2. INTRODUCTION
The most important functions of the
temporomandibular joint (TMJ) are mastication and
speech and are of great interest to dentists,
orthodontists, clinicians, and radiologists.
The TMJ is a ginglymoarthrodial joint, a term that is
derived from ginglymus, meaning a hinge joint,
allowing motion only backward and forward in one
plane, and arthrodia, meaning a joint of which
permits a gliding motion of the surfaces.
3. Temporomandibular joint
Only mobile joint of
skull formed
between head of
mandible and
articular fossa of
temporal bone.
4. Peculiarity of TMJ
1. Bilateral diarthrosis – right & left function together
2. Articular surface covered by fibrocartilage-instead
of hyaline cartilage
3. Only joint in human body to have a rigid endpoint of
closure (that of the teeth making occlusal contact).
5. 4. In contrast to other diarthrodial joints TMJ is
last joint to start develop in- about 7th
week in utero.
5. Develops from two distinct blastema.
i) Temporal.
ii) Condylar.
7. Fibrous capsule
Above to the anterior
edge of the preglenoid
plane.
Posteriorly to the
squamotympanic fissure,
between these to the
edges of the articular
fossa.
Below to the periphery of
the neck of the mandible.
8. Articular disc
Fibrocartilaginous disc dividing joint cavity in upper and
lower compartment.
Shape: Oval
Makes articular surface congruent.
In sagittal section- a thin intermediate zone and thickened
anterior and posterior bands, and its upper surface appears
concavo-convex.
Posteriorly- Attached to a region of loose vascular and
nervous tissue which splits into two laminae, the bilaminar
region.
9.
10. Functions of Articular disc
Stabilize the TMJ.
Makes articular surfaces congruent.
Reduce wear of TMJ.
Aid lubrication of the joint.
11. Lateral ligament of Jaw
Attached above to the articular tubercle on the root
of the zygomatic process of the temporal bone.
It extends downwards and backwards at an angle
of 45° to the horizontal, to attach to the lateral
surface and posterior border of the neck of the
condyle, deep to the parotid gland.
Function: To prevent posterior displacement of the
resting condyle.
12. Sphenomandibular ligament
Medial to, and normally separate from, the
capsule. It is a flat, thin band that descends from
the spine of the sphenoid.
Widens at the lingula of the mandibular foramen.
This part is a vestige of the dorsal end of
Meckel's cartilage.
It is separated from the pharynx by fat and a
pharyngeal vein.
13.
14. Stylomandibular ligament
A thickened band of deep cervical fascia that stretches
from the apex and adjacent anterior aspect of the
styloid process to the angle and posterior border of the
mandible.
Along with sphenomandibular ligament it is
responsible for limitation of mandibular movement.
16. VASCULARIZATION
Predominant vessals supplying tmj are:
Superficial temporal artery from the posterior
Middle meningeal artery from the anterior
Internal maxillary artery from the inferior
18. 1. The Articular zone
Dense fibrous
connective tissue
Poor blood supply
Better ability to
repair
• Good adaption to sliding movement
• Shock absorber
• Less susceptible to the effect of
aging time & breakdown over time.
19. 2. The proliferative
zone
Mainly cellular zone
Undifferentiated
mesenchymal cells
Proliferation &
regeneration
throughout life
20. 3. The cartilagenous
zone
Collagen fibers
arranged in criss -
cross pattern of
bundles
Fibrocartilage
appears in a random
orientation, providing
a three-dimensional
network that offers
resistance against
compressive and
lateral forces.
• Offers considerable resistance against
compressive & lateral forces
• But becomes thinner with age.
21. 4. The calcified zone
Deepest zone
Chondrocytes,
chondroblasts &
osteoblasts
• Active site for remodeling activity as
bone growth proceeds.
22. Movements of TMJ
Rotational / hinge movement in first 20-25mm of
mouth opening
Translational movement after that when the
mouth is excessively opened.
25. Age changes of the TMJ:
Condyle:
Becomes more flattened
Fibrous capsule becomes thicker.
Osteoporosis of underlying bone.
Thinning or absence of cartilaginous zone.
Disk:
Becomes thinner.
Shows hyalinization and chondroid changes.
Synovial fold:
Become fibrotic with thick basement membrane.
Blood vessels and nerves:
Walls of blood vessels thickened.
Nerves decrease in number
26. Age changes lead to:
Decrease in the synovial fluid formation
Impairment of motion due to decrease in the
disc and capsule extensibility
Decrease the resilience during mastication
due to chondroid changes into collagenous
elements
Dysfunction in older people
27. Symmetrical opening
Associated with preparation for incising.
At the start, each mandibular condyle rotates in the lower
joint compartment inside the annulus of its disc.
After a few degrees of opening, the condyle continues
rotating inside its disc, and, in addition, both slide forward
down the articular eminence of the upper joint
compartment.
Without this forward slide, it rapidly becomes impossible to
continue opening the jaw.
28. Eccentric jaw opening
Preparation for power stroke of mastication.
Condyle on the non-working side slide back
and forth during lateral movements.
Temporomandibular and Sphenomandibular
ligament keep condyle firmly against articular
eminence.
29. Eccentric and symmetrical jaw
closing
Jaw closing muscles have a component of
force that forces joint surfaces together.
Joint tissues compressed- ligaments
shortened- no constraint on movements.
Non-working condyle moves further and is
most heavily loaded during power stroke.
31. Various terms have been used to describe
disturbances of the masticatory system.
1934 James costen described group of symptoms
centering around ear and TMJ- Costen syndrome.
1959 Shore introduced TMJ Dysfunction
Ash & Ramfjord- functional TMJ disturbances
Terminology
32. Limited nature of these terms lead to a broader
term- Craniomandibular disorders.
Bell coined the term Temporomandibular
disorders.
Describes both problems associated with the joint
& disturbances associated with function of
masticatory system.
33. Event
Events can be either local or systemic.
Local: a change in proprioceptive input e.g
improper crown.
Can be secondary to trauma- too wide opening of
mouth; unaccustomed use- bruxism.
34. Also deep pain input- alters muscle function by
way of central excitatory effects.
Systemic events; that alter normal function occurs
at a systemic level; emotional stress.
35. Physiologic tolerance
Influenced by local and systemic factors.
Local: orthopedic stability-relation between
mandible and maxilla- good stability; mandible
closes with the condyles in their most superior
and anterior position against posterior slopes of
articular eminence- even contact of all possible
teeth.
Here masticatory system is best able to tolerate
local and systemic events.
36. Poor stability: one way by which occlusal
condition influences symptoms associated with
TMD.
Instability can be in occlusion/joints/both.
Can be genetic/developmental/iatrogenic. Also
related to alterations in normal anatomic form.
38. Etiologic considerations for TMD.
5 major factors associated with TMD
1. Occlusal condition:
2. Trauma
3. Emotional stress
4. Deep pain input
5. Para-functional activities.
39. Occlusal condition
Excessive load on the system due to orthopedic
instability may lead to intracapsular disorders.
2 factors that determine it are: degree of
instability and amount of loading.
Changes can be acute/ sudden or chronic.
40. Trauma
Macro and micro
Macro: sudden face that can result in structural
alterations. Eg blow to face.
Micro : small force applied repeatedly to
structures over a long periods. Bruxism/
clenching.
41. Deep pain input.
Centrally excites a brainstem – produces muscle
response- protective co-contraction.
Functional disorders of masticatory system
2 symptoms: Pain and dysfunction.
42. In case of presence of pain. Evaluated based on
chief complaint
Questions asked
1. Chief complaint.
A. Location of pain.
B. Onset of pain.
• Associated factors.
• Progression.
43. C. Characterstics of pain
1. Quality
2. Behavior
Temporal
Duration
Localization.
3. Intensity of pain
4. Concomitant symptoms.
5. Flow of pain.
44. D. Aggrevating and alleviating factors
1. Function and parafunction
2. Physical modalities
3. Medications
4. Emotional stress
5. Sleep disturbances
E. Past consulations or treatment
II Medical history
III. Review of systems
45. Interpretation;
0- no pain or tenderness
1- uncomfortable on palpation
2- definite discomfort
3- eye tearing/ extreme discomfort
46. Tmj palpation : digital
palpation of joint with
mandible in both static
and dynamic positions.
Finger tips are placed
over the lateral aspects of
joint areas- lateral poles of
condyles passing
downward and forward
felt.
47. Click is a single sound of short duration; if
relatively loud referred to as POP.
Crepitation – multiple gravel like sound – grating
and complicated
Can be done using digits / stethoscope.
48. TMJ DISLOCATION
The mandible can dislocate in the anterior,
posterior, lateral, or superior position.
Anterior dislocations are the most common These
dislocations are classified as acute, chronic
recurrent, or chronic
TMJ dislocation may occur with trauma, extreme
opening of the mouth during yawning, laughing,
singing, vomiting, or dental treatment .
Symmetric mandibular dislocation is most common,
but unilateral dislocation with the jaw deviating to
the opposite side also can occur.
TMJ dislocation is painful and frightening for the
patient.
50. TMJ ANKYLOSIS
Ankylosis of the TMJ
most often results from
trauma or infection.
True bilateral congenital
ankylosis of the TMJ
leads to micrognathia or
“bird face”.
If ankylosis affects only
one side, it produces a
lateral deviation of the
jaw to the non-affected
side, due to the fact that
this side continues its
growth normally.
51. LAB INVESTIGATIONS
1.Blood tests: ESR, CRP for inflammation.
2.Plain radiographs - show gross bony pathology
such as degeneration or trauma.
3.CT or MRI scan of the joint. MRI scan shows the
soft tissues and intra-articular disc well.
4.Ultrasound - this is a useful alternative imaging
technique for monitoring TMJ disorders.
5.Diagnostic nerve block.
6.Arthroscopy.
55. Supportive therapy
Directed toward the reduction of pain and
dysfunction.
Pharmacologic or Physical therapy.
Pharmacologic:
1. Analgesics
2. NSAIDs
3. Corticosteroids
4. Muscle relaxants
5. Antidepressants.
56. Physical therapy. Group of supportive actions, usually
instituted as an adjunct to definitive treatment. 2
types- Modalities & Manual techniques
Modalities: Thermotherapy, ultrasound,
phonophoresis, iontophoresis, laser
Manual techniques: provided by physical therapist; 3
types- soft tissue & joint mobilization, muscle
conditioning.
57. Temporomandibular joint surgery: what
does it mean to the dental practitioner
In March 2011, G Dimitroulis in vincents hospital
melbourne assesed why dental practioners
should be aware of benefits and risks of TMJ
surgeries.
They concluded that all dental practitioners
should be aware of the benefits of TMJ surgery
so that patients do not suffer unnecessarily from
ongoing non-surgical treatments that ultimately
prove to be ineffective in the management of their
condition.
58. Temporomandibular joint problems and
periodontal condition in rheumatoid arthritis
patients
In December 2011, Garib BT1 and Qaradaxi SS in
College of Dentistry, University of Sulaimani,
Kurdistan assesedTemporomandibular joint problems
and periodontal condition in rheumatoid arthritis
patients in relation to their rheumatologic status.
They took plaque index, bleeding index, clinical
attachment loss, radiographic bone loss, tooth loss,
and TMJ problems were assessed in the 2 groups.
They concluded that Patients with advanced RA are
more likely to develop more significant periodontal
and TMJ problems compared with patients with PD
and without RA. There is a great need to instruct
patients with RA to consult a dentist to at least
decrease PD severity.
59. CONCLUSION
It is impossible to comprehend the fine points of occlusion without
an in depth awareness of anatomy ,physiology ,and biomechanics
of the TMJ.
The first requirement for successful occlusal treatment is stable,
comfortable TMJ.
The jaw joints must be able to accept maximum loading by the
elevator muscles with no signs of discomfort.
It is only through an understanding of how the normal, healthy TMJ
functions that we can make sense out of what is wrong when it isn't
functioning comfortably.
This understanding of TMJ is foundational to diagnosis and
treatment.
T- 1st to develop C-rapid growth in dorsolateral dirctn. Blastema is mass of cells for grwtn n regnrtn of body parts.
Squamotympanic-seperates tympanic part or temporal from squamous part.
Congruent is same shape n size.
Meckels cartilage-cartilaginous bar of mand. arch
Limits excessive opening.
STA- arises from ext carotid art. Supllies blood to scalp. Masseteric from mand.nerve innervates masseter and tmj. Auriculo from mand nerve innervates side of head.
Collagen fibers are less susceptible than hyaline cartilage to the effect of ageing and therefore less likely to breakdown over time.
Annulus-ring like anatomical parts
Jaw close-masseter temporalis medial ptery.
Costen synd- loss of hearing tinnitis dizziness headache etc. Mandibular joint neuralgia.
Problems in chewing muscles and associated struc.
Proprio- realted to stimuli.
Central excitatory- exciatatory and depressant effect of pilocarpine due to direct central acion of drug.changes in pH,plasma co2 etc.
Crepitation- crackling sound.
Esr is erythrocyte sedimen rate reveals inflammatory act. Crp is c reactive protein for inflammation.
Meniscectomy- removal of torn meniscus(thin fibre cartilage betwn joints) arthrocentisis-joint aspiration. For arthritis gout etc