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CARDIOLOGY BASICS:
AT THE HEART OF THE
MATTER
Jason M . Chin, DVM
Oradell Animal Hospital
Why is the heart so important?
 It pumps blood to organs to provide:
 Oxygen
 Nutrients
 Removes waste (biproducts of metabolism)
 Provides means for signaling/communication
Perfusion
 Determined by Cardiac Output:
CO = SV x HR
SV = Preload x Contractility
Afterload
 Why are these important to think about?
A & P
A & P
A & P – Electrical Conduction
 Basic Pathway: SA node to AV node to Bundle of
His/Purkinje fibers (ventricles)
A & P – Electrical Conduction
 Systole: The ‘active’, contraction phase of the
heart cycle
 Diastole: The ‘relaxed’, filling phase of the
heart cycle
A & P – Electrical Conduction
The Physical Exam
 History: As important as any physical parameter
 Syncope
 Exercise intolerance
 Difficulty/Labored breathing
 Weight loss (cachexia)
 Observation: What does your patient look like?
 Dyspnea
 Coughing
 Weak
 Abduction of elbows/Extended head
 Cheek puffing/Open mouth breathing
Dyspnea
http://www.youtube.com/watch?v=aqWiWlKv7OE
Dyspnea
http://www.youtube.com/watch?v=Zp7CiC7SXjk
PE – Vital Parameters
 http://www.cvmbs.colostate.edu/clinsci/wing/fir
staid/vital.htm
Dog Cat
Heart Rate 70-160 bpm 160-240 bpm
Respiratory Rate 10-30 bpm 20-30 bpm
Temperature 100.0 – 102.5 F 100.0 – 102.5 F
PE – Vital Parameters
 Pulses
 Strong and Synchronous
 Mucus Membranes
 Pink and Moist
 CRT < 2s
PE - Auscultation
 Cardiac:
 Murmurs
 Arrhythmias
 Pulmonary:
 Crackles (rales)
 Wheezes (rhonchi)
 r/o Upper Airway (referred)
Murmurs
 How do you describe a murmur?
 Intensity (Grade 1-6)
 Location (PMI – point of maximal intensity;
right/left/sternal, apical/basilar)
 Timing (Systolic v. Diastolic v. Continuous)
 Pitch/Frequency (high/med/low, musical/harsh)
 Shape (plateau v. decrescendo v. crescendo-
decrescendo)
Murmurs
 Grading System: Freeman and Levine
 I/VI – Very focal; faint
 II/VI – Faint, but more easily heard than a grade I
 III/VI – Moderately loud and easily heard (over
larger area on same side)
 IV/VI – Loud murmur heard over large area
including the opposite side (discern
between 2 murmurs)
 V/VI – Palpable thrill over PMI
 VI/VI – Audible with stethoscope off of body wall
Murmurs
 http://www.littmann.com/wps/portal/3M/en_US/3M-
Littmann/stethoscope/littmann-learning-institute/heart-
lung-
sounds/?mmmSsoError=1010|Invalid%20Username%2
0and%20Password%20combination.
 Early Systolic
 Pansystolic
Murmurs
 Some Causes…
 Valve insufficiency/regurgitation (Chronic Valve
Disease)
 Valve stenosis
 Septal defects
 Anemia (physiologic murmur)
Lung Sounds
Location of sounds is important to note…
 Normal bronchovesicular sounds
 Crackles (rales)
 Wheezes (rhonchi)
 r/o stridor & stertor (upper airway)
Arrhythmias
 Causes of arrhythmias:
 Primary conduction disturbance
 SA Node, AV Node, fibers (blocks)
 Secondary to heart disease
 Enlarged heart (dilated, thicker, etc)
 Secondary to systemic illness
 Endocarditis, GDV, splenic disease, hyperthyroid,
pheochromocytoma, anemia
 Secondary to electrolyte imbalances
 Potassium, Calcium, Sodium
 Secondary to toxin/drug ingestion
Arrhythmias
 Sinus rhythm (normal)
 Respiratory sinus arrhythmia (normal)
 Faster on inspiration – vagal tone is inhibited
 Tachyarrhythmia
 SVT, V-Tach/AIVR, A-Fib
 Bradyarrhythmia
 AV Blocks, Sick Sinus Syndrome, SA disease, escape
rhythms
 Premature Beats
 APC v. VPC
 Gallop Rhythm
Arrhythmias
 Normal ECG
Arrhythmias
 VPC’s, (Ventricular Premature Contractions)
Arrythmias
 V-Tach (Ventricular Tachycardia)
Arrhythmias
 APC’s (Atrial Premature Contractions)
Arrhythmias
 AV Block (1st Degree & High Grade 2nd
Degree)
Arrhythmias
 A-Fib (Atrial Fibrillation)
Gallop Rhythm
 Gallop (S3 sound)
Types of Cardiac Disease
 Congential Defects (ASD/VSD, PDA, Tetrology
of Fallot)
 Endocardiosis/Chronic Valve Disease
(insufficiency)
 Valvular Stenosis (congenital v. acquired)
 Dilated Cardiomyopathy (dogs)
 Hypertrophic Cardiomyopathy (cats)
 Pulmonary Hypertension
 Systemic Hypertension (primary v. secondary)
Pathophysiology of Disease
 Abnormal blood flow (can be d/t arrhythmia,
cardiomyopathy, etc)  decreased perfusion
 Stimulates RAAS (Renin-Angiotensin-
Aldosterone System)
 Hormones increase preload, vascular tone,
and affect heart myocytes
 Structural changes ensue:
 Eccentric hypertrophy
 Concentric hypertrophy
 Chamber dilation
Pathophysiology of Disease
Congestive Heart Failure (CHF)
 Left sided
 Dyspnea
 Pulmonary Edema
 Nasal fluid/discharge
 Right sided
 Dyspnea
 Ascites
 Jugular Pulses
CHF
 Chest rads (needed for diagnosis)
CHF
CHF
 Treatment:
 Oxygen Therapy
 Lasix (furosemide)
 2mg/kg IM or IV (dog)
 1mg/kg IM or IV (cat)
 Nitroprusside CRI
 Sedation (Torb – beware Ace and Dexdomitor)
Atrial Thromboembolism
 aka “Saddle Thrombus” - Cats!!
 Gallop rhythm, often underlying heart disease
like HCM/RCM/HOCM
 Due to hypercoagulable blood in chambers
(pooling)
 Hind end paralysis (bilateral > unilateral),
usually deep pain negative
 Cold limbs, cyanotic pads
 Painful!!
ATE
 Negative Prognostic Indicators:
 Decreased Temperature, HR
 Bilateral (versus unilateral)
 In CHF
 Treatment:
 Pain management (hydromorphone, fentanyl)
 Oxygen therapy
 Lasix (if in CHF – 1mg/kg)
 +/- Thrombolytic therapy (aspirin, clopidogrel,
heparin)
What does it all mean??
 Do we have lung disease or is it something
else?
 Primary v. secondary diseases
(asthma/pneumonia v. non-cardiogenic pulm
edema/pleural effusion/pericardial effusion v.
CHF)
 Is cardiac disease the underlying cause?
 What diagnostics and treatments are
“We’ve got a code 1 trouble
breathing…”
References
Braunwald, E. Heart Disease: A Textbook of
Cardiovascular Medicine – 5th Ed. Philadelphia:
W.B. Saunders Co., 1997
Fox, Sisson, Moise. Textbook of Canine and Feline
Cardiology: Principles and Clinical Practice – 2nd
Ed. Philadelphia: W.B. Saunders Co., 1999.
Drobatz, J.K. Emergency Management of
Respiratory Distress. In: Proceedings of the
District of Columbia Academy of Veterinary
Medicine; 2004.
Questions?

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Cardiology Basics

  • 1. CARDIOLOGY BASICS: AT THE HEART OF THE MATTER Jason M . Chin, DVM Oradell Animal Hospital
  • 2. Why is the heart so important?  It pumps blood to organs to provide:  Oxygen  Nutrients  Removes waste (biproducts of metabolism)  Provides means for signaling/communication
  • 3. Perfusion  Determined by Cardiac Output: CO = SV x HR SV = Preload x Contractility Afterload  Why are these important to think about?
  • 6. A & P – Electrical Conduction  Basic Pathway: SA node to AV node to Bundle of His/Purkinje fibers (ventricles)
  • 7. A & P – Electrical Conduction  Systole: The ‘active’, contraction phase of the heart cycle  Diastole: The ‘relaxed’, filling phase of the heart cycle
  • 8. A & P – Electrical Conduction
  • 9. The Physical Exam  History: As important as any physical parameter  Syncope  Exercise intolerance  Difficulty/Labored breathing  Weight loss (cachexia)  Observation: What does your patient look like?  Dyspnea  Coughing  Weak  Abduction of elbows/Extended head  Cheek puffing/Open mouth breathing
  • 12. PE – Vital Parameters  http://www.cvmbs.colostate.edu/clinsci/wing/fir staid/vital.htm Dog Cat Heart Rate 70-160 bpm 160-240 bpm Respiratory Rate 10-30 bpm 20-30 bpm Temperature 100.0 – 102.5 F 100.0 – 102.5 F
  • 13. PE – Vital Parameters  Pulses  Strong and Synchronous  Mucus Membranes  Pink and Moist  CRT < 2s
  • 14. PE - Auscultation  Cardiac:  Murmurs  Arrhythmias  Pulmonary:  Crackles (rales)  Wheezes (rhonchi)  r/o Upper Airway (referred)
  • 15. Murmurs  How do you describe a murmur?  Intensity (Grade 1-6)  Location (PMI – point of maximal intensity; right/left/sternal, apical/basilar)  Timing (Systolic v. Diastolic v. Continuous)  Pitch/Frequency (high/med/low, musical/harsh)  Shape (plateau v. decrescendo v. crescendo- decrescendo)
  • 16. Murmurs  Grading System: Freeman and Levine  I/VI – Very focal; faint  II/VI – Faint, but more easily heard than a grade I  III/VI – Moderately loud and easily heard (over larger area on same side)  IV/VI – Loud murmur heard over large area including the opposite side (discern between 2 murmurs)  V/VI – Palpable thrill over PMI  VI/VI – Audible with stethoscope off of body wall
  • 18. Murmurs  Some Causes…  Valve insufficiency/regurgitation (Chronic Valve Disease)  Valve stenosis  Septal defects  Anemia (physiologic murmur)
  • 19. Lung Sounds Location of sounds is important to note…  Normal bronchovesicular sounds  Crackles (rales)  Wheezes (rhonchi)  r/o stridor & stertor (upper airway)
  • 20. Arrhythmias  Causes of arrhythmias:  Primary conduction disturbance  SA Node, AV Node, fibers (blocks)  Secondary to heart disease  Enlarged heart (dilated, thicker, etc)  Secondary to systemic illness  Endocarditis, GDV, splenic disease, hyperthyroid, pheochromocytoma, anemia  Secondary to electrolyte imbalances  Potassium, Calcium, Sodium  Secondary to toxin/drug ingestion
  • 21. Arrhythmias  Sinus rhythm (normal)  Respiratory sinus arrhythmia (normal)  Faster on inspiration – vagal tone is inhibited  Tachyarrhythmia  SVT, V-Tach/AIVR, A-Fib  Bradyarrhythmia  AV Blocks, Sick Sinus Syndrome, SA disease, escape rhythms  Premature Beats  APC v. VPC  Gallop Rhythm
  • 23. Arrhythmias  VPC’s, (Ventricular Premature Contractions)
  • 25. Arrhythmias  APC’s (Atrial Premature Contractions)
  • 26. Arrhythmias  AV Block (1st Degree & High Grade 2nd Degree)
  • 29. Types of Cardiac Disease  Congential Defects (ASD/VSD, PDA, Tetrology of Fallot)  Endocardiosis/Chronic Valve Disease (insufficiency)  Valvular Stenosis (congenital v. acquired)  Dilated Cardiomyopathy (dogs)  Hypertrophic Cardiomyopathy (cats)  Pulmonary Hypertension  Systemic Hypertension (primary v. secondary)
  • 30. Pathophysiology of Disease  Abnormal blood flow (can be d/t arrhythmia, cardiomyopathy, etc)  decreased perfusion  Stimulates RAAS (Renin-Angiotensin- Aldosterone System)  Hormones increase preload, vascular tone, and affect heart myocytes  Structural changes ensue:  Eccentric hypertrophy  Concentric hypertrophy  Chamber dilation
  • 32. Congestive Heart Failure (CHF)  Left sided  Dyspnea  Pulmonary Edema  Nasal fluid/discharge  Right sided  Dyspnea  Ascites  Jugular Pulses
  • 33. CHF  Chest rads (needed for diagnosis)
  • 34. CHF
  • 35. CHF  Treatment:  Oxygen Therapy  Lasix (furosemide)  2mg/kg IM or IV (dog)  1mg/kg IM or IV (cat)  Nitroprusside CRI  Sedation (Torb – beware Ace and Dexdomitor)
  • 36. Atrial Thromboembolism  aka “Saddle Thrombus” - Cats!!  Gallop rhythm, often underlying heart disease like HCM/RCM/HOCM  Due to hypercoagulable blood in chambers (pooling)  Hind end paralysis (bilateral > unilateral), usually deep pain negative  Cold limbs, cyanotic pads  Painful!!
  • 37. ATE  Negative Prognostic Indicators:  Decreased Temperature, HR  Bilateral (versus unilateral)  In CHF  Treatment:  Pain management (hydromorphone, fentanyl)  Oxygen therapy  Lasix (if in CHF – 1mg/kg)  +/- Thrombolytic therapy (aspirin, clopidogrel, heparin)
  • 38. What does it all mean??  Do we have lung disease or is it something else?  Primary v. secondary diseases (asthma/pneumonia v. non-cardiogenic pulm edema/pleural effusion/pericardial effusion v. CHF)  Is cardiac disease the underlying cause?  What diagnostics and treatments are
  • 39. “We’ve got a code 1 trouble breathing…”
  • 40. References Braunwald, E. Heart Disease: A Textbook of Cardiovascular Medicine – 5th Ed. Philadelphia: W.B. Saunders Co., 1997 Fox, Sisson, Moise. Textbook of Canine and Feline Cardiology: Principles and Clinical Practice – 2nd Ed. Philadelphia: W.B. Saunders Co., 1999. Drobatz, J.K. Emergency Management of Respiratory Distress. In: Proceedings of the District of Columbia Academy of Veterinary Medicine; 2004.