PRESENTED BY: PINGALE DIPALI
SHANTARM
PRESENTED BY: PINGALE DIPALI
SHANTARAM
2 ND YEAR MSC NURSING
OBJECTIVES
• Definition.
• Incidence and epidemiology.
• Clinical characteristics.
• Classification.
• Diagnosis.
• Complications.
• Management.
• When more than
one fetus
simultaneously
develops in the
uterus, it is called
multiple
pregnancy.
Terminology vs. number
• Singletons- one fetus
• Twins - two fetuses.
• Triplets - threefetuses.
• Quadruplets - four fetuses.
• Quintuplets- five fetuses.
• sextuplets- sixfetuses.
• Septuplets- seven fetuses.
Types of twins………
DIZYGOTI
C
MONOZYGOTI
C
Dizygotic twins (fraternal)
• Most common represents 2/3 of cases.
Fertilization of more than one egg by more
than one sperm.
• Non identical ,may be of different sex. Two
chorion and two amnion.
• Placenta may be separate or fused. “each
fetus is contained within a complete amniotic-
chorionic membrane “
Dizygotic twins/ BINOVULAR
(fraternal)
Monzygotic twins
• Constitutes 1/3 of twins These twins are multiple
gestations resulting from cleavage of a single,
fertilized ovum.
• The timing of cleavage determines the
placentation of the pregnancy.
• Constant incidence .
• Not affected by heredity.
• Not related to induction of ovulation.
MONOZYGOTIC / UNIOVULAR/
IDENTICAL
MONOZYGOTIC TWINS 23%
75%
1%
<1%
THORACOPAGUS
ISCHIOPAGUSCRANIOPAGUS
RACHYPAGUSPYOPAGUSOMPHALOPAGUS
Important notes:
• 1- Monozygotic twins having same sex &
blood group.
• 2- Process of formation of chorion is earlier
than formation of amnion.
• 3-Dizygotic twins must be
dichorionic/diamniotic.
• 4- There is no dichorionic/ monoamniotic.
HELLIN’S RULE
Twins 1 in 80
Triplets 1 in 80^2
Quadruplets 1 in 80^3
INCIDENCE
Etiology
Ethnic group
Increasing
maternal age
Increasing parity
Family h/o twinning, esp
maternal
Ovulation induction
• Super fecundation: is fertilization of two ova
produced in the same menstrual cycle by two
spermatozoa deposited in two separate acts
of coitus.
• Superfoetation: is fertilization of two ova
produced in two different menstrual cycles by
two separate spermatozoa. Actually, this
cannot occur in human as ovulation is
suppressed once pregnancy occurs.
Maternal Physiological Adaptation
Increase blood volume and cardiac output.
Increase demand for iron and folic acid.
Maternal respiratory difficulty.
Excess fluid retention and edema.
Increase attacks of supine hypotension.
Lie and presentation
• Commonest lie – longitudinal (90%) but
malpresentation are quite common.
• Both vertex (50%)
• First vertex and second breech (30%)
• First breech and second vertex (10%)
• Both breech (10%)
• First vertex and second transverse and so on...
Diagnosis
History
* Family history of multiple pregnancy (wife
and/ or husband).
* Recent intake of ovulatory drugs.
* Increased foetal movement.
Diagnosis
*Inspection
More enlargement of the abdomen.
Palpation:
* Fundal level: higher than that corresponds to the
period of amenorrhoea.
* Fundal, umbilical and first pelvic grips: can detect
multiple foetal poles. At least, 3 poles should be
palpated to diagnose twin pregnancy.
* Foetal limbs: felt as multiple knobs.
Diagnosis
• Auscultation
* Foetal heart sounds: are heard with maximum
intensity in 2 separate points by 2 observers
with a minimum difference of 10 beats per
minute.
* Arnaux sign: occasionally, the superimposition
of two foetal heart sounds produces a
galloping rhythm.
Diagnosis
Ultrasonography
• Diagnosis of twins:
*At 7th week: two separate gestation sacs can be
identified.
*At 8th week: separate foetal bodies can be detected.
* At 12th week: separate heads can be distinguished.
*If routine scanning of all pregnant women is carried out
at 16 weeks twins should rarely be missed.
Diagnosis
Ultrasonography
Detection of:
• Presentations and positions.
• Gestational age.
• Congenital anomalies.
• Polyhydramnios.
• Placental site.
Diagnosis
• X-ray
If ultrasound is not available it can detect foetal
heads and vertebral columns.
DIAGNOSIS OF MULTIPLE PREGNANCY
Positive family history mainly on maternal
side.
Positive history of ovulation induction.
Exaggerated symptoms of pregnancy.
Marked edema of lower limb.
Discrepancy between date and uterine size.
Palpation of many fetal parts.
INVESTIGATION
• Number of sacs. [ before 10 weeks ]
 2 sacs – dichorionic
 Single sac - monochorionic
• Placenta
• Sex
• Intertwin membrane
 thicker and more echogenic in dichorionic
.
• Twin peak / Lambda sign
- characteristic of dichorionic pregnancies
- chorionic tissue between 2 layers of
intertwin membrane at the placental origin
• T Sign – in monochorionic , no chorionic tissue
• If no membrane is seen in between –
monochorionic monoaniotic
Maternal Complications
Antepartum Intrapartum
1.Hyperemesis 1.Dysfunctional labour
2.Hydramnios 2.Malpresentation
3.Pre-eclampsia 3.Operative delivery
4.Pressure symptoms 4.Postpartum
hemorrhage
5.Anaemia 5.Retained Placenta
6.Antepartum
hemorrhage
6.Premature separation
of placenta
Maternal Complications - Antepartum
Hyperemesis – increased β- hCG
Hydramnios – monoamniotic pregnancies, Twin
transfusion syndrome, major cause of prematurity
Pre- eclampsia – 3 times commoner compared to
singleton
Pressure symptoms
Anaemia – increased plasma volume expansion ,
fetoplacental demand for iron increased.
APH – Placenta praevia , Abruptio placenta.
Fetal Complications
Antepartum Intrapartum
1.Prematurity 1.PROM
2.IUGR 2.Cord Prolapse
3.Single fetal demise 3.Abruption in second
twin
4.Twin to Twin transfusion
syndrome
4.Interlocking (rare)
5.Vanishing Twin/abortion
6.Cong.anomalies
7.Conjoined twins
FETAL COMPLICATIONS
 Perinatal mortality: 6 times
 Morbidity: 2- 3 times
 Mono chorionic - morbidity/mortality twice as that of dichorionic.
- additional risk from TTS
 Monoamniotic twins - 50% mortality.
 Main cause of adverse outcome is
1. Prematurity
2. IUGR
 Cerebral palsy, neurodevelopmental impairment, lower IQ scores.
 Monochorionic twins: 1. TTTS
2 .Monoamniotic twinning
3. Conjoined twinning
4. Acardiac fetus
Management
• During pregnancy
* Frequent antenatal visits: to detect early any
complication mentioned before and manage it.
* Proper diet: with prophylactic supplementation of
iron and folic acid.
* Adequate rest: to improve placental blood flow
and avoid preterm labour.
* Prophylactic tocolytics or cerclage: is of no actual
benefit.
Management
• During labour
* Delivery should be in a hospital .
* A team of experienced obstetrician, assistant,
anaesthetist and neonatologist is necessary
for safety.
Management
Delivery of the first twin:
>If it is cephalic: proceed as normal usually
there is no problem.
> If it is breech: caesarean section is safer for
fear of locked twins, although vaginal delivery
may pass without this complication.
>Immediate clamping of the cord is essential
after delivery of the first twin to avoid
bleeding from a uniovular second twin.
Management
Delivery of the second twin: It depends upon its
presentation;
• Longitudinal lie (vertex or breech):
• Transverse or oblique lie:
Management
• Longitudinal lie (vertex or breech):
>Amniotomy is done during uterine contraction
which may be delayed up to 5 minutes.
>If delay is more than 5 minute, start oxytocin
drip.
>Delivery of the second twin is usually easy due
to dilatation of the maternal passages by
delivery of the first twin.
Management
>If there is foetal distress or cord prolapse,
rapid delivery is indicated by:
* breech extraction in breech presentation.
* Forceps delivery in engaged vertex
presentation.
* Vacuum extraction or rarely internal podalic
version and breech extraction may be
indicated in non-engaged head.
Management
• Transverse or oblique lie
• a. External cephalic or podalic version is done
then do amniotomy and deliver the foetus as
cephalic or by breech extraction respectively
or,
• b. Internal podalic version and breech
extraction under general or epidural
anaesthesia.
Management
• Caesarean section is indicated in:
> The first baby is transverse lie.
>Prolapsed pulsating cord or foetal distress in the
first stage.
> Retained second twin when it is;
a. transverse lie,
b. membranes are ruptured,
c. uterus is retracted
d. cervix is not fully dilated.
Management
• Caesarean section is indicated in
> Conjoined twins.
>Triplets or more are safer delivered by C.S.
> Other indications of C.S. as placenta praevia,
contracted pelvis, etc.
Midwifery Management
• Daily weight, abdominal girth monitoring.
• Assessment of nutrition and diet pattern and
modification.
• Health education related to diet, hygiene, and
care.
• Daily monitoring of fetal heart rate.
• Intake and output monitoring.
Nursing theory
• Orem’s self care theory
• Roy’s adaptation theory
• King’s goal attainment theory(safe
confinement)
• Paplau’s interpersonal theory ( postnatal)
• Nightingale’s environmental theory (Baby)
NURSING MANAGEMENT
• 1. Breathing difficulty related to growth of the
two foetuses.
• 2. Hyper emesis related to pressure exerted on
the stomach.
• 3. Fatigue related to exhaustion due to increase
number of vomiting.
• 4. Altered nutrition less than body requirement
related to less intake of food secondary to hyper
emesis.
• 5.Fluid volume deficit related to hyper emesis.
Cont..
• 6. Altered socialization related to
psychological upset due to multiple
pregnancy.
• 7. Anxiety related to outcome of pregnancy.
• 8. knowledge deficit related to the disease
condition, self care and posnatal care of the
baby.
• 9. Fear related to process of labour.
• 10.Ineffective coping mechanism related to
anxiety, fear due to outcome of pregnancy and
process of labour.
• 11. Increased operative inferences related to
number of fetus and position.
• 12. Risk of infection related to repeated per
vaginal examination.
• 13. Risk of trauma related to increased
operative inferences.
• 14.Risk of complications related to multiple
pregnancy.
Multiple pregnancy

Multiple pregnancy

  • 1.
    PRESENTED BY: PINGALEDIPALI SHANTARM
  • 2.
    PRESENTED BY: PINGALEDIPALI SHANTARAM 2 ND YEAR MSC NURSING
  • 3.
    OBJECTIVES • Definition. • Incidenceand epidemiology. • Clinical characteristics. • Classification. • Diagnosis. • Complications. • Management.
  • 4.
    • When morethan one fetus simultaneously develops in the uterus, it is called multiple pregnancy.
  • 5.
    Terminology vs. number •Singletons- one fetus • Twins - two fetuses. • Triplets - threefetuses. • Quadruplets - four fetuses. • Quintuplets- five fetuses. • sextuplets- sixfetuses. • Septuplets- seven fetuses.
  • 6.
  • 7.
    Dizygotic twins (fraternal) •Most common represents 2/3 of cases. Fertilization of more than one egg by more than one sperm. • Non identical ,may be of different sex. Two chorion and two amnion. • Placenta may be separate or fused. “each fetus is contained within a complete amniotic- chorionic membrane “
  • 8.
  • 9.
    Monzygotic twins • Constitutes1/3 of twins These twins are multiple gestations resulting from cleavage of a single, fertilized ovum. • The timing of cleavage determines the placentation of the pregnancy. • Constant incidence . • Not affected by heredity. • Not related to induction of ovulation.
  • 10.
  • 11.
  • 12.
  • 13.
    Important notes: • 1-Monozygotic twins having same sex & blood group. • 2- Process of formation of chorion is earlier than formation of amnion. • 3-Dizygotic twins must be dichorionic/diamniotic. • 4- There is no dichorionic/ monoamniotic.
  • 14.
    HELLIN’S RULE Twins 1in 80 Triplets 1 in 80^2 Quadruplets 1 in 80^3 INCIDENCE
  • 15.
  • 16.
    Increasing parity Family h/otwinning, esp maternal Ovulation induction
  • 17.
    • Super fecundation:is fertilization of two ova produced in the same menstrual cycle by two spermatozoa deposited in two separate acts of coitus. • Superfoetation: is fertilization of two ova produced in two different menstrual cycles by two separate spermatozoa. Actually, this cannot occur in human as ovulation is suppressed once pregnancy occurs.
  • 18.
    Maternal Physiological Adaptation Increaseblood volume and cardiac output. Increase demand for iron and folic acid. Maternal respiratory difficulty. Excess fluid retention and edema. Increase attacks of supine hypotension.
  • 19.
    Lie and presentation •Commonest lie – longitudinal (90%) but malpresentation are quite common. • Both vertex (50%) • First vertex and second breech (30%) • First breech and second vertex (10%) • Both breech (10%) • First vertex and second transverse and so on...
  • 20.
    Diagnosis History * Family historyof multiple pregnancy (wife and/ or husband). * Recent intake of ovulatory drugs. * Increased foetal movement.
  • 21.
    Diagnosis *Inspection More enlargement ofthe abdomen. Palpation: * Fundal level: higher than that corresponds to the period of amenorrhoea. * Fundal, umbilical and first pelvic grips: can detect multiple foetal poles. At least, 3 poles should be palpated to diagnose twin pregnancy. * Foetal limbs: felt as multiple knobs.
  • 22.
    Diagnosis • Auscultation * Foetalheart sounds: are heard with maximum intensity in 2 separate points by 2 observers with a minimum difference of 10 beats per minute. * Arnaux sign: occasionally, the superimposition of two foetal heart sounds produces a galloping rhythm.
  • 23.
    Diagnosis Ultrasonography • Diagnosis oftwins: *At 7th week: two separate gestation sacs can be identified. *At 8th week: separate foetal bodies can be detected. * At 12th week: separate heads can be distinguished. *If routine scanning of all pregnant women is carried out at 16 weeks twins should rarely be missed.
  • 24.
    Diagnosis Ultrasonography Detection of: • Presentationsand positions. • Gestational age. • Congenital anomalies. • Polyhydramnios. • Placental site.
  • 25.
    Diagnosis • X-ray If ultrasoundis not available it can detect foetal heads and vertebral columns.
  • 26.
    DIAGNOSIS OF MULTIPLEPREGNANCY Positive family history mainly on maternal side. Positive history of ovulation induction. Exaggerated symptoms of pregnancy. Marked edema of lower limb. Discrepancy between date and uterine size. Palpation of many fetal parts.
  • 27.
    INVESTIGATION • Number ofsacs. [ before 10 weeks ]  2 sacs – dichorionic  Single sac - monochorionic • Placenta • Sex • Intertwin membrane  thicker and more echogenic in dichorionic .
  • 28.
    • Twin peak/ Lambda sign - characteristic of dichorionic pregnancies - chorionic tissue between 2 layers of intertwin membrane at the placental origin • T Sign – in monochorionic , no chorionic tissue • If no membrane is seen in between – monochorionic monoaniotic
  • 30.
    Maternal Complications Antepartum Intrapartum 1.Hyperemesis1.Dysfunctional labour 2.Hydramnios 2.Malpresentation 3.Pre-eclampsia 3.Operative delivery 4.Pressure symptoms 4.Postpartum hemorrhage 5.Anaemia 5.Retained Placenta 6.Antepartum hemorrhage 6.Premature separation of placenta
  • 31.
    Maternal Complications -Antepartum Hyperemesis – increased β- hCG Hydramnios – monoamniotic pregnancies, Twin transfusion syndrome, major cause of prematurity Pre- eclampsia – 3 times commoner compared to singleton Pressure symptoms Anaemia – increased plasma volume expansion , fetoplacental demand for iron increased. APH – Placenta praevia , Abruptio placenta.
  • 32.
    Fetal Complications Antepartum Intrapartum 1.Prematurity1.PROM 2.IUGR 2.Cord Prolapse 3.Single fetal demise 3.Abruption in second twin 4.Twin to Twin transfusion syndrome 4.Interlocking (rare) 5.Vanishing Twin/abortion 6.Cong.anomalies 7.Conjoined twins
  • 33.
    FETAL COMPLICATIONS  Perinatalmortality: 6 times  Morbidity: 2- 3 times  Mono chorionic - morbidity/mortality twice as that of dichorionic. - additional risk from TTS  Monoamniotic twins - 50% mortality.  Main cause of adverse outcome is 1. Prematurity 2. IUGR  Cerebral palsy, neurodevelopmental impairment, lower IQ scores.  Monochorionic twins: 1. TTTS 2 .Monoamniotic twinning 3. Conjoined twinning 4. Acardiac fetus
  • 34.
    Management • During pregnancy *Frequent antenatal visits: to detect early any complication mentioned before and manage it. * Proper diet: with prophylactic supplementation of iron and folic acid. * Adequate rest: to improve placental blood flow and avoid preterm labour. * Prophylactic tocolytics or cerclage: is of no actual benefit.
  • 35.
    Management • During labour *Delivery should be in a hospital . * A team of experienced obstetrician, assistant, anaesthetist and neonatologist is necessary for safety.
  • 36.
    Management Delivery of thefirst twin: >If it is cephalic: proceed as normal usually there is no problem. > If it is breech: caesarean section is safer for fear of locked twins, although vaginal delivery may pass without this complication. >Immediate clamping of the cord is essential after delivery of the first twin to avoid bleeding from a uniovular second twin.
  • 37.
    Management Delivery of thesecond twin: It depends upon its presentation; • Longitudinal lie (vertex or breech): • Transverse or oblique lie:
  • 38.
    Management • Longitudinal lie(vertex or breech): >Amniotomy is done during uterine contraction which may be delayed up to 5 minutes. >If delay is more than 5 minute, start oxytocin drip. >Delivery of the second twin is usually easy due to dilatation of the maternal passages by delivery of the first twin.
  • 39.
    Management >If there isfoetal distress or cord prolapse, rapid delivery is indicated by: * breech extraction in breech presentation. * Forceps delivery in engaged vertex presentation. * Vacuum extraction or rarely internal podalic version and breech extraction may be indicated in non-engaged head.
  • 40.
    Management • Transverse oroblique lie • a. External cephalic or podalic version is done then do amniotomy and deliver the foetus as cephalic or by breech extraction respectively or, • b. Internal podalic version and breech extraction under general or epidural anaesthesia.
  • 41.
    Management • Caesarean sectionis indicated in: > The first baby is transverse lie. >Prolapsed pulsating cord or foetal distress in the first stage. > Retained second twin when it is; a. transverse lie, b. membranes are ruptured, c. uterus is retracted d. cervix is not fully dilated.
  • 42.
    Management • Caesarean sectionis indicated in > Conjoined twins. >Triplets or more are safer delivered by C.S. > Other indications of C.S. as placenta praevia, contracted pelvis, etc.
  • 43.
    Midwifery Management • Dailyweight, abdominal girth monitoring. • Assessment of nutrition and diet pattern and modification. • Health education related to diet, hygiene, and care. • Daily monitoring of fetal heart rate. • Intake and output monitoring.
  • 44.
    Nursing theory • Orem’sself care theory • Roy’s adaptation theory • King’s goal attainment theory(safe confinement) • Paplau’s interpersonal theory ( postnatal) • Nightingale’s environmental theory (Baby)
  • 45.
    NURSING MANAGEMENT • 1.Breathing difficulty related to growth of the two foetuses. • 2. Hyper emesis related to pressure exerted on the stomach. • 3. Fatigue related to exhaustion due to increase number of vomiting. • 4. Altered nutrition less than body requirement related to less intake of food secondary to hyper emesis. • 5.Fluid volume deficit related to hyper emesis.
  • 46.
    Cont.. • 6. Alteredsocialization related to psychological upset due to multiple pregnancy. • 7. Anxiety related to outcome of pregnancy. • 8. knowledge deficit related to the disease condition, self care and posnatal care of the baby. • 9. Fear related to process of labour.
  • 47.
    • 10.Ineffective copingmechanism related to anxiety, fear due to outcome of pregnancy and process of labour. • 11. Increased operative inferences related to number of fetus and position. • 12. Risk of infection related to repeated per vaginal examination. • 13. Risk of trauma related to increased operative inferences.
  • 48.
    • 14.Risk ofcomplications related to multiple pregnancy.