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Presented by Dr. Anil jangir
Guided by Dr. R.N Gehlot
Common sellar and parasellar tumors
- Pituitary adenoma
- Craniopharyngioma
-Meningioma
-Rathkes cleft cyst
-Chiasmatic glioma
-Dermoid
-Epidermoid
-Germinoma
-Schwannoma
-Metastasis
- Giant aneurysm
Epidermoid cyst
- Well circumscribed
- Non enhancing
- Homogenously hypodense lesion
- CSF signals on T1/T2/FLAIR
- Can cause bone thinning
- Bright on DWI (Most helpful MR sequence for
Making diagnosis )
• (d/d-Arachnoid cyst appears dark like CSF on DWI)
- Central eccentric
Chiasmatic CP angle
Quadrigeminal plate Middle cranial fossa
sylvian fissure
Pituitary adenoma
-Micro adenoma usually enhance later or lesser then normal
pituitary
Suprasellar extension, Inferiorly into sphenoid sinus/clivus
-Isointense to brain parenchyma on T1
*Macro adenoma may contain hemorrhagic or cystic component.
Acute hemorrhage may lead to rapid expansion of the gland and
Cause acute compression of optic chiasma ( pituitary apoplexy)
Hemorrhage appears hyperintense on T1
Pituitary apoplexy
Pituitary apoplexy due to hemorrhage in macroadenoma
Pituitary apoplexy
-Caused by hemorrhagic/ischemic necrosis of pituitary
gland
-pre-existing macroadenoma usually seen
- Sheehan syndrome- acute postpartum ischemic
necrosis
- H/o Medical management (bromocriptine/cabergoline)
of prolactinoma
- Signal intensity on MR depends on clot age
- On T1- heterogeneously iso to hypo
- T2- iso to mildly hyperintense on T2 initially then
hypointense
- Blooming on T2 may seen
- T1 C+ rim enhancement
Rathke’s cleft cyst(RCC)
-Mean age of presentation is 45 years, can be sellar or
suprasellar
Most symptomatic RCC are 5-15mm in diameter, can cause
pituitary dysfunction, visual disturbance and headache.
-On NECT- hypo/iso/hyperdense
-T1- hypo/ hyperintense, T2- most of hyperintense , FLAIR-
hyperintense
-An enhancing rim sign of compressed pituitary (claw sign)can
be sign
-Non enhancing intracystic nodule (dot sign)- pathognomic of
RCC
-Can present with cyst apoplexy caused by sudden intracystic
hemorrhage
Claw sign Dot sign
Fluid dluid level
Fig. A
Child present with
Predominantly cystic
suprasellar mass with
peripheral calcification
on NECT
Fig. B
Fig.C
fig. D
12 year old
boy present
with
suprasellar
mass
Solid ovarian mass differential
diagnosis
-Epithelial tumor- Brenners tumor
-Germ cell tumor- Teratoma, dysgerminoma
-Sex cord tumor- Fibroma, thecoma, Granulosa cell tumor
-Metastatic- krukenberg tumor
-- Signet ring Metastasis to ovary – usually large, b/l solid adnexal
mass showing heterogenous contrast enhancement
Primary gastric cancer metastasize to Liver(m/c), lungs, adrenal and
ovaries.
Can come from stomach (signet ring cells-m/c), Colorectal (2nd m/c),
breast, lung, contralateral ovary, pancreatic, cholangiocarcinoma
On MRI- b/l complex ovarian mass with hypointense solid component
and T1/T2 hyperintense mucin component
Biopsy proven case of signet cell
adenocarcinoma of stomach with b/l ovarian
metastasis(krukenberg tumor)
Brenners tumor
-Usually small solid mass, discovered
incidentally, may present multiloculated
solid-cystic mass
- Amorphous calcification is seen in
solid portion
- On CT solid component is enhance
- On T2WI dense fibrous stromal
component is low signal intensity
Immature teratoma
Have predominant solid component
- May show internal
necrosis/hemorrhage
- More malignant
- Rest of the features are similar to
mature teratoma
Dysgerminoma
- Rare ovarian tumor,
predominantly in young women
-Multilobulated solid mass with
prominent fibrovascular septa
Grsnulosa cell tumor
-can be solid/multiloculated
cystic/completely cystic
-heterogeneously solid due to
intratumoral bleed/necrosis/fibrous
degeneration
No intracystic papillary projection,
no tendency to peritoneal seeding
Fibroma & thecoma
- Benign, fibroma is m/c sex cord tumor,
present as solid mass, ass. With ascites or
meigh syndrome
- homogenous solid mass delayed
enhancement
- Have abundant collagen component so
appears low signals on T1 & very low
signal on T2(diagnostic for fibroma) d/d
– pedunculated uterine leiomyoma
- Having dense scattered calcifications
- Scattered high signal intensity represent
edema or cystic degeneration
Sellar mass and ovarian mass

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Sellar mass and ovarian mass

  • 1. Presented by Dr. Anil jangir Guided by Dr. R.N Gehlot
  • 2. Common sellar and parasellar tumors - Pituitary adenoma - Craniopharyngioma -Meningioma -Rathkes cleft cyst -Chiasmatic glioma -Dermoid -Epidermoid -Germinoma -Schwannoma -Metastasis - Giant aneurysm
  • 3. Epidermoid cyst - Well circumscribed - Non enhancing - Homogenously hypodense lesion - CSF signals on T1/T2/FLAIR - Can cause bone thinning - Bright on DWI (Most helpful MR sequence for Making diagnosis ) • (d/d-Arachnoid cyst appears dark like CSF on DWI) - Central eccentric Chiasmatic CP angle Quadrigeminal plate Middle cranial fossa sylvian fissure
  • 4. Pituitary adenoma -Micro adenoma usually enhance later or lesser then normal pituitary Suprasellar extension, Inferiorly into sphenoid sinus/clivus -Isointense to brain parenchyma on T1 *Macro adenoma may contain hemorrhagic or cystic component. Acute hemorrhage may lead to rapid expansion of the gland and Cause acute compression of optic chiasma ( pituitary apoplexy) Hemorrhage appears hyperintense on T1
  • 5. Pituitary apoplexy Pituitary apoplexy due to hemorrhage in macroadenoma
  • 6. Pituitary apoplexy -Caused by hemorrhagic/ischemic necrosis of pituitary gland -pre-existing macroadenoma usually seen - Sheehan syndrome- acute postpartum ischemic necrosis - H/o Medical management (bromocriptine/cabergoline) of prolactinoma - Signal intensity on MR depends on clot age - On T1- heterogeneously iso to hypo - T2- iso to mildly hyperintense on T2 initially then hypointense - Blooming on T2 may seen - T1 C+ rim enhancement
  • 7. Rathke’s cleft cyst(RCC) -Mean age of presentation is 45 years, can be sellar or suprasellar Most symptomatic RCC are 5-15mm in diameter, can cause pituitary dysfunction, visual disturbance and headache. -On NECT- hypo/iso/hyperdense -T1- hypo/ hyperintense, T2- most of hyperintense , FLAIR- hyperintense -An enhancing rim sign of compressed pituitary (claw sign)can be sign -Non enhancing intracystic nodule (dot sign)- pathognomic of RCC -Can present with cyst apoplexy caused by sudden intracystic hemorrhage
  • 8. Claw sign Dot sign Fluid dluid level
  • 10. Child present with Predominantly cystic suprasellar mass with peripheral calcification on NECT Fig. B
  • 11. Fig.C
  • 12. fig. D 12 year old boy present with suprasellar mass
  • 13. Solid ovarian mass differential diagnosis -Epithelial tumor- Brenners tumor -Germ cell tumor- Teratoma, dysgerminoma -Sex cord tumor- Fibroma, thecoma, Granulosa cell tumor -Metastatic- krukenberg tumor
  • 14. -- Signet ring Metastasis to ovary – usually large, b/l solid adnexal mass showing heterogenous contrast enhancement Primary gastric cancer metastasize to Liver(m/c), lungs, adrenal and ovaries. Can come from stomach (signet ring cells-m/c), Colorectal (2nd m/c), breast, lung, contralateral ovary, pancreatic, cholangiocarcinoma On MRI- b/l complex ovarian mass with hypointense solid component and T1/T2 hyperintense mucin component
  • 15. Biopsy proven case of signet cell adenocarcinoma of stomach with b/l ovarian metastasis(krukenberg tumor)
  • 16. Brenners tumor -Usually small solid mass, discovered incidentally, may present multiloculated solid-cystic mass - Amorphous calcification is seen in solid portion - On CT solid component is enhance - On T2WI dense fibrous stromal component is low signal intensity Immature teratoma Have predominant solid component - May show internal necrosis/hemorrhage - More malignant - Rest of the features are similar to mature teratoma
  • 17. Dysgerminoma - Rare ovarian tumor, predominantly in young women -Multilobulated solid mass with prominent fibrovascular septa Grsnulosa cell tumor -can be solid/multiloculated cystic/completely cystic -heterogeneously solid due to intratumoral bleed/necrosis/fibrous degeneration No intracystic papillary projection, no tendency to peritoneal seeding
  • 18. Fibroma & thecoma - Benign, fibroma is m/c sex cord tumor, present as solid mass, ass. With ascites or meigh syndrome - homogenous solid mass delayed enhancement - Have abundant collagen component so appears low signals on T1 & very low signal on T2(diagnostic for fibroma) d/d – pedunculated uterine leiomyoma - Having dense scattered calcifications - Scattered high signal intensity represent edema or cystic degeneration