Why carbone monoxide is poisonous
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Carbon monoxide is a colorless, odorless, and tasteless gas that is highly toxic to humans. It is produced by the combustion of fuels and can be inhaled in various environments like workplaces, vehicles, homes, and from fires or tobacco smoke. When inhaled, carbon monoxide binds to hemoglobin in the blood over 200 times more than oxygen, reducing the blood's ability to carry oxygen. This leads to oxygen deficiency in the tissues, which can cause suffocation or even death depending on the concentration of carbon monoxide in the blood.
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Why carbone monoxide is poisonous
- 1. WHY CARBONE MONOXIDE IS POISONOUS
- 2. PROPERTIES Carbon monoxide (CO) is a tasteless, nonirritating, odorless, colorless gaseous substance and has 200 times more affinity for haemoglobin than oxygen.
- 3. Sources The main source of CO production is the combustion of fuels. Exposure at the workplace occurs in blast-furnace operations in the steel industry and when gasoline- or propane-powered forklifts, chain-saws, or other machines are used in confined spaces, such as companies, tunnels and mines. Environmental exposure to CO can occur while traveling in motor vehicles; visiting urban locations with heavily traveled roads; or cooking and heating with domestic gas, kerosene, coal, or wood; as well as in fires and by environmental tobacco smoke.
- 4. Health Effect and Mechanism Carbone monoxide when inhaled combines with haemoglobin to form carboxyhaemoglobin. Chemical reaction: Hb + CO → COHb As result, oxygen-carrying capacity of haemoglobin decreases and the blood becomes oxygen-deficient This leads to suffocation and even death depending upon the concentration of CO in the blood.
- 5. References: 1. Benignus, V.A., M.J. Hazucha, M.V. Smith, and P.A. Bromberg. 1994. Prediction of carboxyhemoglobin formation due to transient exposure to carbon monoxide. J. Appl. Physiol. 76(4):1739-1745. 2. Bruce, M.C., and E.N. Bruce. 2006. Analysis of factors that influence rates of carbon monoxide uptake, distribution, and washout from blood and extravascular tissues using a multicompartment model. J. Appl. Physiol. 100(4):1171-1180. 3. Hampson NB, Bodwin D. Toxic CO-ingestions in intentional carbon monoxide poisoning. J Emerg Med 2013; 44:625–630. 4. Goldbaum LR, Orellano T, Dergal E. Mechanism of the toxic action of carbon monoxide. Ann Clin Lab Sci 1976;6:372–376. 5. Turner M, Hamilton-Farrell MR, Clark RJ. Carbon monoxide poisoning: an update. J Accid Emerg Med 1999; 16:92–96.
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