kotturu Basavaraja Virachita. It contains 25 Prakaranas, First 24 Chapters related to Rogas& 25th to Rasadravyas.
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Adv. biopharm. APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMSAkankshaAshtankar
MIP 201T & MPH 202T
ADVANCED BIOPHARMACEUTICS & PHARMACOKINETICS : UNIT 5
APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMS By - AKANKSHA ASHTANKAR
6. GENERAL APPEARANCE
• CONSCIOUSNESS LEVEL (CO2 NARCOSIS, METASTATIC MALIGNANCY)
• COMFORTABLE/DISTRESSED
• INABILITY TO COMPLETE A SENTENCE
• USE OF ACCESSORY MUSCLES OF RESPIRATION (STERNOCLEIDOMASTOID, TRAPEZIUS, SCALENES,
ALAE NASI)
• INTERCOSTAL INDRAWING
• PURSED LIP BREATHING (SEVERE COPD)
• AUDIBLE WHEEZE (INSPIRATORY NOISE)/STRIDOR (EXPIRATORY NOISE)
• HOARSENESS OF VOICE/ WEAKNESS OF VOICE
• SURROUNDINGS (INHALERS, PEAK FLOW METERS, TISSUES, SPUTUM POT, OXYGEN MASK)
7.
8. POSTURE
• SITTING FORWARD WITH ARMS BRACED ON THE TABLE (SO THAT THEY CAN USE
PECTORALIS MAJOR TO PULL THE RIBS OUTWARD)
• LYING TOWARDS THE AFFECTED SIDE IN PNEUMONIA AND PLEURITIS (AUTO
SPLINTING)
18. CYANOSIS
• BLUISH DISCOLOURATION OF THE SKIN THAT IS CAUSED BY INCREASED
AMOUNTS OF REDUCED HEMOGLOBIN IN THE SUBCAPILLARY VENOUS PLEXUS
(MORE THAN 5 G%)
• OR DUE TO ABNORMAL PIGMENTS IN BLOOD (METHB >1.5, SULFHB >0.5 G%)
• CYANOSIS IS ABSENT IN SEVERE ANEMIA AND CO POISONING
19.
20. CYANOSIS IN RESPIRATORY DISEASES
• COPD
• INTERSTITIAL LUNG DISEASE – CYANOSIS ON EXERTION
• ALVEOLAR HYPOVENTILATION
21.
22. CLUBBING
• BULBOUS ENLARGEMENT OF THE DISTAL SEGMENTS OF THE DIGITS DUE TO
INCREASE IN SOFT TISSUE
• IT TAKES 2 TO 3 WEEKS FOR CLUBBING TO MANIFEST
• CLUBBING FIRST APPEARS IN THE INDEX FINGER
• CLUBBING IS SEEN IN 1% OF HOSPITAL ADMISSIONS BUT IS ASSOCIATED WITH A
SERIOUS DIAGNOSIS IN 40% INDIVIDUALS
27. RESPIRATORY CAUSES OF CLUBBING
• CLUBBING DOES NOT OCCUR IN COPD
• HYPERTROPHIC OSTEO ARTHROPATHY – LUNG CANCER, CYSTIC
FIBROSIS,SUPPURATIVE LUNG DISEASES
• CLUBBING, TENDERNESS OF WRISTS AND ANKLES
• SUBPERIOSTEAL NEW BONE FORMATION
35. BLOOD PRESSURE
• PULSUS PARADOXUS
• INSPIRATORY FALL OF SBP >10 MM HG
• OMINOUS SIGN IN OBSTRUCTIVE LUNG DISEASE
36. RESPIRATION
• RATE (12 – 16/MIN)
• TACHYPNEA (RATE MORE THAN 20 IS ABNORMAL IN ADULTS)
• 15 TO 20 BREATHS PER MIN MAYBE DUE TO ANXIETY
• DIFFERENT FROM DYSPNEA
• BRADYPNEA
• APNEA (CENTRAL APNEA/ OBSTRUCTIVE APNEA)
• TYPE OF RESPIRATION
• ABDOMINOTHORACIC
• THORACOABDOMINAL
• THORACIC – DIAPHRAGMATIC PARALYSIS, PERITONITIS, ASCITES
• ABDOMINAL – PLEURITIS, LUNG COLLAPSE
• PATTERN OF RESPIRATION
• PARADOXICAL RESPIRATION AND THORACO-ABDOMINAL ASYNCHRONY
37.
38.
39. • CHEYNE STOKES BREATHING
• SEVERELY ILL PATIENTS
• SEVERE HEART FAILURE
• NARCOTIC DRUG POISONING
• NEUROLOGICAL DISORDERS
• SLEEP, HEALTHY ADULTS IN ALTITUDE
• DUE TO ABNORMAL FEEDBACK FROM CAROTID CHEMORECEPTORS TO
RESPIRATORY CENTRE
44. OTHER SYSTEM EXAMINATION
• MUSCULOSKELETAL SYSTEM (TO R/O CONNECTIVE TISSUE DISEASES)
• CARDIOVASCULAR SYSTEM (TO R/O PULMONARY HYPERTENSION AND COR
PULMONALE)
• ABDOMEN (ASCITES CAN CAUSE HEPATIC HYDROTHORAX)
45. REFERENCES
• MACLEOD’S CLINICAL EXAMINATION 14TH ED.
• HUTCHISON’S CLINICAL METHODS 24TH ED.
• ALAGAPPAN MANUAL OF PRACTICAL MEDICINE, 5TH ED.
• P.J.MEHTA’S PRACTICAL MEDICINE
• HARRISON PRINCIPLES OF INTERNAL MEDICINE 20TH ED.
• FISHMAN’S PULMONARY DISEASES AND DISORDERS, 5TH ED.
• MURRAY & NADEL TEXTBOOK OF RESPIRATORY MEDICINE 6TH ED.