Periodontal pathogensis
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This document outlines the pathogenesis of periodontal disease. It begins by defining pathogenesis as the origination and development of disease. It then defines periodontal disease as an infection of the gums, periodontal ligament and alveolar bone. Healthy gums are described as pink, firm and not inflamed or swollen, while gum diseases like gingivitis and periodontitis are defined. The document goes on to describe the inflammatory response in the periodontium, including microbial virulence factors from bacteria and host-derived inflammatory mediators like cytokines and prostaglandins. Clinical signs of periodontal disease are provided.
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Pathogenesis of plaque associated periodontal disease
This document provides an overview of the pathogenesis of periodontal disease. It begins with definitions of pathogenesis and periodontitis. Key points include: plaque bacteria initiate inflammatory responses leading to tissue damage; the host immune response determines susceptibility; and the transition from gingivitis to periodontitis involves a shift from localized to widespread inflammation and bone/tissue loss. Histopathological changes are described at each disease stage. The roles of bacterial virulence factors and host inflammatory mediators such as cytokines are discussed.
Hormonal changes in female patients and periodontal diseases
Hormonal fluctuations in females can impact periodontal disease. Puberty, menstruation, pregnancy, menopause, and oral contraceptive use can all influence the gingival tissues and affect susceptibility to periodontal disease. Pregnancy in particular increases sex hormone levels like estrogen and progesterone, which can directly impact the periodontium and immune response, resulting in conditions like pregnancy gingivitis. Periodontal disease has also been associated with adverse pregnancy outcomes like preterm birth and preeclampsia. Proper oral hygiene and treatment during stages of hormonal change in females can help reduce risks to oral and overall health.
GINGIVAL INFLAMMATION
BASIC CONCEPTS OF INFLAMMATION-STAGES OF INFLAMMATION-ALL ABOUT GINGIVAL INFLAMMATION-CLINICAL FEATURES AND STAGES OF GINGIVITIS-HOW TO MANAGE-ALL IN ONE-FOR B.D.S LEVEL PROJECTS AND SEMINARS
Gingivitis
Gingivitis is defined as the inflammation of gingival tissue.Gingival inflammation has two components: the acute
inflammatory component, with vasodilation, edema, and
polymorphonuclear infiltration, and the chronic inflammatory
component, with B and T lymphocytes and capillary
proliferation forming a granulomatous response.
Gingival diseases in children
Children are subject to a wide variety and severity of gingival diseases. Children should be routinely examined for the presence of periodontal infection signs to aid in early detection, diagnosis and required treatment. Most of the times, children are evaluated only for hard tissue examination , leaving out soft tissue examination.
Classification of disseases
This document summarizes various gingival and periodontal diseases. It describes diseases caused by dental plaque, such as gingivitis, and modified by factors like medications, malnutrition, and systemic diseases. It also discusses periodontitis and other conditions like necrotizing gingivitis/periodontitis, endo-perio lesions, and developmental deformities that can affect the gingiva and periodontium. Diagnostic methods and typical treatments are mentioned for several conditions.
027.necrotizing ulcerative periodontitis NUP
This document discusses different types of necrotizing ulcerative periodontitis including non-AIDS type and AIDS-associated type. It also discusses refractory periodontitis caused by abnormal host response, resistant bacteria, failure to remove plaque, and smoking. Microbial complexes associated with refractory periodontitis include Porphyromonas gingivalis, Treponema denticola, and Tannerella forsythia. Treatment involves antimicrobial therapy and local drug delivery. The document also discusses periodontitis caused by systemic diseases that impair neutrophil function such as Papillon-Lefèvre syndrome, Chédiak-Higashi syndrome, and Down syndrome.
Necrotizing periodontal disease
Necrotizing periodontal disease (NPD) is a rapidly destructive inflammatory disease caused by plaque bacteria that affects the gingiva and periodontium. It exists in acute and chronic forms and can range from superficial necrotic gingival lesions to more severe necrotizing stomatitis involving deeper tissues. Predisposing factors include poor oral hygiene, systemic diseases like HIV/AIDS, and malnutrition. It is characterized by painful ulcerated and necrotic gingival tissue covered with a non-coherent slough. Without treatment, the lesions can progress rapidly and cause significant bone and tissue destruction.
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Pathogenesis of plaque associated periodontal disease
This document provides an overview of the pathogenesis of periodontal disease. It begins with definitions of pathogenesis and periodontitis. Key points include: plaque bacteria initiate inflammatory responses leading to tissue damage; the host immune response determines susceptibility; and the transition from gingivitis to periodontitis involves a shift from localized to widespread inflammation and bone/tissue loss. Histopathological changes are described at each disease stage. The roles of bacterial virulence factors and host inflammatory mediators such as cytokines are discussed.
Hormonal changes in female patients and periodontal diseases
Hormonal fluctuations in females can impact periodontal disease. Puberty, menstruation, pregnancy, menopause, and oral contraceptive use can all influence the gingival tissues and affect susceptibility to periodontal disease. Pregnancy in particular increases sex hormone levels like estrogen and progesterone, which can directly impact the periodontium and immune response, resulting in conditions like pregnancy gingivitis. Periodontal disease has also been associated with adverse pregnancy outcomes like preterm birth and preeclampsia. Proper oral hygiene and treatment during stages of hormonal change in females can help reduce risks to oral and overall health.
GINGIVAL INFLAMMATION
BASIC CONCEPTS OF INFLAMMATION-STAGES OF INFLAMMATION-ALL ABOUT GINGIVAL INFLAMMATION-CLINICAL FEATURES AND STAGES OF GINGIVITIS-HOW TO MANAGE-ALL IN ONE-FOR B.D.S LEVEL PROJECTS AND SEMINARS
Gingivitis
Gingivitis is defined as the inflammation of gingival tissue.Gingival inflammation has two components: the acute
inflammatory component, with vasodilation, edema, and
polymorphonuclear infiltration, and the chronic inflammatory
component, with B and T lymphocytes and capillary
proliferation forming a granulomatous response.
Gingival diseases in children
Children are subject to a wide variety and severity of gingival diseases. Children should be routinely examined for the presence of periodontal infection signs to aid in early detection, diagnosis and required treatment. Most of the times, children are evaluated only for hard tissue examination , leaving out soft tissue examination.
Classification of disseases
This document summarizes various gingival and periodontal diseases. It describes diseases caused by dental plaque, such as gingivitis, and modified by factors like medications, malnutrition, and systemic diseases. It also discusses periodontitis and other conditions like necrotizing gingivitis/periodontitis, endo-perio lesions, and developmental deformities that can affect the gingiva and periodontium. Diagnostic methods and typical treatments are mentioned for several conditions.
027.necrotizing ulcerative periodontitis NUP
This document discusses different types of necrotizing ulcerative periodontitis including non-AIDS type and AIDS-associated type. It also discusses refractory periodontitis caused by abnormal host response, resistant bacteria, failure to remove plaque, and smoking. Microbial complexes associated with refractory periodontitis include Porphyromonas gingivalis, Treponema denticola, and Tannerella forsythia. Treatment involves antimicrobial therapy and local drug delivery. The document also discusses periodontitis caused by systemic diseases that impair neutrophil function such as Papillon-Lefèvre syndrome, Chédiak-Higashi syndrome, and Down syndrome.
Necrotizing periodontal disease
Necrotizing periodontal disease (NPD) is a rapidly destructive inflammatory disease caused by plaque bacteria that affects the gingiva and periodontium. It exists in acute and chronic forms and can range from superficial necrotic gingival lesions to more severe necrotizing stomatitis involving deeper tissues. Predisposing factors include poor oral hygiene, systemic diseases like HIV/AIDS, and malnutrition. It is characterized by painful ulcerated and necrotic gingival tissue covered with a non-coherent slough. Without treatment, the lesions can progress rapidly and cause significant bone and tissue destruction.
Gingivitis
This document provides a classification system for periodontal pathology, dividing it into gingival diseases and periodontitis. It describes various forms of gingival disease including those induced by dental plaque, modified by systemic factors like pregnancy or diabetes, drug-induced forms from medications like phenytoin, and non-plaque induced lesions from conditions such as linear gingival erythema. It also outlines different types of periodontitis including chronic, aggressive, periodontitis associated with systemic diseases, necrotizing forms, abscesses, and lesions associated with endodontic problems. Developed deformities and occlusal trauma are additionally discussed.
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*definition
*classification
*Signs and Symptoms: Increased GCF, Gingival Bleeding, Color change, Consistency, Surface texture (STIPPLING), Position of Gingiva, Gingival Contour, Size.
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张结 3 1 1
Periodontal diseases affect the tissues around the teeth and range from gingivitis to periodontitis. About 60-90% of people have some form of periodontal disease depending on age. Periodontal diseases are caused by bacterial dental plaque which leads to inflammation if not removed regularly. The bacteria can damage gum tissue, bone, and ligaments supporting the teeth. Periodontal diseases are classified based on severity from gingivitis to chronic periodontitis with bone loss. Treatment focuses on improving oral hygiene through regular cleaning to control the bacterial plaque.
Periodontal disease
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Chronic periodontitis (updated)
chronic periodontitis
History
Introduction
Definition
Classification
Overall Characteristics
Clinical Features
Disease Distribution
Disease Severity
Clinical Course Of Chronic Periodontitis
Progression Of Chronic Periodontitis
Prevalence
Pathogenesis
Risk factors for Chronic Periodontitis
Treatment
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Acute necrotising ulcerative gingivitis is a rare condition characterized by necrosis of the gingiva and interdental papillae. It frequently occurs during times of stress and poor oral hygiene. The condition is caused by fusiform bacillus and Borrelia vincentii bacteria. Clinically, the gingiva becomes painful and develops punched-out ulcers. It can spread to other oral tissues and rarely the skin, causing further complications.
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This document summarizes information on periodontal abscesses. It begins by defining periodontal abscesses as localized acute bacterial infections confined to the tissues of the periodontium. It then discusses the classification, microbiology, pathogenesis, predisposing factors, diagnosis, and differential diagnosis of periodontal abscesses. Regarding treatment, it states that periodontal abscesses were historically a main reason for tooth extraction, but today the main therapeutic approaches discussed are drainage and debridement, systemic antibiotics, and periodontal surgical procedures in the chronic phase.
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This document provides information on gingival and periodontal diseases. It discusses various types of gingival diseases like gingivitis, acute gingival diseases, gingival enlargement. It also covers different types of periodontal diseases like chronic periodontitis, aggressive periodontitis. Periodontal diseases are infectious diseases that cause destruction of tissues supporting the teeth. Nearly 75% of adults suffer from some form of periodontal disease. Early detection and treatment can help most people keep their teeth for life.
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This document discusses chronic periodontitis, the most common form of periodontitis. It is defined as an infectious disease resulting in inflammation and destruction of the tissues supporting the teeth. The key clinical features are plaque, gingivitis, attachment loss, bone loss, and inflammation. Chronic periodontitis is considered a site-specific disease where inflammation and bone loss occur at specific sites due to local plaque accumulation. The severity is classified as slight, moderate, or severe based on the amount of attachment loss. Risk factors include poor plaque control, systemic diseases, environmental/behavioral factors like smoking, and genetics. Treatment involves improving oral hygiene, nonsurgical treatments like scaling and root planing, and sometimes surgery.
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This document defines and classifies gingival enlargement according to its etiology and pathologic changes. It describes inflammatory, drug-induced, idiopathic, and enlargements associated with systemic diseases. Chronic and acute inflammatory enlargements are discussed. Specific drugs that commonly cause drug-induced gingival enlargement are identified along with their mechanisms and prevalence. Systemic diseases like leukemia and granulomatous diseases that can cause gingival enlargement are outlined. Benign and malignant neoplastic enlargements are also defined. The document concludes with descriptions of false enlargements and various treatment methods for gingival enlargement.
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This document discusses the clinical features of gingivitis. It begins by defining gingivitis as inflammation of the gingiva and describes how plaque bacteria can damage gingival tissues. It then covers the different types of gingivitis based on duration and distribution. Key signs of gingivitis that are discussed include gingival bleeding, color changes, changes in consistency, size, surface texture, position and contour. Specific conditions like gingival recession are also explained in terms of definition, classification, etiology and clinical significance.
Intro eight gingival and periodontal diseases
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Gum infection, also known as periodontal infection, refers to an inflammatory condition that affects the tissues surrounding and supporting the teeth. It primarily involves the gums (gingiva) and can progress to affect the bone and other structures that hold the teeth in place.
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This document provides a classification system for periodontal pathology, dividing it into gingival diseases and periodontitis. It describes various forms of gingival disease including those induced by dental plaque, modified by systemic factors like pregnancy or diabetes, drug-induced forms from medications like phenytoin, and non-plaque induced lesions from conditions such as linear gingival erythema. It also outlines different types of periodontitis including chronic, aggressive, periodontitis associated with systemic diseases, necrotizing forms, abscesses, and lesions associated with endodontic problems. Developed deformities and occlusal trauma are additionally discussed.
Gingivitis presentation
All about gingivitis
*definition
*classification
*Signs and Symptoms: Increased GCF, Gingival Bleeding, Color change, Consistency, Surface texture (STIPPLING), Position of Gingiva, Gingival Contour, Size.
Treatment consisits of scaling and root planing. The more inflamed a gingival unit appears clinically, the better the chances of therapeutic measures resulting in a return to normal gingival health
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This document summarizes various causes of non-plaque induced gingivitis, including specific bacterial, viral, and fungal infections that can cause gingival inflammation and lesions. It describes conditions such as herpetic gingivostomatitis caused by the herpes simplex virus and candidosis caused by Candida albicans. It also discusses genetic causes like hereditary gingival fibromatosis and systemic diseases that can involve the gingiva, such as lichen planus, pemphigoid, and pemphigus vulgaris. Allergic reactions to dental materials and oral hygiene products are another potential cause of non-plaque gingivitis covered.
张结 3 1 1
Periodontal diseases affect the tissues around the teeth and range from gingivitis to periodontitis. About 60-90% of people have some form of periodontal disease depending on age. Periodontal diseases are caused by bacterial dental plaque which leads to inflammation if not removed regularly. The bacteria can damage gum tissue, bone, and ligaments supporting the teeth. Periodontal diseases are classified based on severity from gingivitis to chronic periodontitis with bone loss. Treatment focuses on improving oral hygiene through regular cleaning to control the bacterial plaque.
Periodontal disease
Periodontal disease is a chronic inflammatory disease of the gums and tissues surrounding the teeth. It is caused by plaque bacteria, including Porphyromonas gingivalis and Actinobacillus actinomycetemcomitans. Left untreated, it can lead to tooth loss. Symptoms include red, swollen, bleeding gums; bad breath; and loose teeth. It is diagnosed through examination of the gums and bone measurements. Treatment involves deep cleaning or surgery to remove bacterial infection. Daily brushing and flossing is the best prevention method.
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chronic periodontitis
History
Introduction
Definition
Classification
Overall Characteristics
Clinical Features
Disease Distribution
Disease Severity
Clinical Course Of Chronic Periodontitis
Progression Of Chronic Periodontitis
Prevalence
Pathogenesis
Risk factors for Chronic Periodontitis
Treatment
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Acute necrotising ulcerative gingivitis is a rare condition characterized by necrosis of the gingiva and interdental papillae. It frequently occurs during times of stress and poor oral hygiene. The condition is caused by fusiform bacillus and Borrelia vincentii bacteria. Clinically, the gingiva becomes painful and develops punched-out ulcers. It can spread to other oral tissues and rarely the skin, causing further complications.
Treatment of gingival enlargement - by Dr Harshavardhan Patwal
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This document summarizes information on periodontal abscesses. It begins by defining periodontal abscesses as localized acute bacterial infections confined to the tissues of the periodontium. It then discusses the classification, microbiology, pathogenesis, predisposing factors, diagnosis, and differential diagnosis of periodontal abscesses. Regarding treatment, it states that periodontal abscesses were historically a main reason for tooth extraction, but today the main therapeutic approaches discussed are drainage and debridement, systemic antibiotics, and periodontal surgical procedures in the chronic phase.
Chronic periodontitis.pptxby shrish
Chronic periodontitis is an infectious disease that causes inflammation of the tissues supporting the teeth and progressive bone and attachment loss. It typically affects people aged 30-35 and causes variable pocket depths and bone loss patterns, including horizontal and vertical loss. Symptoms may include pain, sensitivity, and food impaction. Risk factors include poor oral hygiene, smoking, diabetes, and genetic factors. Treatment involves improving oral hygiene, nonsurgical therapies like scaling and root planing, and sometimes surgery.
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This document provides information on gingival and periodontal diseases. It discusses various types of gingival diseases like gingivitis, acute gingival diseases, gingival enlargement. It also covers different types of periodontal diseases like chronic periodontitis, aggressive periodontitis. Periodontal diseases are infectious diseases that cause destruction of tissues supporting the teeth. Nearly 75% of adults suffer from some form of periodontal disease. Early detection and treatment can help most people keep their teeth for life.
Chronic periodontitis
This document discusses chronic periodontitis, the most common form of periodontitis. It is defined as an infectious disease resulting in inflammation and destruction of the tissues supporting the teeth. The key clinical features are plaque, gingivitis, attachment loss, bone loss, and inflammation. Chronic periodontitis is considered a site-specific disease where inflammation and bone loss occur at specific sites due to local plaque accumulation. The severity is classified as slight, moderate, or severe based on the amount of attachment loss. Risk factors include poor plaque control, systemic diseases, environmental/behavioral factors like smoking, and genetics. Treatment involves improving oral hygiene, nonsurgical treatments like scaling and root planing, and sometimes surgery.
Gingival Enlargement
This document defines and classifies gingival enlargement according to its etiology and pathologic changes. It describes inflammatory, drug-induced, idiopathic, and enlargements associated with systemic diseases. Chronic and acute inflammatory enlargements are discussed. Specific drugs that commonly cause drug-induced gingival enlargement are identified along with their mechanisms and prevalence. Systemic diseases like leukemia and granulomatous diseases that can cause gingival enlargement are outlined. Benign and malignant neoplastic enlargements are also defined. The document concludes with descriptions of false enlargements and various treatment methods for gingival enlargement.
Periodontal diseases iii / dental implant courses by Indian dental academy
The document defines and describes key periodontal diseases: Chronic Periodontitis, Aggressive Periodontitis, and Necrotizing Ulcerative Periodontitis. Chronic Periodontitis is the most common form and involves plaque-induced inflammation that causes progressive bone and attachment loss. Aggressive Periodontitis affects otherwise healthy individuals and causes rapid destruction, while Necrotizing Ulcerative Periodontitis involves necrosis, ulceration and pain between teeth with potential systemic involvement. The document provides details on etiology, clinical features, classifications and prevalence for each disease.
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The document discusses the classification and features of periodontal disease. It covers topics such as marginal periodontitis, juvenile periodontitis, trauma from occlusion, and periodontal atrophy. It also describes the periodontal pocket in detail, including its pathogenesis, morphology, contents, and relationship to bone loss. The extension of inflammation from the gingiva to the supporting periodontal tissues is discussed as well.
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This document discusses the clinical features of gingivitis. It begins by defining gingivitis as inflammation of the gingiva and describes how plaque bacteria can damage gingival tissues. It then covers the different types of gingivitis based on duration and distribution. Key signs of gingivitis that are discussed include gingival bleeding, color changes, changes in consistency, size, surface texture, position and contour. Specific conditions like gingival recession are also explained in terms of definition, classification, etiology and clinical significance.
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Treatment of gingival enlargement - by Dr Harshavardhan Patwal
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Periodontal diseases iii / dental implant courses by Indian dental academy
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This document provides an overview of gingival and periodontal diseases. It defines key terms like gingivitis, periodontitis, inflammation and describes the signs of inflammation. It discusses the differences between gingivitis and periodontitis and classifies them as mild, moderate or advanced. The main causes are identified as bacterial plaque accumulation and predisposing factors like calculus. Proper patient education and preventive measures like effective plaque removal and balanced diet are important for prognosis.
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Periodontal pathogensis
- 2. Javaria Baig 3rd year BDS Batch 18 Periodontal Pathogenesis
- 3. Outlines What is Pathogenesis? What is periodontal disease? Healthy gums Gum diseases Inflammatory response in periodontium Inflammatory mediators Clinical signs Model of periodontitis
- 4. Whatis pathogenesis? Pathos ( suffering), Genesis ( generation) Pathogenesis is origination and development of disease.
- 5. Whatis periodontal disease? Periodontal diseases are infections of the structures around the teeth, which include the gums, periodontal ligament and alveolar bone.
- 7. HealthyGums Pink in appearance Not swollen Not inflamed Firmly attached to bone Minimal bleeding on probing
- 9. GUMdiseases Gingivitis is an inflammation of the gums caused by a bacterial infection. Periodontitis is inflammation of the gums and supporting structures of the teeth.
- 10.
- 11. Inflammatory responsein periodontium They are divided into two: Microbial virulence factors Host derived inflammatory mediatiors
- 13. Microbial virulence factors Lipopolysaccharide ( endotoxins) Bacterial enzyme ( directly damage tissue) Noxious products ( ammonia and H2S) Microbial invasion ( P.gingivalis, Aggregatibacter actinomycetemcomitans, fusobacterium nucleatum) Fimbriae ( stimulate immune response) Bacterial deoxyribonucleic acid Extracellar deoxyribonucleic acid
- 15. Hostderived inflammatory mediators Cytokines ( inflammatory mediators) Prostaglandins ( vasodilation) Matrix metalloproteinases ( degrade extra cellular matrix molecules)
- 17. Inflammatory mediators Interleukin 1 Tumor necrosis factor Interleukin 6 Prostaglandin E2 Matrix metalloproteinase Anti inflammatory cytokines Chemokines
- 19. Clinicalsignsof periodontal disease Gingivitis Swollen gums Red gums at margins Bleeding on probing Bad breath Tooth sensitivity Pressure change Pocket formation Bone loss around tooth Abscesses formation
- 21. Reference