More Related Content
Similar to Gall stones
Similar to Gall stones (20)
More from Tagore medical College
More from Tagore medical College (20)
Recently uploaded
Recently uploaded (20)
Gall stones
- 1. BILIARY TRACT PATHOLOGY Dr.SundaresanDr.Sundaresan PathologyPathology Loyola collegeLoyola college
- 4. VAST MAJORITYVAST MAJORITY REMAINREMAIN ASYMPTOMATICASYMPTOMATIC FOR DECADESFOR DECADES TOGETHERTOGETHER
- 9. TYPES CHOLESTEROL STONES – 80%CHOLESTEROL STONES – 80% CRYSTYALLINECRYSTYALLINE CHOLESTEROLCHOLESTEROL MONOHYDRATEMONOHYDRATE PIGMENT STONESPIGMENT STONES
- 11. WHO ARE AT RISK ?
- 15. BILE SUPERSATURATED WITHBILE SUPERSATURATED WITH CHOLESTEROLCHOLESTEROL GB HYPOMOTILITY PROMOTESGB HYPOMOTILITY PROMOTES NUCLEATIONNUCLEATION ACCELERATED NUCLEATIONACCELERATED NUCLEATION MUCUS HYPERSECRETION TRAPSMUCUS HYPERSECRETION TRAPS CRYSTALS AND PERMITSCRYSTALS AND PERMITS AGGREATION.AGGREATION.
- 17. PREGNANCYPREGNANCY RAPID WEIGHT LOSSRAPID WEIGHT LOSS TPNTPN PROLONGED FASTINGPROLONGED FASTING SPINAL CORD INJURYSPINAL CORD INJURY CONDITIONS PROMOTING DEFFECTIVE GB EMPTYING
- 18. CHOLESTEROL STONES PURITY OF STONES VARIESPURITY OF STONES VARIES FROM 100% DOWN TOFROM 100% DOWN TO AROUND 50%AROUND 50% CA CARBONATE, PHOSPHATECA CARBONATE, PHOSPHATE AND BILIRUBINAND BILIRUBIN MULTIPLE STONESMULTIPLE STONES SURFACES ROUNDED ORSURFACES ROUNDED OR FACETEDFACETED RADIOLUCENT 80%RADIOLUCENT 80%
- 22. PIGMENT STONES INFECTIONINFECTION –– E.COLI ,E.COLI , ASCARIS LUMBRICOIDES, LIVERASCARIS LUMBRICOIDES, LIVER FLUKEFLUKE INTRAVASCULARINTRAVASCULAR HEMOLYSISHEMOLYSIS
- 24. BLACK - STERILE ROOM ,BLACK - STERILE ROOM , BROWN - INFECTED GBBROWN - INFECTED GB 50-75% OF BLACK STONES ARE50-75% OF BLACK STONES ARE RADIO-OPAQUERADIO-OPAQUE
- 29. ASYMPTOMATIC 70 TO 80%ASYMPTOMATIC 70 TO 80% BILIARY PAINBILIARY PAIN CLINICAL FEATURES
- 32. SIZE DOES MATTER HERE VERY SMALL STONES ORVERY SMALL STONES OR GRAVEL ARE THEGRAVEL ARE THE DANGEROUS STONESDANGEROUS STONES
- 40. ACUTE CHOLECYSTITIS MC REASON FORMC REASON FOR EMERGENCYEMERGENCY CHOLECYSTECTOMYCHOLECYSTECTOMY
- 43. STONES REMAIN THE PRIMARY CAUSE OF ACUTE EPISODE
- 48. ACUTE ACALCULOUS CHOLECYSTITIS POST OP STATE AFTER MAJOR SxPOST OP STATE AFTER MAJOR Sx SEVERE TRAUMASEVERE TRAUMA SEVERE BURNSSEVERE BURNS MULTISYSTEM ORGAN FAILUREMULTISYSTEM ORGAN FAILURE SEPSISSEPSIS PROLONGED IV HYPERALIMENTATIONPROLONGED IV HYPERALIMENTATION POSTPARTUM STATEPOSTPARTUM STATE
- 51. CHRONIC CHOLECYSTITIS SEQUEL TO REPEATED BOUTS OFSEQUEL TO REPEATED BOUTS OF ACUTE EPISODESACUTE EPISODES ROKITANSKY- ASCHOFF SINUSESROKITANSKY- ASCHOFF SINUSES PORCELAIN GBPORCELAIN GB XANTHOGRANULOMATOUSXANTHOGRANULOMATOUS CHOLECYSTITISCHOLECYSTITIS HYDROPS OF THE GALL BLADDERHYDROPS OF THE GALL BLADDER
- 63. WATCH OUT FOR THESE COMPLICATIONS CHOLANGITIS OR SEPSISCHOLANGITIS OR SEPSIS PERFORATION AND LOCAL ABSCESSPERFORATION AND LOCAL ABSCESS FORMATIONFORMATION RUPTURE WITH DIFFUSERUPTURE WITH DIFFUSE PERITONITISPERITONITIS FISTULA AND ILEUSFISTULA AND ILEUS AGGRAVATES PRE EXISTINGAGGRAVATES PRE EXISTING MEDICAL ILLNESSMEDICAL ILLNESS
- 64. CLINICAL FEATURES RUQ PAINRUQ PAIN MOST PATIENTS ARE FREE OF JAUNDICEMOST PATIENTS ARE FREE OF JAUNDICE ATTACK SUBSIDES IN 7 TO 10 DAYSATTACK SUBSIDES IN 7 TO 10 DAYS ACUTE ACALCULOUS ONE PREDISPOSEDACUTE ACALCULOUS ONE PREDISPOSED TO GANGRENE AND PERFORATIONTO GANGRENE AND PERFORATION ACUTE EMPHYSEMATOUS CHOLECYSTITISACUTE EMPHYSEMATOUS CHOLECYSTITIS