The document summarizes various abnormalities and diseases that can affect the dental pulp and periapical tissues, including:
1) Pulp calcification, which involves mineralization within the pulp chamber or root canals and can occur as denticles, pulp stones, or diffuse linear calcifications.
2) Resorption of teeth, which can be physiological for deciduous teeth but pathological for permanent teeth.
3) Diseases of periapical tissues including periapical abscesses, granulomas, radicular cysts, phoenix abscesses, and condensing osteitis. These conditions are responses to dental infection and inflammation and can develop from other lesions if left untreated.
3. Pulp Calcification
Cause
no clear-cut etiology
no relation between
inflammation + irritation
• since pulp calcification
can be found in unerupted
teeth
6. (1) Denticles
believed to form as a result
of epitheliomesenchymal
interaction within
developing pulp
form during period of root
development
occur in root canal + pulp
chamber adjacent to furcation
areas of multirooted teeth
7. (2) Pulp Stones
believed to develop around
central nidus of pulp tissue
examples:
collagen fibril
ground substance
formed within coronal portions
of pulp
8. (2) Pulp Stones
may arise as part of age-
related or local pathologic
changes
most develops after tooth
formation is completed
usually free or attached
some instances, may be embedded
9. (3) Diffuse Linear
Calcifications
doesn’t demonstrate lamellar
organization of pulp stones
exhibit areas of:
fine
fibrillar
irregular calcification
may be present in pulp
chamber or canals
frequency increases with age
10. (3) Diffuse Linear
Calcifications
Clinical Significance:
very little clinical significance
except insofar as they may
obstruct endodontic treatment
11. (3) Diffuse Linear
Calcifications
Clinical Significance:
discovered on radiograph
only as radioopacity
may cause pain from
mild pulpal neuralgia to
severe excruciating pain
resembling tic douloureux
• as denticle may impinge
on nerve of pulp
12. (3) Diffuse Linear
Calcifications
Clinical Significance:
difficulty may be encountered
in extirpating pulp
during root canal therapy
13. (3) Diffuse Linear
Calcifications
Treatment & Prognosis
No treatment is required
14. Resorption of the Teeth
deciduous teeth are progressively
loosened
result of progressive
resorption of roots
physiological process arising
from pressure of underlying
successors
resorption of permanent is
always pathological
15. Resorption of the Teeth
Pathology
pressure is probably main
factor
resorption is mainly carried
out by osteoclast
humoral mediators, such
as prostgalndins
• may contribute to resorption
17. Idiopathic Resorption
Internal Resorption
pink spot
curious + uncommon
condition
dentin is resorbed from
within the pulp
18. Idiopathic Resorption
(1) Internal Resorption
tends to be localized
well-defined rounded area
of rediolucency in crown
can affect any part of teeth
NO signs until pulp is
opened + allows access to
infection
25. Idiopathic Resorption
(2) External Resorption
usually a limited area of
root is attacked from
external surface near
amelocemental junction
• resorption goes on until
pulp is reached
26. Idiopathic Resorption
(2) External Resorption
often preferentially
destroys root before
penetrating the pulp
27. Idiopathic Resorption
(2) External Resorption
accessible defects may be
amenable to restoration
with mineral trioxide or
other materials
long term success in infrequent;
unpredictable
28. Idiopathic Resorption
(2) External Resorption
Pathology
• vascular granulation
tissue replaces part
or periodontal ligament
or pulp
• osteoclasts border the
affected dentin or enamel
29. Idiopathic Resorption
(2) External Resorption
Treatment
• usually untreatable
• if a pink spot in an incisor
tooth is noticed at an early
stage
endodontic treatment should
be carried out before
30. Idiopathic Resorption
(2) External Resorption
Treatment
• resorption of teeth may
result from pressure
exerted by impacted teeth
indication for removal
of unerupted teeth
33. (1) Periapical Abscess
also known as Dento-alveolar
Abscess; Alveolar Abscess
acute or chronic supporative
process of dental periapical
region
usually arises as a result of
infection
34. (1) Periapical Abscess
abcess ay develop directly
as an acute apical periodontitis
following an acute pulpitis
but more commonly it
originates in an area of
chronic infection
35. (1) Periapical Abscess
Clinical Feature
presents features of
acute inflammation of
apical peridontium
tooth is extremely painful
slightly extruded from its
socket
36. (1) Periapical Abscess
Clinical Feature
chronic periapical
abscess generally presents
no clinical features
mild, circumscribed area
of suppuration that shows
little tendency to spread from
local area
37. (1) Periapical Abscess
Radiographic Feature
except for SLIGHT thickening
of periodontal membrane
no roentgenographic
evidence of its presence
chronic abscess, developing
in a periapical granuloma
• radioluscent area at apex
39. (1) Periapical Abscess
Histopathologic Features
area of suppuration is
composed chiefly of central
area of disintegrating
polymorphonuclear
leukocytes
dilation of blood vessels
in periodontal ligament
40. (1) Periapical Abscess
Histopathologic Features
tissue surrounding area
of suppuration contains
serous exudate
41. (1) Periapical Abscess
Treatment & Prognosis
drainage must be
established
• open pulp chamber
• extract the tooth
42. (1) Periapical Abscess
Treatment & Prognosis
under some circumstances
tooth may be retained
• root canal therapy
43. (1) Periapical Abscess
Treatment & Prognosis
left untreated, spread
of infection
• osteomyelitis
• cellulitis
• bacterimia
• formation of fistulous
tract opening on skin
or oral mucosa
44. (2) Periapical Granuloma
also known as Apical
Periodontitis
one of the most common
sequeala of pulpitis
localized mass of chronic
granulation tissue
response to infection
45. (2) Periapical Granuloma
Clinical Features
1st evidence; spread beyond
confines of tooth pulp
may be noticeable sensitivity
of involved tooth to
percussion
mild pain when biting or
chewing on solid food
46. (2) Periapical Granuloma
Clinical Features
some cases tooth feels
elongated in its socket
sensitivity is due to
• hyperemia
• edema
• inflammation of apical
periodontal ligament
47. (2) Periapical Granuloma
Radiographic Features
earliest evidence,
thickening of ligament at root
apex
proliferation of granulation
tissue
concomitant resorption of bone
continue
48. (2) Periapical Granuloma
Radiographic Features
appear as a radiolucent
area of variable size
seemingly attached to
root apex
some cases, well
circumscribed lesion
• definitely demarcated
from surrounding bone
49. (2) Periapical Granuloma
Histologic Features
arises as chronic process
from onset
does not pass through an
acute phase
50. (2) Periapical Granuloma
Histologic Features
begins as:
• hyperemia
• edema of periodontal
ligament with infiltration
of chronic inflammatory cells
chiefly lymphocytes
plasma cells
51. (2) Periapical Granuloma
Histologic Features
inflammation + locally
increased vascularity
of tissue
• induce resorption
of supporting bone
adjacent to this area
52. (2) Periapical Granuloma
Histologic Features
as bone is resorbed
• proliferation of fibroblast
+ endothelial cells
• formation of more tiny
vascular channels
• numerous delicate connective
tissue fibrils
53. (2) Periapical Granuloma
Treatment & Prognosis
extraction of involved
teeth
under certain conditions,
root canal therapy with or
without subsequent
apicoectomy
55. (2) Periapical Granuloma
Treatment & Prognosis
left untreated, may
undergo transformation
into an apical periodontal
cyst
• proliferation of epithelial
rests in the area
56. (3) Radicular Cyst
also known as Apical
Periodontal Cyst;
Periapical Cyst;
Root End Cyst
common
not inevitable sequela of
periapical granuloma originating
as a result of:
bacterial infection
necrosis of dental pulp
following carious involvement of tooth
57. (3) Radicular Cyst
Pathogenesis
initial reaction leading
to cyst formation
• proliferation of epithelial
rest in the periapical
area involved by granuloma
• epithelial proliferation
follows an irregular pattern of
growth
58. (3) Radicular Cyst
Clinical Features
asymptomatic
present no clinical evidence
of their presence
seldom painful or even
sensitive to percussion
59. (3) Radicular Cyst
Clinical Features
represents chronic
inflammatory process
• develops only over
a long period of time
60. (3) Radicular Cyst
Radiographic Features
identical with periapaical
granuloma
since the lesion is a chronic
progressive one developing
in a pre-existing granuloma
• cyst may be of greater
size than granuloma
• due to longer duration
61. (3) Radicular Cyst
Radiographic Features
occasionally, exhibits
thin, radioopaque line
around the periphery
of radiolucent area
• indicates reaction of
bone to slowly expanding
mass
71. (4) Phoenix Abscess
can occur immediately
following root canal treatment
another cause is due to untreated
necrotic pulp (chronic apical
periodontitis)
result of inadequate debridement
during endodontic procedure
72. (4) Phoenix Abscess
Bacteriology
Staphylococci are frequently
associated with pus formation
• produce enzyme called
coagulase
• causes fibrin formation
• helps in walling off of lesion
74. (4) Phoenix Abscess
Clinical Features
when palpated clinically
• superficial abscess is
fluctuant
offending tooth is carious
+ mobile
symptoms of acute inflammation
• swelling
• fever
75. (4) Phoenix Abscess
Treatment
repeating endodontic
treatment with improved
debridement
tooth extraction
antibiotics may be indicated
to control a spreading or
systemic infection
76. (5) Condensing Osteitis
also known as Chronic
Focal Sclerosing Osteomyelitis
unusual reaction of bone
occuring in instances of
extremely high tissue resistance
or in cases of low grade infection
77. (5) Condensing Osteitis
Clinical Features
occurs in almost young
person before the age of
20 years old
commonly affected is
mandibular 1st molar
with large carious lesion
80. (5) Condensing Osteitis
Clinical Features
associated with non vital
teeth or teeth undergoing
process of degeneration
tooth is usually asymptomatic
some cases, pain or tenderness
• percussion
• palpation
81. (5) Condensing Osteitis
Radiographic Features
well circumscribed
radiopaque mass of
sclerotic bone surrounding
extending below apex of
one or more roots
82. (5) Condensing Osteitis
Histologic Features
dense mass of bony trabeculae
with little interstitial
marrow tissue
83. (5) Condensing Osteitis
Histologic Features
dense mass of bony trabeculae
with little interstitial
marrow tissue
chronic inflammatory cells;
plasma cells, lymphocytes
are seen scanty in bone
marrow
84. (5) Condensing Osteitis
Treatment & Prognosis
endodontic treatment
extraction
surgical removal of sclerotic
should not be attempted
unless symptomatic
85. References:
Books
Cawson, R.A: Cawson’s Essentials of Oral
Oral Pathology and Oral Medicine,
8th Edition
• (page 70-72)
Ghom, Ali & Mhaske, Shubhangi: Textbook of
Oral Pathology
• (pages 429-433)
Neville, et. al: Oral and Maxillofacial Pathology
3rd Edition
• (pages 127-138)
Shafer, et al: A textbook of Oral Pathology,
3rd Edition