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EKG Interpretation
Amir Aziz Alkatiri
Objectives
■ The Basics
■ Interpretation
■ Clinical Pearls
■ Practice Recognition
The Normal Conduction System
Lead Placement
aVF
All Limb Leads
Precordial Leads
EKG Distributions
■ Anteroseptal: V1, V2, V3, V4
■ Anterior: V1–V4
■ Anterolateral: V4–V6, I, aVL
■ Lateral: I and aVL
■ Inferior: II, III, and aVF
■ Inferolateral: II, III, aVF,
and V5 and V6
Waveforms
Interpretation
■ Develop a systematic approach to
reading EKGs and use it every time
■ The system we will practice is:
■ Rate
■ Rhythm (including intervals and blocks)
■ Axis
■ Ischemia
■ Hypertrophy
■ Electrolyte imbalance
■ Other abnormalities
ECG Interpretation
What is your approach to reading an ECG?
•Rate
•Rhythm
•Axis
•Hypertrophy
•Intervals
•P wave
•QRS complex
•ST segment – T wave
Rate
■ Rule of 300- Divide 300 by the number
of boxes between each QRS = rate
Number of
big boxes
Rate
1 300
2 150
3 100
4 75
5 60
6 50
Rate
■ HR of 60-100 per minute is normal
■ HR > 100 = tachycardia
■ HR < 60 = bradycardia
What is the heart rate?
(300 / 6) = 50 bpm
www.uptodate.com
Rhythm
■ Sinus
■ Originating from
SA node
■ P wave before
every QRS
■ P wave in same
direction as QRS
What is this rhythm?
Normal sinus rhythm
Intervals
What is the normal PR interval?
•0.12 to 0.20 s (3 - 5 small squares). Short PR – Look for Wolff-
Parkinson-White. Long PR – 1st Degree AV block
What is the normal QRS?
•< 0.12 s duration (3 small squares). Long QRS - look for bundle
branch block, ventricular pre-excitation, ventricular pacing or
ventricular tachycardia
What is the normal QTc (QT/square root of RR)?
•< 0.42 s. Long QTc can lead to torsades to pointes.
Normal Intervals
■ PR
■ 0.20 sec (less than one
large box)
■ QRS
■ 0.08 – 0.10 sec (1-2
small boxes)
■ QT
■ 450 ms in men, 460 ms
in women
■ Based on sex / heart rate
■ Half the R-R interval with
normal HR
P wave
■ Always positive in lead I and II
■ Always negative in lead aVR
■ < 3 small squares in duration
■ < 2.5 small squares in amplitude
■ Commonly biphasic in lead V1
■ Best seen in leads II
P Waves
Evaluate the shape, height and width of P waves.
•Multiple morphologies Wandering pacemaker
or Multifocal atrial tachycardia
•Notched (M-shaped) P-wave in I and II, > 0.12 s
P-mitrale seen in severe left atrial enlargement
Right Atrial Enlargement
■ Tall (> 2.5 mm), pointed P waves (P Pulmonale)
Left Atrial Enlargement
■ Notched/bifid (‘M’ shaped) P wave (P mitrale in
limb leads)
P Pulmonale
P Mitrale
Blocks
■ AV blocks
■ First degree block
■ PR interval fixed and > 0.2 sec
■ Second degree block, Mobitz type 1
■ PR gradually lengthened, then drop QRS
■ Second degree block, Mobitz type 2
■ PR fixed, but drop QRS randomly
■ Type 3 block
■ PR and QRS dissociated
What is this rhythm?
First degree AV block
PR is fixed and longer than 0.2 sec
What is this rhythm?
Type 1 second degree block (Wenckebach)
What is this rhythm?
Type 2 second degree AV block
Dropped QRS
What is this rhythm?
3rd degree heart block (complete)
The QRS Axis
▪ Represents the overall direction of the heart’s activity
▪ Axis of –30 to +90 degrees is normal
The Quadrant Approach
■ QRS up in I and up in aVF = Normal
What is the axis?
Normal- QRS up in I and aVF
Ischemia
■ Usually indicated by ST changes
■ Elevation = Acute infarction
■ Depression = Ischemia
■ Can manifest as T wave changes
■ Remote ischemia shown by q waves
What is the diagnosis?
Acute inferior MI with ST elevation
in leads II, III, aVF
What do you see in this EKG?
ST depression II, III, aVF, V3-V6 = ischemia
Lateral MI
Reciprocal changes
Inferolateral MI
ST elevation II, III, aVF
ST depression in aVL, V1-V3 are reciprocal changes
Anterolateral / Inferior Ischemia
LVH, AV junctional rhythm, bradycardia
Hypertrophy
▪ Add the larger S wave of V1 or V2 in
mm, to the larger R wave of V5 or V6.
▪ Sum is > 35mm = LVH
Hyperkalemia
Tall, narrow and symmetric T waves
Hypokalemia
U waves
Can also see PVCs, ST depression, small T waves
Wolff-Parkinson-White Syndrome
Short PR interval <0.12 sec
Prolonged QRS >0.10 sec
Delta wave
Can simulate ventricular hypertrophy, BBB and previous MI
Brugada Syndrome
RBBB or incomplete RBBB in V1-V3 with convex ST elevation
Brugada Syndrome
■ Autosomal dominant genetic mutation
of sodium channels
■ Causes syncope, v-fib, self terminating
VT, and sudden cardiac death
■ Can be intermittent on EKG
■ Most common in middle-aged males
■ Can be induced in EP lab
■ Need ICD
Arrythmias
■ Tachycardia
■ Bradycardia
Atrial Flutter with Variable Block
Sawtooth waves
Typically at HR of 150
Torsades de Pointes
Notice twisting pattern
Treatment: Magnesium 2 grams IV
Supraventricular Tachycardia
Narrow complex, regular; retrograde P waves, rate <220
Retrograde P waves
Ventricular Tachycardia
Right Bundle Branch Block
V1: RSR prime pattern with inverted T wave
V6: Wide deep slurred S wave
Left Bundle Branch Block
Monophasic R wave in I and V6, QRS > 0.12 sec
Loss of R wave in precordial leads
QRS T wave discordance I, V1, V6
Consider cardiac ischemia if a new finding
Thank You
Any Questions?

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EKG_Interpretation FK UNRI(2).pptx