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NURSING CARE
DIABETES MELLITUS
Diabetes
What is diabetes?
What population of patients would
be
at risk for developing diabetes?
Diabetes Mellitus
 Commonly referred to as diabetes.
 Diabetes “Greek word”- Siphon implies a lot of
urine.
 Mel “Latin word”- Honey.
 Metabolic disorder in which there are High
blood sugar levels over a prolonged period.
Anatomy
Diabetes
 Normal Pathophysiology
 Need to consider how insulin works
 insulin continuously released: during “fasting
periods”, the pancreas continuously releases a
small amount of insulin along with glucagon.
Together a constant level of glucose in the blood
is maintained by stimulating the release of
glucose from the liver.
Diabetes
 In diabetes
 the body’s ability to respond to insulin may
decrease
 the pancreas may stop producing insulin. This in
turn leads to hyperglycemia leads to other
acute metabolic complications
 Diabetes is a heterogeneous group of diseases
involving disruption of metabolism of
carbohydrates, fats, and protein.
Diabetes
 Classification of diabetes mellitus
Type I
 may occur at any age
 usually thin
 abrupt onset
 family history?
Diabetes
 Type II
 >age 30
 often obese
 few classic symptoms
 insulin resistant
 Impaired glucose tolerance
 plasma glucose levels higher than normal, but not
diagnostic for diabetes 2 hr plasma glucose >140
mg/dl & < 200 mg/dl
Diabetes
 Impaired fasting glucose
 fasting plasma glucose > 110 mg/dl & < 126 mg/dl
 Gestational diabetes
 has onset or discovery of glucose tolerance during
pregnancy
Diabetes
 Clinical Manifestations
 Insulin deficiency or decreased insulin activity glucose not
used properly
 results in frequent urination (polyuria), and thirst
(polydipsia)
 without insulin the patient may experience hunger
(polyphagia)
 the body will turn to other energy sources besides glucose:
first fat and then protein
Diabetes
 Diagnostic studies
 normal blood glucose range: 70-110 mg/dl
 urine tests not sufficient for a dx of diabetes
 fasting blood glucose of > 126 mg/dl
 glycosylated hemolobin
Diabetes
 Nutritional therapy
 Goals of nutritional therapy
 maintenance of as near-normal blood glucose levels
 achievement of optimal serum lipid levels
 provision of adequate calories for maintaining or
attaining reasonable weights, normal growth &
development rates
 prevention and treatment of acute complications
 improvement of overall health through optimal nutrition
Diabetes
 Nutritional therapy
 Type I
 based on patient’s usual food intake with insulin
therapy
 eat at consistent times, synchronized with the action of
their insulin
 monitor blood glucose levels and adjust as needed
Diabetes
 Nutritional therapy
 Type II
 achieving glucose, lipid, and blood pressure goals
 weight loss is desirable
 regular exercise
 monitor blood glucose level
Diabetes
 Drug therapy
4 types of insulin; things to consider
 how soon the insulin starts working (onset)
 when it works the hardest (peak time)
 how long it lasts in your body (duration)
Insulin
 Rapid-acting insulin:
 onset: 15 minutes after injection
 peak: 30-90 minutes later
 duration: may last as long as 5 hours
 Short-acting:
 onset: 30 minutes after injection
 peak:2 to 4 hours
 duration: 4 to 8 hours
Insulin
 Intermediate-acting
 onset: 2 to 6 hours
 peak: 4 to 14 hours
 duration: 14 to 20 hours
 Long-acting
 onset: 6 to 14 hours
 peak: 10 to 16 hours
 duration: 20 to 24 hours
Insulin and Oral Agents
 Problems with insulin therapy
 allergic reactions
 lipodystrophy
 Somogyi effect and dawn phenomenon
 Oral medications
 Other drugs affecting blood glucose levels
Nursing Management: Diabetes
 Assessment:
 Subjective data
 past health information
 family history
 medications
 surgery and other treatments
 Health-perception-health management
 + family history, malaise
Nursing Management: Diabetes
 Nutritional-metabolic
 weight
 thirst and hunger
 Nausea and vomiting
 poor healing compliance with diet
 Elimination
 constipation or diarrhea
 frequent urination, incontinence, nocturia
 skin infections
Nursing Management: Diabetes
 Activity-exercise
 muscle weakness, fatigue
 Cognitive-perceptual
 abdominal pain, headache, blurred vision,
numbness or tingling of extremities, pruritis
 Sexuality-reproductive
 impotence, frequent vaginal infections, decreased
libido
Nursing Management: Diabetes
 Coping-stress
 depression
 apathy
 irritability
 Value-belief
 commitment to lifestyle changes involving diet,
medication, and activity patterns
Nursing Management: Diabetes
 Insulin therapy
 assessment of patient’s use of and response to
insulin therapy
 education of the patient regarding administration,
adjustment to, and side effects of insulin
Nursing Management: Diabetes
 Oral agents
 nursing responsibilities similar to those taking
insulin
 Personal hygiene
 dental
 skin care
 Medical identification and travel
 Follow-up nursing management
Methods of Insulin Delivery
 Pens
 Jet injectors
 Insulin pumps—insulin is delivered at .5-2
units/hour. Most common risk of insulin pump
therapy is ketoacidosis.
 Implantable devices
 Transplantation of pancreatic cells
Complications of Diabetes
 Diabetic Ketoacidosis
 Etiology
 undiagnosed diabetes
 inadequate treatment of existing diabetes
 insulin not taken as prescribed
 change in diet, insulin, or exercise regimen
Complications of Diabetes
 Diabetic Ketoacidosis
 Assessment
 dry mouth, thirst, abdominal pain, N & V, confusion,
lethargy, flushed dry skin, eyes appear sunken, breath
odor of ketones, rapid, weak pulse, labored breathing,
fever, urinary frequency, serum glucose > 300 mg/dl,
glucosuria and ketonuria
Complications of Diabetes
 Diabetic Ketoacidosis
 Nursing interventions
 Initial
 ensure patent airway
 O2
 establish IV access and begin fluid resuscitation
begin continuous IV insulin
 identify history of diabetes, time of last food, and
time/amount of last insulin injection
Complications of Diabetes
 Diabetic Ketoacidosis
 Nursing interventions
 ongoing monitoring
 monitor VS, LOC, cardiac rhythm, O2 saturation, and urine
output
 assess breath sounds
 monitor serum glucose and serum potassium
 anticipate possible administration of sodium bicarbonate
with severe acidosis (pH < 7.0)
Complications of Diabetes
 Hyperglycemic Hyperosmolar Nonketosis
 occurs in a patient who has some insulin to
prevent DKA but not enough to prevent severe
hyperglycemia, osmotic diuresis, and extracellular
fluid depletion
 usually is a history of inadequate fluid intake,
increasing mental depression and polyuria
 HHNK constitutes a medical emergency
Complications of Diabetes
 Hyperglycemic Hyperosmolar Nonketosis
 Nursing management
 administration of a rapid-acting insulin
 administration of IV fluid
 assessment of mental status
 I & O
 assessment of blood glucose levels
 assessment of blood and urine for ketones
 electrocardiogram monitoring
Complications of Diabetes
 Hypoglycemia
 clinical manifestations
 blood glucose <50 mg/dl
 cold, clammy skin
 numbness of fingers , toes, mouth
 emotional changes, HA, nervousness, seizures, coma,
faintness, dizziness
 changes in vision
 hunger
 unsteady gait, slurred speech
Complications of Diabetes
 Hypoglycemia
 causes
 alcohol intake with food
 too little food - delayed, omitted, inadequate intake
 diabetic medication or food taken at wrong time
 loss of weight with change of medication
 use of B-blockers
Complications of Diabetes
 Hypoglycemia
 nursing management
 immediate ingestion of 5-20 g of simple carbohydrates
 ingestion of another 5-20 g of simple carbohydrates in
15 min if no relief obtained
 contact physician if no relief obtained
 collaborate with physician
 prevention is the key
Complications of Diabetes
 Hyperglycemia
 clinical manifestations
 elevated blood sugar
 increase urination
 increase in appetite followed by lack of appetite
 weakness, fatigue
 blurred vision, HA
 nausea and vomiting, abdominal cramps
 glycosuria
 progression to DKA or HHNK
Complications of Diabetes
 Hyperglycemia
 causes
 too much food
 too little or no diabetes medication
 inactivity
 emotional, physical stress
 poor absorption of insulin
Complications of Diabetes
 Hyperglycemia
 nursing management
 notify physician
 continuance of diabetes medication as ordered
 frequent checking of blood and urine specimens and
recording of results
 prevention is key
Chronic Complications
 Macroangiopathy
 Microangiopathy
 Peripheral Vascular Disease
 Diabetic Retinopathy
 Nephropathy
 Neuropathy
 Skin changes
Teaching Plan
 Education is critical
 Simple Pathophysiology
 Treatment modalities
 Recognition, treatment and prevention of acute complications
 When to call the doctor
 Foot care, eye care, general hygiene, risk factor management
Teaching patients to
administer insulin
 Storing insulin (may not refrigerate if used within
one month). Prefilled syringes should be stored
standing up.
 Syringes
 Concentrations of insulin
 Mixing insulins
 Do not rotate area to area, use same anatomic area
 No need to aspirate
THANK YOU ! 

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  • 2. Diabetes What is diabetes? What population of patients would be at risk for developing diabetes?
  • 3. Diabetes Mellitus  Commonly referred to as diabetes.  Diabetes “Greek word”- Siphon implies a lot of urine.  Mel “Latin word”- Honey.  Metabolic disorder in which there are High blood sugar levels over a prolonged period.
  • 5. Diabetes  Normal Pathophysiology  Need to consider how insulin works  insulin continuously released: during “fasting periods”, the pancreas continuously releases a small amount of insulin along with glucagon. Together a constant level of glucose in the blood is maintained by stimulating the release of glucose from the liver.
  • 6. Diabetes  In diabetes  the body’s ability to respond to insulin may decrease  the pancreas may stop producing insulin. This in turn leads to hyperglycemia leads to other acute metabolic complications  Diabetes is a heterogeneous group of diseases involving disruption of metabolism of carbohydrates, fats, and protein.
  • 7. Diabetes  Classification of diabetes mellitus Type I  may occur at any age  usually thin  abrupt onset  family history?
  • 8. Diabetes  Type II  >age 30  often obese  few classic symptoms  insulin resistant  Impaired glucose tolerance  plasma glucose levels higher than normal, but not diagnostic for diabetes 2 hr plasma glucose >140 mg/dl & < 200 mg/dl
  • 9. Diabetes  Impaired fasting glucose  fasting plasma glucose > 110 mg/dl & < 126 mg/dl  Gestational diabetes  has onset or discovery of glucose tolerance during pregnancy
  • 10. Diabetes  Clinical Manifestations  Insulin deficiency or decreased insulin activity glucose not used properly  results in frequent urination (polyuria), and thirst (polydipsia)  without insulin the patient may experience hunger (polyphagia)  the body will turn to other energy sources besides glucose: first fat and then protein
  • 11. Diabetes  Diagnostic studies  normal blood glucose range: 70-110 mg/dl  urine tests not sufficient for a dx of diabetes  fasting blood glucose of > 126 mg/dl  glycosylated hemolobin
  • 12. Diabetes  Nutritional therapy  Goals of nutritional therapy  maintenance of as near-normal blood glucose levels  achievement of optimal serum lipid levels  provision of adequate calories for maintaining or attaining reasonable weights, normal growth & development rates  prevention and treatment of acute complications  improvement of overall health through optimal nutrition
  • 13. Diabetes  Nutritional therapy  Type I  based on patient’s usual food intake with insulin therapy  eat at consistent times, synchronized with the action of their insulin  monitor blood glucose levels and adjust as needed
  • 14. Diabetes  Nutritional therapy  Type II  achieving glucose, lipid, and blood pressure goals  weight loss is desirable  regular exercise  monitor blood glucose level
  • 15. Diabetes  Drug therapy 4 types of insulin; things to consider  how soon the insulin starts working (onset)  when it works the hardest (peak time)  how long it lasts in your body (duration)
  • 16. Insulin  Rapid-acting insulin:  onset: 15 minutes after injection  peak: 30-90 minutes later  duration: may last as long as 5 hours  Short-acting:  onset: 30 minutes after injection  peak:2 to 4 hours  duration: 4 to 8 hours
  • 17. Insulin  Intermediate-acting  onset: 2 to 6 hours  peak: 4 to 14 hours  duration: 14 to 20 hours  Long-acting  onset: 6 to 14 hours  peak: 10 to 16 hours  duration: 20 to 24 hours
  • 18. Insulin and Oral Agents  Problems with insulin therapy  allergic reactions  lipodystrophy  Somogyi effect and dawn phenomenon  Oral medications  Other drugs affecting blood glucose levels
  • 19. Nursing Management: Diabetes  Assessment:  Subjective data  past health information  family history  medications  surgery and other treatments  Health-perception-health management  + family history, malaise
  • 20. Nursing Management: Diabetes  Nutritional-metabolic  weight  thirst and hunger  Nausea and vomiting  poor healing compliance with diet  Elimination  constipation or diarrhea  frequent urination, incontinence, nocturia  skin infections
  • 21. Nursing Management: Diabetes  Activity-exercise  muscle weakness, fatigue  Cognitive-perceptual  abdominal pain, headache, blurred vision, numbness or tingling of extremities, pruritis  Sexuality-reproductive  impotence, frequent vaginal infections, decreased libido
  • 22. Nursing Management: Diabetes  Coping-stress  depression  apathy  irritability  Value-belief  commitment to lifestyle changes involving diet, medication, and activity patterns
  • 23. Nursing Management: Diabetes  Insulin therapy  assessment of patient’s use of and response to insulin therapy  education of the patient regarding administration, adjustment to, and side effects of insulin
  • 24. Nursing Management: Diabetes  Oral agents  nursing responsibilities similar to those taking insulin  Personal hygiene  dental  skin care  Medical identification and travel  Follow-up nursing management
  • 25. Methods of Insulin Delivery  Pens  Jet injectors  Insulin pumps—insulin is delivered at .5-2 units/hour. Most common risk of insulin pump therapy is ketoacidosis.  Implantable devices  Transplantation of pancreatic cells
  • 26. Complications of Diabetes  Diabetic Ketoacidosis  Etiology  undiagnosed diabetes  inadequate treatment of existing diabetes  insulin not taken as prescribed  change in diet, insulin, or exercise regimen
  • 27. Complications of Diabetes  Diabetic Ketoacidosis  Assessment  dry mouth, thirst, abdominal pain, N & V, confusion, lethargy, flushed dry skin, eyes appear sunken, breath odor of ketones, rapid, weak pulse, labored breathing, fever, urinary frequency, serum glucose > 300 mg/dl, glucosuria and ketonuria
  • 28. Complications of Diabetes  Diabetic Ketoacidosis  Nursing interventions  Initial  ensure patent airway  O2  establish IV access and begin fluid resuscitation begin continuous IV insulin  identify history of diabetes, time of last food, and time/amount of last insulin injection
  • 29. Complications of Diabetes  Diabetic Ketoacidosis  Nursing interventions  ongoing monitoring  monitor VS, LOC, cardiac rhythm, O2 saturation, and urine output  assess breath sounds  monitor serum glucose and serum potassium  anticipate possible administration of sodium bicarbonate with severe acidosis (pH < 7.0)
  • 30. Complications of Diabetes  Hyperglycemic Hyperosmolar Nonketosis  occurs in a patient who has some insulin to prevent DKA but not enough to prevent severe hyperglycemia, osmotic diuresis, and extracellular fluid depletion  usually is a history of inadequate fluid intake, increasing mental depression and polyuria  HHNK constitutes a medical emergency
  • 31. Complications of Diabetes  Hyperglycemic Hyperosmolar Nonketosis  Nursing management  administration of a rapid-acting insulin  administration of IV fluid  assessment of mental status  I & O  assessment of blood glucose levels  assessment of blood and urine for ketones  electrocardiogram monitoring
  • 32. Complications of Diabetes  Hypoglycemia  clinical manifestations  blood glucose <50 mg/dl  cold, clammy skin  numbness of fingers , toes, mouth  emotional changes, HA, nervousness, seizures, coma, faintness, dizziness  changes in vision  hunger  unsteady gait, slurred speech
  • 33. Complications of Diabetes  Hypoglycemia  causes  alcohol intake with food  too little food - delayed, omitted, inadequate intake  diabetic medication or food taken at wrong time  loss of weight with change of medication  use of B-blockers
  • 34. Complications of Diabetes  Hypoglycemia  nursing management  immediate ingestion of 5-20 g of simple carbohydrates  ingestion of another 5-20 g of simple carbohydrates in 15 min if no relief obtained  contact physician if no relief obtained  collaborate with physician  prevention is the key
  • 35. Complications of Diabetes  Hyperglycemia  clinical manifestations  elevated blood sugar  increase urination  increase in appetite followed by lack of appetite  weakness, fatigue  blurred vision, HA  nausea and vomiting, abdominal cramps  glycosuria  progression to DKA or HHNK
  • 36. Complications of Diabetes  Hyperglycemia  causes  too much food  too little or no diabetes medication  inactivity  emotional, physical stress  poor absorption of insulin
  • 37. Complications of Diabetes  Hyperglycemia  nursing management  notify physician  continuance of diabetes medication as ordered  frequent checking of blood and urine specimens and recording of results  prevention is key
  • 38. Chronic Complications  Macroangiopathy  Microangiopathy  Peripheral Vascular Disease  Diabetic Retinopathy  Nephropathy  Neuropathy  Skin changes
  • 39. Teaching Plan  Education is critical  Simple Pathophysiology  Treatment modalities  Recognition, treatment and prevention of acute complications  When to call the doctor  Foot care, eye care, general hygiene, risk factor management
  • 40. Teaching patients to administer insulin  Storing insulin (may not refrigerate if used within one month). Prefilled syringes should be stored standing up.  Syringes  Concentrations of insulin  Mixing insulins  Do not rotate area to area, use same anatomic area  No need to aspirate
  • 41. THANK YOU !