Each examining system can be described using four elements;
- looking/inspection
- feeling/palpation
- tapping/percussion
- listening/auscultation
- assessment of function
Brief Presentation on clinical examination of Respiratory System with Report of Normal case
references:
macleod's clinical examination 13th edition
hutchinson clinical methods
R Alagappan - Manual of Practical Medicine, 4th Edition
Brief Presentation on clinical examination of Respiratory System with Report of Normal case
references:
macleod's clinical examination 13th edition
hutchinson clinical methods
R Alagappan - Manual of Practical Medicine, 4th Edition
Brief Presentation on clinical examination of Cardio Vascular System with Report of Normal case
references:
macleod's clinical examination 13th edition
hutchinson clinical methods
COMPLETE EXAMINATION OF RESPIRATORY SYSTEM IN PEDIATRICS. IT HAS BEEN SUMMARIZED FROM ALL WELL KNOWN 32 BOOKS UNDER GUIDANCE OF ONE OF THE BEST PEDIATRIC DOCTORS AND PROFESSORS .
BY DR. SURAJ R. DHANKIKAR.
Cyanosis
Cyanosis refers to a bluish color of the skin and mucous membranes resulting from an increased quantity of reduced hemoglobin/deoxyhemoglobin or abnormal hemoglobin derivatives, in the small blood vessels of those areas.
Explanation of what splenomegaly is in relation to its dimension deviation from normal spleen.Classification of splenomegaly according to it's size in adult and pediatric. The causes of splenomegaly along with the symptom that would manifest as a result of this anomaly. Lastly, diagnosis of splenomegaly
Diabetes Mellitus: Presentation and CLinical ExaminationPranab Chatterjee
The presentation which won the Best Paper award at the Students' Paper Presentation in Rhapsody 2010. This paper was presented by Dr. Rimesh Pal Medical College Kolkata, 3rd Professional MBBS Student.
Brief Presentation on clinical examination of Cardio Vascular System with Report of Normal case
references:
macleod's clinical examination 13th edition
hutchinson clinical methods
COMPLETE EXAMINATION OF RESPIRATORY SYSTEM IN PEDIATRICS. IT HAS BEEN SUMMARIZED FROM ALL WELL KNOWN 32 BOOKS UNDER GUIDANCE OF ONE OF THE BEST PEDIATRIC DOCTORS AND PROFESSORS .
BY DR. SURAJ R. DHANKIKAR.
Cyanosis
Cyanosis refers to a bluish color of the skin and mucous membranes resulting from an increased quantity of reduced hemoglobin/deoxyhemoglobin or abnormal hemoglobin derivatives, in the small blood vessels of those areas.
Explanation of what splenomegaly is in relation to its dimension deviation from normal spleen.Classification of splenomegaly according to it's size in adult and pediatric. The causes of splenomegaly along with the symptom that would manifest as a result of this anomaly. Lastly, diagnosis of splenomegaly
Diabetes Mellitus: Presentation and CLinical ExaminationPranab Chatterjee
The presentation which won the Best Paper award at the Students' Paper Presentation in Rhapsody 2010. This paper was presented by Dr. Rimesh Pal Medical College Kolkata, 3rd Professional MBBS Student.
An ECG (electrocardiogram) records the electrical activity of your heart at rest. It provides information about your heart rate and rhythm and shows if there is an enlargement of the heart due to high blood pressure (hypertension) or evidence of a previous heart attack (myocardial infarction).
Questions and Answers related to ECG and illustration. Short assignment with diagram and images
Radiation Diagnosis of Disease of Heart and Blood VesselsFaizan Siddiqui
General Principal Includes :
Ischemia testing
Maximal workload testing (VO2max)
Contractile reserve testing
Viability testing
Before the test:
Assess the pretest probability
Know sensitivity and specificity
Are there therapeutic consequences?
Contains 10 images related to topic "Biochemistry of blood" for printing as project or assignment.
Perfectly describe the synthesis and degradation of blood and how the oxygen and carbon dioxide is being transported in cellular and tissue level, formation of blood and much more :)
Fa1zanS: Disorder of exchange of chromoprotein (Detail review : Explainaton o...Faizan Siddiqui
Detailed review : Explainaton of its PPT topic in details, focusing on the above topics :
Disorder of exchange of Chromoprotein (endogenous pigment).
Disorder of exchange of nucleic acids.
Disorder of mineral exchange.
Pathological calcification.
Formation of Stone.
Fa1zanS: Methods of studying biochemical properties of microorganismFaizan Siddiqui
Methods of studying biochemical properties of microorganism, how they are cultured in laboratory and how they play key role in maintaining the equilibrium of our environment, their role in energy production, agriculture, ecological balance. Media: Providing Nutrients in the Laboratory. Incubation, Inspection, and Identification. Methods of cultivation
Fa1zanS: Disorder of exchange of Chromoprotein (endogenous pigment). Disorder...Faizan Siddiqui
Disorder of exchange of Chromoprotein (endogenous pigment).
Disorder of exchange of nucleic acids.
Disorder of mineral exchange.
Pathological calcification.
Formation of Stone.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
1. General Examination of The
Patient.
Submitted to:
Mr. Turldaliev Sir
Subject:
General Medicine
Prepared By:
Mohd Faizan Siddiqui
Group:
19-A
OSH STATE UNIVERSITY
INTERNATIONAL MEDICAL FACULTY
2. Introduction
DOCTORS SHOULD BE OBSERVANT,LIKE A DETECTIVE;
“Ethan Hunt or James Bond”
Look at the patients general appearance…at the face ,hands
and body
Each examining system can be described using four elements;
- looking/inspection
- feeling/palpation
- tapping/percussion
- listening/auscultation
- assessment of function
3. First impressions…..
Decide how sick is your patient?
Is He/She well ,sitting up and talking?
Or ill, totally not aware of his/her
surroundings?
4. VITAL SIGNS
PULSE
BLOOD PRESSURE
TEMPERATURE
RESPIRATORY RATE
Should be assessed immediately once you
discover that your patients unwell.
They provide important basic physiological
information.
5. Weight,body habitus and posture
Obesity,BMI >30.
Any wasting of muscles?
Tall or short?
Always observe when the patient walks into
the examination room.
6. hydration
Mild-2.5L deficit
-mild thirst,dry mucous membranes,concentrated urine
• Moderate – 4L deficit
-as above with moderate thirst,reduced skin turgor(especially the
arms,forehead,chest and abdomen) , tachycardia
• Severe – 6L
-great thirst,reduced skin turgor and decreased eyeball pressure
-collapsed veins,sunken eyes,postural hypotension,oligu
7. FACIAL
Specific diagnosis can be made by just
looking at a patient’s face.
Some facial characteristics are so typical of
certain diseases that they immediately
suggest the diagnosis, so called diagnostic
facies.
13. JAUNDICE
It is the yellowish discolouration of a patient’s
skin and sclerae that results from
hyperbilirubinemia.
It happens when the serum bilirubin level
rises twice above the normal upper limit.
It is deposited in the tissues of the body that
contains elastin.
15. CYANOSIS
Blue discolouration of the skin and mucous membranes;it is
due to the presence of deoxygenated haemoglobin in the
superficial blood vessels.
Occurs when there is more than 50g/L of deoxygenated
haemoglobin in the capillary blood.
Types-central and peripheral
Central cyanosis- abnormal amount of deoxygenated
haemoglobin in the arteries and that a blue discolouration is
present in parts of the body with good circulation.eg;tongue.
Peripheral cyanosis-occurs when blood supply to a particular
part of body is reduced,eg;lips in cold weather becomes blue
but the tongue is spared.
17. Causes of cyanosis
Central cyanosis
1)Decreased arterial oyygen
saturation.
-high altitude
-lung disease
-right to left cardiac shunt
2)Polycythaemia
3)Haemoglobin
abnormalities;methaemoglo
binemia,sulphaemoglobinem
ia
Peripheral cyanosis
1)All the causes of central
cyanosis
2)Exposure to cold
3)Reduced cardiac output
-left ventricular failure
-shock
4)Arterial or venous obstruction
18. PALLOR
Deficiency of haemoglobin can produce
pallor of the skin.
Should be noticeable especially in the
mucous membranes of the sclerae if the
anaemia is severe- Hb of less than 7g/L.
Facial pallor can also be seen in patients with
shock,due to the reduction of cardiac output.
These patients usually appear cold and
clammy and significantly hypotensive.
19. Causes of anaemia
MICROCYTIC ANAEMIA
1)Iron deficiency anaemia
-chronic bleeding
-malabsorption
-hookworm
-pregnancy
2)Thalassemia minor
3)Sideroblastic anaemia
4)Longstanding anaemia of chronic blood loss
20. Macrocytic anaemia
Megaloblastic bone marrow
1)Vitamin B12 defiency due to
-pernicious anaemia
-gastrectomy
-tropical sprue
-ileal disease;crohns disease,ileal resection
-fish tapeworm
-poor diet in vegetarians
21. Macrocytic anaemia
2) Folate deficiency due to
-dietary defiency in alcoholics
-malabsorption
-increased cell turnover eg;pregnancy,leukemia,chronic
haemolysis
-anti folate drugs –
phenytoin,methotrexate,sulphasalazine
non megaloblastic bone marrow
-alcohol,cirrohis of the
liver,hypothyroidism,myelodysplastic syndrome
28. Pigmentation in the mouth
Heavy metals-
lead,bismuth,iron;haemochromatosis there is
blue grey pigmentation in the hard palate
Drugs-antimalarials,OCPs(brown/black
pigmentation anywhere in the mouth)
Addisons disease
Peutz-jeghers syndrome
Malignant melanoma
34. NECK;lymphadenopathy,goitre
During palpation of lymph nodes the
following features should be considered;
SITE
-Localised or generalised?
-palpable lymph node areas are;
Epitrochlear,axillary,cervical and
occipital,supraclavicular,para-aortic,inguinal
and popliteal.
35. NECK;lymphadenopathy,goitre
SIZE
CONSISTENCY
-hard are suggestive of carcinoma
-soft may be normal
-rubbery may be due to lymphoma
TENDERNESS
-Acute infection of inflammation
FIXATION
-If fixed to the underlying structures its most likely malignant
OVERLYING SKIN
-if inflammed then its suggestive of infection,teethered suggests
carcinoma.
39. localised
Local or acute infection
Metastasis from carcinoma or other solid
tumour
Lymphoma especially hodgkin’s disease
40. NAILS
CLUBBING
-Increase in the soft tissue of the distal part of the fingers or toes.
CAUSES
1)Cardiovascular
-cyanotic congenital heart disease,IE
2) Respiratory
-lung carcinoma
-bronchiectasis,lung abscess,emphyema
-lung fibrosis
3)Gastrointestinal
-cirrohis,IBS,Coeliac disease
4)Thyrotoxicosis
5)Familial