Introduction to ArtificiaI Intelligence in Higher Education
aphasia in brief
1. APHASIA : A CASE
STUDY
BY : POOJA TOMAR & Mrs. KALYANI
SINGH
2. What is Aphasia…?
Aphasia is “ an acquired impairment of language
processes underlying receptive and expressive
modalities and caused by damage to areas of
the brain which are primarily responsible for
the language function “ ( By Davis 1983 ).
Aphasia is the “loss of verbal communication
due to an acquired lesion of the nervous
system involving one or more aspects of the
processes of comprehending and producing
verbal messages” ( Basso and Cubelli, 1999 ).
3. Incidence and Prevalence
• The incidence estimated that there are
80,000 new cases of aphasia per year
(National Stroke Association, 2008).
• Prevalence estimates that approximately
1 million people, or 1 in 250 , suffer from
aphasia.
4. Signs and Symptoms
• Verbal Expression Impairments
• Auditory Comprehension
Impairments
• Reading Comprehension
Impairments (Alexia)
• Written Language Impairments
(Agraphia)
5. APHASIA ETIOLOGIES
• Aphasia is caused by damage to the language centers of the brain. In
most people, these language centers are located in the left
hemisphere, but aphasia can also occur as a result of damage to the
right hemisphere; this is often referred to as crossed aphasia to
denote that the right hemisphere is language dominant in these
individuals. Common causes of aphasia are
CVA ( stroke )
– ischemic: blockage that disrupts blood flow to a region of the brain
– hemorrhagic: a ruptured blood vessel that damages surrounding
brain tissue
traumatic brain injury
brain tumors
brain infections
other neurological diseases (e.g., dementia).
6.
7. CLASSIFICATION• There are several classification system based on main characteristics are :
Functional anatomy
Clinical features
Associated condition
• According to wernicke’s 1874 based on antomical lesion, there are 3 types of
aphasia motor aphasia, sensory aphasia and conduction aphasia.
• According to wernicke’s theme model 1884classification system there are 7
types of aphasia :
1. Motor aphasia : subcortical motor aphasia , cortical motor aphasia and
transcortical motor aphasia
2. Conduction aphasia
3. Sensory aphasia : subcortical sensory aphasia , cortical sensory aphasia and
trans cortical sensory aphasia
• According to Goodglass classification 1974 there are 8 types of aphasia
which are generally used in administering the WAB , these are broca’s
aphasia , wernicke’s aphasia, global aphasia , anomic aphasia, transcortical
motor aphasia, transcortical sensory aphasia, conduction aphasia and
isolation aphasia.
8. CLASSIFICATION OF APHASIA
Fluent
Non-
Fluent
Wernicke’s Aphasia
Conduction Aphasia
Transcortical sensory
Aphasia
Anomic Aphasia
Broca’s Aphasia
Transcortical motor
Aphasia
Isolation Aphasia
Global Aphasia
Given by Goodglass( 1974)
10. Type of Aphasia Lesion localization
Broca’s area Posterior-Inferior frontal (area 44, 45,
sometimes 46 )
Wernicke’s Aphasia Posterior- Superior Temporal ( area 22 )
Conduction Aphasia Arcuate Fasciculus- supramarginal gyrus
Anomic Aphasia Inferior – parietal ( angular gyrus )
Global Aphasia Perisylvian region ( middle cerebral
artery territory )
Transcortical Motor aphasia Anterior & superior to broca’s area (
supplementary motor area )
Transcortical sensory Aphasia Watershed areas of middle & posterior
cerebral artery
Isolation Aphasia Watershed areas of middle, anterior &
posterior cerebral artery
11. ASSESSMENT
• Purpose of assessment : Generally , methods of
assessment vary greatly, depending on the examiner’s
goal. There are six general types of evaluation purpose
may be distinguished in aphasic assessment :
a) Screening procedures
b) Diagnostic assessment
c) Descriptive testing in rehabilitation and counselling
d) Progressive evaluation
e) Assessment of functional and pragmatic
communication; and
f) Assessment of related disorders
12. • Assessment of aphasia is a comprehensive
examination of speech & language
characteristics along with other behavioral
and medical condition.
• Assessment can involve many phases such as
reviewing medical records, a patient and
family interview and a bedside screening ,
formal testing procedures.
• Formal testing involves a comprehensive
language test battery to assess patient
linguistic and communication impairments.
13. Language Test Batteries
Basically assess two primary input
modalities & three output modalities,
language test batteries generally assess
four modalities, these are :
Speech
Auditory comprehension
Reading
Writing
14. Contemporary tests and
methods
Clinical
bedside
examinati
-on
Screening
tests:
bedside
evaluation
screening test
•Aphasia
screening test
•Frenchay
aphasia
screening test
Test of
specific
aspects of
language
behaviour :
•Boston
naming test
•Token test
•Discourse
comprehension
test
Function-
-al
communi
cation :
•Communicati
ve
effectiveness
index
•Communicati
on activity of
daily living
Diagnost
-ic
examina
tion :
• PICA
• WAB
• BDAE
• MTDDA
• MAE
• BASA
15. Treatment
Sometimes aphasia will improve on its own without treatment, but speech
and language therapy is usually recommended.
For people with aphasia, speech and language therapy aims to:
• help you communicate to the best of your ability
• help restore as much of your speech and language as possible
• find alternative ways of communicating
Evidence suggests speech and language therapy is more effective if it's
started as soon as possible.
Other treatments:
• Medication – such as piracetam, bifemelane, piribedial, bromocriptine
and idebenone
• Transcranial magnetic stimulation – where an electromagnet placed on
the scalp is stimulated for a short time using an electric current to
stimulate parts of the brain affected by aphasia
18. PATIENT BACKGROUND HISTORY
Client was a employee in railway , he is 68 years
old , Client had history of stroke( CVA ) twice in
his life, the 1st episode took place in 2010 & the
2nd was in 2015 & client also had a history of
paralysis attack after 3 months of 2nd stroke and
after that paralysis attack his family members
experienced that there are some changes in his
voice and had mild phonatory gap in vocal cords ,
client’s medical treatment is going on at central
hospital new Delhi in cardiology and neurological
department .
19. Assessment for Aphasia & allied disorders :
• Case Name: XYZ
• Age / sex : 68 yrs/ male
• Case No. : 1069/12/16
• Present complaint as stated by family members :
a) Speech & language : can not speak properly
b) Other behavior : not present
• Language prefered ( speaking/ writing ) : client
prefers speaking
20. • Medical History :
Etiology : stroke ( CVA )
Site of lesion : temporo-parietal region in the left hemisphere
Type of lesion :left MCA infarct
Any previous strokes : first episode in the year 2010
Physical illness / deficit : no
Visual defects : yes ( poor vision )
Hearing defects : no
Sensory motor function ( sensation, strength, range ) : sensation
is present, strength is decreased, & range is restricted.
Surgery : client has gone through two cardiac surgery.
Reports : from kalawati reports client has good comprehension
& poor verbal expression & from LHMC reports client is having
mild phonatory gap leading to hoarseness.
21. • Present status :
General physical condition : client appears to be normal
Personality & behavior :socially adequate personality & behavior
Hobbies & interests : client show interest in listening to songs &
reading newspaper
Management of activities of daily living : client complete his
tasks by himself including cooking , washing cloths.
• Ongoing treatment :
Medical : In central hospital new delhi ( in cardiology &
neurology department )
Physiotherapy : no
Occupational therapy : no
Psychological counseling : no
Other rehabilitation : yes client takes speech therapy.
22. • Oral peripheral examination :
Lips : lateral movements is affected ,
protrusion is normal , alternate movement is
affected deviated towards right side.
Tongue : Normal in appearance & function.
Soft palate : Normal in appearance & function.
Vegetative skills : all vegetative skills are
normal except blowing , client can resist air in
the mouth but he has difficulty in blowing.
23. • Communication behavior including speech &
language :
Mode of communication : mode is both verbal &
non verbal. In verbal, client has a limited
vocabulary and paraphasic speech & non verbally,
he uses gestures and pointing.
Linguistic behavior ( verbal, written in terms of
content, form & use of language comprehension
and production ) : written expression is poor,
reading text and reading commands are poor and
verbal output is limited.
24. • Other areas :
Attention / perception : Adequate
Motivation : Adequate
Thinking : above average
Memory : Intact
Material facilitating response : Adequate but non verbal
only.
Modality ( visual, auditory or tactile ) : vision is poor &
other modalities are present.
Cueing ( verbal, gestural, visual-printed/ written ) : yes
through verbal or gestural sometimes.
Arithmetic : fair arithmetic skills
• Language test administered & results : ( Token test/ WAB/
BDAE / PICA / Border Reading Test ) :
WAB was administered and aphasia quotient & cortical
quotient were calculated for :-
27. Scoring of Token test
• Part A ( 7 points possible ) = 7
• Part B ( 8 points possible ) = 8
• Part C ( 12 points possible ) = 12
• Part D ( 16 points possible ) = 12
• Part E ( 24 points possible ) = 18
• Part F ( 96 points possible ) = 49
TOTAL SCORES : 163
Scores obtained : 106
28. • Diagnostic formulation : case came to NISHD
with complaint of aphasia, he expresses
verbally through common words and phrases (
paraphasic error is present ) and non verbally
through pointing and gestures, client’s
vocabulary consists of common objects, client
has fair auditory comprehension.
• Provisional diagnosis : Broca’s aphasia
29. • Recommendation :
Speech and language therapy at nearest
center / NISHD
Speech and language stimulation at home
Aphasia therapy
Follow up
30. VOICE ASSESSMENT
• Brief history of the problem : client had dinner and he was normal for 10
minutes, after 10 minutes he had stroke first episode took place in 2010 &
again the 2nd episode took place in 2015 . The voice problem is started
after 2nd stroke that was on 15th august 2015.
• Medical history : CVA in tempo-parietal region in left hemisphere & had
left MCA infarction.
Client get irritation/ pain in the throat- while speaking after 2nd stroke
Onset of problem was sudden
The problem of voice is static
Client indulge in excessive & loud speaking & had a habbit of clearing of
throat 6-7 times in a day
Other development ( physical , secondary sexual development ) are
normal
Client has history of phonatory gap.
31. • Speech mechanism ( structure & function ) :
structurally all articulators are normal expect lips,
alternate & lateral movement are affected & jerks
present and all vegetative skills are present but
blowing is affected.
• Perceptual assessment :
Pitch : pitch range is limited, habitual pitch is
high , & pitch breaks are also present
Loudness : very loud
Quality : hoarsness
Phonation duration : for [ a ] is 7.4 sec for [ i ] is 8
sec & for [ u ] is 5 sec.
32. • Diagnostic formulation : client came to NISHD
with the complaint of unclear voice as a post sign
of aphasia, client has normal physical &
secondary sexual development , oral peripheral
examination reveals that there is restriction in lip
movement & blowing is affected, on perceptually
assessment of voice, client has limited pitch
range , loudness is very loud & quality of voice is
hoarse voice
• Provisional diagnosis : phonatory gap with
hoarseness
• Recommendation :
vocal hygiene
Voice therapy at NISHD/ nearnest center
Follow up