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Secondary hyperaldosteronism
1.
SECONDARY HYPERALDOSTERONISM ALIYA RAHMAN
2.
DEFINITION Common clinical
condition occurs secondary to some cause which stimulate RAAS. It is either physiological or pathological
3.
ETIOLOGY Physiologic causes
– Salt depletion- inadequate salt intake, excessive salt through kidney or GI Pregnancy- progesterone stimulate aldosterone production pathologic causes- Hypoperfusion of kidney – nephrotic syndrome, liver cirrhosis, CHF, diuretic therapy
4.
Renal ischemia-
malignant hypertension,RAS High renin production by juxta glomerular apparatus
5.
DIAGNOSIS Plasma sodium
level- normal or near normal Plasma potassium level – low Plasma renin and aldosterone- elevated
6.
MANAGEMENT TREAT UNDERLYING
CAUSE Salt depletion- normal saline infusion Nephrotic syndrome, cirrohsis of liver CHF, -SPIRONOLACTONE+ LOOP DIURETIC Renal ischemia- ACE INHIBITORS RAS- TRANSLUMINAL ANGIOPLASTY Diuretic therapy- use potassium sparing diuretics.
7.
THANK YOU
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