The document discusses a study that found propagermanium, an organic germanium compound, reduces atherosclerosis in mice by inhibiting macrophage infiltration. Propagermanium inhibits the MCP-1/CCR2 pathway, which plays an important role in macrophage recruitment during plaque formation. The study showed propagermanium treated mice had significantly smaller atherosclerotic lesions and fewer macrophages in plaques than untreated mice. By suppressing macrophage infiltration through the MCP-1 receptor, propagermanium may prevent atherosclerosis and be a potential drug for treating vulnerable plaque.