BILIARY TRACT
PATHOLOGY
Dr.SundaresanDr.Sundaresan
PathologyPathology
Loyola collegeLoyola college
VAST MAJORITYVAST MAJORITY
REMAINREMAIN
ASYMPTOMATICASYMPTOMATIC
FOR DECADESFOR DECADES
TOGETHERTOGETHER
TYPES
CHOLESTEROL STONES – 80%CHOLESTEROL STONES – 80%
CRYSTYALLINECRYSTYALLINE
CHOLESTEROLCHOLESTEROL
MONOHYDRATEMONOHYDRATE
PIGMENT STONESPIGMENT STONES
WHO ARE AT RISK ?
STORY BEHIND THESE
CHOLESTEROL
STONES
BILE SUPERSATURATED WITHBILE SUPERSATURATED WITH
CHOLESTEROLCHOLESTEROL
GB HYPOMOTILITY PROMOTESGB HYPOMOTILITY PROMOTES
NUCLEATIONNUCLEATION
ACCELERATED NUCLEATIONACCELERATED NUCLEATION
MUCUS HYPERSECRETION TRAPSMUCUS HYPERSECRETION TRAPS
CRYSTALS AND PERMITSCRYSTALS AND PERMITS
AGGREATION.AGGREATION.
PREGNANCYPREGNANCY
RAPID WEIGHT LOSSRAPID WEIGHT LOSS
TPNTPN
PROLONGED FASTINGPROLONGED FASTING
SPINAL CORD INJURYSPINAL CORD INJURY
CONDITIONS PROMOTING DEFFECTIVE
GB EMPTYING
CHOLESTEROL STONES
PURITY OF STONES VARIESPURITY OF STONES VARIES
FROM 100% DOWN TOFROM 100% DOWN TO
AROUND 50%AROUND 50%
CA CARBONATE, PHOSPHATECA CARBONATE, PHOSPHATE
AND BILIRUBINAND BILIRUBIN
MULTIPLE STONESMULTIPLE STONES
SURFACES ROUNDED ORSURFACES ROUNDED OR
FACETEDFACETED
RADIOLUCENT 80%RADIOLUCENT 80%
PIGMENT STONES
INFECTIONINFECTION –– E.COLI ,E.COLI ,
ASCARIS LUMBRICOIDES, LIVERASCARIS LUMBRICOIDES, LIVER
FLUKEFLUKE
INTRAVASCULARINTRAVASCULAR
HEMOLYSISHEMOLYSIS
BLACK - STERILE ROOM ,BLACK - STERILE ROOM ,
BROWN - INFECTED GBBROWN - INFECTED GB
50-75% OF BLACK STONES ARE50-75% OF BLACK STONES ARE
RADIO-OPAQUERADIO-OPAQUE
CHOLESTEROLOSIS
STRAWBERRY GALLBLADDERSTRAWBERRY GALLBLADDER
ASYMPTOMATIC 70 TO 80%ASYMPTOMATIC 70 TO 80%
BILIARY PAINBILIARY PAIN
CLINICAL FEATURES
SIZE DOES MATTER HERE
VERY SMALL STONES ORVERY SMALL STONES OR
GRAVEL ARE THEGRAVEL ARE THE
DANGEROUS STONESDANGEROUS STONES
COMPLICATION
EMPYEMAEMPYEMA
PERFORATIONPERFORATION
FISTULAFISTULA
CHOLANGITISCHOLANGITIS
OBSTRUCTIVE CHOLESTASISOBSTRUCTIVE CHOLESTASIS
PANCREATITISPANCREATITIS
ACUTE
CHOLECYSTITIS
MC REASON FORMC REASON FOR
EMERGENCYEMERGENCY
CHOLECYSTECTOMYCHOLECYSTECTOMY
STONES REMAIN THE
PRIMARY CAUSE OF ACUTE
EPISODE
ACUTE ACALCULOUS CHOLECYSTITIS
POST OP STATE AFTER MAJOR SxPOST OP STATE AFTER MAJOR Sx
SEVERE TRAUMASEVERE TRAUMA
SEVERE BURNSSEVERE BURNS
MULTISYSTEM ORGAN FAILUREMULTISYSTEM ORGAN FAILURE
SEPSISSEPSIS
PROLONGED IV HYPERALIMENTATIONPROLONGED IV HYPERALIMENTATION
POSTPARTUM STATEPOSTPARTUM STATE
ACUTE ACALCULOUS
CHOLECYSTITIS
RESULT FROM
ISCHAEMIA
CHRONIC CHOLECYSTITIS
SEQUEL TO REPEATED BOUTS OFSEQUEL TO REPEATED BOUTS OF
ACUTE EPISODESACUTE EPISODES
ROKITANSKY- ASCHOFF SINUSESROKITANSKY- ASCHOFF SINUSES
PORCELAIN GBPORCELAIN GB
XANTHOGRANULOMATOUSXANTHOGRANULOMATOUS
CHOLECYSTITISCHOLECYSTITIS
HYDROPS OF THE GALL BLADDERHYDROPS OF THE GALL BLADDER
WATCH OUT FOR THESE
COMPLICATIONS
CHOLANGITIS OR SEPSISCHOLANGITIS OR SEPSIS
PERFORATION AND LOCAL ABSCESSPERFORATION AND LOCAL ABSCESS
FORMATIONFORMATION
RUPTURE WITH DIFFUSERUPTURE WITH DIFFUSE
PERITONITISPERITONITIS
FISTULA AND ILEUSFISTULA AND ILEUS
AGGRAVATES PRE EXISTINGAGGRAVATES PRE EXISTING
MEDICAL ILLNESSMEDICAL ILLNESS
CLINICAL FEATURES
RUQ PAINRUQ PAIN
MOST PATIENTS ARE FREE OF JAUNDICEMOST PATIENTS ARE FREE OF JAUNDICE
ATTACK SUBSIDES IN 7 TO 10 DAYSATTACK SUBSIDES IN 7 TO 10 DAYS
ACUTE ACALCULOUS ONE PREDISPOSEDACUTE ACALCULOUS ONE PREDISPOSED
TO GANGRENE AND PERFORATIONTO GANGRENE AND PERFORATION
ACUTE EMPHYSEMATOUS CHOLECYSTITISACUTE EMPHYSEMATOUS CHOLECYSTITIS
LAP
CHOLESCYSTECTOMY

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