Unifying themes of major receptor superfamilies: Provide and explain 2 examples of each of the following processes, making reference to ion channels, G-coupled protein receptors, tyrosine kinase receptors and/or nuclear receptors. You can provide examples from different receptors types for different processes How binding of an agonist to its receptor initiates a pharmacodynamic response (biochemical/molecular mechanism). How receptor activation activates intracellular signaling pathways and/or effector molecules (cellular mechanism). How different types of receptors within the same family can, despite a high degree of homology and shared functionality, generate distinct pharmacodynamic effects (i.e. modulate different signaling pathways in different ways or in different cells/tissues). How receptors and/or receptor-mediated signaling are down-regulated following continued activation by an endogenous or xenobiotic agonist (desensitization).
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cell signaling is part of any communication process that governs basic activities of cells and coordinates multiple-cell actions. The ability of cells to perceive and correctly respond to their microenvironment is the basis of development, tissue repair, and immunity, as well as normal tissue homeostasis
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Units started into production during the current manth 12000 Units com.docxCharlesWXCMacDonalda
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Unifying themes of major receptor superfamilies- Provide and explain 2.docx
1. Unifying themes of major receptor superfamilies: Provide and explain 2 examples of each of the
following processes, making reference to ion channels, G-coupled protein receptors, tyrosine
kinase receptors and/or nuclear receptors. You can provide examples from different receptors
types for different processes How binding of an agonist to its receptor initiates a
pharmacodynamic response (biochemical/molecular mechanism). How receptor activation
activates intracellular signaling pathways and/or effector molecules (cellular mechanism). How
different types of receptors within the same family can, despite a high degree of homology and
shared functionality, generate distinct pharmacodynamic effects (i.e. modulate different signaling
pathways in different ways or in different cells/tissues). How receptors and/or receptor-mediated
signaling are down-regulated following continued activation by an endogenous or xenobiotic
agonist (desensitization).
