This document discusses arthropod-borne infections like dengue fever. It provides classifications of arthropod-transmitted diseases including viral, bacterial and parasitic. It then focuses on dengue fever, describing the epidemiology, signs and symptoms, pathogenesis and phases. It discusses the dengue virus and its four serotypes. Prevention and control is through early diagnosis, treatment, community participation and mosquito control efforts like source reduction and active surveillance.
Now a days.All the World is facing a serious problem..Dengue
so i make a presentation on dengue to prevent and aware from dengue...and if you have dengue faver then which types of treatment you use for your Health.
Now a days.All the World is facing a serious problem..Dengue
so i make a presentation on dengue to prevent and aware from dengue...and if you have dengue faver then which types of treatment you use for your Health.
Dengue & Chikungunya - All You Need To Know!Akshit Arora
A presentation on Dengue & Chikungunya and preventive measures! Received via one Instant Messenger application. Don't know about the credibility whether it's actually from the WHO or not! But good enough for education.
Dengue fever is the fastest emerging arboviral infection spread
by Aedes mosquitoes with major public health consequences in
over 100 tropical and sub-tropical countries in South-East Asia,
the Western Pacific, and South and Central America. Up to 2.5
billion people globally live under the threat of dengue fever and its
severe forms—dengue hemorrhagic fever (DHF) or dengue shock
syndrome (DSS). More than 75% of these people, or approximately
1.8 billion, live in the Asia-Pacific Region. As the disease spreads to
new geographical areas, the frequency of the outbreaks is increasing
along with changing disease epidemiology. It is estimated that 50
a million cases of dengue fever occur worldwide annually and half a
million people suffering from DHF require hospitalization each year,
a very large proportion of whom (approximately 90%) are children
less than five years old. About 2.5% of those affected with dengue
die of the disease.
Dengue & Chikungunya - All You Need To Know!Akshit Arora
A presentation on Dengue & Chikungunya and preventive measures! Received via one Instant Messenger application. Don't know about the credibility whether it's actually from the WHO or not! But good enough for education.
Dengue fever is the fastest emerging arboviral infection spread
by Aedes mosquitoes with major public health consequences in
over 100 tropical and sub-tropical countries in South-East Asia,
the Western Pacific, and South and Central America. Up to 2.5
billion people globally live under the threat of dengue fever and its
severe forms—dengue hemorrhagic fever (DHF) or dengue shock
syndrome (DSS). More than 75% of these people, or approximately
1.8 billion, live in the Asia-Pacific Region. As the disease spreads to
new geographical areas, the frequency of the outbreaks is increasing
along with changing disease epidemiology. It is estimated that 50
a million cases of dengue fever occur worldwide annually and half a
million people suffering from DHF require hospitalization each year,
a very large proportion of whom (approximately 90%) are children
less than five years old. About 2.5% of those affected with dengue
die of the disease.
A zoonosis is an infectious disease that has jumped from a non-human animal to humans. Zoonotic pathogens may be bacterial, viral or parasitic, or may involve unconventional agents and can spread to humans through direct contact or through food, water or the environment
COMMUNITY HEALTH NURSING-II
HEALTH PLANNING POLICIES AND
PROBLEMS.To address the unmet needs for contraception, health care infrastructure and health personnel and to provide integrated service delivery for basic reproductive and child health care.
To bring the TFR to replacement level by 2010, through vigorous implication of inter-sectorial operational strategies.
To bring the TFR to replacement level by 2010, through vigorous implication of inter-sectorial operational strategies.
To achieve a stable population by 2045, at a level consistent with the requirements of sustainable economic growth, social development and environmental protection
Address the unmet needs for basic reproductive and child health services, supplies and infrastructure.
Make school education up to age 14 free and compulsory, and reduce drop outs at primary and secondary school levels to below 20 percent for both boys and girls.
Reduce infant mortality rate to below 30 per 1000 live births.Reduce maternal mortality ratio to below 100 per 100,000 live births.
Achieve universal immunization of children against all vaccine preventable diseases
Promote delayed marriage for girls, not earlier than age 18 and preferably after 20 years of age.
Achieve 80 percent institutional deliveries and 100 percent deliveries by trained persons.
Achieve universal access to information/counseling, and services for fertility regulation and contraception with a wide basket of choices.
Achieve 100 per cent registration of births, deaths, marriage and pregnancyContain the spread of Acquired Immunodeficiency Syndrome (AIDS), and promote greater integration between the management of reproductive tract infections (RTI) and sexually transmitted infections (STI) and the National AIDS Control Organisation.
Prevent and control communicable diseasesIntegrate Indian Systems of Medicine (ISM) in the provision of reproductive and child health services, and in reaching out to households.
Promote vigorously the small family norm to achieve replacement levels of TFR.
Bring about convergence in implementation of related social sector programs so that family welfare becomes a people centered programme.Decentralized planning and programme implementation
Convergence of service delivery at village level
Empowering women for improved health and nutrition
Child health and survival
Meeting the unmet needs for family welfare services
Underserved population groups(urban slums, tribals, hill areas, adolescents)
Diverse health care providers
Collaboration with and commitments from non government organisations and private sector
Mainstreaming Indian systems of medicine and Homeopathy
A National Commission on Population, presided over by the Prime Minister, will have the Chief Ministers of all states and UTs, and the Central Minister in charge of the Department of Family Welfare and other concerned Central Ministries and Departments reputed demographers, public health professionals, and NGOs as members.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
2. ARTHROPODINFECTIONS
• Arthropods forms a major group of disease vector with mosquitoes, flies, sand
flies ,lice, fleas , ticks and mites transmitting a huge number of diseases.
• Many such vectors are hematophagous which feed on blood at some or at all
aspects of life.
• Arthropod - A Greek Word Meaning Jointeleg(arthron:joint,pod:foot)
• It Is An Invertebrate Animal Having An Exoskeleton, A Segmented Body And
Paired Body Appendages. VECTOR • A Vector Is An Organism That Does Not
Cause Disease In Itself ,But Does Spread The Parasite To Other Life Forms.
4. DENGUE
• Is An Acute Infectious Viral Disease, Also Known As Breakbone Fever.
• It Is AArthropod-borne (Arboviral ) Illness In Human .
• It Is Caused By Infection With 1 Of The 4 Serotypes Of Dengue Virus
(Denv1,2,3,4), Which Is A Flavivirus (A Genus Of Single-stranded Nonsegmented
Rna Viruses).
• Once Affected Persons Acquire Long-life Serotype Specific Immunity.
• Dengue Is Transmitted By Mosquitoes Of The Genus
5. • Dr Benjamin rush A Professor Of Chemistry And Medical Theory At The
UPEN, During The Philadelphia Epidemic 1779 -1780 , First Described The
Dramatic Symptoms Of Dengue As Break Bone Fever.
• A Small Percentage Of Persons Who Have Previously Been Infected By
One Dengue Serotype Develop Bleeding And Endothelial Leak Up On
Infection With Another Dengue Serotype. This Syndrome Is Termed Dengue
Hemorrhagic Fever (DHF). Also Been Termed Dengue Vasculopathy.
• Vascular Leakage In These Patients Results In Hemoconcentration And
Serious Effusions And Can Lead To Circulatory Collapse. 4
• This, IN Conjunction With Severe Hemorrhagic Complications, Can Lead
To Dengue Shock Syndrome(dss), Which Poses A Greater Fatality Risk Than
Bleeding .
6. • EPIDEMIOLOGY
• In India First Outbreak Of Dengue Was Recorded In 1812. A Double Peak Hemorrhagic Fever
Epidemic Occurred In India For The First Time In Calcutta Between July 1963 & March 1964
In New Delhi, Outbreaks Of Dengue Fever Reported INC
• 5,00,000 Cases Of DHF Require Hospitalization Each Year, Of Which A Very
Large Proportion Are Children. At Least 2.5% Of Cases Die Without Proper
Treatment.
• A Rapid Rise In Urban Populations Is Bringing Greater Numbers Of People
Into Contact With This Vector, Especially In Areas That Are Favourable For
Mosquito Breeding, E.G. Where Household Water Storage Is Common And
Where Solid Waste Disposal Services Are Inadequate.
• Increased Worldwide Distribution Of Disease Seen After World War I
8. ETIOLOGY
• Dengue Is Transmitted By Infected Female Mosquito.
• A.Egypti Is A Primarily A Daytime Feeder. It Lives Around Human Habitation.
• Lays Eggs And Produces Larvae Preferentially In Artificial Containers . Only The
Female Aedes Mosquito Bites As It Needs The Protein In Blood To Develop Its Eggs.
• The Mosquito Becomes Infective Approximately 7 Days After It Has Bitten A
Person Carrying The Virus.
9. • This Is The Extrinsic Incubation Period, During Which Time The Virus
Replicates In The Mosquito And Reaches
• The mosquitoes Remains Infected For The Remainder Of Its Life. The Life Span
Of AAegypti Is Usually 21 Days But Ranges From 15 To 65 Days.
• The Mosquito Can Lay Eggs About 3 Times In Its Lifetime, And About 100
Eggs Are Produced Each Time.
• The Eggs Can Lie Dormant In Dry Conditions For Up To About 9 Months,
After Which They Can Hatch If Exposed To Favourable Conditions, I.E. Water
And Food.
10. • Is Primarily Transmitted By Aedes Mosquitoes, Particularly A. Aegypti.
• These Mosquitoes Usually Live Between The Latitudes Of 35° North And 35°
South Below An Elevation Of 1,000 Metres (3,300 Ft).
• They Bite Primarily During The Day.
• Other Aedes Species That Transmit The Disease Include A. Albopictus, A.
Polynesiensis And A. Scutellaris.
• Humans Are The Primary Host Of The Virus, But It Also Circulates In
Nonhumanprimates.
11. • An infection Can Be Acquired Via A Single Bite.
• A Female Mosquito That Takes A Blood Meal From A Person
Infected With Dengue Fever Becomes Itself Infected With The Virus In
The Cells Lining Its Gut.
• About 8–10 Days Later, The Virus Spreads To Other Tissues
Including The Mosquito's Salivary Glands And Is Subsequently
Released Into Its Saliva.
12. PATHOPHYSIOLOGY
• TRANSMISSION CYCLE OF DENGUE VIRUS BY THE MOSQUITO AEDES AEGYPTI
BEGINS WITH A DENGUE- INFECTED PERSON. THIS PERSON WILL HAVE VIRUS
CIRCULATING IN THE BLOOD—A VIREMIA THAT LASTS FOR ABOUT FIVE DAYS.
• DURING THE VIREMIC PERIOD, AN UNINFECTED FEMALE AEDES AEGYPTI
MOSQUITO BITES THE PERSON AND INGESTS BLOOD THAT CONTAINS DENGUE
VIRUS.
• THEN, WITHIN THE MOSQUITO, THE VIRUS REPLICATES DURING AN EXTRINSIC
INCUBATION PERIOD OF EIGHT TO TWELVE DAYS.
14. TYPES OF VIRUS
• There Are Four TYPES Of The Virus, Which Are Called Serotypes,
And These Are Referred To As DENV- 1, DENV-2, DENV-3 And
DENV-4.
• All Four Serotypes Can Cause The Full Spectrum Of Disease.
• Infection With One Serotype Is Believed To Produce Lifelong
Immunity To That Serotype But Only Short Term Protection Against
The Others.
15. MECHANISM
• When A Mosquito Carrying Dengue Virus Bites A Person,
• The Virus Enters The Skin Together With The Mosquito's Saliva.
• It Binds To And Enters White Blood Cells, And Reproduces Inside The Cells While
They Move Throughout The Body
• In Severe Infection, The Virus Production Inside The Body Is Greatly Increased, And
Many More Organs (Such As The Liver And The Bone Marrow) Can Be Affected,
• And Fluid From The Bloodstream Leaks Through The Wall Of Small Blood Vessels
Into Body Cavities.
• As a Result, Less Blood Circulates In The Blood Vessels, And The Blood Pressure
Becomes So Low That It Cannot Supply Sufficient Blood To Vital Organs.
• Furthermore, Dysfunction Of The Bone Marrow Leads To Reduced Numbers Of
Platelets, Which Are Necessary For Effective Blood Clotting; This Increases The Risk
Of Bleeding, The Other Major Complication Of Dengue Fever
•
16. SIGNS AND SYMPTOMPS
• Asymptomatic Or Mild Symptoms Such As An Uncomplicated Fever( 80%)
• More Severe Illness (5%),
• In A Small Proportion It Is Life-threatening.
• The Incubation Period (Time Between Exposure And Onset Of Symptoms) Ranges From 3–14
Days, But Most Often It Is 4–7 Days.
• Symptoms Of Dengue Are
• Sudden-onset Fever,
• Headache (Typically Located Behind The Eyes),
• Muscle And Joint Pains, And A Rash.
• The Alternative Name For Dengue, "Break-bone Fever", Comes From The Associated Muscle
And Joint Pains
17. •
• Symptoms Include
• A Characteristic Skin Rash That Is Similar To Measles.
• In A Small Proportion Of Cases The Disease Develops Into The Life-
threatening Dengue Hemorrhagic Fever,
• Resulting In Bleeding, Low Levels Of Blood Platelets And Blood Plasma
Leakage, Or Into Dengue Shock Syndrome, Where Dangerously Low Blood
Pressure Occurs.
20. FEBRILE PHASE
• High Fever, Often Over 40 °C (104 °F),biphasic In Nature Breaking And Then
Returning For One Or Two Days Generalized Pain Headache; Usually Lasts
Two To Seven Days.
• Rash Occurs In The First Or Second Day Of Symptoms As Flushed Skin,
• Or Later In The Course Of Illness (Days 4–7), As A Measles-like Rash.
• Some Petechiae
• (Small Red Spots That Do Not Disappear When The Skin Is Pressed, Which Are
Caused By Broken Capillaries) Can Appear At This Point,
• As May Some Mild Bleeding From The Mucous Membranes Of The Mouth And
Nose.
21. CRITICAL PHASE
• Critical Phase, Which Follows The Resolution Of The High Fever And Typically
Lasts One To Two Days.
• During This Phase There May Be Significant Fluid Accumulation In The
Chest And Abdominal Cavity Due To Increased Capillary Permeability And
Leakage. This Leads To Depletion Of Fluid From The Circulation And
Decreased Blood Supply To Vital Organs.[9]
• Organ Dysfunction And Severe Bleeding, Typically From The
Gastrointestinal Tract Shock (Dengue Shock Syndrome) And Hemorrhage
(Dengue Hemorrhagic Fever) Occur In Less Than 5% Of All Cases Of Dengue,
However Those Who Have Previously Been Infected With Other Serotypes Of
Dengue Virus ("Secondary Infection") Are At An Increased Risk.
22. RECOVERY PHASE
• Resorption Of The Leaked Fluid Into The Bloodstream
• This Usually Lasts Two To Three Days.
• Severe Itching And A Slow Heart Rate
• During This Stage, A Fluid Overload State May Occur;
• If It Affects The Brain, It May Cause A Reduced Level Of Consciousness Or
Seizures.
23. DENGUE HEMORRHAGIC FEVER(DHF)
• Dengue Hemorrhagic Fever Was Subdivided Further Into Grades I–IV.
• Grade I Is The Presence Only Of Easy Bruising Or A Positive Tourniquet Test In
Someone With Fever,
• Grade II Is The Presence Of Spontaneous Bleeding Into The Skin And Elsewhere,
• Grade III Is The Clinical Evidence Of Shock,
• And Grade IV Is Shock So Severe That Blood Pressure And Pulse Cannot Be
Detected.
• Grades III And IV Are Referred To As "Dengue Shock Syndrome".
•
24. LABORATORY DIAGNOSIS
• Reported Symptoms And Physical Examination;
• Low White Blood Cell Count,
• Followed By Low Platelets And Metabolic Acidosis.
• In Severe Disease, Plasma Leakage Results In Hemo concentration (As
Indicated By A Rising Hematocrit)
• Hypo albuminemia.
• Pleural Effusions Or Ascites Can Be Detected By Physical Examination When
Large But The Demonstration Of Fluid On Ultrasound May Assist In The
Early Identification Of Dengue Shock Syndrome.
26. ACTIVE SURVEILLANCE
It Is An important component of a dengue prevention programme.
The goal should be to provide an early warning or predictive
capability for epidemic transmission, that emergency mosquito
control programme can be initiated
Hospital used an sentinel sites should includes all those upon the
patients who have severe infectious diseases in the community
27. MOSQUITO CONTROL
Prevention And Control Depends On The Control Of The mosquito
vector in and around home, where most transmission occurs.
The most effective way to control the mosquito that transmits
dengue is larval source reduction
Elimination or cleaning of water holding containers that serves as
the larval habit in domestic environment
28. COMMUNITY PARTICIPATION
Now a days , emphasis is on the community based approaches.
Prevention programmes require extensive health education to
achieve community participation.
29. PREVENTION OF DENGUE IN TRAVELLERS
Staying In Screened or air conditioned room
Spraying the rooms with aerosol bound insecticides to kill adult mosquitoes
indoor.
using a repellent containing DEET( dimethyl meta-toluamide) on exposed skin
wearing protective clothing (long sleeved clothing and pants)
Use mosquito net to prevent you from mosquito bites.
30. NURSING MANAGEMENT
Thorough assessment of the patient, provide adequate bed rest
Continuous watch on vital signs
Hydrate with adequate fluids like milk, fruit juices and ORS
Tepid sponging when applicable
Administered antipyretics as prescribed, should not exceed more
than 4g per day
Eliminate the mosquito breeding place and use mosquito net to
prevent mosquito bite