PERITONITIS
 By Mohammed Shadabul Haqh
 16901087
 Contact : 056-176-4224
 Email : mohammed.16901087@rakmhsu.ac.ae
Map of the presentation
 Case study
 Paths to peritonitis
 Microbiology of peritonitis
 Clinical features
 Investigations
 Imaging
 Management
 Special forms of peritonitis
 References
 A 42 year-old male presented to Hospital for evaluation of worsening
abdominal pain, nausea and vomiting starting 3 days prior to
presentation. On admission, his history was remarkable for four similar
prior episodes over the previous five years that lasted between 3 and 5
days. He denied any constipation, obstipation or associated
hematemesis, fevers, chills or urinary symptoms. During the first
episode five years ago, he was evaluated at an outlying health center
and diagnosed with peptic ulcer disease and was managed with
omeprazole intermittently . His past medical and surgical history was
non contributory and he had no allergies and he denied alcohol intake
or tobacco use. His HIV serostatus was negative approximately one year
prior to presentation.
CASE
 On examination he was afebrile, with a heart rate of 120
beats/min, blood pressure 135/78 mmHg and respiratory
rate of 22/min. Abdominal examination revealed mild
distension with generalized guarding and marked rebound
tenderness in the epigastrium. There were no palpable
masses and bowel sounds were absent. Full blood count
and serum chemistry was unavailable . Erect and supine
abdominal and chest radiographs were normal however,
abdominal ultrasonography revealed free fluid throughout
the abdomen and pelvis
Paths to peritonitis
 GI perforation
 Transmural translocation ( bacterial translocation)
 Exogenous contamination
 Female GUT infection
Microbiology
 Gram negative bacteria
 Clostridium perfringens
 chlamydia spp
Clinical features
 Abdominal pain – worse on movement, coughing, deep inspiration
 Constitutional upset (AMF)
 GI upset
 Raised pulse rate
 Tenderness = guarding, rigidity, rebound
 Rectal or vaginal tenderness ( pelvic peritonitis)
 Absent bowel sounds
 SIRS and MODS (later stages )
INVESTIGATIONS
 Baseline urea and electrolytes (U&Es) for treatment.
 Full blood count for white cell count (WCC).
 Serum amylase
 Urine dipstix for urinary tract infection.
IMAGING
 XRAY
 CT
 ULTRASOUND
MANAGEMENT
 General care of the patient
 Specific treatment of the cause
 ANTIBIOTICS
 ANALGESIA
 CORRECTION OF FLUID LOSS AND CIRCULATING VOLUME
SPECIAL FORMS OF PERITONITIS
 Bile peritonitis
 Spontaneous bacterial
 Primary pneumococcal peritonitis
 TB peritonitis
Reference
 Bailey and love short practice of surgery
 27th edition
 Pg : 1048 – 1052
THANK YOU …………
 Questions are warmly welcomed

Peritonitis

  • 1.
    PERITONITIS  By MohammedShadabul Haqh  16901087  Contact : 056-176-4224  Email : mohammed.16901087@rakmhsu.ac.ae
  • 2.
    Map of thepresentation  Case study  Paths to peritonitis  Microbiology of peritonitis  Clinical features  Investigations  Imaging  Management  Special forms of peritonitis  References
  • 3.
     A 42year-old male presented to Hospital for evaluation of worsening abdominal pain, nausea and vomiting starting 3 days prior to presentation. On admission, his history was remarkable for four similar prior episodes over the previous five years that lasted between 3 and 5 days. He denied any constipation, obstipation or associated hematemesis, fevers, chills or urinary symptoms. During the first episode five years ago, he was evaluated at an outlying health center and diagnosed with peptic ulcer disease and was managed with omeprazole intermittently . His past medical and surgical history was non contributory and he had no allergies and he denied alcohol intake or tobacco use. His HIV serostatus was negative approximately one year prior to presentation. CASE
  • 4.
     On examinationhe was afebrile, with a heart rate of 120 beats/min, blood pressure 135/78 mmHg and respiratory rate of 22/min. Abdominal examination revealed mild distension with generalized guarding and marked rebound tenderness in the epigastrium. There were no palpable masses and bowel sounds were absent. Full blood count and serum chemistry was unavailable . Erect and supine abdominal and chest radiographs were normal however, abdominal ultrasonography revealed free fluid throughout the abdomen and pelvis
  • 6.
    Paths to peritonitis GI perforation  Transmural translocation ( bacterial translocation)  Exogenous contamination  Female GUT infection
  • 7.
    Microbiology  Gram negativebacteria  Clostridium perfringens  chlamydia spp
  • 8.
    Clinical features  Abdominalpain – worse on movement, coughing, deep inspiration  Constitutional upset (AMF)  GI upset  Raised pulse rate  Tenderness = guarding, rigidity, rebound  Rectal or vaginal tenderness ( pelvic peritonitis)  Absent bowel sounds  SIRS and MODS (later stages )
  • 9.
    INVESTIGATIONS  Baseline ureaand electrolytes (U&Es) for treatment.  Full blood count for white cell count (WCC).  Serum amylase  Urine dipstix for urinary tract infection.
  • 10.
  • 13.
    MANAGEMENT  General careof the patient  Specific treatment of the cause  ANTIBIOTICS  ANALGESIA  CORRECTION OF FLUID LOSS AND CIRCULATING VOLUME
  • 14.
    SPECIAL FORMS OFPERITONITIS  Bile peritonitis  Spontaneous bacterial  Primary pneumococcal peritonitis  TB peritonitis
  • 15.
    Reference  Bailey andlove short practice of surgery  27th edition  Pg : 1048 – 1052
  • 16.
    THANK YOU ………… Questions are warmly welcomed

Editor's Notes

  • #7 Peritonitis is simply defines as inflammation of the peritoneum and maybe localized or generalized. Peritonitis Is used without qualification acute bacterial peritonitis Gi – perforated ulcer, appendix, diverticulum Transmural translocation – no perforation … pancreatitis , ischemic bowel Ex contamination -