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General Examination of The
Patient.
Submitted to:
Mr. Turldaliev Sir
Subject:
General Medicine
Prepared By:
Mohd Faizan Siddiqui
Group:
19-A
OSH STATE UNIVERSITY
INTERNATIONAL MEDICAL FACULTY
Introduction
 DOCTORS SHOULD BE OBSERVANT,LIKE A DETECTIVE;
“Ethan Hunt or James Bond”
 Look at the patients general appearance…at the face ,hands
and body
 Each examining system can be described using four elements;
- looking/inspection
- feeling/palpation
- tapping/percussion
- listening/auscultation
- assessment of function
First impressions…..
 Decide how sick is your patient?
 Is He/She well ,sitting up and talking?
 Or ill, totally not aware of his/her
surroundings?
VITAL SIGNS
 PULSE
 BLOOD PRESSURE
 TEMPERATURE
 RESPIRATORY RATE
 Should be assessed immediately once you
discover that your patients unwell.
 They provide important basic physiological
information.
Weight,body habitus and posture
 Obesity,BMI >30.
 Any wasting of muscles?
 Tall or short?
 Always observe when the patient walks into
the examination room.
hydration
 Mild-2.5L deficit
-mild thirst,dry mucous membranes,concentrated urine
• Moderate – 4L deficit
-as above with moderate thirst,reduced skin turgor(especially the
arms,forehead,chest and abdomen) , tachycardia
• Severe – 6L
-great thirst,reduced skin turgor and decreased eyeball pressure
-collapsed veins,sunken eyes,postural hypotension,oligu
FACIAL
 Specific diagnosis can be made by just
looking at a patient’s face.
 Some facial characteristics are so typical of
certain diseases that they immediately
suggest the diagnosis, so called diagnostic
facies.
Important diagnostic facies
 Acromegaly
 Cushingnoid
 Down syndrome
 Hippocratic
 Marfanoid
 Myxoedemetous
 Thyrotoxic
 parkinsonism
acromegaly
Acromegaly hands
Downs syndrome
Cushing’s syndrome
JAUNDICE
 It is the yellowish discolouration of a patient’s
skin and sclerae that results from
hyperbilirubinemia.
 It happens when the serum bilirubin level
rises twice above the normal upper limit.
 It is deposited in the tissues of the body that
contains elastin.
jaundice
CYANOSIS
 Blue discolouration of the skin and mucous membranes;it is
due to the presence of deoxygenated haemoglobin in the
superficial blood vessels.
 Occurs when there is more than 50g/L of deoxygenated
haemoglobin in the capillary blood.
 Types-central and peripheral
 Central cyanosis- abnormal amount of deoxygenated
haemoglobin in the arteries and that a blue discolouration is
present in parts of the body with good circulation.eg;tongue.
 Peripheral cyanosis-occurs when blood supply to a particular
part of body is reduced,eg;lips in cold weather becomes blue
but the tongue is spared.
cyanosis
Causes of cyanosis
 Central cyanosis
1)Decreased arterial oyygen
saturation.
-high altitude
-lung disease
-right to left cardiac shunt
2)Polycythaemia
3)Haemoglobin
abnormalities;methaemoglo
binemia,sulphaemoglobinem
ia
 Peripheral cyanosis
1)All the causes of central
cyanosis
2)Exposure to cold
3)Reduced cardiac output
-left ventricular failure
-shock
4)Arterial or venous obstruction
PALLOR
 Deficiency of haemoglobin can produce
pallor of the skin.
 Should be noticeable especially in the
mucous membranes of the sclerae if the
anaemia is severe- Hb of less than 7g/L.
 Facial pallor can also be seen in patients with
shock,due to the reduction of cardiac output.
These patients usually appear cold and
clammy and significantly hypotensive.
Causes of anaemia
 MICROCYTIC ANAEMIA
1)Iron deficiency anaemia
-chronic bleeding
-malabsorption
-hookworm
-pregnancy
2)Thalassemia minor
3)Sideroblastic anaemia
4)Longstanding anaemia of chronic blood loss
Macrocytic anaemia
 Megaloblastic bone marrow
1)Vitamin B12 defiency due to
-pernicious anaemia
-gastrectomy
-tropical sprue
-ileal disease;crohns disease,ileal resection
-fish tapeworm
-poor diet in vegetarians
Macrocytic anaemia
2) Folate deficiency due to
-dietary defiency in alcoholics
-malabsorption
-increased cell turnover eg;pregnancy,leukemia,chronic
haemolysis
-anti folate drugs –
phenytoin,methotrexate,sulphasalazine
non megaloblastic bone marrow
-alcohol,cirrohis of the
liver,hypothyroidism,myelodysplastic syndrome
Normochromic anaemia
 Bone marrow failure
-aplastic anaemia
-ineffective haematopoiesis
-infiltration
• Anaemia of chronic disease
-chronic inflammation
-liver disease
-malignancies,chronic renal failure
• Haemolytic anaemia
Oral cavity
 The teeth and breath
 Check the oral cavity looking for
 MOUTH ULCERS
-Aphtous,drugs and trauma
-gastrointestinal disease;inflammatory bowel
disease,coeliac disease
-rheumatological;Behcets syndrome,reiter
-erythema multiforme
-infections;herpes zoster,simplex,syphilis,tuberculosis
Oral Cavity
Behcets ulcers
Gum hypertrophy
 Phenytoin
 Pregnancy
 Scurvy(vitamin C deficiency;gums become
swollen,spongy,red and bleeds easily)
 Gingivitis;smoking
 leukemia
Gum hypertrophy
Pigmentation in the mouth
 Heavy metals-
lead,bismuth,iron;haemochromatosis there is
blue grey pigmentation in the hard palate
 Drugs-antimalarials,OCPs(brown/black
pigmentation anywhere in the mouth)
 Addisons disease
 Peutz-jeghers syndrome
 Malignant melanoma
Pigmentation in the mouth
HAIR
 ALOPECIA
 Non-scarring
-alopecia areta
-scalp ring worm
-traction alopecia
• Scarring
-burns,radiation,lupoid erythema,sarcoidosis
Alopecia areata
Traction alopecia
Alopecia totalis
NECK;lymphadenopathy,goitre
 During palpation of lymph nodes the
following features should be considered;
 SITE
-Localised or generalised?
-palpable lymph node areas are;
Epitrochlear,axillary,cervical and
occipital,supraclavicular,para-aortic,inguinal
and popliteal.
NECK;lymphadenopathy,goitre
 SIZE
 CONSISTENCY
-hard are suggestive of carcinoma
-soft may be normal
-rubbery may be due to lymphoma
TENDERNESS
-Acute infection of inflammation
FIXATION
-If fixed to the underlying structures its most likely malignant
OVERLYING SKIN
-if inflammed then its suggestive of infection,teethered suggests
carcinoma.
Cervical lymphadenopathy
CAUSES OF LYMPHADENOPATHY
 GENERALISED
-lymphoma
-leukemia
-infections
-viral;infectious mononucleosis,CMV,HIV
-bacterial;tuberculosis,syphilis
-protozoal;toxoplasmosis
-connective tissue disease
-infitration;sarcoidosis
-drugs;phenytoin
localised
 Local or acute infection
 Metastasis from carcinoma or other solid
tumour
 Lymphoma especially hodgkin’s disease
NAILS
 CLUBBING
 -Increase in the soft tissue of the distal part of the fingers or toes.
 CAUSES
1)Cardiovascular
-cyanotic congenital heart disease,IE
2) Respiratory
-lung carcinoma
-bronchiectasis,lung abscess,emphyema
-lung fibrosis
3)Gastrointestinal
-cirrohis,IBS,Coeliac disease
4)Thyrotoxicosis
5)Familial
Clubbing
psoriasis
THANKYOU
Prepared By:
Mohd Faizan Siddiqui
Group:
19-A

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General Examination of The Patient.

  • 1. General Examination of The Patient. Submitted to: Mr. Turldaliev Sir Subject: General Medicine Prepared By: Mohd Faizan Siddiqui Group: 19-A OSH STATE UNIVERSITY INTERNATIONAL MEDICAL FACULTY
  • 2. Introduction  DOCTORS SHOULD BE OBSERVANT,LIKE A DETECTIVE; “Ethan Hunt or James Bond”  Look at the patients general appearance…at the face ,hands and body  Each examining system can be described using four elements; - looking/inspection - feeling/palpation - tapping/percussion - listening/auscultation - assessment of function
  • 3. First impressions…..  Decide how sick is your patient?  Is He/She well ,sitting up and talking?  Or ill, totally not aware of his/her surroundings?
  • 4. VITAL SIGNS  PULSE  BLOOD PRESSURE  TEMPERATURE  RESPIRATORY RATE  Should be assessed immediately once you discover that your patients unwell.  They provide important basic physiological information.
  • 5. Weight,body habitus and posture  Obesity,BMI >30.  Any wasting of muscles?  Tall or short?  Always observe when the patient walks into the examination room.
  • 6. hydration  Mild-2.5L deficit -mild thirst,dry mucous membranes,concentrated urine • Moderate – 4L deficit -as above with moderate thirst,reduced skin turgor(especially the arms,forehead,chest and abdomen) , tachycardia • Severe – 6L -great thirst,reduced skin turgor and decreased eyeball pressure -collapsed veins,sunken eyes,postural hypotension,oligu
  • 7. FACIAL  Specific diagnosis can be made by just looking at a patient’s face.  Some facial characteristics are so typical of certain diseases that they immediately suggest the diagnosis, so called diagnostic facies.
  • 8. Important diagnostic facies  Acromegaly  Cushingnoid  Down syndrome  Hippocratic  Marfanoid  Myxoedemetous  Thyrotoxic  parkinsonism
  • 13. JAUNDICE  It is the yellowish discolouration of a patient’s skin and sclerae that results from hyperbilirubinemia.  It happens when the serum bilirubin level rises twice above the normal upper limit.  It is deposited in the tissues of the body that contains elastin.
  • 15. CYANOSIS  Blue discolouration of the skin and mucous membranes;it is due to the presence of deoxygenated haemoglobin in the superficial blood vessels.  Occurs when there is more than 50g/L of deoxygenated haemoglobin in the capillary blood.  Types-central and peripheral  Central cyanosis- abnormal amount of deoxygenated haemoglobin in the arteries and that a blue discolouration is present in parts of the body with good circulation.eg;tongue.  Peripheral cyanosis-occurs when blood supply to a particular part of body is reduced,eg;lips in cold weather becomes blue but the tongue is spared.
  • 17. Causes of cyanosis  Central cyanosis 1)Decreased arterial oyygen saturation. -high altitude -lung disease -right to left cardiac shunt 2)Polycythaemia 3)Haemoglobin abnormalities;methaemoglo binemia,sulphaemoglobinem ia  Peripheral cyanosis 1)All the causes of central cyanosis 2)Exposure to cold 3)Reduced cardiac output -left ventricular failure -shock 4)Arterial or venous obstruction
  • 18. PALLOR  Deficiency of haemoglobin can produce pallor of the skin.  Should be noticeable especially in the mucous membranes of the sclerae if the anaemia is severe- Hb of less than 7g/L.  Facial pallor can also be seen in patients with shock,due to the reduction of cardiac output. These patients usually appear cold and clammy and significantly hypotensive.
  • 19. Causes of anaemia  MICROCYTIC ANAEMIA 1)Iron deficiency anaemia -chronic bleeding -malabsorption -hookworm -pregnancy 2)Thalassemia minor 3)Sideroblastic anaemia 4)Longstanding anaemia of chronic blood loss
  • 20. Macrocytic anaemia  Megaloblastic bone marrow 1)Vitamin B12 defiency due to -pernicious anaemia -gastrectomy -tropical sprue -ileal disease;crohns disease,ileal resection -fish tapeworm -poor diet in vegetarians
  • 21. Macrocytic anaemia 2) Folate deficiency due to -dietary defiency in alcoholics -malabsorption -increased cell turnover eg;pregnancy,leukemia,chronic haemolysis -anti folate drugs – phenytoin,methotrexate,sulphasalazine non megaloblastic bone marrow -alcohol,cirrohis of the liver,hypothyroidism,myelodysplastic syndrome
  • 22. Normochromic anaemia  Bone marrow failure -aplastic anaemia -ineffective haematopoiesis -infiltration • Anaemia of chronic disease -chronic inflammation -liver disease -malignancies,chronic renal failure • Haemolytic anaemia
  • 23. Oral cavity  The teeth and breath  Check the oral cavity looking for  MOUTH ULCERS -Aphtous,drugs and trauma -gastrointestinal disease;inflammatory bowel disease,coeliac disease -rheumatological;Behcets syndrome,reiter -erythema multiforme -infections;herpes zoster,simplex,syphilis,tuberculosis
  • 26. Gum hypertrophy  Phenytoin  Pregnancy  Scurvy(vitamin C deficiency;gums become swollen,spongy,red and bleeds easily)  Gingivitis;smoking  leukemia
  • 28. Pigmentation in the mouth  Heavy metals- lead,bismuth,iron;haemochromatosis there is blue grey pigmentation in the hard palate  Drugs-antimalarials,OCPs(brown/black pigmentation anywhere in the mouth)  Addisons disease  Peutz-jeghers syndrome  Malignant melanoma
  • 30. HAIR  ALOPECIA  Non-scarring -alopecia areta -scalp ring worm -traction alopecia • Scarring -burns,radiation,lupoid erythema,sarcoidosis
  • 34. NECK;lymphadenopathy,goitre  During palpation of lymph nodes the following features should be considered;  SITE -Localised or generalised? -palpable lymph node areas are; Epitrochlear,axillary,cervical and occipital,supraclavicular,para-aortic,inguinal and popliteal.
  • 35. NECK;lymphadenopathy,goitre  SIZE  CONSISTENCY -hard are suggestive of carcinoma -soft may be normal -rubbery may be due to lymphoma TENDERNESS -Acute infection of inflammation FIXATION -If fixed to the underlying structures its most likely malignant OVERLYING SKIN -if inflammed then its suggestive of infection,teethered suggests carcinoma.
  • 36.
  • 38. CAUSES OF LYMPHADENOPATHY  GENERALISED -lymphoma -leukemia -infections -viral;infectious mononucleosis,CMV,HIV -bacterial;tuberculosis,syphilis -protozoal;toxoplasmosis -connective tissue disease -infitration;sarcoidosis -drugs;phenytoin
  • 39. localised  Local or acute infection  Metastasis from carcinoma or other solid tumour  Lymphoma especially hodgkin’s disease
  • 40. NAILS  CLUBBING  -Increase in the soft tissue of the distal part of the fingers or toes.  CAUSES 1)Cardiovascular -cyanotic congenital heart disease,IE 2) Respiratory -lung carcinoma -bronchiectasis,lung abscess,emphyema -lung fibrosis 3)Gastrointestinal -cirrohis,IBS,Coeliac disease 4)Thyrotoxicosis 5)Familial
  • 42.
  • 44.
  • 45. THANKYOU Prepared By: Mohd Faizan Siddiqui Group: 19-A