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BURN
Department of Surgery
ELWA Hospital
Dr. J. Nutai Kolleh, Intern
Outline
• Overview
• Classification of Burns
• Burn Depth
• Prognosis
• Resuscitation
• Transfusion
• Inhalation Injury and Ventilator Management
• Treatment of the Burn Wound
• Nutrition
• Complications in Burn Care
• Rehabilitation
• Prevention
• Radiation Burns
Overview
Burn refer to a wound that result from tissue
damage by caused by
• Thermal
• Electrical
• Chemical
• Radiation.
4
Skin Anatomy
• Skin Layers
• Epidermis
• Dermis
• Subcutaneous tissue
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5
Skin Form & Function
• Largest body organ
• More than just a passive covering
• Functions:
• Sensation
• Protection
• Temperature regulation
• Fluid retention
• Metabolic (vit D synthesis and excretory function)
• Social( self-image, social image)
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Initial Evaluation
Initial evaluation of the burned patient
involves four crucial assessments:
•airway management
•evaluation of other injuries
•estimation of burn size
•and diagnosis of CO and cyanide
poisoning.
Initial Evaluation
• Perioral burns and singed nasal hairs necessitate
further evaluated for mucosal injury,
• Signs of impending respiratory compromise may
include a hoarse voice, wheezing, or stridor;
• subjective dyspnea is a particularly concerning
symptom and should trigger prompt elective
endotracheal intubation.
Initial Evaluation
• Burned patients should be first considered trauma
patients, especially when details of the injury are
unclear.
• Primary survey
• An early and comprehensive secondary survey must
be performed on all burn patients, but especially
those with a history of associated trauma such as
with a motor vehicle collision
Estimating Burn Size
• Superficial or first-degree burns should not be
included when calculating the %TBSA, and
thorough cleaning of soot and debris is mandatory
to avoid confusing
• Wallace rule of nine
• Laud Browder
• Rule of the Palm
Metabolic Poisoning
• Carbon Monoxide (CO) affinity for hemoglobin is
approximately 200 – 250 times more than oxygen
• Administration of 100% oxygen is the gold standard
for treatment of CO poisoning
• Hydrogen cyanide toxicity patients may have a
persistent lactic acidosis or ST elevation on
electrocardiogram (ECG).
• Treatment consists of sodium thiosulfate,
hydroxocobalamin, and 100% oxygen
Classification of Burn
• Burns are commonly classified as thermal,
electrical, or chemical burns, with thermal
burns consisting of flame, contact, or scald
burns.
• Flame burns are not only the most common
cause but also the highest cause of mortality
Electrical Burn
• Electrical burns are classified as low-voltage ( < 1000
volts) and high-voltage injuries (1000 volts and higher).
• There may be instant death from cardiac or respiratory
arrest or ventricular fibrillation from alteration of the
action potentials of nerves and muscles.
• Cardiac arrhythmia
• Compartment syndrome
• Rhabdomyolysis
• fasciotomies should be performed even in cases of
moderate clinical suspicion
Chemical Burn
• Chemical burns are less common but potentially severe
burns.
• Careful removal and irrigation for minimum of 30mins
• Lye powder should be swept away to avoid activating
the aluminum hydroxide with water
• Formic acid has been known to cause hemolysis and
hemoglobinuria, and hydrofluoric acid causes
hypocalcemia.
• Calcium-based therapies are the mainstay of treating
hydrofluoric acid burns
Burn Depth
• Superficial (First degree) – Involves only
epidermis, Red, Heal in ~7 days
• Partial Thickness (Second degree) – Extends into
the dermis, Salmon pink Heal in ~7 to 21 days
• Full Thickness (Third degree) Through epidermis,
dermis into underlying structures Thick, dry, Pearly
gray or charred black,
• Fourth degree burn – Involves underlying
structures , same finding as 3rd degree burn with
involved bone, muscle and tendon.
Burn Depth
• Zone of coagulation – of necrosis in the center is
irreversibly damaged.
• ischemia or stasis where vascular spasms and
intravascular micro-thrombi result in compromised
perfusion with potential to progressive conversion
to tissue death.
• Zone of hyperemia, heal with minimal or no
scarring and is most like a superficial or first-degree
burn.
Prognosis
• The Baux score (mortality risk equals age plus
%TBSA) was used for many years to predict
mortality in burns.
• Analysis of multiple risk factors for burn mortality
has validated age and %TBSA as the strongest
predictors of mortality.
• However, age and burn size, as well as inhalation
injury, continue to be the most robust indicators for
burn mortality
Resuscitation
Google Images
Resuscitation
• For pediatrics, the simplest approach is to deliver a
weight-based maintenance IV fluid with glucose
supplementation in addition to the calculated
resuscitation fluid with lactated Ringer’s.
• The burn (and/or inhalation injury) drives an
inflammatory response that leads to capillary leak.
• Continuation of fluid volumes should depend on the
time since injury, urine output, and mean arterial
pressure (MAP).
Resuscitation
Other adjuncts
• High-dose ascorbic acid (vitamin C) may decrease
fluid volume requirements and ameliorate
respiratory embarrassment during resuscitation.
• Plasmapheresis may also decrease fluid
requirements in patients who require higher
volumes than predicted to maintain adequate urine
output and MAP
Transfusion
• A large multicenter study of blood
transfusions in burn patients found that
increased numbers of transfusions were
associated with increased infections and
higher mortality in burn patients, even when
correcting for burn severity.
Inhalation Injury and
Ventilator Management
• Inhalation injury decreases lung compliance and
increases airway resistance work of breathing
• Treatment of inhalation injury consists primarily of
supportive care.
• Aggressive pulmonary toilet and routine use of
nebulized bronchodilators such as albuterol are
recommended
• Inhaled nitric oxide may also be useful as a last
effort in burn patients with severe lung injury who
are failing other means of ventilatory support.
A classic study by Navar et al :
• burned patients with inhalation injury
required an average of 5.76 mL/kg/% burn,
vs. 3.98 mL/kg/% burn for patients without
inhalation injury, and this has been
corroborated by subsequent studies
Treatment of Burn Wound
Exposure method
• Allow daily inspection and any cracks in the eschar are
dressed with Vaseline gauzeor or sofra tulle
• In full thickness burns, exposure end as soon as the
eschar is broken
Dressing
• Occlusive dressing prevents bacterial contamination,
reduces pain and evaporative fluid loss and increases
the rate of re-epitheliazation.
• Providone-iodine: effective against gram positive
organisms and fungi. It is painful and causes excessive
drying of eschar.
• Honey: Honey prevent bacterial contamination and
promote healing
Google Images
Treatment of Burn Wound
• Early excision and skin grafting - Classically between
3-7 days
• Larger burn – meshed autografted skin
• Areas of cosmetic importance – use nonmeshed sheet
grafts ensure optimal results
• For excision of burns in areas such as the face, eyelids,
or hands, a pressurized water dissector may offer more
precision but is time consuming.
• Xenograft and cadaveric graft function as well as
allograft for temporary wound coverage.
• Amniotic Membrane
Burn in Specific Areas
Face
• Adequate cleaning of the face, exposure dressing with
antibacterial lotions/ creams.
Neck:
• Exposure treatment is ideal
• Flexion contracture – keep the neck extended by placing
a pillow under the shoulders.
• soft and hard neck collars and fabricated splints can be
use later
• Circumferential burn can form a tourniquet effect as the
edema progresses.
• Escharotomies are rarely needed within the first 8 hours
following injury and should not be performed unless
indicated
Burn in Specific Areas
• Axillae:
• Keep the arm in abduction.
• bulky axillary dressings are useful.
• Negative pressure wound therapy (NPWT), may benefit
patient requiring excision and grafting.
• Crucifix splint may be used in children
The Hands:
• dressed the fingers and the web spaces separately with
Vaseline gauze.
• Splint wrist in extension, Metacarpophalangeal joint and
90 degree flexion, interphalangeal joint in extension
Burn in Specific Areas
Perinerum
• Treated by exposure
• In children gallows splint ensures adequate
immobilization and exposure.
• Adults are nursed in supine position with the hip
joints in abduction.
The legs and feet: These must be elevated to
encourage venous drainage.
Nutrition
• Nutritional support may be more important in
patients with large burns than in any other patient
population
• Adjuncts such as metoclopramide promote
gastrointestinal motility.
• if other measures for gastric feeding are
unsuccessful, advancing the tube into the small
bowel with nasojejunal feeding can be attempted.
Nutrition
• Harris-Benedict equation, is a commonly used
formula in nonburned population.
• The Harris-Benedict equation may be inaccurate in
burns of less than 40% TBSA, and in these patients,
the Curreri formula may be more appropriate.
• This formula estimates caloric needs to be 25
kcal/kg/d plus 40 kcal/%TBSA/d.
Nutrition
• β-Blocker use in pediatric patients decreases heart rate
and resting energy expenditure
• There may be benefits to β-blockade in adult patients.
• Insulin may have a metabolic benefit in burn patients
• Oral hypoglycemic and prevent hyperglycemia and
prevent muscle catabolism.
• A recent double-blind, randomized study of
oxandrolone showed decreased length of stay, improved
hepatic protein synthesis, and no adverse effects on
endocrine function,
Causes of death in burn patients
• Hypovolaemic shock in the
first 48 hours.
Renal failure
• (i) Pre-renal from
hypovolaemia .
• (ii) Acute tubular necrosis from
myogtobinuria or
hemoglobinuria
• Sepsis may be a local infection
or septicemia often with
endotoxic shock.
• Tetanus may also occur.
Respiratory tract infection
accounts for 50 % of bum
sepsis and 50% of deaths.
• Pulmonary problems: Laryngo-
tracheo-bronchitis, laryngeal
oedema, pulmonary oedema,
pneumonia
• Some studies have reported an
incidence of over 50% of multi-
Organ failure in deaths after bum
injury.
Complications
• Early complications and
include;
1. HypovolaemicShock
Infections
• 3. Gastrointestinal problems
• Acute gastric dilatation,
Paralytic ileus, Curling's
ulcers, Liver damage
• 4. Cardio-respiration problems
• Respiratory obstruction
• Tracheo-bronchitis
• Pneumonia, atelectasis
• 5. Genito-urinary problems
• Renal failure
• Cystitis, pyelonephritis,
Calculi
• 6. Vascularproblems
• Thrombophlebitis,
• Deep venous thrombosis
• Pulmonary embolism
• 7. Anemia
• Disseminated intravascular
coagulation (DIC)
Complications
• Late complications of burns are usually related to
consequences of poor healing of the burn wound and
include
• Unstable scars and chronic burn wounds.
• Burn scar hypertrophy and Keloids
• Burn scar contractures and deformities
• Dyschromic scars (hypo-, hyper-, & de-pigmentation)
• Bum Scar metaplasia and Marjolin's ulcers
• Loss of body parts.
• Psychiatric problems including PTSD
Rehabilitation
• Patients should be taught exercises they can do
themselves to maintain full range of motion.
• Psychological rehabilitation is equally important in
the burn patient.
• Depression, posttraumatic stress disorder, concerns
about image, and anxiety are essential.
Radiation
• Radioactive material results in both acute injury from
immediate exposure and more prolonged injury from
delayed exposure to radioactive fallout or
contamination.
• The explosion results in a direct pressure wave and an
indirect wind drag
• After initial evaluation and decontamination by
removing clothing, a useful way to estimate exposure is
by determining the time to emesis.
• Patients who do not experience emesis within 4 hours of
exposure are unlikely to have severe clinical effects.
• Irrigation fluid should be collected to prevent
radiation spread into the water supply.
Prevention
•Burns is preventable, the common
denominator in most instances being
ignorance, carelessness and poverty
•Smoke alarms are known to decrease
mortality from structural fires.
References
• Schwartz's Principle of Surgery, 10th edition
• Baja’s Principles and Practice of Surgery, 5th
edition

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BURN

  • 1. BURN Department of Surgery ELWA Hospital Dr. J. Nutai Kolleh, Intern
  • 2. Outline • Overview • Classification of Burns • Burn Depth • Prognosis • Resuscitation • Transfusion • Inhalation Injury and Ventilator Management • Treatment of the Burn Wound • Nutrition • Complications in Burn Care • Rehabilitation • Prevention • Radiation Burns
  • 3. Overview Burn refer to a wound that result from tissue damage by caused by • Thermal • Electrical • Chemical • Radiation.
  • 4. 4 Skin Anatomy • Skin Layers • Epidermis • Dermis • Subcutaneous tissue https://slideplayer.com/slide/ 5754434/
  • 5. 5 Skin Form & Function • Largest body organ • More than just a passive covering • Functions: • Sensation • Protection • Temperature regulation • Fluid retention • Metabolic (vit D synthesis and excretory function) • Social( self-image, social image) https://slideplayer.com/slide/ 5754434/
  • 6. Initial Evaluation Initial evaluation of the burned patient involves four crucial assessments: •airway management •evaluation of other injuries •estimation of burn size •and diagnosis of CO and cyanide poisoning.
  • 7. Initial Evaluation • Perioral burns and singed nasal hairs necessitate further evaluated for mucosal injury, • Signs of impending respiratory compromise may include a hoarse voice, wheezing, or stridor; • subjective dyspnea is a particularly concerning symptom and should trigger prompt elective endotracheal intubation.
  • 8. Initial Evaluation • Burned patients should be first considered trauma patients, especially when details of the injury are unclear. • Primary survey • An early and comprehensive secondary survey must be performed on all burn patients, but especially those with a history of associated trauma such as with a motor vehicle collision
  • 9. Estimating Burn Size • Superficial or first-degree burns should not be included when calculating the %TBSA, and thorough cleaning of soot and debris is mandatory to avoid confusing • Wallace rule of nine • Laud Browder • Rule of the Palm
  • 10.
  • 11. Metabolic Poisoning • Carbon Monoxide (CO) affinity for hemoglobin is approximately 200 – 250 times more than oxygen • Administration of 100% oxygen is the gold standard for treatment of CO poisoning • Hydrogen cyanide toxicity patients may have a persistent lactic acidosis or ST elevation on electrocardiogram (ECG). • Treatment consists of sodium thiosulfate, hydroxocobalamin, and 100% oxygen
  • 12. Classification of Burn • Burns are commonly classified as thermal, electrical, or chemical burns, with thermal burns consisting of flame, contact, or scald burns. • Flame burns are not only the most common cause but also the highest cause of mortality
  • 13. Electrical Burn • Electrical burns are classified as low-voltage ( < 1000 volts) and high-voltage injuries (1000 volts and higher). • There may be instant death from cardiac or respiratory arrest or ventricular fibrillation from alteration of the action potentials of nerves and muscles. • Cardiac arrhythmia • Compartment syndrome • Rhabdomyolysis • fasciotomies should be performed even in cases of moderate clinical suspicion
  • 14. Chemical Burn • Chemical burns are less common but potentially severe burns. • Careful removal and irrigation for minimum of 30mins • Lye powder should be swept away to avoid activating the aluminum hydroxide with water • Formic acid has been known to cause hemolysis and hemoglobinuria, and hydrofluoric acid causes hypocalcemia. • Calcium-based therapies are the mainstay of treating hydrofluoric acid burns
  • 15. Burn Depth • Superficial (First degree) – Involves only epidermis, Red, Heal in ~7 days • Partial Thickness (Second degree) – Extends into the dermis, Salmon pink Heal in ~7 to 21 days • Full Thickness (Third degree) Through epidermis, dermis into underlying structures Thick, dry, Pearly gray or charred black, • Fourth degree burn – Involves underlying structures , same finding as 3rd degree burn with involved bone, muscle and tendon.
  • 16. Burn Depth • Zone of coagulation – of necrosis in the center is irreversibly damaged. • ischemia or stasis where vascular spasms and intravascular micro-thrombi result in compromised perfusion with potential to progressive conversion to tissue death. • Zone of hyperemia, heal with minimal or no scarring and is most like a superficial or first-degree burn.
  • 17. Prognosis • The Baux score (mortality risk equals age plus %TBSA) was used for many years to predict mortality in burns. • Analysis of multiple risk factors for burn mortality has validated age and %TBSA as the strongest predictors of mortality. • However, age and burn size, as well as inhalation injury, continue to be the most robust indicators for burn mortality
  • 19. Resuscitation • For pediatrics, the simplest approach is to deliver a weight-based maintenance IV fluid with glucose supplementation in addition to the calculated resuscitation fluid with lactated Ringer’s. • The burn (and/or inhalation injury) drives an inflammatory response that leads to capillary leak. • Continuation of fluid volumes should depend on the time since injury, urine output, and mean arterial pressure (MAP).
  • 20. Resuscitation Other adjuncts • High-dose ascorbic acid (vitamin C) may decrease fluid volume requirements and ameliorate respiratory embarrassment during resuscitation. • Plasmapheresis may also decrease fluid requirements in patients who require higher volumes than predicted to maintain adequate urine output and MAP
  • 21. Transfusion • A large multicenter study of blood transfusions in burn patients found that increased numbers of transfusions were associated with increased infections and higher mortality in burn patients, even when correcting for burn severity.
  • 22. Inhalation Injury and Ventilator Management • Inhalation injury decreases lung compliance and increases airway resistance work of breathing • Treatment of inhalation injury consists primarily of supportive care. • Aggressive pulmonary toilet and routine use of nebulized bronchodilators such as albuterol are recommended • Inhaled nitric oxide may also be useful as a last effort in burn patients with severe lung injury who are failing other means of ventilatory support.
  • 23. A classic study by Navar et al : • burned patients with inhalation injury required an average of 5.76 mL/kg/% burn, vs. 3.98 mL/kg/% burn for patients without inhalation injury, and this has been corroborated by subsequent studies
  • 24. Treatment of Burn Wound Exposure method • Allow daily inspection and any cracks in the eschar are dressed with Vaseline gauzeor or sofra tulle • In full thickness burns, exposure end as soon as the eschar is broken Dressing • Occlusive dressing prevents bacterial contamination, reduces pain and evaporative fluid loss and increases the rate of re-epitheliazation. • Providone-iodine: effective against gram positive organisms and fungi. It is painful and causes excessive drying of eschar. • Honey: Honey prevent bacterial contamination and promote healing
  • 26. Treatment of Burn Wound • Early excision and skin grafting - Classically between 3-7 days • Larger burn – meshed autografted skin • Areas of cosmetic importance – use nonmeshed sheet grafts ensure optimal results • For excision of burns in areas such as the face, eyelids, or hands, a pressurized water dissector may offer more precision but is time consuming. • Xenograft and cadaveric graft function as well as allograft for temporary wound coverage. • Amniotic Membrane
  • 27.
  • 28. Burn in Specific Areas Face • Adequate cleaning of the face, exposure dressing with antibacterial lotions/ creams. Neck: • Exposure treatment is ideal • Flexion contracture – keep the neck extended by placing a pillow under the shoulders. • soft and hard neck collars and fabricated splints can be use later • Circumferential burn can form a tourniquet effect as the edema progresses. • Escharotomies are rarely needed within the first 8 hours following injury and should not be performed unless indicated
  • 29.
  • 30.
  • 31. Burn in Specific Areas • Axillae: • Keep the arm in abduction. • bulky axillary dressings are useful. • Negative pressure wound therapy (NPWT), may benefit patient requiring excision and grafting. • Crucifix splint may be used in children The Hands: • dressed the fingers and the web spaces separately with Vaseline gauze. • Splint wrist in extension, Metacarpophalangeal joint and 90 degree flexion, interphalangeal joint in extension
  • 32. Burn in Specific Areas Perinerum • Treated by exposure • In children gallows splint ensures adequate immobilization and exposure. • Adults are nursed in supine position with the hip joints in abduction. The legs and feet: These must be elevated to encourage venous drainage.
  • 33. Nutrition • Nutritional support may be more important in patients with large burns than in any other patient population • Adjuncts such as metoclopramide promote gastrointestinal motility. • if other measures for gastric feeding are unsuccessful, advancing the tube into the small bowel with nasojejunal feeding can be attempted.
  • 34. Nutrition • Harris-Benedict equation, is a commonly used formula in nonburned population. • The Harris-Benedict equation may be inaccurate in burns of less than 40% TBSA, and in these patients, the Curreri formula may be more appropriate. • This formula estimates caloric needs to be 25 kcal/kg/d plus 40 kcal/%TBSA/d.
  • 35. Nutrition • β-Blocker use in pediatric patients decreases heart rate and resting energy expenditure • There may be benefits to β-blockade in adult patients. • Insulin may have a metabolic benefit in burn patients • Oral hypoglycemic and prevent hyperglycemia and prevent muscle catabolism. • A recent double-blind, randomized study of oxandrolone showed decreased length of stay, improved hepatic protein synthesis, and no adverse effects on endocrine function,
  • 36. Causes of death in burn patients • Hypovolaemic shock in the first 48 hours. Renal failure • (i) Pre-renal from hypovolaemia . • (ii) Acute tubular necrosis from myogtobinuria or hemoglobinuria • Sepsis may be a local infection or septicemia often with endotoxic shock. • Tetanus may also occur. Respiratory tract infection accounts for 50 % of bum sepsis and 50% of deaths. • Pulmonary problems: Laryngo- tracheo-bronchitis, laryngeal oedema, pulmonary oedema, pneumonia • Some studies have reported an incidence of over 50% of multi- Organ failure in deaths after bum injury.
  • 37. Complications • Early complications and include; 1. HypovolaemicShock Infections • 3. Gastrointestinal problems • Acute gastric dilatation, Paralytic ileus, Curling's ulcers, Liver damage • 4. Cardio-respiration problems • Respiratory obstruction • Tracheo-bronchitis • Pneumonia, atelectasis • 5. Genito-urinary problems • Renal failure • Cystitis, pyelonephritis, Calculi • 6. Vascularproblems • Thrombophlebitis, • Deep venous thrombosis • Pulmonary embolism • 7. Anemia • Disseminated intravascular coagulation (DIC)
  • 38. Complications • Late complications of burns are usually related to consequences of poor healing of the burn wound and include • Unstable scars and chronic burn wounds. • Burn scar hypertrophy and Keloids • Burn scar contractures and deformities • Dyschromic scars (hypo-, hyper-, & de-pigmentation) • Bum Scar metaplasia and Marjolin's ulcers • Loss of body parts. • Psychiatric problems including PTSD
  • 39. Rehabilitation • Patients should be taught exercises they can do themselves to maintain full range of motion. • Psychological rehabilitation is equally important in the burn patient. • Depression, posttraumatic stress disorder, concerns about image, and anxiety are essential.
  • 40. Radiation • Radioactive material results in both acute injury from immediate exposure and more prolonged injury from delayed exposure to radioactive fallout or contamination. • The explosion results in a direct pressure wave and an indirect wind drag • After initial evaluation and decontamination by removing clothing, a useful way to estimate exposure is by determining the time to emesis. • Patients who do not experience emesis within 4 hours of exposure are unlikely to have severe clinical effects.
  • 41. • Irrigation fluid should be collected to prevent radiation spread into the water supply.
  • 42. Prevention •Burns is preventable, the common denominator in most instances being ignorance, carelessness and poverty •Smoke alarms are known to decrease mortality from structural fires.
  • 43. References • Schwartz's Principle of Surgery, 10th edition • Baja’s Principles and Practice of Surgery, 5th edition

Editor's Notes

  1. Unexpected neurologic symptoms should raise the level of suspicion, and an arterial carboxyhemoglobin level must be obtained because pulse oximetry can be falsely elevated. 100% oxygen reduces the half-life of CO from 250 minutes in room air to 40 to 60 minutes Cyanide inhibits cytochrome oxidase, which is required for oxidative phosphorylation.15
  2. Late complications, may arise several months or years after the accident. (i) Cataract especially when the head is a contact point. (ii) Spinal cord transecuon leading to paraplegia or quadriplegia, Abnormal gait, impotence, bladder dysfunction a (iv) Convulsion, (v) Intractable headache, (vi) Deformities.
  3. Offending agents can be systemically absorbed and may cause specific metabolic derangements.
  4. 1st degree burn Tender, Blanches under pressure, Possible swelling, no blisters, 2nd degree burn Moist, shiny Painful, Blisters may be present 3rd degree burn May bleed from vessel damage, Painless, Hair pulls out easily, Require grafting
  5. The zone of coagulation is the most severely burned portion and is typically in the center of the wound
  6. of early bum wound excision and closure with skin or its substitute that has played the most pivotal rote in the survival of the burned patient mitant illness make the outlook worse. Outcome is also worse at the extremes of age ( < 3 years and > 60 years) and those with severe base deficit in the first 24 hou
  7. The concept behind continuous fluid requirements is simple. The burn (and/or inhalation injury) drives an inflammatory response that leads to capillary leak; as plasma leaks into the extravascular space, crystalloid administration maintains the intravascular volume
  8. Children they do not have sufficient glycogen stores to maintain an adequate glucose level in response to the inflammatory response.
  9. Inhalation injury and pneumonia combined increases mortality by up to 60% over burns alone. Smoke inhalation causes injury in two ways: by direct heat injury to the upper airways and inhalation of combustion products into the lower airways. increased fluid requirement during resuscitation most common
  10. eliminates pain from multiple dressing changes, reduces infection and allows the patient to be mobilized more rapidly. The strategy of early excision and grafting in burned patients revolutionized survival outcomes in burn care Not only did it improve mortality, but early excision also decreased
  11. Nursing the patient in a recliner with elevation at 30 to 45 degrees head-up facilitates resolution of the edema over a few days. Warning signs of impending compartment syndrome may include paresthesias, pain, decreased capillary refill, and progression to loss of distal pulses; in an intubated patient,
  12. Digital escharotomies do not usually result in any meaningful salvage of functional tissue and are not recommended
  13. The fingers must be observed for adequacy of circulation The limbs must be elevated and physical and occupational hand therapy started immediately.